cover
Contact Name
Raymond Pranata
Contact Email
raymond_pranata@hotmail.com
Phone
+6282112918892
Journal Mail Official
ijc@inaheart.org
Editorial Address
Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
Location
Kota adm. jakarta barat,
Dki jakarta
INDONESIA
Indonesian Journal of Cardiology
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Articles 11 Documents
Search results for , issue "Vol. 28, No. 1 Januari 2007" : 11 Documents clear
Perkembangan Kedokteran Kardiologi di Indonesia Layanan, Pendidikan Dokter Spesialis dan Pendidikan Lanjutan di Bidang Kardiologi Muhammad Munawar
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.15

Abstract

Layanan Jantung Anak di Indonesia
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.16

Abstract

Low Cardiac Output Syndrome pada Anak Pasca Operasi Jantung Terbuka PW Busro; EM Marwali; N Budiwardhana; CG Koto; PS Roebiono; AU Rahajoe; GM Harimurti; D Fakhri; J Rahmat
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.17

Abstract

Background. A Low cardiac output syndrome (LCOS) was defined as poor perfusion due to transient myocardial dysfunction.The purpose of this study was to identify patients at risk for the development of LCOS in pediatric after cardiac surgery.Methods and results.The patient characteristics that were independent predictors of LCOS were identified among472 consecutive pediatric pa-tients who underwent cardiac surgery at the National Cardiovascular Center – Indonesia between January 2005 and December2005. The overall preva-lence of LCOS was 15.43 % (n= 73). Logistic regression analyses identified  eight independent predictors of LCOS and calculated the factor-ad-justed odds ratiosassociated with each predictor: (1) residual lesion (odds ratio 141.98); (2) complexity score(odds ratio 1.74);(3) Cardiopulmonary bypass (CPB) time (odds ratio1.01); (4) preoperative Intensive Care Unit (ICU) (odds ratio 8.51); (5) preoperative Congestive Heart Failure (CHF) (odds ratio 3.14); (6) bleeding (oddsratio 24.88); (7) arrhythmia (odds ratio 4.78); and (8) pulmonary hypertension (odds ratio3.75). The opera-tive mortality rate was higherin patients in whom LCOS developed than in those in whom it did not develop (39.72% versus 0.75%, p<0.001). Mean basic complexity score was 6.25 with mortality rate 6.76, and the overall performance was 5.83.Conclusions. Compared to STS and EACTS, the performance of National Cardiovascular Center– Indonesia was still lower. LCOS caused longer time of intubation time, ICU and hospital stay. There were eight independents predictor that can be used to predicts LCOS in pediatric patients after open heart surgery. Patients at high risk for the develop-ment of low cardiacoutput syndrome should be the focus of more inten-sive management to prevent the development of LCOS.
Natrium Urin Petanda Surrogate Brain Natriuretic Peptide Plasma Pasien Infark Miokard dengan Elevasi ST? Januar W Martha; Hariadi Hariawan; S Taofan; Nani Hersunarti; Bambang B Siswanto
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.263

Abstract

Background. High level of Brain Natriuretic Peptide (BNP) was suspected as a predictor of adverse events in acute ST Elevation Myocardial Infarc-tion (STEMI). Unfortunately, the cost and availability of BNP assay had hampered its use. Natriuresis is one of the hallmark effect of raised BNP and measuring urinary sodium might offer insight to the plasma BNP. This study aims to search for any correlation between measured plasma BNP and urinary sodium in patients presenting with acute STEMI.Methods. In an observational descriptive analytic study, we selectively in-cluded patients presenting with acute STEMI and checked for plasma BNP and urinary sodium. All patients had no symptoms or therapy of heart failure prior to admission, had no prior MI, had no valvular abnormalities, and had normal renal function. Plasma BNP was tested using immunoassay method from Abbot Diagnostics, while urinary sodium with ionic specific electrode method. Specimen for plasma BNP were taken at the admission while urinary sodium in the next morning, taking account of urine volume. Results. There were 17 patients, 15 were (82.4%) men, with age 55.1+8.2 years old, onset of STEMI 18.6+2.3 hours, and left ventricular ejection fraction (LVEF) 47.6+11.6%. The urinary sodium was 85.1+34.3 mEq/L, and plasma BNP 449.7+48.8 pg/ml. Pearson’s correlation and linear re-gression analysis showed positive correlation between urinary sodium and plasma BNP (r = 0.71). In multivariate analysis, plasma BNP (p<0.01) and LVEF (p<0.05) were the major influencing factors for urinary sodium level.Conclusion.This study revealed a strong correlation between plasma BNP and urinary sodium in patients presenting with acute STEMI. While mea-surement of urinary sodium cannot replace plasma BNP, it might actually reflect plasma BNP level.
Kadar Ischemia Modified Albumin pada pasien Angina Pektoris Stabil yang menjalani Percutaneous Coronary Intervention Hariadi Hariawan; Otte J Rachman; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.264

Abstract

Background. Ischemia Modified Albumin (IMA) is a biomarker of myocardial ischemia. Blood levels of IMA rise in patients who develop ischemia during percutaneous coronary intervention (PCI). This study evaluated the relation between IMA levels and variables of procedure during coronary angioplasty such as number of balloon inflations, inflation pressure and duration of inflations in patients with stable angina pectoris who underwent PCI. Methods and results. Twenty three consecutive patients (21 men and 2 women), range of age 41 to 80 years old with stable angina pectoris underwent elective PCI between March to April 2006. Blood samples were drawn from femoral or radial artery sheath 10 minutes before PCI and 10 minutes after the last balloon inflation for IMA levels measurement. IMA levels increased positive from 95.26+8.44U/mL before PCI to a level 135.07+18.80U/mL after PCI (p<0.0001). All of the patients used stents except in 2 cases. There are significant correlation between elevation of IMA levels with number of balloon inflation (r=0.59;p=0.003), total pressure inflation (r=0.48;p=0.02) and total duration of balloon inflation (r=0.48;p=0.02).Conclusion. Ischemia Modified Albumin (IMA) levels significantly elevated after percutaneous coronary angioplasty (PCI). There were positive correlation between numbers of inflations, total pressure and total duration of balloon inflations.
Distorsi Terminal Komplek QRS pada Infark Miokard Akut Inferior sebagai prediktor kejadian High-degree AV Block A Sewianto; M Kasim; M Yusak
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.265

Abstract

Background. ST elevation in acute myocardial infarction can predict the size of infarction, response to reperfusion therapy, and prognosis. Birnbaum et al. showed that early QRS distortion is a reliable prediction of the devel-opment of advanced AV block among patients receiving thrombolytic therapy for inferior wall acute myocardial infarction. Distortion of the ter-minal portion of the QRS in inferior wall acute myocardial infarction based on J point / R wave ratio of more than 50% in at least two leads of the inferior leads (II, III, and aVF). Although transient, development of heart block during inferior infarction is associated with a high in-hospital mortal-ity rate, even they received thrombolytic therapy. Some studies showed ethnic differences in electrocardiogram amplitudes. The aims of this study is to evaluate the correlation between early QRS terminal distortion and high degree AV block among our patients with inferior acute myocardial infarction treated by thrombolytic.Methods and results. This study is a cross-sectional study on patients with inferior wall acute myocardial infarction treated by thrombolytic at National Cardiovascular Center – Harapan Kita, Jakarta during January 2000 until December 2004, that fulfill inclusion and exclusion criteria. They were divided into two groups, those with QRS distortion and without QRS dis-tortion. Correlation between the two groups were analyzed by t test, chi-square and multivariate regression analysis. There are 186 patients, ages between 37 until 72 years old, mostly men (89%) which are 93 patients with QRS distortion and 93 patients without QRS distortion. Those two groups were comparable. With univariate analysis, the group with QRS distortion have higher ST segment deviation (9,61±3,67 vs 7,76±3,53, p=0,001) and higher risk of failed thrombolytic (74,2%vs60,2%, p=0,042).With multivariate regression analysis, there is a significant correlation be-tween QRS distortion and high-degree AV block (OR 2,5; 95% CI 1,04-6,01; p=0,04), most AV block happened during hospitalization.Conclusions. Patients with distortion of the QRS terminal portion in in-ferior acute myocardial infarction and treated by thrombolytic have a higher risk of high-degree AV block during hospitalization, compared to patients without QRS distortion.
Prediktor Kegagalan Intervensi Koroner Perkutan pada Oklusi Total Koroner Kronik di Pusat Jantung Nasional, Indonesia I Gumiwang; Muhammad Munawar; RWM Kaligis
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.266

Abstract

Background. Percutaneous Coronary Intervention (PCI) in patients with Chronic Total Coronary Occlusion (CTO) is associated with higher rate of failure and higher rate of complication compared to non-CTO angioplasty. Improvement in technique, logistic and patient’s selection method lead to a better success rate. Identification of predictor of failure could be an important step in patient selection. The aim of the study is to know the predictors of failure of PCI in patients with CTO.Methods and results. A retrospective analysis of clinical and angiographic data of 78 consecutive eligible CTO patients who underwent PCI selected in series of 1205 total occluded vessel of 3654 angiographic patients in the year of 2005 in our catheterization laboratory. We analysed 25 variables, 12 clinical variables (age, sex, family history, smoking, diabetes mellitus, hypertension, history of myiocardial infarction, history of coronary bypass operation, age of occlusion >1 year, severe angina and poor left ventricle systolic dysfunction)and 13 angiographic variables (true CTO, CTO location, ostial lesion, calcification, tortousity, non-tapered type, side branch type, bridging collateral, diffuse disease, vessel diameter < 3mm, CTO length > 15mm, multi-lesion and multi vessel disease)by univariate and multivariate analysis (logistic regression) in association between 21 cases of procedural failure group and 57 cases of procedural success group. Procedural success was achieved in 57 patients (73%) and complication occured in one patient (1%). Majority of patients are male with mean age 55 year. Presence of calcification is the only predictor identified by univariate analysis (OR 3,28. p 0,04. 95%CI 1.05-10,18). Multivariate analysis identified multivessel disease (OR 7,1. p 0,07 .95%CI 0,85-59,21) and diffuse disease (OR 2,7. p 0,06 .95%CI 0,93-8,08) as predictors of procedural failure.Conclusions. We identified multivessel disease and diffuse disease as two predictors of procedural failure of PCI in our series of CTO patients with 73% success rate and 1% complication rate in the year of 2005.
Pengaruh Terapi Oksigen Hiperbarik terhadap kadar Vascular Endothelial Growth Factor (VEGF) pada pasien Ulkus Diabetik MR Akbar; RWM Kaligis; M Kasim
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.267

Abstract

Background. Hyperbaric oxygen therapy (HBOT) increase endothelial oxygenation and stimulates vascular endothelial growth factor(VEGF) as the most specific and potent growth factor for angiogenesis, and increases wound healing process. The aim of the study is to know if five days HBOT can increase the level of VEGF in diabetic ulcer patients. Methods and results. Clinical experimental study was conducted on 12 diabetic ulcer patients who received HBOT 30 minutes, 3 times a day for 5 days (HBOT group) and 10 diabetic ulcer patients as a control group who did not receive HBOT (non-HBOT group). The VEGF level in both groups was measured on days 1 and 5. In HBOT group the mean level of VEGF on day 1 was 1241.325 + 237.6533 pg/ml and became 1244.458 + 264.5641 pg/ml (p = 0.583) on day 5, while in non-HBOT group the mean level of VEGF on day 1 was 1262.350 + 227.9603 pg/ml and became 1112.460 + 220.3795 pg/ml (p = 0.093) on day 5. There were no signifi-cant differentiation of VEGF level between HBOT group and non-HBOT group both on day 1 (p = 1) and day 5 (p = 0.872) Conclusions. Hyperbaric oxygen therapy for 5 days did not increase the VEGF level of diabetic ulcer patients.
Stenosis Mitral Kongenital: Parachute Mitral Valve Laporan 2 kasus NIR Asnil; Ganesja M Harimurti; Poppy S Roebiono; Anna Ulfah Rahajoe; J Rachmat
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.268

Abstract

Mitral stenosis kongenital merupakan penyakit jantung bawaan (PJB) yang jarang terjadi. Laporan-laporan terdahulu menyebutkan angka insiden sekitar 0,4-0,8% dari seluruh pasien PJB. Keadaan ini dapat terjadi tersendiri (isolated), atau disertai dengan PJB lainnya, seperti pada  Shone’s complex,atau sebagai bagian dari Hypoplastic Left Heart Syndrome.
Tatalaksana sindroma Wolf-Parkinson-White terselubung pada pasien dengan Alat Pacu Jantung Daf Juzar; Yoga Yuniadi; Muhammad Munawar
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.269

Abstract

Wolf-Parkinson-White syndrome is part of the supraventricular tachycardias, which are relatively common, often repetitive, occasionally persistent and rarely life threat-ening. Management of atrioventricular reciprocating tachycardia (AVRT) in patients with permanent pacemaker (PPM) is rarely being discussed. We would like to report our experience in the management of two cases with AVRT, one on VVIR and another one on AAI. In the first case catheter ablation of the atrioven-tricular juction was performed, while in the second case arrhythmia was termi-nated using burst pacing.

Page 1 of 2 | Total Record : 11


Filter by Year

2007 2007


Filter By Issues
All Issue Vol 46 No 4 (2025): October - December, 2025 Vol 46 No 3 (2025): July - September, 2025 Vol 46 No 2 (2025): April - June, 2025 Vol 46 No 1 (2025): January - March, 2025 Vol 45 No Suppl_A (2024): Vol 45 No Suppl_A (2024): Abstracts of the 6th Indonesian Intensive & Acut Vol 45 No 4 (2024): Online First - Indonesian Journal of Cardiology April-June 2021 Vol 45 No 3 (2024): July - September, 2024 Vol 45 No 2 (2024): April - June, 2024 Vol 45 No 1 (2024): January - March, 2024 Vol 45 No Suppl_C (2024): Abstracts of the 33rd Annual Scientific Meeting of the Indonesian Heart As Vol 45 No Suppl_B (2024): Abstracts of the 11th Annual Scientific Meeting of the Indonesian Heart Rh Vol 44 No 4 (2023): Indonesian Journal of Cardiology: October - December 2023 Vol 44 No 3 (2023): Indonesian Journal of Cardiology: July - September 2023 Vol 44 No 2 (2023): Indonesian Journal of Cardiology: April - June 2023 Vol 44 No 1 (2023): Indonesian Journal of Cardiology: January - March 2023 Vol 44 No Suppl_A (2023): Abstracts of the 32nd Annual Scientific Meeting of the Indonesian Heart As Vol 44 No Suppl_B (2023): Abstracts of the 10th Annual Scientific Meeting of the Indonesian Heart Rh Vol 43 No 4 (2022): Indonesian Journal of Cardiology: October - December 2022 Vol 43 No 3 (2022): Indonesian Journal of Cardiology: July - September 2022 Vol 43 No 2 (2022): Indonesian Journal of Cardiology: April - June 2022 Vol 43 No Suppl_B (2022): Abstracts of the 6th InaPrevent (2022) Vol 43 No 1 (2022): Indonesian Journal of Cardiology: January - March 2022 Vol 43 No Suppl_C (2022): Abstracts of the 31st Annual Scientific Meeting of the Indonesian Heart As Vol 43 No Suppl_D (2022): Abstracts of the 9th Annual Scientific Meeting of the Indonesian Heart Rhy Vol 43 No Supplement (2022): Abstracts of the 5th Indonesian Intensive and Acute Cardiovascular Care Vol 42 No 4 (2021): Indonesian Journal of Cardiology: October - December 2021 Vol 42 No 3 (2021): Indonesian Journal of Cardiology: July - September 2021 Vol 42 No 2 (2021): Indonesian Journal of Cardiology: April - June 2021 Vol 42 No 1 (2021): Indonesian Journal of Cardiology: January - March 2021 Vol 42 No Supplement (2021): Abstracts of the 5th InaPrevent in Conjunction with the 1st InTension S Vol 42 No Supplement (2021): Abstracts of the 13th Indonesian Society of Interventional Cardiology A Vol 42 No Supplement (2021): Abstracts of the 8th Annual Scientific Meeting of the Indonesian Heart Vol 41 No 4 (2020): Indonesian Journal of Cardiology: October - December 2020 Vol 41 No 3 (2020): Indonesian Journal of Cardiology: July - September 2020 Vol 41 No 2 (2020): Indonesian Journal of Cardiology: April - June 2020 Vol 41 No 1 (2020): Indonesian Journal of Cardiology: Januari - Maret 2020 Vol 41 No Suppl_A (2020): Abstracts of the 29th Annual Scientific Meeting of the Indonesian Heart As Vol 41 No Suppl_B (2020): Abstracts of the 12th Indonesian Society of Interventional Cardiology Annu Vol 40 No 4 (2019): Indonesian Journal of Cardiology: October-December 2019 Vol 40 No 3 (2019): Indonesian Journal of Cardiology: July-September 2019 Vol 40 No 2 (2019): Indonesian Journal of Cardiology: April-June 2019 Vol 40 No 1 (2019): Indonesian Journal of Cardiology: January-March 2019 Vol 39 No 4 (2018): Indonesian Journal of Cardiology: October-December 2018 Vol 39 No 3 (2018): Indonesian Journal of Cardiology: July-September 2018 Vol 39 No 2 (2018): Indonesian Journal of Cardiology: April-June 2018 Vol 39 No 1 (2018): January - March 2018 Vol 39 No 1 (2018): January - March 2018 Vol 39 No Suppl_A (2018): Indonesian Journal of Cardiology Supplement_A Vol 39 No Suppl_A (2018): Indonesian Journal of Cardiology Supplement_A Vol 39 No Suppl_B (2018): Indonesian Journal of Cardiology Supplement_B Vol 39 No Suppl_B (2018): Indonesian Journal of Cardiology Supplement_B Vol 38 No 4 (2017): October - December 2017 Vol 38 No 4 (2017): October - December 2017 Vol 38 No 3 (2017): July - September 2017 Vol 38 No 3 (2017): July - September 2017 Vol. 38, No. 2 April-June 2017 Vol. 38, No. 2 April-June 2017 Vol. 38, No. 1 Januari-Maret 2017 Vol. 37, No. 4 Oktober - Desember 2016 Vol. 37, No. 3 Juli - September 2016 Vol. 37, No. 2 April - Juni 2016 Vol. 37, No. 1 Januari - Maret 2016 Vol. 36, No. 4 Oktober - Desember 2015 Vol. 36, No. 3 Juli - September 2015 Vol. 36, No. 2 April - Juni 2015 Vol. 36, No. 1 Januari - Maret 2015 Vol. 35, No. 4 Oktober - Desember 2014 Vol. 35, No. 3 Juli - September 2014 Vol. 35, No. 2 April - Juni 2014 Vol. 35, No. 1 Januari - Maret 2014 Vol. 34, No. 4 Oktober - Desember 2013 Vol. 34, No. 3 Juli - September 2013 Vol. 34, No. 2 April - Juni 2013 Vol. 34, No. 1 Januari - Maret 2013 Vol. 33, No. 4 Oktober - Desember 2012 Vol. 33, No. 3 Juli - September 2012 Vol. 33, No. 2 April - Juni 2012 Vol. 33, No. 1 Januari - Maret 2012 Vol. 32, No. 4 Oktober - Desember 2011 Vol. 32, No. 3 Juli - September 2011 Vol. 32, No. 2 April - Juni 2011 Vol. 32, No. 1 Januari - Maret 2011 Vol. 31, No. 3 September - Desember 2010 Vol. 31, No. 2 Mei - Agustus 2010 Vol. 31, No. 1 Januari - April 2010 Vol. 30, No. 3 September - Desember 2009 Vol. 30, No. 2 Mei - Agustus 2009 Vol. 30, No. 1 Januari - April 2009 Vol. 29, No. 3 September - Desember 2008 Vol. 29, No. 2 Mei - Agustus 2008 Vol. 29, No. 1 Januari - April 2008 Vol. 28, No. 6 November 2007 Vol. 28, No. 5 September 2007 Vol. 28, No. 4 Juli 2007 Vol. 28, No. 3 Mei 2007 Vol. 28, No. 2 Maret 2007 Vol. 28, No. 1 Januari 2007 Vol. 26, No. 1 Januari - Maret 2002 More Issue