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Raymond Pranata
Contact Email
raymond_pranata@hotmail.com
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+6282112918892
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ijc@inaheart.org
Editorial Address
Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
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Dki jakarta
INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 3 Documents
 1 
Search results for , issue "Vol. 26, No. 1 Januari - Maret 2002" : 3 Documents clear
Efek Enhanced External Counterpulsation Terhadap Perbaikan Kontraktilitas Miokard yang masih viabel pada penderita penyakit jantung koroner Utojo Lubiantoro; Fadilah Supari; Lies Dina Liastuti
Jurnal Kardiologi Indonesia Vol. 26, No. 1 Januari - Maret 2002
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v26i1.271

Abstract

Objective: To obtain data about the effect of EECP on dysfunctional but viable myocardial contractility in patient with coronary heart disease.Methods: This is an experimental study which include twenty male coronary heart disease but viable myocardium. Before EECP treatment, all subjects under 2D echocardiographic examination and myocardial contractility was measured as WMSI. Patients is given EECP 1 hour daily (5-6 times a week) for a total of 36 hours. Immediately after EECP treatment, WMSI is measured again for each subject. The value between before and after EECP treatment is analyzed with Wilcoxon signed rank test. Significant result if p < 0,05.Results: WMSI decreased after EECP treatment from 1,58+0,39 to 1,53+0,40, but the difference was not significant.Conclusion: EECP does not improve dysfunctional but viable myocardial contractility in patients with coronary heart disease
Perubahan Nilai Aggregasi Platelet Pada Penderita Sindrom Koroner Akut dengan Terapi Enoxaparine dibandingkan dengan Terapi Heparin Tak Terfraksinasi Ridwan Sofyansyah; Otte J Rachman
Jurnal Kardiologi Indonesia Vol. 26, No. 1 Januari - Maret 2002
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v26i1.272

Abstract

Tujuan dan Latar Belakang: Pengaruh heparin tak terfraksinasi (UFH) terhadap platelet dapat menyebabkan platelet inhabitasi atau ativasi. Menginhibsi platelet melalui ikatan heparin - AT III dapat mengurangi produksi tromblin. Pengaruh heparin pada fungsi platelet dapat meningkatkan produksi tomboxan A2 dan inhibitasi produksi dari prostacyclin I2. Percobaan ini didesain untuk meneliti pengaruh UFH atau enoxaparine pada aggregasi platelet pada pasien dengan sindrom koroner akut.Metoda: Penelitian ini melibatkan 33 pasien dengan sindrom koroner, 17 pasien menerima UFH dan 16 pasien menerima enoxaparine. Penelitian ini merupakan penelitian acak yang terbuka. Penelitian aggregasi platelet dilakukan terhadap semua pasien sebelum dan setelah 5 hari pemberian UFH atau enoxaparine. Peneliti menggunakan program komputer SPSS versi 10\0 untuk menganalisa data.Hasil: Aggregasi platelet menurun pada kelompok enoxaparun dari 29,5+11,8% ke 21,6+10,3%, p=0,04 (dengan 5 um ADP) dan dari 44,1+16% ke 33,6+10,8%, p=0,04 (dengan 10 um ADP). Dalam kelompok UFH dari 25,7+13,5% ke 22,6+12,5%, p=0,46 (dengan 5 um ADP) dan 39,2%+14,4% ke 37,4+14,3%, p=0,45 (dengan 10 um ADP).Kesimpulan: Terjadi penurunan aggregasi platelet pada pasien dengan sindroma koroner akut yang mendapatkan terapi enoxaparine tapi tidak pada pasien yang mendapatkan terapi UFH.
Pengaruh Angioplasti Koroner Terhadap perubahan Dispersi QT Pasca Infark Miokard Pudjo Harasto; Rossana Barack; Anasthasia Sari; Otte J Rachman
Jurnal Kardiologi Indonesia Vol. 26, No. 1 Januari - Maret 2002
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v26i1.273

Abstract

Latar Belakang: Artimia ventrikular adalah penyebab yang paling umum dari kematian jantung mendadak dalam pasca infark miokard (IM). Banyak tanda-tanda dari kematian jantung mendadak pasca infark miokard yang telah dikenali. Salah satu dari tanda-tanda ini adalah dispersi QT yang meningkat pada kejadian iskhemia. Penelitian terdahulu mengungkapkan korelasi antara dispersi QT dan artimia ventrikular. Kami mengevaluasi pengaruh dari angioplasti koroner pada dispersi QT pada penderita pasca-IM.Metoda dan Hasil: Kami mengukur QT dan mengoreksi dispersi QT (QTc) sebelum angiopati, 24 jam dan 33 hari setelah prosedur. 37 Pasien diikutsertakan dalam penelitian ini, tetapi hanya 24 pasien yang dapat dianalisa. Garis acuan dispersi QT adalah 101,67+38,75 (115,02+36,82) ms. Berlainan halnya dengan dispersi QT, dispersi QT yang terkoreksi menurun secara bermakna menjadi 94,50+36,05 ms (p=0,018 dibanding garis acuan) setelah 24 jam. Evaluasi selanjutnya ditemukan bahwa dispersi QT tidak berubah terhadap garis acuan setelah 33 hari.Kesimpulan: Disimpulkan bahwa dispersi QT (QTc) pada pasien pasca-IM tidak menurun setelah angioplasti dengan berhasil.

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