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Agus Prima
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Jl. DR. Wahidin Sudiro Husodo No.243B, Kembangan, Klangonan, Kec. Kebomas, Kabupaten Gresik, Jawa Timur 61124
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The Journal of Society Medicine (JSOCMED)
Published by PT. CoinReads Media Prima
ISSN : -     EISSN : 29645565     DOI : https://doi.org/10.47353/jsocmed.v2i1
Core Subject : Health, Science,
Health Professions Immunology & microbiology Medicine & Pharmacology Neuroscience
The Journal of Society Medicine (JSOCMED) | ISSN (e): 2964-5565 is a leading voice in the Indonesia and internationally for medicine and healthcare. Published continuously, JSOCMED features scholarly comment and clinical research. JSOCMED is editorially independent from and its The Editor-in-Chief (EIC) is Prof. dr. Aznan Lelo, PhD, SpFK. JSOCMED offers many attractive features for authors, including free online access to all research articles, online publication ahead of print, and online responses to articles published as Quick Comments. In addition, as befitting a publication of the Journal of Society Medicine, JSOCMED implements best practice in scientific publishing with an open peer review process, declarations of competing interests and funding, full requirements for patient consent and ethical review, and statements of guarantorship, contributorship, and provenance.
Arjuna Subject : Kedokteran - Kedokteran (Lain-Lain)
Articles 2 Documents
 1 
Search results for , issue "articles in press" : 2 Documents clear
Hypertension as a Risk Factor for Diabetic Peripheral Neuropathy in Patients with Type 2 Diabetes Mellitus: A Narrative Review Widha Zhafira Dewi; Yudhisman Imran
Journal of Society Medicine Articles in Press
Publisher : CoinReads Media Prima

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Abstract

Introduction: Type 2 diabetes mellitus (T2DM) is a chronic metabolic disease frequently complicated by diabetic peripheral neuropathy (DPN), which substantially impairs the quality of life and predisposes patients to foot ulceration and amputation. Hypertension, a highly prevalent comorbidity in T2DM, is an important modifiable risk factor for DPN. This review aims to synthesize the current evidence on the association between hypertension and DPN in patients with T2DM and elucidate the underlying pathophysiological mechanisms. Methods: A narrative literature review was conducted using electronic databases, including PubMed and Google Scholar, to identify clinical, epidemiological, and experimental studies that examined the relationship between blood pressure and DPN in patients with diabetes. Results: Observational studies across diverse populations consistently demonstrated that coexisting hypertension is associated with a higher prevalence and greater severity of DPN, and multivariate analyses identified hypertension as an independent risk factor after adjusting for age, diabetes duration, and glycemic control. Mechanistically, hypertension acts synergistically with chronic hyperglycemia to aggravate endothelial dysfunction, oxidative stress, inflammation, and structural remodeling of the vasa nervorum, resulting in endoneurial hypoxia, demyelination, and accelerated nerve degeneration. Experimental and imaging studies have corroborated more severe nerve injury when both conditions coexist. Conclusion: Hypertension is a modifiable contributor to the development and progression of DPN in patients with T2DM. Blood pressure control, alongside glycemic management, should be integrated into DPN prevention strategies, and interventional studies are warranted.
A Diagnostic Dilemma Between Brain Metastasis and Intracranial Tuberculoma in a 60-Year-Old Man with Metastatic Carcinoma of Unidentified Primary and Sputum-Positive Pulmonary Tuberculosis: A Case Report Amalia Noor Zafira Nasution
Journal of Society Medicine Articles in Press
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Abstract

Introduction: Intracranial tuberculoma and brain metastasis present with overlapping clinical and neuroimaging features. This distinction is critical because their diagnostic pathways and treatments differ fundamentally, and the dilemma is amplified when metastatic carcinoma and active tuberculosis coexist in the same patient. Case Description: A 60-year-old Indonesian man presented with decreased consciousness, a one-year history of chronic headache, right hemiparesis, abducens and facial nerve palsies, fever, weight loss, and long-standing left cervical and supraclavicular lymphadenopathy. Histopathology of a cervical lymph node demonstrated metastatic carcinoma without an identified primary site. Head computed tomography revealed multiple hypodense lesions with vasogenic edema, initially suggestive of metastases, whereas contrast-enhanced neck magnetic resonance imaging incidentally showed multiple enhancing intracranial lesions, including a 12.4 × 10.6 × 11.3 mm left temporal lesion, interpreted as favoring tuberculoma. Sputum examination was positive for tuberculosis despite an unremarkable chest radiograph. Antituberculosis therapy with corticosteroids and supportive neurological management was initiated, and the Glasgow Coma Scale improved from 12 to 15. Intracranial biopsy and lumbar puncture were not available. The patient was discharged on day 7 but died at home 5 days later, leaving the intracranial diagnosis unconfirmed. Conclusion: Coexisting metastatic carcinoma and sputum-positive tuberculosis create genuinely competing etiologies for enhancing brain lesions. Neither extracranial cytology nor sputum positivity can establish the nature of an intracranial lesion. Tissue or microbiological confirmation, a systematic primary-tumor workup, and multidisciplinary interpretation should be pursued whenever feasible to avoid premature diagnostic closure.

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