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All Journal BALI MEDICAL JOURNAL (BMJ) Medicina journal of internal medicine INDONESIAN JOURNAL OF BIOMEDICAL SCIENCES E-Jurnal Medika Udayana Majalah Ilmiah Fisioterapi Indonesia E-Jurnal Udayana Medica WMJ (Warmadewa Medical Journal) The Indonesian Biomedical Journal ISM (Intisari Sains Medis) : Jurnal Kedokteran Bioscientia Medicina : Journal of Biomedicine and Translational Research Review of Primary Care Practice and Education (Kajian Praktik dan Pendidikan Layanan Primer) Indonesian Journal of Clinical Pathology and Medical Laboratory (IJCPML) International Journal of Nasopharyngeal Carcinoma PREPOTIF : Jurnal Kesehatan Masyarakat Bioscientia Medicina : Journal of Biomedicine and Translational Research Warmadewa Minesterium Medical Journal Journal of Global Pharma Technology Seminar Ilmiah Nasional Teknologi, Sains, dan Sosial Humaniora (SINTESA) Physical Therapy Journal of Indonesia Biosaintifika: Journal of Biology & Biology Education
I Wayan Putu Sutirta Yasa
Bagian Patologi Klinik Fakultas Kedokteran Universitas Udayana
Agriculture, Biological Sciences & Forestry Biochemistry, Genetics & Molecular Biology Computer Science & IT Decision Sciences, Operations Research & Management Education Health Professions Immunology & microbiology Library & Information Science Medicine & Pharmacology Neuroscience Nursing Public Health Social Sciences

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Journal : E-Jurnal Medika Udayana

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IMMUNOPATOGENESIS INFEKSI VIRUS DENGUE IBN Dwipayana Manuaba; I Wayan Putu Sutirtayasa; DAP Rasmika Dewi
E-Jurnal Medika Udayana vol 2 no 10 (2013):e-jurnal medika udayana
Publisher : Universitas Udayana

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Abstract

Dengue , adalah penyakit arboviral tersering yang tersebar di seluruh dunia. Dengue disebabkan oleh infeksi 1 dari 4 serotipe virus dengue. Virus dengue berasal dari keluarga Flaviviridae , genus Flavivirus ( virus RNA ikatan-tunggal tidak ber-segmen).Virus dengue menular ke manusia melalui gigitan nyamuk Aedes betina yang telah ter-infeksi. Virus dengue (DEN) mempunyai 4 serotipe (DEN-1 , DEN-2 , DEN-3 , DEN-4) . infeksi dari salah satu tipe virus bisa tanpa gejala di mayoritas kasus yang terjadi atau bisa menimbulkan gejala klinis. Mulai dari yang ringan dengan gejala seperti flue (yang biasa disebut demam dengue [DF] ) , bentuk yang lebih parah (yang biasa disebut demam berdarah [DHF]) , dan yang hingga menimbulkan gejala syok ( dengue syok sindrom [DSS]). Penyebab dari perkembangan dan mekanisme yang terlibat dalam patofisiologi penyakit infeksi DENV tidak bisa dengan jelas didefinisikan. Immunopatogenesis infeksi virus dengue membahas tentang jalan masuk DENV , aktivasi berlebihan imune yang menyimpang dan menginduksi autoantibody , respon humoral ,peningkatan dependen antibody
DIAGNOSE OF THYPOID FEVER WITH WIDAL TEST I Made Tomik Nurya Wardana; Sianny Herawati; I Wayan Putu Sutirta Yasa
E-Jurnal Medika Udayana vol 3 no 2 (2014):e-jurnal medika udayana
Publisher : Universitas Udayana

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Abstract

Thypoid fever is one of infection systemic disease cause by Salmonella enterica serotype typhi, thisbacterium is member of family Enterobacteriaciae. Everyone with thypoid fever bring thebacterium in blood stream and gastrointestinal sytem so that it can transmit the infection to othersdirectly through water or food that has been contaminated. Laboratory examination to establish adiagnosis of typhoid fever is with conventional methods, namely the culture of the bacterium andserological test Widal and non-conventional method, namely Polimerase Chain Reaction (PCR),Enzyme Immunoassay Dot (EID) and Enzyme-Linked Immunosorbent Assay (ELISA). The definitivediagnosis of typhoid fever is with isolation of Salmonella enterica serotype typhi from blood, urineor other body fluids. It is often not possible in developing countries because of inadequatebakteriologik facilities on many small hospitals, while typhoid fever is an endemic disease in thecountry. With this kind of situation, the diagnosis should be enforced by connecting thecorresponding clinical symptoms with typhoid fever and the presence of antibody titer is increasingsignificantly in the blood against antigens O or H antigens of Salmonella enterica serotype typhi(Widal test).
PREVALENSI INFEKSI DENGUE BERDASARKAN HASIL PEMERIKSAAN NS1 ANTIGEN PADA PASIEN DI NIKI DIAGNOSTIC CENTER TAHUN 2011-2012 D.P.G. Jananuraga Maharddhika; I Wayan Putu Sutirta Yasa
E-Jurnal Medika Udayana vol 4 no 12(2015):e-jurnal medika udayana
Publisher : Universitas Udayana

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PREVALENCE OF DENGUE INFECTION BASED ON NS1 ANTIGEN EXAMINATION IN PATIENTS AT NIKI DIAGNOSTIC CENTER DURING 2011-2012Dengue is a disease caused by infection with dengue virus, which is transmitted by mosquitoes and can cause a variety of clinical manifestation, either asymptomatic or mild symptomatic to fatal. To diagnose accurately, early examination is needed. Nonstructural protein 1 (NS1) antigen is one examination that is used as a new biomarker in the early diagnosis of dengue infection. Several studies have been conducted expressing the importance of NS1 antigen as a biomarker because it can be detected before antibodies are formed.The study design was retrospective study by taking the data of patients undergoing examination dengue NS1 antigen in Niki Diagnostic Center in 2011-2012. The sample selection is done by consecutive sampling. The results obtained from the medical records are then presented descriptively with tables and narration.Patients who did a dengue checkup of NS1 antigen in Niki Diagnostic Center in 2011 amounted to 140 people with 63 men (45.0%) and 77 women (55.0%), whereas in 2012 amounted to 285 people with 166 men (58.2%) and 119 women (41.8%). For the age group with the most number of examination of dengue NS1 antigen is infant 0-5 years consists of 74 persons (52.9%) in 2011 and 161 people (58.2%) in 2012.On examination of dengue NS1 antigen in 2011, resulted in positive dengue infection as many as 18 of the 140 people (12.9%) with the largest group of dengue infection consists of 66.7% women and27.8%adulthood18-40 years. In 2012 examination dengue NS1 antigen positive results obtained dengue infection as many as 34 of 285 people (11.9%) with the largest group of dengue infection of 61.8% male and 47.1% adulthood 18-40 years.
PREVALENSI PENYAKIT GINJAL KRONIK STADIUM 5 YANG MENJALANI HEMODIALISIS DI RSUD BADUNG PERIODE TAHUN 2017-2018 I.B.GDE ANANTA MAHESVARA; WAYAN PUTU SUTIRTA YASA; AAN. NGURAH SUBAWA
E-Jurnal Medika Udayana Vol 9 No 7 (2020): Vol 9 No 07(2020): E-Jurnal Medika Udayana
Publisher : Universitas Udayana

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24843/MU.2020.V09.i7.P06

Abstract

ABSTRAK Penyakit ginjal kronik (PGK) merupakan masalah kesehatan masyarakat global dengan prevalensi daninsidensi gagal ginjal yang meningkat, prognosis yang buruk dan biaya terapi yang tinggi. Stadium PGK dibagi menjadi 5, berdasarkan laju filtrasi glomerulus (LFG) yang masih dapat dihasilkan ginjal, dimana halini mencerminkan fungsi ginjal. Untuk pasien dengan PGK stadium 5, terapi hemodialisis (HD) merupakansalah satu pilihan utama yang sering digunakan. Tujuan dilakukannya penelitian ini adalah untukmengetahui prevalensi dan karakteristik pasien dengan PGK stadium 5 yang menjalani HD di RSUDBadung periode tahun 2017-2018. Rancangan penelitian yang digunakan pada penelitian ini adalahpenelitian deskriptif dengan pendekatan retrospektif cross-sectional study. Sampel penelitian diambil daricatatan medis populasi terjangkau secara consecutive sampling. Penelitian dilaksanakan di Instalasi RekamMedis RSUD Badung dengan sumber data yang diambil selama 1 tahun, yaitu dari Januari tahun 2018sampai dengan Desember 2018. Pada penelitian ini didapatkan prevalensi PGK stadium 5 yang menjalaniHD di RSUD Badung sebesar 0,0003%. Pasien PGK stadium 5 yang paling banyak menjalani HD adalahsebagai berikut: pasien dengan jenis kelamin laki-laki, pasien berusia 50-59 tahun, pasien yang menjalaniHD selama <12 bulan, pasien dengan diagnosis etiologis pielonefritis kronik (PNC), pasien dengan aksesvaskular arteriovenous fistula (AVF), pasien dengan kadar hemoglobin (HB) 7-10 g/dL, pasien dengankadar kalsium (Ca) <8,4 mg/dL, pasien dengan kadar fosfat (PO4) >5,5 mg/dL, pasien dengan kadar produkCa x PO4 <55 mg2/dL2, dan pasien dengan kadar asam urat <7 mg/dL.Kata Kunci: PGK, stadium 5, hemodialisis, RSUD Badung ABSTRACTChronic kidney disease (CKD) is a global public health problem with increased prevalence and incidence of kidney failure, bad prognosis and high cost of therapy. CKD is divided into 5 stages, based onthe glomerular filtration rate (GFR) that the kidneys can still produce, which reflects to kidney function.For patients with stage 5 CKD, hemodialysis (HD) therapy is one of the main choices that are often used.The purpose of this study was to determine the prevalence and characteristics of patients with stage 5 CKDwho underwent HD on Badung General Hospital in the year of 2017-2018. The research design used in thisstudy was a descriptive study with a retrospective cross-sectional study approach. The study sample wastaken from population medical records by consecutive sampling. The study was carried out in the MedicalRecord Installation of Badung General Hospital with data sources taken for 1 year, from January 2018 toDecember 2018. In this study, the prevalence of stage 5 CKD which underwent HD in Badung GeneralHospital was 0.0003%. Stage 5 CKD patients who underwent the most HD were as follows: patients with male gender, patients aged 50-59 years, patients underwent HD for <12 months, patients with etiological diagnosis of chronic pyelonephritis (PNC), patients with AVF vascular access, patients with hemoglobin (HB) levels 7-10 g/dL, patients with calcium (Ca) <8.4 mg/dL, patients with phosphate levels (PO4) >5.5 mg/dL, patients with product levels Ca x PO4<55 mg2/dL2, and patients with uric acid level <7 mg/dL. Keywords: CKD, stadium 5, hemodialysis, Badung General Hospital
HUBUNGAN ANTARA UJI ANTIGEN NON STRUCTURAL 1 (NS1) DENGAN KEJADIAN TROMBOSITOPENIA PADA KASUS DEMAM DENGUE (DD)/DEMAM BERDARAH DENGUE (DBD) DIRUMAH SAKIT ARI CANTI, GIANYAR, BALI TAHUN 2016 Ni Wayan Ari Anindita Sari; I Wayan Putu Sutirta Yasa
E-Jurnal Medika Udayana Vol 9 No 12 (2020): Vol 9 No 12(2020): E-Jurnal Medika Udayana
Publisher : Universitas Udayana

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24843/MU.2020.V09.i12.P17

Abstract

ABSTRAK Demam dengue (DD)/demam berdarah dengue (DBD) merupakan penyakit yang disebabkan virus dengue dan disebarkan melalui gigitan nyamuk Aedes aegypti. Pada DD/DBD terjadi kelainan hematologi seperti penurunan kadar trombosit di bawah normal (trombositopenia) yaitu <150.000 terkait infeksi virus dengue. Pada kondisi ini, apabila pasien tidak segera tertangani dapat jatuh ke kondisi dengue shock syndrome. Adanya suatu uji NS1 antigen sebagai rapid diagnostic test kasus DD/DBD, maka penurunan trombosit yang terjadi dapat diperkirakan dan ditangani lebih dini. Penelitian bertujuan untuk mengetahui hubungan hasil uji NS1 antigen terhadap kejadian trombositopenia pada DD/DBD. Rancangan penelitian analitik dengan pendekatan cross-sectional. Subjek dikelompokkan menjadi 2 yaitu kelompok pasien dengan hasil uji NS1 antigen positif 599 subjek dan hasil uji NS1 antigen negatif 176 subjek. Kemudian dilakukan penghitungan jumlah pasien yang sudah maupun tidak mengalami trombositopenia dari masing-masing kelompok subjek yang didapatkan dari hasil rekam medis pasien. Hasil menunjukkan jumlah pasien dengan hasil uji NS1 antigen positif dan trombositopenia sebanyak 564(94,2%) pasien, tidak trombositopenia sejumlah 35(5,8%) pasien. Pasien hasil uji NS1 antigen negatif dan trombositopenia sebanyak 78(44,3%) pasien, tidak trombositopenia 98(55,7%) pasien. Berdasarkan hasil analisis uji chi-square dengan tingkat kemaknaan 5% diperoleh p=0,001, tingkat nilai keeratan hubungan 0,554. Disimpulkan, adanya hubungan signifikan (p<0,05) antara hasil uji NS1 antigen terhadap kejadian trombositopenia pada subjek yang diteliti dengan tingkat keeratan hubungan sedang (0,40-0,599). Arah hubungan kedua variabel positif artinya apabila terdapat hasil uji NS1 antigen positif maka risiko trombositopenia semakin tinggi. Kata Kunci : demam dengue, demam berdarah dengue, trombositopenia, dengue shock syndrome, rapid diagnostic test, NS1 antigen.
DIAGNOSIS LABORATORIK ANEMIA DEFISIENSI BESI Dina Sophia Margina; Sianny Herawati; I W P Sutirta Yasa
E-Jurnal Medika Udayana vol 3 no 1 (2014):e-jurnal medika udayana
Publisher : Universitas Udayana

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Abstract

Iron deficiency anemia is a decreament of iron level in the body. Iron deficiency anemiais oftenly seen, especially in the tropical countries or the third world country thatassociated with social economic rate. Iron deficiency anemia happens in more than onethird world’s population. Iron deficiency anemia can be caused by chronic hemorrhage,low intake of iron, absorption disturbance, and increasement of demand. To diagnose irondeficiency anemia, laboratoric examination is needed. The treatment can be causaltreatment, iron supplementation and another additional treatment.
ANEMIA SEL SABIT Gede Agus Suwiryawan; I Wayan Putu Sutirta Yasa; DAP Rasmika Dewi
E-Jurnal Medika Udayana vol 2 no 9 (2013):e-jurnal medika udayana
Publisher : Universitas Udayana

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Abstract

Anemia is a form of blood disorder most often occurs in the community. Sickle cell anemia is anemia due to hemoglobinopathy caused by a change in the amino acid-6 of the ? globin chain. Sickle cell anemia is common in tropical areas of Africa and some parts of the region Saudi Arabia, India and the Mediterranean as well as black people in America. In addition there are also careers in various European countries. In pathophysiology, there are amino acid change from glutamic acid to valine in the ?-globin chain that causes red blood cells become sickle-shaped when deoxygenatied, but still be able to return to its normal shape when experiencing oxygenation. When the red blood cell membrane have been amended, the polymerization of red blood cells has become irreversible. Clinical picture seen in sickle cell anemia can be divided into two, namely: acute and chronic. Diagnosis can be done is to distinguish between heterozygous or homozygous sickle cell. Treatment provided in accordance with the clinical picture appears. Treatment that can be done is by blood transfusion, bone marrow transplant, anti-sickling drug delivery, and drug delivery to trigger the synthesis of HbF. Treatment still in the development stage is to use stem cells.
GAMBARAN HASIL PEMERIKSAAN KADAR HBA1C PADA PENDERITA DIABETES MELLITUS DI LABORATORIUM RUMAH SAKIT UMUM SURYA HUSADHA TAHUN 2013 Airin Que; I Wayan Putu Sutirta Yasa; A.A. Wiradewi Lestari
E-Jurnal Medika Udayana vol 4 no 12(2015):e-jurnal medika udayana
Publisher : Universitas Udayana

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Abstract

THE HBA1C RESULT TEST OF PATIENTS WITH DIABETES MELLITUS IN THE LABORATORY OF SURYA HUSADHA HOSPITAL 2013Diabetes Mellitus is a group of metabolic diseases characterized by hyperglycemia. In 2010, an estimated 285 million people suffer from diabetes worldwide and in 2030 an estimated 438 million people will suffer from diabetes worldwide. Effect caused by hyperglycemia affect the vascular system and long-term health, it takes the diagnosis and management of diabetes mellitus quickly and accurately. The diagnosis of diabetes can be enforced through a lot of things, one of which is a measurement of HbA1C (Glycated Hemoglobin atau Glycosylated Hemoglobin) levels. On examination of HbA1c does not require patients to fast, the results were not influenced by short-term lifestyle of the patient, because the patient's HbA1C describe long-term (2-3 months) blood sugar and blood sugar control patients. Indonesian Society of Endocrinology (PERKENI) in 2006 has categorized the results of laboratory tests HbA1C levels into 3 variables, good control (HbA1c levels <6.5%), moderate control (HbA1c levels of 6.5% - 8%), and poor control (HbA1c levels ? 8%). This study aims to describe the results of HbA1C levels in people with diabetes mellitus in the Laboratory of Surya Husadha Hospital during the year 2013. This study used cross sectional design, non experimental data obtained retrospectively from Surya Husadha Hospital Laboratory. The sample was all the result of HbA1C level among patients who were tested in the Surya Husadha Hospital Laboratory in 2013 with total 173 people. The result of this study was from 173 people who done the HbA1C level test in the Surya Husdha Hospital Laboratory in 2013, the results HbA1C levels of disease control, showed that good control was 64 people (36%), moderate controls was 46 people (25.8%) and poor control was 68 people (38.2%). Characteristics of patients according to gender, the male gender was 110 people (61.8%) and female gender was 68 persons (38.2%).
ANALISIS PERBEDAAN KADAR BLOOD UREA NITROGEN (BUN) DAN KADAR SERUM CREATININE (SC) PADA PENDERITA DIABETES MELITUS TIPE 2 DENGAN PROFIL LIPID NORMAL DAN DISLIPIDEMIA DI RUMAH SAKIT UMUM PUSAT SANGLAH PERIODE JANUARI-DESEMBER 2014 Made Ayu Widyaningsih; AA Wiradewi Lestari; I Wayan Putu Sutirta Yasa
E-Jurnal Medika Udayana Vol 5 No 10 (2016): E-jurnal medika udayana
Publisher : Universitas Udayana

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Abstract

T2DM and dyslipidemia are the two conditions that are often found together. Blood sugar levels which are not controlled can lead to the increasing of fatty substances level in the blood and accelerate the arteriosclerosis. The consequences of dyslipidemia and inflammation will cause a decrease in blood supply to the kidneys, interfere the glomerulus filtration rate (GFR), and then followed by increased blood urea nitrogen (BUN) and serum creatinine (SC). Goal of this study to prove the existence of differences in levels of BUN and SC in patients with T2DM with normal lipid profiles and dyslipidemia in Sanglah Hospital. A cross-sectional analytic study was completed with a total sample of 160 T2DM patients treated at Sanglah Hospital. Data were collected through consecutive sampling method from the medical record of January-December 2014 and analys with Mann Whitney Test.The mean differences of BUN levels are significant between patients with normal lipid profile compare with hypertriglyceridemia (p = 0.002), hypercholesterolemia (p = 0.005), and hyper-LDL (p = 0.013) in T2DM patients. There are also significant mean differences of SC levels among patients with normal lipid profiles compare with hypertriglyceridemia (p = 0.000), and hypercholesterolemia (p = 0.006) in T2DM patients. Our study highlights that there are significant differences of BUN levels in T2DM patients with normal lipid profile compare with hypertriglyceridemia, hypercholesterolemia, and hyper-LDL patients and significant differences of SC levels in T2DM patients with normal lipid profile compare with patients who have hypertriglyceridemia and hypercholesterolemia (p <0.05).  
HUBUNGAN JUMLAH TRANSFUSI DARAH DAN PENGGUNAAN KELASI BESI DENGAN KADAR FERITIN PADA PASIEN TALASEMIA Fatqur Rochman; Ni Kadek Mulyantari; I WP Sutirtayasa
E-Jurnal Medika Udayana Vol 8 No 9 (2019): Vol 8 No 9 (2019): E-Jurnal Medika Udayana
Publisher : Universitas Udayana

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Abstract

Talasemia merupakan salah satu kelainan keturunan pembentukan darah yang menyebabkanjumlah kematian cukup signifikan (3,4%.) pada usia di bawah 5 tahun di dunia. Hingga kinimanajemen utama anemia pada pasien talasemia adalah transfusi darah. Pemberian transfusiyang sering akan meningkatkan kadar besi dalam tubuh sehingga membutuhkan obat kelasi besi.Penelitian ini bertujuan untuk mengetahui hubungan antara jumlah transfusi darah yangdiberikan dan penggunaan kelasi besi terhadap kadar feritin. Penelitian ini menggunakan rancangan studi potong lintang. Jumlah sampel penelitian adalah 25 sampel yang merupakanpasien talasemia di RSUP Sanglah dari Januari 2014 hingga Juni 2015. Pemilihan sampeldilakukan dengan teknik total sampling. Penelitian ini dilaksanakan dengan mengambil datasekunder dari rekam medis pasien talasemia. Pada hasil analisis, terlihat secara umumpeningkatan jumlah transfusi akan meningkatkan rerata kadar feritin . Dari penelitian ini dapatdisimpulkan bahwa terdapat hubungan yang signifikan antara jumlah transfusi darah yangdiberikan dengan kadar feritin pasien talasemia (p=0.003). Pada analisis hubungan antara kadarferitin dan ada atau tidaknya pemberian kelasi besi mendapatkan hasil yang tidak signifikansecara statistik (p>0.664). Penelitian ini diharapkan dapat dipakai untuk pembelajaran dan dapatdijadikan dasar untuk dilakukan penelitian lain yang serupa di masa depan. Kata kunci: talasemia, transfusi darah, obat kelasi besi dan kadar feritin.
Co-Authors Adhitya, Putu Gde Surya Agung Nova Mahendra Agung Wiwiek Indrayani Airin Que Ali Djamhuri Amaral, Meriana Barreto Ana Rahmawati Anak Agung Gede Sudewa Djelantik Anak Agung Ngurah Subawa Anak Agung Wiradewi Lestari Anak Agung Wiradewi Lestari, Anak Agung Anom Suardika Astini, Dewa Ayu Agung Alit Suka Bagus Komang Satriyasa Bastianus Alfian Juatmadja Benny Supono Calvin Jonathan Cokorda Agung Wahyu Purnamasidhi Cokorda Istri Dewiyani Pemayun D.P.G. Jananuraga Maharddhika Desak Gde Diah Dharma Santhi Dewa Ayu Putu Rasmika Dewi Dewi, Ni Kadek Ari Kusuma Dharayani, Luh Anandita Dharma Santhi, Desak Gde Diah Dina Sophia Margina Divasta, I G Mahapraja Fatqur Rochman Feliciano Pinto, Feliciano Ferbian Milas Siswanto Gde Ary Putra Kamajaya Gede Agus Eka Tirta Putra Gede Agus Suwiryawan Gusti Ngurah Sutapa Hamsu Kadriyan Haneetha Yogarajah I A Putri Wirawati I Dewa Made Sukrama I Gde Raka Widiana I Gde Suranaya Pandit I Gede Juliarta I Gede Widhiantara I Gst Agung Dwi Mahasurya I Gusti Agung Dyah Ambarawati I Gusti Agung Ngurah Radhitya Wijaya Radhitya Wijaya I Gusti Kamasan Arijana I Gusti Made Aman I Kadek Arya Candra I Kadek Septiawan I Ketut Agus Somia I Ketut Suastika I Ketut Suwiyoga I Made Ady Wirawan I Made Bagus Cahya Wibawa I Made Bakta I Made Dwikayana I Made Jawi I Made Oka Adi Parwata I Made Sudarmaja I Made Tomik Nurya Wardana I NYOMAN MANTIK ASTAWA I Nyoman Wande I Nyoman Wande I W. Sudarsa I Wayan Gede Artawan Eka Putra I Wayan Niryana I Wayan Rosiana I Wayan Sumardika I Wayan Wita I. B. T. Wibawa Manuaba, I. B. T. Wibawa I. B. Tjakra Wibawa Manuaba I. K. Widiana, I. K. I.A.A. Widhiartini I.B.GDE ANANTA MAHESVARA IBN Dwipayana Manuaba Ida Ayu Kemala Wasita Manuaba Ida Bagus Made Suryatika Ida Bagus Ngurah Ida Bagus Putra Manuaba Ida Bagus Verry Kusumaningrat Ida Bagus Wayan Kardika Ida Kurniawati, Ida IGA Ayu Ratih Pradnyadewi Ignatius Ferdi Yuatmadja Indah Pramita Indrayani, Pande Kadek Putri Rahayu Intan Astariani Kamayoga, I Dewa Gede Alit Karta Sawenda Ketut Siki Kawiyana Ketut Suega Ketut Suryana Ketut Tuti Parwati Merati Ketut Widyani Astuti Lesmana, Cokorda Agung Bagus Jaya Luh Putu Sukma Diyanti Made Ayu Widyaningsih Made Dyah Khrisnadewi MADE RATNA SARASWATI . Manuaba, Ida Bagus Tjakra Wibawa Melania Antonia Barreto Cerqueira Michael Christian Widjaja Milaviwanda, Luh Komang Ayu N. K. Niti Susila, N. K. Naw, Sin War Ni Gusti Ayu Putu Lestari Santika Dewi Ni Kadek Mulyantari Ni Kadek Nita Utami Ni Ketut Susilawati Ni Ketut Susilawati Ni Luh Candra Mas Ayuni Ni Luh Wayan Pani Ambarasari Ni Made Dharma Laksmi Ni Made Linawati Ni Nyoman Mahartini Ni Nyoman Mahartini Ni Nyoman Mahartini Ni Putu Sriwidyani Ni Putu Widya Nandasari Ni Wayan Ari Anindita Sari Ocktaviana Saputri, Legis Pande Ayu Naya Kasih Permatananda Pande Komang Gede Bayu Wikrama Permatasari, Anak Agung Ayu Putri Poniman, S. Prabasari, Pande Visca Gayatri pradnya wibawa, komang budhi Pratiwi, Cokorda Agung Pujawan, I Made Naris Purnamawati, Susy Puspa Negara, Anak Agung Gede Angga Putu Angga Wiradana Putu Astawa Putu Budhiastra Putu Diani Wirayanti Raka-Sudewi A. A. Romdhoni, Achmad Chusnu Rudi Wisaksana S. Herawati, S. Sagung Novita Widyaningrat Santosa, I Gusti Ngurah Putra Eka Sianny Herawati Sri Maliawan Sri Wahyuni Suryawisesa, Ida Bagus Made Swari, Kadek Gyna Yadnya Thaha .. Tirtha Yasa, I Nyoman Wawan Tjokorda Gde Agung Suwardewa Tjokorda Gede Oka Tri Pramartha, I Nyoman Valari, Putu Kavita Krisnina Wayan Suardana Widiyanti, I Gusti Ayu Wijaya Kusuma Yasa, I Nyoman Wawan Tirtha Yayang Christian Yenny Kandarini