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All Journal Indonesian Journal of Clinical Pathology and Medical Laboratory (IJCPML) Indian Journal of Forensic Medicine & Toxicology
Ferdy Royland Marpaung
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Biochemistry, Genetics & Molecular Biology Health Professions Medicine & Pharmacology Neuroscience Public Health

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Gitelman Syndrome in Post-Partum Pre-Eclampsia in Dr. Soetomo Hospital, Surabaya, Indonesia Erix Agus Hasudungan Telaumbanua; Ferdy Royland Marpaung
Indian Journal of Forensic Medicine & Toxicology Vol. 15 No. 3 (2021): Indian Journal of Forensic Medicine & Toxicology
Publisher : Institute of Medico-legal Publications Pvt Ltd

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.37506/ijfmt.v15i3.15746

Abstract

Gitelman Syndrome (GS) is an autosomal recessive tubular kidney disorder. The disorder is rare with a prevalence estimated to be 1 in 40,000 individuals. We closely observed a 28-year-old female with complaints of stiffness and cramps on both hands and feet for 2 weeks and became worse in 3 days before admittance, nausea (+), vomiting (+), shortness of breath (+). Medical records showed that patient underwent post SC 32nd day because of severe pre-eclampsia + IUGR + Oligohydramnion. Physical examination showed GCS 456, blood pressure 140/80 mmHg, heart rate 96x/minute, respiration rate 24x/minute, temperature 37 oC. Laboratory results showed Hb 9.4 g/dl, MCV 86.8 fL, MCH 29.6 pg, MCHC 34.1 g/dL, WBC 7.83x103/uL, PLT 483x103/uL, potassium 2.8 mmol/L, sodium 138 mmol/L, chloride 95 mmol/L, calcium 8.3 mg/dL, magnesium 1.1 mg/dL, phosphate 3 mg/dL, BUN 8 mg/dL, serum creatinin 0.76 mg/dL, urinary calcium 488.4 mg/24 hours, urinary creatinine 488.4 mg/24 hours, proteinuria +1, and blood gas test suggested mixed metabolic alkalosis and respiratory acidosis. Gitelman Syndrome is characterized by hypokalemia, hypomagnesemia, metabolic alkalosis and hypocalciuria. The diagnosis of GS is generally relatively late because of its difficulty to be clinically categorized; therefore, a more comprehensive diagnosis approach is needed. The diagnosis of Gitelman Syndrome is based on clinical symptoms and laboratorial abnormalities and can be confirmed by genetic testing.
GLYCATED ALBUMIN AND HBA1C IN DIABETIC NEPHROPATHY (Albumin Glikat HbA1c dan Penyakit Nefropati Diabetik) Elvan Dwi Widyadi; Jusak Nugraha; Ferdy Royland Marpaung
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 22, No 3 (2016)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v22i3.1242

Abstract

Nefropati diabetik merupakan komplikasi kronis yang sering terjadi di pasien penyakit Diabetus Melitus (DM), perubahan statusglikemik harus diketahui lebih awal untuk mencegah komplikasi lebih lanjut. HbA1c sering digunakan sebagai pengendali glikemikjangka panjang (tiga/3 bulan). Albumin terglikasi merupakan tolok ukur baru yang dikembangkan untuk mengendalikan glikemikdalam waktu yang lebih singkat (2 minggu). Penelitian ini bertujuan untuk mengetahui kenasaban Albumin Glikat (GA) terhadapHbA1c di pasien nefropati diabetik. Dari 89 pasien DM yang diteliti, 34 didiagnosis sebagai diabetes nefropati. Kadar HbA1c diukurdengan metode Turbidimetric Inhibition Immunoassay (TINIA) dan GA diperiksa secara enzimatik. Analisis statistik dilakukan denganmenggunakan kenasaban Pearson, yang bermakna pada p<0,005. Cut off untuk GA: adalah 16% dan untuk HbA1c: 6,5%. RerataGA: adalah 20,73% dan 7,42% HbA1c. Pada penelitian ini, dengan uji Pearson diketahui kenasaban yang baik antara GA dan HbA1c(r=0,785, p-nilai<0,0001). Albumin Glikat memiliki kenasaban yang kuat terhadap HbA1c. Oleh karena itu, GA dapat digunakan untukmendeteksi indeks glikemik dalam jangka waktu singkat (2 minggu) di pasien pengidap nefropati diabetik.
Co-Authors Elvan Dwi Widyadi Erix Agus Hasudungan Telaumbanua Jusak Nugraha