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Raymond Pranata
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ijc@inaheart.org
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Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
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INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 13 Documents
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Search results for , issue "Vol. 31, No. 2 Mei - Agustus 2010" : 13 Documents clear
Sel Punca Pada Penyakit Kardiovaskular: Kembali ke Bench Suko Adiarto
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.137

Abstract

Ungkapan from bench to bedside sesungguhnya merupakan dambaan setiap peneliti yang bergerak dalam ilmu-ilmu dasar kardiovaskular, dimana penemuan yang mereka hasilkan dari laboratorium pada tingkat kultur sel sampai uji efektitivitasnya pada hewan coba dengan model penyakit kardiovaskular dapat men-emukan aplikasi klinik yang bermanfaat. Tidak semua temuan yang secara teoritis menjanjikan pada tingkat laboratorium maupun hewan coba dapat menunjukan efektivitas yang diharapkan pada uji klinik. Bahkan, ketika suatu modalitas yang secara meyakinkan menunjukkan potensi terapetik yang baik pada pe-nelitian laboratorium atau hewan coba, namun gagal pada tingkat uji klinik seringkali diuji kembali pada penelitian laboratorium untuk mencari pemecahan dari kegagalan pada uji klinik tersebut.
Hubungan antara dispersi qt pasca bedah pintas arteri koroner dengan kejadian kardiovaskular mayor Reynold Agustinus; Yoga Yuniadi; Budhi Setianto
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.138

Abstract

Introduction:Coronary artery bypass graft (CABG) improves prognosis in patient with coronary artery disease. However, some patients might experi-ence major adverse cardiovascular events. An increase in QT dispersion is predictor of sudden cardiac death and associated with an increased risk of major adverse cardiovascular events (MACE) after successful percutaneous coronary intervention in patients with acute myocardial infarction. This study was aimed to evaluate the correlation between QT dispersion and MACE after isolated CABG.Methods: This was a retrospective cohort study of post-CABG patients using medical record analysis in National Cardiovascular Center Harapan Kita Jakarta during period of January until December 2007 with 1 year fol-low up. QT dispersion was defined as difference between the longest and the shortest QT interval of standard 12-lead ECG on the fifth or the seventh post operative day. It was assessed manually and blinded to clinical data then divided into two groups (QT d <60 and QT d > 60).Results: Of 517 patients who had undergone isolated CABG, 343 were eligible to be analyzed. There are 303 men and 40 women (mean age 58,0 ± 7,9 years) with mean QT dispersion 55,7 ± 19,9 ms. The incidences of MACE were 43 (12.5%) comprised with 19 (5,5%) heart failure, 11 (3,2%) non fatal acute coronary syndrome, 2 (0,6%) stroke and 11 (3,2%) subject died due to cardiac or non cardiac caused. QT dispersion > 60 ms related with total MACE (RR 3.58; [95 % CI 1.93 – 6.65], p< 0.01), non fatal acute coronary syndrome (RR 9.41 [95% CI 2,03 – 43,58], p = 0.004) and acute heart failure (RR 4.56 (95% CI 1.73 – 12.00, p = 0.002).Conclusion: QT dispersion > 60 ms in subject undergone isolated CABG had increased risk of total MACE, acute heart failure, and non fatal acute coronary syndrome during 1 year follow up.
Nilai prognostik dispersi QT pasca bedah pintas koroner Budhi Setianto
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.139

Abstract

Hubungan antara dispersi QT pascabedah pintas arteri koroner dengan kejadian kardiovaskuler telah diteliti akhir-akhir ini oleh Agustinus dkk dari De-partemen Kardiologi dan Kedokteran Vaskuler FKUI di Rumah Sakit Jantung dan Pembuluh Darah Hara-pan Kita secara restropektif dengan kesimpulan yang menarik perhatian untuk diberikan komentar. Bahwa dispersi QT > 60 milidetik pada subyek pasca BPAK telah meningkatkan risiko terjadinya kejadian kar-diovaskular mayor, gagal jantung akut dan sindroma koroner akut non fatal dalam pemantauan selama 1 tahun.Melihat desain penelitian yang tidak mengikut sertakan dispersi QT pra BPAK, merupakan kes-engajaan penulis agar lebih fokus pada penelitian prognosis pasca BPAK dengan dengan dispersi QT sebagai tolok ukurnya. Telah diketahui sebelumnya peranan BPAK dalam memperbaiki dispersi QT dalam arti memperpendek dispersi QT.
N-terminal probrain natriuretic peptide sebagai petanda adanya hipertrofi ventrikel kiri pada pasien hipertensi dibandingkan left ventricle mass index pada ekokardiografi Mohammad Ariful Munir; Djanggan Sargowo
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.140

Abstract

Background: Some research reports the usefulness of the N-terminal proBrain natriuretic peptide (NT proBNP) as a diagnostic tool for left ven-tricular hypertrophy due to increased levels of NT proBNP in hypertensive patients with left ventricular hypertrophy.Objective:To determine the role of NT pro-BNP in terms of diagnostic tool in hypertensive patients with left ventricular hypertrophy, knowing the value of sensitivity, specificity, positive predictive value, negative predictive value of NT proBNP and levels of NT proBNP in hypertensive patients with left ventricular hypertrophy.Methods: Cross sectional observational study conducted in outpatient clinic RSSA Malang from July 2008 until December 2008. This study mea-sures the level of NT proBNP and echocardiography examinations on 79 hypertensive patients. Pearson correlation test was used to analyze the correlation between NT proBNP with left ventricle mass index(LVMI). Chisquare test was used to determine the value of sensitivity, specificity, positive predictive value, negative predictive value NT proBNP compared with echocardiography. All calculations using a= 5%, 95% confidence intervals and considered significant if p <0.05.Results: The level of NT proBNP left ventricular hypertrophy group were significantly higher than non-left ventricular hypertrophy (797,31±546,08 v 56,09±29,81 pg/ml). There is significant positive correlation between LVMI with NT proBNP (p=0.006, r=0.306). NT proBNP had a sensitivity of 60%, specificity 65.5%, 75% positive predictive value, negative predic-tive value 48.7% and AUC (area under curve) of 0.649. Conclusion: The level of NT proBNP increased in hypertensive patients with left ventricular hypertrophy and there is significant positive correlation between LVMI with NT proBNP. NT proBNP less sensitive and specific than echocardiography to determine left ventricular hypertrophy.
Biomarker untuk hipertrofi ventrikel kiri Arieska Ann Soenarta
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.141

Abstract

Dampak peningkatan kronis tekanan darah arterial terhadap jantung merupakan respons dari ventrikel kiri terhadap beban tekanan yang berlebihan (pressure overload) dan rangsangan neurohumoral. Disamping hipertrofi sel-selotot jantung (cardiac myocyte) yang mengakibatkan hipertrofi ventrikel kiri (LVH) juga terjadi perubahan-perubahan pada komponen non cardiac myocyte otot jantung berupa apoptosis, fibrosis, dan perubahan-perubahan mikro-sirkulasi yang dipengaruhi hormon, growth factors, cy-tokines, molekul-molekul proinflammatory, sehingga terjadi pathologic myocardial remodeling.Remodeling otot jantung dapat disebabkan infark miokard, beban tekanan (pressure overload) maupun beban volume (volume overload), dan cardiomyopa-thy. Remodeling miokard akibat hipertensi mencakup peningkatan apoptosis miosit, fibrosis interstitial and perivasculer dan perubahan-perubahan mikrosirkulasi sehingga terjadi perubahan struktur maupun fungsi miokard berupa hipertrofi ventrikel kiri, peningkatan massa ventrikel kiri dan disfungsi ventrikel
Troponin I untuk memprediksi kejadian Kardiovaskular mayor Pasca bedah pintas arteri koroner Siti Elkana Nauli; Ganesja M Harimurti; Nani Hersunarti
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.142

Abstract

Background. CABG has impact in morbidity and mortality. Early detection for which patients will develop these outcomes should be done, especially in patients with low risk for morbidity and mortality after CABG. Elevated troponin I level is common after CABG but its prognostic value still confus-ing due to its lack of cut-off point. This phenomenon has an independent prognostic value for short and long term outcomes. The aim of this of this study was to find out the cut off value of troponin I which could predict MACE 30-day after elective isolated CABG. Methods. This is a retrospective cohort study included 284 patients after elective CABG. The CKMB level was measured on the 1st and 12nd hour after CABG, while the troponin I level was measured on 8th hours after CABG. We used the ROC curve for troponin I cut-off point. MACE in this research were heart failure or worsening heart failure, low cardiac output syndrome, and death in 30-day after elective isolated CABG.Results.There were 284 patients who were included in this research, 20,7% had MACE during 30 day after CABG. From bivariate analysis, his-tory of heart failure before CABG, ejection fraction less than 50% before operation, and troponin I level = 5 ng/ml was significantly predicts 30-days MACE after CABG. While from the multivariate analysis showed history of heart failure and troponin I level = 5 ng/ml was significantly increases MACE with OR 3 and 6,88. Troponin I was an independent predictor of MACE with sensitivity 70%.Conclusion.Troponin I was an independent predictor for 30-days MACE after elective isolated CABG with cut-off value 5 ng/ml.
Mencari kadar Troponin I yang bernilai prognostik Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.143

Abstract

Kejadian nekrosis miokard yang ditandai oleh pelepasan biomarker jantung, hampir selalu terjadi sekitar tindakan bedah pintas arteri koroner (BPAK). Berbagai hal seperti trauma langsung akibat manipulasi bedah atau iskemia miokard akibat kardioproteksi yang inadekuat, trombosis arteri koroner maupun kegagalan akut graft adalah mekanisme kerusakan miokard pada BPAK.Beberapa mekanisme itu tidak dapat dihindari kejadianya oleh karena itu amat pent-ing menentukan ambang batas pelepasan biomarker jantung pasca BPAK yang berakibat jelek terhadap prognosis. Beberapa penelitian terdahulu telah men-coba menghubungkan kadar troponin T pasca BAPAK terhadap prognosis selama perwatan di rumah sakit maupun terhadap prognosis jangka panjang. Studi lain juga meneliti nilai prediksi troponin I pasca BPAK terhadap komplikasi selama perawatan maupun luaran jangka panjang.
Intervensi Koroner Perkutan Primer Doni Firman
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.144

Abstract

Penyakit Kardiovaskular masih merupakan pembunuh nomor satu di Indonesia maupun di dunia. Dari data di Amerika setiap tahun 1,2 juta orang mengalami infark miokard dan kira-kira sepertiganya merupakan infark miokard dengan ST elevasi.Dari seluruh orang yang mengalami infark miokard di Amerika, 25-35% nya meninggal sebelum mendapat perawatan, sebagian besar karena Fibrilasi Ventrikel. Pada kelompok yang mendapat perawatan, angka kematian turun dari 11.2% di tahun 1990 menjadi 9.4% di tahun 1999. Hal tersebut dikarena-kan adanya tindakan reperfusi pada Infark dengan ST elevasi, baik dengan fibrinolitik maupun Intervensi Koroner Perkutan ( IKPP ). Dari analisa National Registry of Myocardia Infarction angka kematian di rumah sakit pada pasien yang mendapat reperfusi adalah sekitar 5.7%, jauh lebih rendah dibandingkan dengan pasien yang tidak mendapat reperfusi walau-pun sebenarnya kandidat yaitu 14.8%
Terapi anti-koagulan pada pasien pasca bedah katup Ali Syahputra; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.145

Abstract

The development of valvular surgery has enabled many patients to lead active and useful lives. Prosthetic heart valves improve quality of life and survival of patients with severe valvular heart disease, but the need for antithrombotic therapy to prevent thrombotic complications in valve recipients poses challenges for clinicians and patients. Advances in antithrombotic therapy and valve technologies are likely to improve the management of patients with heart valves surgery.
Implantasi hibrid alat terapi resinkronisasi jantung Alexander Edo Tondas; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 31, No. 2 Mei - Agustus 2010
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v31i2.146

Abstract

Recent trials have proven the clinical and functional benefits of cardiac resynchronization therapy (CRT) by biventricular pacing in patients (pts) with severe heart failure and intraventricular conduction delay, principally left bundle branch block (LBBB). However, placement of the transvenous left ventricular lead of CRT device is unsuccessful in 5–10% of patients and a further 20% fail to respond. For these groups, epicardial left ventricular lead placement is one alternative A 75-year-old male patient diagnosed as non ischemic dilated cardiomyopa-thy patient NYHA Class III-IV meeting the clinical initiation criteria of QRS duration > 120 msec, and low ejection fraction (=35%), with worsening symptoms despite one year of medical therapy.A hybrid approach of LV lead implantation by mini thoracostomy and conventional implantation of the RA and RV leads were performed because the coronary sinus cannot be accessed transvenously due to small caliber even after angioplasty. The patient responded quite well to CRT and was discharged with stable he-modynamics, and NYHA Functional Class II.

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