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Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
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INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 11 Documents
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Search results for , issue "Vol. 34, No. 3 Juli - September 2013" : 11 Documents clear
LAA Closure: Where we stand now? Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.334

Abstract

  Insiden fibrilasi atrium diperkirakan akan makin meningkat dimasa yang akan datang. Stroke merupakan persoalan besar pada fibrilasi atrium dengan risiko hingga 5 kali lebih besar dibandingkan pasien tanpa fibrilasi atrium. Bahkan risiko stroke tersebut tidak berbeda baik pada fibrilasi atrium yang paroksismal maupun persisten. Terlebih lagi penderita fibrilasi atrium bila mengalami stroke umumnya mempunyai gejala yang lebih berat, disabilitas yang lebih parah serta rekurensi yang lebih sering dibandingkan pasien non fibrilasi atrium.  Oleh karena itu stratifikasi risiko stroke pada pasien fibrilasi atrium menjadi sangat penting untuk pencegahan stroke yang lebih baik dan terarah. Skor CHA2DS2VaSc terbukti efektif sebagai panduan pemilihan pasien fibrilasi atrium yang mendapat terapi antikoagulan. Antikoagulan baik antagonis vitamin K (warfarin) maupun antikoagulan oral baru (direct antithrombin atau anti faktor Xa) secara signifikan dapat menurunkan kejadian stroke dengan komplikasi perdarahan yang kecil.
Risiko Gagal Ginjal Akut Pasca Bedah Jantung yang memerlukan Terapi Pengganti Ginjal Kontinyu Juweni Joe; Zuswahyuda Samsu; April Retno; Rita Zahara; Ronggo Prakoso; Ardiyan A
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.335

Abstract

Background: Acute renal failure is a rare but serious complication followingcardiac surgery and associated with increased mortality and morbidity.Objective:To identify factors associated with mortality and mortality ofpatients with acute renal failure after cardiac surgery treated with continuousrenal replacement therapy.Method: This was a cohort retrospective study on cardiac surgery patientswho developed acute renal failure requiring renal replacement therapy aftersurgery in Harapan Kita National Cardiac Center between January 2011and April 2012. Data was retrieved from medical record and consistedof pre-operative, intra-operative, and post-operative variables. Risk factoridentification was done using multivariate logistic regression analysis,whereas relative risk analysis was applied to know the association betweenrisk factor and morbidity. Direct or indirect effect of variables on renal failurewas analyzed using Barttlet’s and anti-image correlation test.Results: A total of 110 cases were obtained during the study period; 70(63.3%) among them were men. Patients mean age was 57.6 years. Preoperativerenal failure, New York Heart Association Functional ClassificationClass (NYHA) class IV, critical condition, coronary revascularization surgeryand bleeding, post-operative anemia, bleeding and venous saturation <65%showed a trend of mortality and morbidity rate between 0.1 and 9.1. TheKeiser-Meyer-Olkin (KMO) value and Barttlet’s test showed that re-surgery,bleeding and low inotropic score resulted in 31.63% probability of havingpost-operative renal failure.Conclusion: Re-surgery, bleeding and inotropic use may result in postoperativerenal failure.
Skrining Oportunistik untuk Mendeteksi Diabetes Melitus yang Baru Terdiagnosis Sri R Paputungan; Fabiola MS Adam; John MF Adam
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.336

Abstract

Background. It is estimated that 50% of the diabetic patients are undiagnosed.Opportunistic screening is one of the screening method, to detectnewly diagnosed diabetes mellitus. The aim of this study is to detect theundiagnosed diabetes mellitus by screening in the clinical setting.Subjects and Methods. Subjects were form the EIDEG screening fordiabetes mellitus. The procedure is a two step screening, first using thereflectance meter for capillary blood sugar, followed by confirmation testin the laboratory. Capillary blood sugar was divided into 3 groups, < 100mg/dL as normal, 100-199 mg/dl possible diabetes, and ? 200 mg/dL suspecteddiabetes. For group two, an OGTT was performed, and for groupthree, only FPG. Diabetes mellitus was diagnosed if FPG ? 126 mg/dl andor 2 hour OGTT ?200 mg/dl.Results. During the screening, 4737 subjects can be screened, only 1654completed the screening. Diabetes mellitus was diagnosed in 240 subjectsor 14.5%. There were more females compared to males, 52,9% and 47,1%subsequently, most were at the age ?50 years. More diabetic patients werediagnosed by OGTT compared to FPG only.Conclusions. This study showed that opportunistic screening may detectmore diabetic patients. It is suggested that this screening procedure canbe used by every clinicians in their daily practice.
Perbedaan Rasio Kadar Serum MMP-9/TIMP- 1 pada Kejadian Infark Miokard Akut Elevasi Segmen ST (IMA-EST) dan Sindroma Koroner Akut Non Elevasi Segmen-ST (SKA-NEST) Basuki Rahmat; Lucia Kris Dinarti; Irmalita I; Budi Yuli Setianto
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.337

Abstract

Background: Differences between the pathogenesis of ST-Elevation Myocardialinfarction (STEMI) and Non-ST Elevation Acute Coronary Syndrome(NSTE-ACS) had yet unknown. Matrix metalloproteinase-9 (MMP-9) as thematrix degradation enzyme secreted by inflammatory cells play a role in thepathogenesis of plaque rupture. MMP-9 proteolytic activity is inhibited byspecific inhibitors of the Tissue Inhibitor of metalloproteinase-1 (TIMP-1).MMP-9/TIMP-1 ratio describes the actual proteolytic activity of MMP-9.This ratio may distinguish the pathogenesis of STEMI and NSTE-ACS.Objective: To examine the difference serum level ratio MMP-9/TIMP-1 inpatients with STEMI and NSTE-ACS.Methods and subjects: This is a cross-sectional study which recruits patientsconsecutively with ACS admitted to ICCU of Dr. Sardjito General Hospital Yogyakartawithin 24 h onset. Acute infection, chronic inflammation, acute stroke,kidney failure requiring renal replacement therapy, chronic heart failure, liver cirrhosis,acute exacerbation of COPD and pneumonia, thromboembolic disease,malignancy, pregnancy and the use of steroids and steroid anti-inflammatorydrugs are excluded. Serum levels of MMP-9 and TIMP-1 examined using themethod of sandwich enzyme-linked immunosorbent assay (ELISA).Results: The total of 60 subjects with STEMI patients 31 (51.7%) and NSTEACS29 (48.3%). Level of serum MMP-9/TIMP-1 ratio is significantly higher inSTEMI compared to NSTE-ACS (1.106 ± 0.065 vs. 1.046 ± 0.057, p <0.001).MMP-9/TIMP-1 ratio in serum is an independent factor for STEMI (p = 0.003)followed by blood sugar level (p = 0.013) and MMP-9 (p = 0.033). Interestingly,patients with serum MMP-9/TIMP-1 ratio> 1.0639 has a prevalence riskof 1.7 times having STEMI (p = 0.039; KI95% from 1.040 to 8.508). Levels ofserum MMP-9/TIMP-1 ratio significantly higher in STEMI compared to NSTEMIgroup (p = 0.003) and in the STEMI and UAP group (0.026), but did not differsignificantly in NSTEMI and UAP group (p = 0.045).Conclusion: High levels of serum MMP-9/TIMP-1 ratio in patients withSTEMI than NSTEACS may explain the role of serum MMP-9/TIMP-1 ratioin differentiating the pathogenesis of STEMI and NSTE-ACS.(J Kardiol Indones. 2013;34:160-6)
Permanent Pacemaker Implantation Through Persistent Left Superior Vena Cava: Safe for Normal Heart Position and Dextrocardia Erika Maharani; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.338

Abstract

Permanent pacemaker implantation in patient with persistent left superior vena cava (LSVC) has been reported in literature. Some authors has also been reported pacemaker implantation in patient with dextrocardia. Both of this situation has a challenging situation in placing the lead in right ventricle and risks of lead dislocation. We report two cases of successful pacemakers implantation,one with mirror image dextrocardia, in patient with persistent LSVC.
Membedakan Takikardia Ventrikel dan Takikardia Supraventrikular Dengan Aberansi Pada Takikardia Dengan Kompleks QRS Lebar Andi Haryanto; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.340

Abstract

Wide complex tachycardia is a quite common rhythm found in ECG. Basicly there are 3 arrhytmia that can cause wide QRS complex tachycardia, which are: Ventricle tachycardia (VT) which is the most common (80%), Supraventricular tachycardia (SVT) with abberancy (15-20%), and Atrioventriculare Reentrant Tacycardia (AVRT) with antidromic conduction (1–6 %). Correct diagnosis in differentiating SVT with aberancy and VT is important, due to the different patophysiology and different mechanism they present. Thus the therapy and management will be different, and miss treatment proven to be fatal.          Since ECG is still the main modality to provide the diagnosis in wide QRS complex tachycardia, many effort were done including the creation of algorhythms to help establish the diagnosis for wide complex tachycardia. The oldest and most widely used algorhythm is the Brugada algorhythm with respectable sensitivity and specificity. In 2007 Vereckei et al proposed a new algorhythm for differentiating VT and SVT with abberancy, and in 2008 Vereckei renew his previous algorhythm into only using single aVR lead to differentiate VT and SVT with abberancy, which was made solely based on the differences in the direction and velocity of the impulse. The latest method was again proposed by Brugada in 2010 which was called the ultrasimple Brugada criterion evethough there still haven’t many research that discuss the accuracy of such criteria.
Sindroma Pre Eksitasi Asimtomatik: Ablasi Versus Konservatif Dian Andina Munawar; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.341

Abstract

Wolff-Parkinson-White (WPW) is a sporadic and familial abnormality. Epidemiologicaldata indicate that 0.1% to 0.3% of the general populationhave ECG findings suggesting that during sinus rhythm. The risk of suddencardiac death is around 0.25%. Patients with WPW syndrome is oftensymptomatic because of cardiac arrhythmias. Sometimes the arrhythmiacan be life-threatening, and leads to sudden cardiac death. However, thereis still controversy in management of individuals with asymptomatic WPWpattern. Despite radiofrequency catheter ablation remains the first linetherapy of the WPW syndrome, the risk of complications of this procedureis almost the same as the risk of sudden cardiac death in asymptomaticWPW. Therefore, an algorithm to determine the appropriate managementof asymptomatic WPW patients is needed.
Nilai Prediktif Mri Kardiak Pasca Stemi Peran Late Enhancement Irnizarifka I; Sony Hilal Wicaksono; Ario Soeryo Kuncoro; Manoefris Kasim
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.342

Abstract

Forum Pencitraan Kardiovaskular edisi sebelumnya membahas tentang peran Magnetic Resonancec Imaging (MRI) kardiak pada kasus Penyakit Jantung Koroner dalam kondisi akut maupun kronik. Dalam forum kali ini, kami sajikan contoh kasus penggunaan MRI kardiak pada pasien pasca Infark Miokard Akut Elevasi ST (IMAEST) yang dilakukan pemeriksaan MRI kardiak saat perawatan sebagai stratifikasi risiko pasien pasca IMAEST. Penyakit kardiovaskular masih menjadi penyebab morbiditas dan mortalitas tertinggi di dunia, dengan penyakit arteri koroner (PAK) sebagai manifestasi utamanya.1 Dengan semakin berkembangnya manajemen serangan jantung akut, setidaknya 70% pasien yang dirawat di rumah sakit dengan infark miokard akut (IMA) berhasil melewati fase akutnya. Namun, konsekuensi jangka panjang pasca serangan akut tetap dapat terjadi meskipun intervensi fase akut telah dilakukan. Dengan demikian, ketepatan waktu dalam mendiagnosis IMAEST menjadi sangat penting.
Tips Dan Trik: Mengenali Tamponade Rina Ariani; Amiliana M Soesanto
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.343

Abstract

Tamponade merupakan kondisi klinis mengancam jiwa dimana fungsi jantung terganggu oleh penekanan dari ruang pericardium, baik oleh cairan, massa, bekuan darah, ataupun kombinasi antara ketiganya. Fisiologi tamponade terjadi akibat terganggunya hemodinamik akibat peningkatan tekanan intra pericardium sehingga mengganggu pengisian ruang jantung. Tamponade dapat terjadi akibat adanya volume efusi yang cukup besar atau terbentuknya efusi dalam waktu yang singkat. Perlu diingat bahwa efusi yang besar dapat tidak menimbulkan tamponade bila terakumulasi bertahap sehingga memungkinkan rongga perikard meregang dan beradaptasi. Harus pula digaris bawahi diagnosis tamponade merupakan diagnosis berdasarkan klinis namun ekokardiografi seringkali dapat mengenali adanya tamponade dengan lebih dini.
Torsades de Pointes pada pasien Hipokalemia: Peranan Afterdepolarization pada mekanisme Takiaritmia Alexander Tondas; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 34, No. 3 Juli - September 2013
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v34i3.344

Abstract

KasusSeorang wanita usia 56 tahun, dengan riwayat gagaljantung dalam pengobatan dengan obat diuretik furosemid, mengalami muntaber selama 3 hari terakhir. Saat tiba di UGD, EKG dengan irama sinus tiba-tiba mengalami degenerasi menjadi takikardia QRS lebar polimorfik saat perekaman. Presentasi hemodinamik pasien menjadi tidak stabil sehingga diputuskan untuk dilakukan kardioversi elektrik. Setelah defibrilasi, irama jantung kembali menjadi irama sinus dan hasil laboratorium menujukkan hipokalemia berat (2,4 meq/L). Setelah koreksi potassium dan perawatan, pasien membaik kemudian dipulangkan. Apa mekanisme tercetusnya torsades de pointes pada pasien ini?

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