Hendiperdana, Mochamad Rizky
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Acute Anterior Reinfarction Complicating with Transient Symptomatic Total Atrioventricular Block Hendiperdana, Mochamad Rizky; Firdaus, Isman
Jurnal Kardiologi Indonesia Vol 43 No 3 (2022): Indonesian Journal of Cardiology: July - September 2022
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.1216

Abstract

Background Acute stent thrombosis is a frequent cause of myocardial infarct (MI) after stent placement. Total atrioventricular (AV) block is frequently become the conductive disturbance complication of acute reinfarct. Inferior MI has Low long-term mortality and greater reversibility than anterior MI which has higher in-hospital and long-term mortality. Case Illustration A 44-year-old man, came to emergency department Cardiovascular Centre Harapan Kita with altered mental status since 12 hours ago. PPCI stenting at proximal LAD of his acute anterior MI 2 days ago. Patient had acute stent thrombosis then underwent urgent PCI at referring hospital. Patient present with blood pressure 57/30, heart rate 20 -30 with TAVB rhythm. Laboratory showed increased serum lactate level 5.2. Patient was diagnosed with Total AV block caused by MI. Patient was planned for emergency temporary pacemaker (TPM) implantation. After 24 hours close monitoring, the patient intrinsic rhythm resolved with spontaneous recovery. Patient was hemodynamically stable until discharge. Discussion Stent thrombosis of proximal stent of LAD will cause TAVB because of the source of the distal portion of the AV node is originating from septal branch of LAD. It is caused by extensive necrosis with higher in-hospital and long-term mortality, often culminated in permanent pacemaker. However, spontaneous recovery of TAVB into sinus rhythm take place. This could be caused by transient reversible ischemia of infra nodal region of AV node which supplied by septal perforator branch Conclusion This case reporting a complete atrioventricular block during the course of acute anterior reinfarct and had spontaneous resolution of the AV block. Mechanisms of spontaneous resolution of complete AV block in the setting of acute MI is associated transient ischemia after occlusion of proximal LAD.This article has a related Erratum.
Tatalaksana Farmakologi takiaritmia supraventrikel pada pasien dengan pembesaran jantung kanan: Serial kasus Hendiperdana, Mochamad Rizky
Jurnal Kardiologi Indonesia Vol 46 No 1 (2025): January - March, 2025
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.1750

Abstract

Background Right atrial (RA) enlargement is a common finding in patients with pulmonary hypertension (PH). Supraventricular arrhythmia (SVA) is common in PH patients with RA enlargement. Treatment of SVA should be aggressive since it can cause hemodynamic worsening consequences because RA function plays an important role in right heart function. Case Illustration Three cases of SVA in underlying right heart enlargement with preserved ventricular function that successfully managed by pharmacological cardioversion according to the guidelines. The first case describes atrial flutter with right bundle branch block (RBBB) morphology which successfully converted to sinus rhythm by amiodarone (class III antiarrhythmic drug) administration, meanwhile the second and third cases demonstrate paroxysmal SVA that converted to sinus rhythm by diltiazem (class IV antiarrhythmic drug) administration. Conclusion Supraventricular arrhythmia is a frequent arrhythmia that occurs in pulmonary hypertensive and right heart dilation patients. The tachyarrhythmia in this patient population tolerated poorly and led to hemodynamic perturbation. Pharmacological cardioversion is one of the effective approaches to alleviate patient symptoms with significant clinical improvement.
An Acute Anterior Reinfarction Complicating with Transient Symptomatic Total Atrioventricular Block Hendiperdana, Mochamad Rizky; Firdaus, Isman
Jurnal Kardiologi Indonesia Vol 46 No 4 (2025): October - December, 2025
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.2007

Abstract

In “An Acute Anterior Reinfarction Complicating with Transient Symptomatic Total Atrioventricular Block” (Indonesian Journal of Cardiology, 43(3), 130-6. https://doi.org/10.30701/ijc.1216), there is an error noted. An error has been found in the PDF version of this article. The DOI printed in the PDF is incorrect. The correct DOI is https://doi.org/10.30701/ijc.1216. The error occurs only in the PDF; the DOI listed in the article metadata is already correct. In the original published version of this article, there is an error in the author’s name. The author’s name has been changed to “Mochamad Rizky Hendiperdana” from the previous “Rizky Hendiperdana.”The publisher apologizes for any inconvenience caused by this error.DOI of original article: https://doi.org/10.30701/ijc.1216
Myocardial Remission in High Burden Outflow Tract Premature Ventricular Complex-Induced Cardiomyopathy after Radiofrequency Catheter Ablation: Case Report Hendiperdana, Mochamad Rizky; Maharani, Erika
Jurnal Kardiologi Indonesia Online First
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.1844

Abstract

Background: Premature Ventricular Complex (PVC)-induced Cardiomyopathy (PVC-CMP) is a spectrum of dilated cardiomyopathy. Case Illustration: A 51-year-old female patient who was diagnosed with high suspicion of PVC-CMP underwent successful 3D mapping radiofrequency catheter ablation with a good result. Post-ablation 24-hour ECG-Holter evaluation showed a significant reduction of PVC burden. Echocardiographic evaluation 5 months post-ablation showed improvement in left ventricular systolic function parameters. The presence of high-burden PVC with a typical outflow tract origin could raise suspicion of a specific PVC-CMP aetiology. PVC burden emerged as a major predictor of the development of CMP. Several criteria can be used to identify PVC-CMP. Our case met those descriptive criteria, increasing the likelihood of PVC-CMP. Conclusion: PVC-CMP should be considered in patients with dilated cardiomyopathy who are accompanied by frequent outflow tract origin PVC (> 10 % burden). Early recognition of PVC-CMP is essential, as removal of the primary aetiology improves ventricular structure and function.