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All Journal Jurnal Kesehatan Reproduksi Jurnal Neuroanestesi Indonesia
Johansyah, Theodorus Kevin Putra
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Biochemistry, Genetics & Molecular Biology Education Health Professions Medicine & Pharmacology Neuroscience Nursing Public Health

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Status Epilepticus Following Labor in a Remote Area: A Case Report of Puerperal Sepsis Silalahi, Andre Martua; Johansyah, Theodorus Kevin Putra; Gusti, Agustinus; Siswantoro, Cahyo Wicaksono
JURNAL KESEHATAN REPRODUKSI Vol 10, No 3 (2023)
Publisher : Fakultas Kedokteran, Kesehatan Masyarakat dan Keperawatan UGM

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.22146/jkr.87457

Abstract

Background: Puerperal sepsis is one of the five leading causes of maternal mortality worldwide, commonly occurs in low to middle income countries. Higher incidence is observed in rural areas where diagnostic and therapeutic interventions may be limited.Case: A a 21-year-old woman who developed a fever, abdominal pain, and foul-smelling vaginal discharge five days after giving birth. The patient convulsed five times in total before being admitted. She was diagnosed with status epilepticus caused by sepsis-associated encephalopathy and puerperal sepsis, which was successfully treated with antibiotics and supportive care.Discussion: Pregnancy and the puerperium period involve significant changes in maternal physiology and immune function. The Society of Obstetric Medicine Australia and New Zealand (SOMANZ) has proposed Obstetric-modified quick sepsis related organ failure assessment (omqSOFA) for early recognition of maternal sepsis. An score of 2 or higher has predictive value for maternal sepsis.Conclusion: Early recognition, appropriate treatment, and close monitoring are crucial for improving outcomes in patients with puerperal sepsis.
Dormant No More: The Neurogical Impact of Herpes Simplex Virus Reactivation Following Traumatic Brain Injury Johansyah, Theodorus Kevin Putra; Soetomo, Cindy Thiovany; Tiffany, Tiffany; Maliawan, Sri
Jurnal Neuroanestesi Indonesia Vol 14, No 1 (2025)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24244/jni.v14i1.659

Abstract

Herpes simplex viruses (HSV), including HSV-1 and HSV-2, are neurotropic viruses capable of establishing lifelong latency in sensory ganglia and reactivating under various triggers, including traumatic brain injury (TBI). TBI induces secondary injury cascades such as neuroinflammation, excitotoxicity, and blood-brain barrier disruption, which create a conducive environment for HSV reactivation. The reactivation of HSV after TBI, particularly HSV-1 and HSV-2, can lead to significant neurological consequences, including encephalitis, cognitive decline, and the development of neurodegenerative diseases like Alzheimers disease and Chronic Traumatic Encephalopathy. Current therapeutic approaches focus on antiviral agents like acyclovir and valacyclovir, which manage acute HSV infection but are less effective in preventing long-term neurological damage. Emerging research highlights the potential of anti-inflammatory and neuroprotective strategies to complement antiviral therapies, aiming to reduce the neuronal damage caused by viral reactivation and inflammation. However, gaps remain in understanding the precise mechanisms linking TBI-induced neuroinflammation to HSV reactivation and its long-term impact on neurological health. This review synthesizes the current literature on the pathophysiology of HSV reactivation following TBI, and their contributions to acute and chronic neurological outcomes
Co-Authors Gusti, Agustinus Silalahi, Andre Martua Siswantoro, Cahyo Wicaksono Soetomo, Cindy Thiovany Sri Maliawan Tiffany, Tiffany