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Sakina, Adriani
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Health Professions Medicine & Pharmacology Public Health

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Diagnosis Klinis, Tatalaksana, dan Pencegahan Chlamydial Conjunctivitis Sakina, Adriani
Cermin Dunia Kedokteran Vol 46, No 7 (2019): CME - Continuing Medical Education
Publisher : PT. Kalbe Farma Tbk.

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (1172.334 KB) | DOI: 10.55175/cdk.v46i7.455

Abstract

Konjungtivitis merupakan salah satu penyakit mata yang umum. Penyebab konjungtivitis antara lain alergi, zat kimiawi, reaksi imun, dan infeksi. Salah satu agen infeksi penyebab konjungtivitis adalah Chlamydia. Infeksi Chlamydia mata dibagi menurut klasifikasi Jones, meliputi Blinding trachoma, Non-blinding trachoma, dan Paratrachoma. Diagnosis Chlamydial Conjunctivitis ditegakkan berdasarkan klinis. Berbagai pemeriksaan laboratorium dilakukan saat survei epidemiologis atau penelitian.Conjunctivitis is a common eye disease. The etiology can be allergy, chemical reaction, immunology reaction, and infection. One of the infection agent is Chlamydia. Chlamydial eye infection is classified according to Jones Classification into 3 group : Blinding trachoma, Non-blinding trachoma, and Paratrachoma. Dagnosis of chlamydial conjunctivitis is based on the clinical condition. Laboratory tests are only used in epidemiology survey or research.
Infark Miokard pada Pasien dengan Infeksi HIV: Laporan Kasus: Laporan Kasus Sakina, Adriani; Rahmah, Fitri
Cermin Dunia Kedokteran Vol 53 No 02 (2026): Kedokteran Umum
Publisher : PT Kalbe Farma Tbk.

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.55175/cdk.v53i02.1554

Abstract

Introduction: The association of myocardial infarction and HIV infection is influenced by many risk factors, either the specific risk factors of cardiovascular and metabolic disease, or HIV-related risk factors related to the immunologic process and the effects of antiretrovirals (ARV). Case: A 43-year-old man on ARV therapy presented with chest pain as heaviness under the left breastbone and referred to the shoulder to the back for 24 hours. The ECG showed ST-segment elevation in leads II, III, and aVF. The patient underwent intensive cardiac care and receivedaspirin therapy with a loading dose of 160 mg, followed by a maintenance dose of 80 mg daily, clopidogrel therapy with a loading dose of 300 mg, followed by a maintenance dose of 75 mg daily, and intravenous anticoagulant enoxaparin injection therapy, 75 mg twice daily. Thepatient also finally stopped smoking. Discussion: Myocardial infarction in people with HIV is a multifactorial condition influenced by various risk factors, chronic inflammation associated with HIV, endothelial dysfunction, and the effects of certain ARV therapies. The combination of tenofovir-based ARVs and efavirenz has not been shown to increase the risk of myocardial infarction and even has a protective effect on lipid profiles. Conclusion: In general, management of myocardial infarction in HIV-infected patients did not differ from that of general population;one important issue being the drug interaction of ARV with antiplatelet agents and statins. The prognosis of HIV-infected patients who had myocardial infarction did not differ from that of those without. However, acute myocardial infarction recurrence in the HIV-infected population is higher than general population.
Co-Authors Fitri Rahmah