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I Made Rama Putra
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Medicine & Pharmacology

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KORELASI ANTARA KONSUMSI ALKOHOL DAN FRACTIONAL URIC ACID CLEARANCE (FUAC) PADA POPULASI SUKU BALI DI DESA PENGLIPURAN, KUBU, BANGLI Rama Putra, I Made; Raka Putra, Tjokorda
journal of internal medicine Vol. 11, No. 3 September 2010
Publisher : journal of internal medicine

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Abstract

Hyperuricemia is a term for serum uric acid (UA) level above normal limit, cause by overproduction or underexcretion.Fractional Uric Acid Clearance (FUAC) can determine both conditions. One of many factors affect to FUAC is alcohol.A cross sectional analytic study perform on Balinesse people in Penglipuran Village, to know correlation between alcoholconsumption and FUAC. We arrange a questionaire with anthropometric data, physical and laboratory examinations, to collectdata on September 2009. Blood and urine examinations perform in Laboratorium Klinik Prodia Denpasar. Data was analyzed bycomputer software, result in charactericstic descriptions, Kolmogorov-Smirnov tests for data normality, Spearman!s correlationto analyze dependent variable and independent variable, and multiple linear analysis, with 95% conÞ dent interval (CI) andsigniÞ cance value p < 0.05.Ninety people fulÞ lled inclusions criteria, consist of 30 (33.3 %) female and 60 (66.7%) male. Mean age was 42.40 ± 13.91years, mean UA serum were 5.19 ± 1.40 mg/dl, with 79 people (88.9 %) normouricemia and 11 people (11.1 %) hyperuricemia.Alcohol consumption prevalence was 47.8%. Mean FUAC were 7.92 ± 2.97%. Spearman!s correlation coeÞ cients (r) were -0.24;p = 0.02 between alcohol consumption and FUAC, while Ancova multivariate result none of variable signiÞ cant to FUAC, butalcohol consumption. In conclusion, there is a signiÞ cant correlation between alcohol consumption and FUAC.
THROMBOPHILIA KARENA DEFISIENSI PROTEIN C DAN PROTEIN S Rama Putra, I Made; Suega, Ketut
journal of internal medicine Vol. 11, No. 2 Mei 2010
Publisher : journal of internal medicine

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Protein C is a natural anticoagulant, inhibits thrombin generation. Protein C de! ciency, is associated with a variablyincreased risk of thrombosis. Incidence of symptomatic protein C de! ciency is approximately 1 in 16,000 to 1 in 32,000 persons,similar in men and women. Morbidity in protein C de! ciency greatly increases with advancing age, when patients are at greaterrisk for thrombotic events such DVT, PE, postphlebitic and blindness secondary to vitreous hemorrhages may occur in patientswith these severe conditions. The etiology can be inherited and acquired in a number of clinical scenarios include meningococcalseptic shock; DIC; liver disease; or chemotherapy. Diagnostic base on personal history of venous thrombosis, thromboembolicevents, abnormal laboratory coagulation tests. The laboratory workup should include haemostatic parameters, coagulationsystem such aPTT, PT, ! brinogen level and D-dimer test. A decreased protein C activity level is required. Imaging studies areappropriate for assessing the presence of thrombotic disease in a particular vessel. The treatment depends largely on a particularpatient?s disease manifestations. Surgical treatment may be appropriate in some circumstances.We reported, a male Balinese 62 years old came with complain red vision and dull feeling since a moth prior toadmission. No historical medical problem found. Physical exam found hypertension and laboratory ! nding showed slightdyslipidemia. Ophthalmologic examination revealed BRVO (Branch Retinal Vein Occlusion) ocular sinistra. Hypercoagulablaestate are suspected. Hematologic exam revealed light hyperagregation and low level of protein C and protein S. Treated withheparin 5000 unit intravenously followed by drip 1000 unit per hour for 3 day continued with warfarin 4 mg OD reach INR 1.5? 2.5. Good resulted base on patient?s good compliance.
Co-Authors Ketut Suega Tjokorda Raka Putra