Spinal anesthesia commonly induces hemodynamic alterations, particularly post–spinal anesthesia hypotension resulting from sympathetic blockade of efferent preganglionic vasomotor fibers. This sympathetic inhibition reduces vascular tone and venous return, leading to a decline in systolic blood pressure. Leg elevation is a simple, non‑pharmacological maneuver that may enhance venous return through gravitational redistribution of blood toward the central circulation. This study aimed to determine the effectiveness of 30° and 45° leg elevation in increasing blood pressure among patients experiencing hypotension after spinal anesthesia. A quasi‑experimental design employing a Three‑Group Pretest–Posttest Control Group Design was used. A total of 30 respondents were selected through purposive sampling and allocated into three groups. Respondents met the inclusion criteria of post–spinal anesthesia hypotension (80 mmHg < systolic ≤ 100 mmHg). The independent variables were 30° leg elevation and 45° leg elevation, while the dependent variable was systolic blood pressure. Statistical analyses included normality testing, homogeneity testing, paired t‑test, independent t‑test, and one‑way ANOVA. The results demonstrated a significant difference in systolic blood pressure following the application of 30° and 45° leg elevation, with a p‑value of 0.000 (p < 0.005) and a mean difference of 12.3 mmHg. The 45° leg elevation produced a greater increase in systolic blood pressure compared to the 30° elevation. In conclusion, leg elevation at 45° is more effective in increasing systolic blood pressure among post–spinal anesthesia hypotensive patients. A higher elevation angle facilitates greater gravitational blood flow toward the heart, thereby enhancing venous return and improving systolic blood pressure.Keywords: 30° leg elevation; 45° leg elevation; blood pressure; hypotension; spinal anesthesia