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Hubungan Dehidrasi Terhadap Memori Segera/Atensi Mochamad Bahrudin; Annisa Bunga Nafara
Saintika Medika Vol. 15 No. 1 (2019): JUNI 2019
Publisher : Universitas Muhammadiyah Malang

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.22219/sm.Vol15.SMUMM1.8487

Abstract

Dehidrasi adalah kondisi kekurangan atau kehilangan cairan dari seluruh kompartemen tubuh. Kekurangan cairan minimal 1% dapat mempengaruhi fungsi kognitif otak termasuk penurunan kemampuan daya ingat jangka pendek. Penelitian ini untuk mengetahui hubungan dehidrasi terhadap memori segera/atensi. Rancangan penelitian dengan True Experimental, Pre test and post Test with Control Group Design dengan dua kelompok, kontrol dan kelompok perlakuan (induksi dehidrasi ringan dengan treadmil 5,6 km/jam selama 40 menit, kemudian istirahat 20 menit). Instrumen yang digunakan adalah WAIS Digit Span Backward. Analisa data menggunakan Uji Chi-Square, Mann-Whitney Test, Nonparametric Correlations Spearman’s dan uji Regression. Dari penelitian ini didapatkan hasil prosentase penurunan memori segera/atensi pada kelompok tidak dehidrasi 16,1%, kelompok dehidrasi.adalah 30,6% dan Pada Uji Chi-Square signifikansi P= 0,02. uji NP Nonparametric Correlations Spearman’s signifikansi P=0.004 artinya ada korelasi antara beratnya dehidrasi dengan penurunan memori segera/Atensi dan pada uji regressi didapat korelasi negatif artinya semakin besar nilai dehidrasi maka semakin berat derajat penurunan memori segera/Atensi. Dari penelitian ini didapatkan hubungan dehidrasi terhadap memori segera/atensi dengan korelasi negatif.
Acute Malignant Transformation as A Rare Complication of Middle Cerebral Artery Infarction Annisa Bunga Nafara; Ika Yulieta Margaretha Permatasari
Magna Neurologica Vol. 4 No. 1 (2026): January
Publisher : Department of Neurology Faculty of Medicine Universitas Sebelas Maret

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20961/magnaneurologica.v4i1.2644

Abstract

Background: Malignant ischemic stroke is a stroke characterized by extensive acute edema resulting in a space-occupying lesion. This transformation occurs in 10% of ischemic strokes with a mortality rate up to 80%. Hence, it is crucial to early detection and timely treatment. Case: A 51-year-old male was diagnosed with ischemic stroke, presented with NIHSS 11 and ASPECTS 4. Within 26 hours, the patient's level of consciousness declined progressively from a GCS of 15 to a GCS of 10. Serial brain imaging using CT scan and MRI revealed infarct expansion, a space-occupying lesion, and further midline shift. During decompressive craniectomy, extensive edema was found without hemorrhage, suggesting malignant ischemic stroke. After 6 months, the patient had undergone cranioplasty, with no significant complaints, but left hemiparesis remained. Discussion: Malignant ischemic stroke occurs within 5 days after onset. Diagnosis of malignant complication should be considered in ischemic stroke patients with younger age, higher NIHSS, not receiving thrombolysis, neurological status decline in 4-6 hours after onset, wide hypoattenuation in MCA territory, and signs of progressive space-occupying lesion in brain imaging. Management of malignant ischemic stroke consists of managing intracranial pressure with pharmacology and decompressive craniectomy. Conclusion: Malignant transformation is a rare complication of ischemic stroke. Early and accurate diagnosis is crucial to determine the prognosis. Pharmacological therapy and decompressive craniectomy surgery are considered life-saving therapies, but are not able to reduce morbidity in the patient.