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All Journal Jurnal Indonesia Sosial Teknologi
Pramarta Y. Dwiputra
Department of Internal Medicine Buleleng Regional General Hospital Bali, Indonesian
Computer Science & IT Economics, Econometrics & Finance Environmental Science Law, Crime, Criminology & Criminal Justice Social Sciences

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A Case of Tuberculous Peritonitis Accompanied By Tuberculous Pleuritis I Larasati A.Wahyu; Pramarta Y. Dwiputra
Jurnal Indonesia Sosial Teknologi Vol. 5 No. 4 (2024): Jurnal Indonesia Sosial Teknologi
Publisher : Publikasi Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.59141/jist.v5i4.1009

Abstract

Tuberculous peritonitis is a form of extrapulmonary tuberculosis, a peritoneal or visceral inflammation caused by Mycobacterium tuberculosis. The disease is rarely independent but is usually a continuation of the tuberculosis process elsewhere, especially pulmonary tuberculosis. We report a case of TB peritonitis accompanied by TB pleurisy, a 29-year-old female patient with complaints of an enlarged abdomen, heartburn, fever, diarrhoea, and decreased appetite. Treatment history was Acitral, Zinc, and Metronidazole. On physical examination, it was found that the general condition was weak, and the axilla temperature was 39.5ºC. Thorax examination: decreased vesicular sound on the left chest. Abdominal examination found distension, epigastric tenderness, undulation, shifting dullness, checkerboard phenomenon, and increased bowel noise. Laboratory examination of complete blood within normal limits. Complete stool; yellow color, mucus (+), leukocytes 4-6/LPB, bacteria (+). The thorax photo showed left pleural effusion, BOF 3 position: ascites. Abdominal ultrasound results: thickening of the peritoneum, ascites, suspected TB peritonitis. Results of ascites and pleural fluid analysis: rivalta (+) and Adenosine Deaminase (ADA) increased. From anamnesis, physical examination and supporting examination can be established to diagnose TB peritonitis and TB pleuritis, followed by OAT therapy and FDC for 12 months. From this case, it can be concluded that clinical and supporting examinations (radiology) are needed to diagnose correctly, and body fluid analysis examinations can help confirm the diagnosis.
Differences The Incidence of Hyperuricemia in Families with Gouty Arthritis and Families Without Gouty Arthritis in Buleleng Regional General Hospital, Bali, Indonesian Larasati A.Wahyu; Pramarta Y. Dwiputra
Jurnal Indonesia Sosial Teknologi Vol. 5 No. 4 (2024): Jurnal Indonesia Sosial Teknologi
Publisher : Publikasi Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.59141/jist.v5i4.1010

Abstract

The most common manifestation of hyperuricemia is gout. Hyperuricemia is the leading cause of gout. It is about five times more common than gout. The heritability of hyperuricemia and gout is about 73%, and 40-50% of patients have a family history of gout. Indonesia, especially Bali, has a high prevalence of hyperuricemia in families with gout at 26.6%. Therefore, we performed this study to look for differences in the incidence of hyperuricemia in families with gouty arthritis and families without gouty arthritis. In an unpaired case-control study, which is the target population is Balinese, the target population is Balinese individuals with gouty arthritis who come to the Internal Medicine Polyclinic of Buleleng Regional General Hospital. The controls in this study were Balinese individuals without gouty arthritis; there were 41 samples who made a pedigree and checked serum uric acid at one time. Of the total 41 case-control samples used, the incidence of hyperuricemia in families with gouty arthritis is 65.9%, and in families without gouty arthritis, is 29.3%, with an Odd Ratio (OR) value of 2.25 with a 95% confidence interval, p-value = 0.002. There were differences in hyperuricemia incidence in families with gouty arthritis and families without gouty arthritis with a p-value = 0.002. Hyperuricemia in families with gouty arthritis is 2.25 times (OR) higher than in families without gouty arthritis.
Co-Authors I Larasati A.Wahyu Larasati A.Wahyu