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Raymond Pranata
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Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
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INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 13 Documents
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Search results for , issue "Vol. 32, No. 1 Januari - Maret 2011" : 13 Documents clear
Manfaat Suplemen Antioksidan: Fakta atau Fiksi ? Faisal Baraas
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.113

Abstract

Peranan suplemen antioksidan masih tetap kontroversial sampai saat ini. Boleh dikatakan, belum ada rilis hasil penelitian tentang manfaat antioksidan bagi  penderita penyakit jantung koroner dalam jurnal-jurnal terbaru beberapa tahun belakangan ini sebagai hasil penelitian klinis mutakhir yang bisa dianggap konklusif. Manfaat antioksidan masih tetap kontroversial dan memerlukan penelitian lebih lanjut dengan disain penelitian yang lebih scrutinized dan analisis yang lebih kritis.Tidak ada isu lain di bidang terapi kardiovaskuler yang lebih kontroversial saat ini, melainkan isu tentang peranan antioksidan itu. Seorang pejabat FDA menyebutkan bahwa efektivitas antioksidan dalam pengobatan penyakit jantung memang masih tetap belum konklusif, walau pun sudah banyak penelitian yang dilakukan. Sebagian penelitian itu memang sudah menunjukkan hasil yang positif, tapi sebagian lagi masih tetap menjadi bahan perdebatan yang panjang.Maka pertanyaan, “perlukah suplemen antioksidan diberikan secara rutin pada pasien dengan kelainan koroner – karena manfaatnya dan keamanannya?”, belum bisa dijawab dengan pasti dan masih tetap mengambang.
Hubungan Matriks Metaloproteinase-9 (MMP-9) Dengan Troponin-I (cTn-I) pada Infark Miokard dengan ST-Elevasi (STEMI) dan Sindrom Koroner Akut Tanpa ST-Elevasi (NSTEACS) Budi Yuli Setianto; Indwiani Astuti; Bambang Irawan; Sofia Mubarika
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.114

Abstract

Background: Acute coronary syndrome (ACS) is due to plaque rupture or erosion. Plaque rupture or erosion occurs because of the extra-cellular matrix destruction by an MMP (matrix metalloproteinase). Troponin I (cTn-I) is a biomarker that will increase in ACS with myocardial necrosis.Objective: To determine levels of MMP-9 difference between STEMI and NSTEACS and the relationship between levels of MMP-9 and cTn-I levels between the two groups.Methods: The sample examination performed in 80 patients with ACS (39 STEMI and 41 with NSTE-ACS) prior to the act of intravenous thrombolysis or coronary intervention.Analysis of MMP-9 levels relationship and cTn-I using Spearman test, and analysis of the cut-off relationship MMP-9 in the STEMI and NSTEACS groups were tested by Chi square.Results: Group STEMI had higher levels of MMP-9 and significantly higher compared with NSTE-ACS group (p = 0.002). Spearman correlation test showed a significant and positive correlation between MMP-9 and troponin-I between the two groups (p = 0.003 and r = 0.33).Conclusion: Increased levels of MMP-9 were significantly higher in STEMI compared with NSTE-ACS and its association with elevated levels of tro-ponin-I, provide information about the role of MMP-9 against the severity of heart muscle damage that occurred.
Dapatkah Matrix Metalloproteinase Memprediksi Luasnya Kerusakan Miokard? Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.115

Abstract

Trombosis arteri merupakan dasar terjadinya sindrom koroner akut. Sebagian besar trombosis terjadi pada robekan plak pada bagian  fibrous cap yang tipis. Plak aterom yang memiliki fibrous cap yang tipis dan mudah mengalami robekan sering disebut sebagai vulnerable plaque. Penting dicatat bahwa derajat stenosis plak yang mengalami robekan seringkali hanya  ringan atau sedang saja.3 Para peneliti berhipotesis bahwa kehilangan komponen matriks khususnya serabut kolagen pada fibrous cap menyebabkan penipisan cap. Hal ini akan memudahkan terjadinya robekan baik secara spontan maupun sebagai respon terhadap trigger hemodinamik ata yang lainya.Matrix metalloproteinases (MMPs) agaknya memainkan peran penting dalam remodeling plak aterosklerotik, walaupun berbagai bukti menunjukkan hubungan peningkatan MMP dengan terjadinya instabilitas plak masih kontroversial.
Pengaruh Komposisi Asupan Makan terhadap Komponen Sindrom Metabolik pada Remaja Djanggan Sargowo; Sri Andarini
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.116

Abstract

Obesity as the component of metabolic syndrome and its associated disor-ders are a growing epidemic across the world. Several genetic, behavioral, and physiological factors play a role in etiology of obesity. Behavior factor such as high-carbohydrate and high-fat diet is important as this factor, but not the sedentary factor, is still having possibility to be altered. This study is performed to explain the influence of food intake to the components of metabolic syndrome. The method used is case-control analysis of population-based, epidemiological surveys using the metabolic syndrome definition of International Diabetes Federation. The results suggest that the total cholesterol and waist circumference components have higher pathway co-efficiency than other components. The compositions of food intake causing the metabolic syndrome are total  calories followed by fat and carbohydrate, consecutively. From these results, reduction of total calorie and fat can be recommended as dietary modification that reduces the risk of developing metabolic syndrome.
Pengaruh Makanan Pada Sindrom Metabolik Sutomo Kasiman
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.117

Abstract

Sejak 15 sampai 20 tahun terakhir ini sejumlah perubahan yang berhubungan dengan resistensi insulin termasuk hipertensi, obesitas, hiperinsulinemia, hipertrigliseridemia dan HDL yang rendah sudah dipahami dengan baik. Reaven yang menyatakan bahwa perubahan itu disebut sebagai sindrom metabolik yang bukan suatu penyakit tetapi merupakan sekumpulan kelainan metabolisme dimana penyebab utama sindrom ini saling berinteraksi, yaitu obesitas dan kerentanan metabolisme endogen. The National Cholesterol Education Program (NCEP) Adult Treatment Panel III (ATP III) menyatakan bahwa diagnosis sindrom metabolik harus memenuhi 3 atau lebih faktor risiko yaitu obesitas abdomen, trigliserida, kadar HDL, tekanan darah dan kadar gula darah puasa.
Hubungan Waktu Pemberian Ebtifibatide dengan TIMI Flow Infarct Related Artery pada Intervensi Koroner Perkutan Primer Abdul Hakim Alkatiri; Yoga Yuniadi; Dicky A Hanafy; Doni Firman; Sunarya Soerianata
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.118

Abstract

Background. Optimal administration timing of Glicoprotein IIb/IIIa inhibitor in STEMI patients undergoing Primary PCI is controversial. Several stud -ies have shown that early administration of eptifibatide, which is given to patients with pain awitan of < 90 minutes will improved TIMI grade flow of infarct related coronary artery (IRA). However, significant numbers of patient arrived in emergency room/PCI center with pain awitan of > 3 hours. This study was aimed to evaluated effect of eptifibatide administra-tion timing to TIMI grading flow in first angiographic IRA during primary PCI in patients with STEMI.Methods and result. Of 116 consecutive STEMI patients who underwent primary PCI, 79 patients received ebtifibatide < 90 minutes (Group 1) and 37 patients received eptifibatide > 90 minutes (Group 2) before first angiographic of IRA. There were no significant differences of TIMI 3 flow proportion after PCI between the groups (86.1% vs 83.8% for Group 1 and 2 respectively, p = 0.745). Group 2 showed more frequent TIMI 2 flow (18.9% vs 5.1%, p = 0.036) but tend to have less frequent TIMI 0 flow (56.8 % vs 67.1%, p = 0.281).Conclusion. Patients who received eptifibatide > 90 minutes before first angiographic IRA during primary PCI achieved more appropriate TIMI flow as compare to that received eptifibatide < 90 minutes.
Penghambat GPIIb/IIIa pada Intervensi Koroner Perkutan Primer: Kapan dan Dimana? Doni Firman
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.119

Abstract

Prosedur Intervensi Koroner Perkutan Primer (IKPP) merupakan prosedur pilihan pada pasien STEMI dengan awitan kurang dari 12 jam. Tetapi banyak hal yang masih belum terjawab di antaranya pada pasien dengan awitan dini (2 atau 3 jam) apakah pihannya langsung dirujuk ke RS dengan fasilitas IKPP tetapi dengan konsekuensi adanya keterlambatan atau langsung diberikan trombolitik. Masalah kedua adalah jenis terapi yang diberikan saat transportasi ke rumah sakit rujukan apakah antitrombin heparin, bivalirudin, penghambat GP IIb/IIIa atau trombolitik? Masalah ketiga adalah IKPP tidak selalu berhasil memberi reperfusi. Masalah lain adalah keberhasilan angiografis yang tidak disertai bukti reperfusi yang diharapkan pada pemeriksaan dengan modalitas imajing.
Intoksikasi Digoksin: Bagaimana Cara Mengenali Dan Penatalaksanaannya Dewi H Suprobo; Bambang B Siswanto; Yoga Yuniadi; Ganesja M Harimurti
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.120

Abstract

Background: Digitalis is the oldest compound in cardiovascular medicine that continues to be used in contemporary clinical practice. It is one of the most frequently prescribed medications and has historically been implicated as one of the most common causes of adverse drug reactions. Despite the fact that digitalis preparations have been used  therapeutically for more than 200 years, diagnosis of digoxin toxicity remains difficult. Signs and symptoms associated with toxicity are nonspecific, as are electrocardiographic changes, and the “therapeutic” and “toxic” concentrations overlap.Objective: to present a case report of digoxin intoxication and to review the diagnosis and management of the disease.Summary: We have reported a case related to intoxication of a drug that is one of the most frequently prescribed medications and has historically been implicated as one of the most common causes of adverse drug reactions. This fact is reasonable since digoxin has a narrow margin of safety, where at the therapeutic dosage digoxin could induce intoxication. Moreover the response to this drug is influenced by many factors. Although the general manifestation of digoxin intoxication is not specified, but in patient who are in digoxin therapy with clinical manifestation of digoxin intoxication, we have to put digoxin intoxication as one of our differential diagnosis. The diagnosis is supported by the ECG manifestation and confirmed by the examination of serum digoxin level. Eventhough, normal digoxin level could also induce intoxication. The initial management of digoxin intoxication is early recog -nition that a dysrhythmia and/or noncardiac manifestation may be related to digitalis intoxication and stop the digoxin therapy. Immunotherapy, in hemodynamically stable or unstable patients, is a first-line therapy.
Implantasi Pacu Jantung Permanen Melalui Vena Kava Superior Kiri Persisten Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.121

Abstract

Seorang anak perempuan, 5 tahun mengalami blok AV total pasca operasi VSD perimembranous besar sehingga direncanakan pemasangan pacu jantung permanen. Pada saat punksi vena subklavia kanan wire peelaway sheath tidak dapat masuk ke atrium kanan.Injeksi kontras melalui abocath memperlihatkan oklusi vena cava superior kemungkinan oleh thrombus yang terorganisasi. Samar-samar terlihat kontras mengisi vena kana superior kiri  persisten melalui kolateral dari vena kava superior kanan (Gambar 1). Maka diputuskan untuk melakukan pemasangan pacu jantung menetap (PJM) melalui vena subklavia kiri ke alur keluar ventrikel kanan (RVOT= right ventricle outflow tract) melalui vena kava superior kiri persisten.
Hubungan Obstructive Sleep Apnea dengan Kardiovaskular Debi Febriani; Faisal Yunus; Budhi Antariksa; Hananto Andrianto
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.122

Abstract

Breathing Disorders during sleep seen as potential factors for some Cardio -vascular diseases in last decade. Apnea syndrome during sleep divided into 3 types there are central type, obstruction type and mix type. Obstructive Sleep Apnea (OSA) is the most frequently encountered, marked with apnea episode and hypopnea. There are 4 mechanism relationship of OSA with cardiovascular disease those are mechanical, hemodynamic, neurohor -monal and inflammation. Cardiovascular disease that related to OSA are hypertension, heart failure, acute coronary syndrome, arythmias, stroke, sudden cardiac death. Proper management pro ven improve cardiovascular parameters and increase quality of life.

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