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All Journal Medula JPM (Jurnal Pengabdian Masyarakat) Ruwa Jurai
Jausal, Annisa Nuraisa
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Education Health Professions Medicine & Pharmacology Neuroscience Nursing Public Health Social Sciences

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Kerusakan, alkohol, kesehatan Kerusakan Lambung Akibat Konsumsi Alkohol: Dampak dan Penanganannya Madina Ghassan Nebraska; Jausal, Annisa Nuraisa; Wulan, Anggraeni Janar; Rudiyanto, Waluyo
Medula Vol 14 No 3 (2024): Medula
Publisher : CV. Jasa Sukses Abadi

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.53089/medula.v14i3.984

Abstract

Alcohol consumption has spread across various social strata of society, especially among teenagers. Health issues caused by alcohol are often overlooked. This article delves into the damage to the stomach caused by alcohol consumption. Alcohol can damage the gastric mucosa layer and disrupt the balance of stomach acid, leading to various conditions such as gastritis, gastric ulcers, and even gastric cancer. Additionally, alcohol can affect the movement of food and fluids in the digestive tract, increasing the risk of acid reflux, which can worsen stomach conditions. The mechanisms of stomach damage by alcohol involve the direct toxic effects of alcohol itself and the chronic inflammation it induces. Although this damage is often not immediately felt, its long-term effects can lead to serious complications. Management of alcohol-induced stomach damage involves lifestyle modifications, such as reducing or ceasing alcohol consumption, and medical treatment to alleviate symptoms and accelerate healing. To prevent further damage, it is important to raise awareness of the risks of alcohol to stomach health and promote a healthy lifestyle. Understanding the impact and management of stomach damage caused by alcohol consumption is crucial in preventing more serious complications.
Mekanisme Hiperglikemia Kronis terhadap Densitas Mineral Tulang Dwiputri, Maliya Finda; Larasati, TA; Jausal, Annisa Nuraisa; Carolia, Novita
Medula Vol 14 No 12 (2024): Medula
Publisher : CV. Jasa Sukses Abadi

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.53089/medula.v14i12.1438

Abstract

Hyperglycaemia or high levels of glucose in the blood is the hallmark of diabetes mellitus. Diabetes mellitus type 2 (DMT2) significantly impacts bone health, leading to a condition known as diabetic bone disease, characterized by decreased bone mineral density and an increased risk of fractures. This condition arises from metabolic disturbances triggered by hyperglycemia, insulin resistance, and inflammatory factors that compromise bone structure. Research indicates that over 35% of DMT2 patients experience reduced bone density, with approximately 20% meeting osteoporosis criteria. The pathophysiology involves several mechanisms, including the accumulation of advanced glycation end-products (AGEs), which hinder bone formation and increase resorption. Insulin deficiency further exacerbates this issue by impairing osteoblast function and reducing the synthesis of key bone matrix proteins like collagen and osteocalcin. Treatment options such as metformin have shown potential in improving bone health, while other medications like sulfonylureas and SGLT-2 inhibitors may have detrimental effects on bone density. Therefore, careful management of diabetes is essential to mitigate the risks of osteoporosis and fractures in patients with DMT2. Ongoing research is needed to deepen the understanding of diabetic bone disease and optimize treatment strategies to protect bone health in these patients.
Co-Authors Anggraeni Janar Wulan, Anggraeni Janar Dwiputri, Maliya Finda Endah Setyaningrum, Endah Madina Ghassan Nebraska Marcellia, Selvi Novita Carolia Rosa, Emantis Rudiyanto, Waluyo TA Larasati