<![CDATA[COVID-19 is caused by acute respiratory syndrome coronavirus 2 (SARS-CoV-2) of the betacoronavirus genus. Immune dysregulation in hypertensive patients makes cases more susceptible to infection with SARS-CoV-2. In addition, in humans, ACE2 is widely expressed in many organs, including heart, kidney, liver, intestine, and pulmonary alveolar epithelial cells. However, that 83% of cells expressing ACE2 are found alveolar cells and the large lung surface area makes SARS-CoV-2 highly susceptible to inhaled viruses. Therefore, it is plausible that SARS-CoV-2 is more likely to damage the lung tissue of hypertensive patients, leading to an increase in the number of severe. The main imaging modality of choice is a chest X-ray, in patients with COVID-19, features such as ground-glass opacification, infiltrates, peribronchial thickening, focal consolidation, pleural effusion, and atelectasis can be found. Chest X-ray is less sensitive than CT scan, because about 40% of cases do not find abnormalities on chest X-ray. The type of research method used is descriptive observational. The type of data used in this study is quantitative data with secondary data as the source of data used. The population in the study used was patients diagnosed with COVID-19 at the Jakarta Hospital for the March - December 2020 period. From this study, it was found that the largest number of COVID-19 patients with hypertension had pneumonia as many as 51 patients (54.8%). In this analysis, a P value of 0.873 means P 0.05 which indicates that there is no relationship between blood pressure and the incidence of pneumonia in COVID-19 patients.]]>
Copyrights © 2022
