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Contact Name
Raymond Pranata
Contact Email
raymond_pranata@hotmail.com
Phone
+6282112918892
Journal Mail Official
ijc@inaheart.org
Editorial Address
Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
Location
Kota adm. jakarta barat,
Dki jakarta
INDONESIA
Indonesian Journal of Cardiology
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Articles 712 Documents
Hubungan Morfologi Ventrikel Kiri Orang Terlatih dengan Respons Hipoksia di Ruang Udara Bertekanan Rendah Adhantoro Rahadyan; Andang H Joesoef; Dolly R.D Kaunang
Jurnal Kardiologi Indonesia Vol. 29, No. 2 Mei - Agustus 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i2.188

Abstract

Background. The higher the altitude, the higher the risk to hypoxia exposure. Good fitness status of airplane pilot has been thought of great importance to encounter hypoxic risk. Meanwhile, intensive endurance training results in increase of left ventricular mass and change of cardiac function due to down regulation of beta adrenergic receptor. It is not clear whether the difference in myocardial morphology due to endurance training will cause difference tolerance in hypoxia responses.Objectives. To compare hypoxia response of trained and untrained men and to investigate association of left ventricle morphology and hypoxia responses.Methods. This is a cross-sectional study involving 128 trained and 25 untrained men. They underwent medical examination in Saryanto Institute of Aerospace Medicine, Jakarta. Echocardiography and treadmill test ex-amination were performed. The time of useful consciousness (TUC) was measured using 25,000 feet hypobaric stimulation chamber.Results. Six subjects were excluded due to incomplete data. The mean age of trained men was 22 ± 1.0 years. TUC of trained (n=122) and untrained (n=25) men were 232 ± 64 sec and 260 ± 51 sec, respectively (p=0.01). To evaluate for association between echocardiographic parametric of ventricle morphology and TUC, all subjects were divided into TUC < 4 min and TUC = 4 min. Parameters related to TUC are LVEDD and LVMI. TUC < 4 min related to larger LVEDD and LVMI. Conclusion. Trained men had shorter TUC compared to untrained men. Shorter TUC related to larger LVEDD and LVMI.
Respon Hipoksia Orang Terlatih di Ruang Udara Bertekanan Rendah Herman Mulijadi
Jurnal Kardiologi Indonesia Vol. 29, No. 2 Mei - Agustus 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i2.189

Abstract

Hipoksia merupakan suatu fenomena khusus dalam dunia penerbangan dan masih menjadi perhatian bagi keselamatan penerbangan. Pada umumnya kita menganggap seseorang yang mempunyai tingkat kesamaptaan aerobik yang tinggi ( terlatih aerobik) maka akan makin tahan terhadap kondisi hipoksia. Tetapi pada latihan paparan hipoksia menggunakan RUBR, didapatkan beberapa peserta latihan dengan riwayat latihan aerobik yang intensif ternyata lebih rentan terhadap hipoksia. Oleh karena itu penelitian ini penting untuk menjawab mengapa pada peserta latihan yang terlatih aerobik tersebut menjadi lebih rentan pada paparan hipoksia.
Implantasi Drug-eluting Stentatas Indikasi Off-label: Pengalaman dan Luaran Klinis di Pusat Jantung Nasional Harapan Kita David D Ariwibowo; Daniel Tobing; Sunanto Ng; Muhammad Munawar; Sunarya A Soerianata
Jurnal Kardiologi Indonesia Vol. 29, No. 2 Mei - Agustus 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i2.190

Abstract

Background. Food and Drug Administration (FDA) of Unites States set the indication of drug-eluting stent (DES) on the product label (on-label indication). In the clinical practice, many patients received DES implantation outside the indication (off-label indication). Several “real-world” register showed off-label implantation reach prevalence of 50-60% with wide-ranging clinical outcomes.Objectives. To report the frequency, procedural complications, stent thrombosis (ST) and major cardiovascular events (MACE) of DES implantation with off-label indication in the National Cardiac Center Harapan Kita.Methods. This retrospective cohort study included patients who underwent coronary intervention with one or more DES (sirolimus or paclitaxel) implantation at 2006. Subjects were grouped into on-label and off-label indication. Subjects were followed-up until mid 2008.Results. 196(59%) of total patients who received DES implantation in 2006 in PJNHK are on off-label indications. The most frequent off-label indication was long-lesion intervention. There were more patients with previous myocardial infarction, PCI and CABG in off-label group. No significant difference of procedural complication rates between on-label and off-label group (2.2% vs. 2.6%, P =0.57). There was no significant difference of stent thrombosis rate. There was higher incident of MACE in off-label than on-labelgroup (17.9% vs. 8.6%, P =0.03). This difference was most contributed by incident of cardiac death. The adjusted hazard ratio of off-label was 1.7 (95% CI 0.8 – 3.6; P = 0.16); with several confounding factors including previous cerebrovascular disease, creatinine clearance <60 mL/min, non-elective PCI and double antiplatelet cessation. Conclusion. Off-label indication was quite frequent but has non-significant difference of TVR and ST rate compared to on-label indication. There was higher incident of MACE in off-label group, but this may be influenced by several clinical confounders.
Drug Eluting Stent dan pemakaian Off-label Otte J Rachman
Jurnal Kardiologi Indonesia Vol. 29, No. 2 Mei - Agustus 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i2.191

Abstract

Revaskulasisasi koroner dengan PCI (Percutaneous Coronary Intervention) merupakan suatu cara yang sudah diterima sebagai alternatif bedah dalam mengatasi Penyakit Jantung Koroner (PJK). Pemakaian Stent mempertinggi keberhasilan PCI namun tidak berhasil menghilangkan “the Achilles heel of PCI” berupa restenosis yang mengganjal keberhasilan jangka dekat dan panjang. Stent dengan penyalut obat (DES) berhasil mengurangi insidens restenosis menjadi amat kecil sehingga meningkatkan keberhasilan PCI dalam mengatasi PJK.Pemakaian DES kemudian amat meningkat dan menimbulkan kejadian fatal yang disebabkan karena kematian karena terjadinya trombosis. Sejak FDA di Amerika sekitar bulan Februari 2003 mengizinkan pemakaian DES dengan obat Sirolimus dan Paclitaxel, pemakaian DES diindikasikan hanya pada penderita2 tertentu dengan lesi de novodengan panjang < 30 mm dan diameter antara 2.5 sampai 3.5 mm sesuai dengan data2 yang didapat pada penelitian awal.
Ablasi Radiofrekuensi pada Kepak Atrium Tipikal Setelah Pemasangan Amplatzer Septal Occluder : Suatu Laporan Kasus Siska Suridanda Danny; Yoga Yuniadi; Amir Aziz Alkatiri; Faris Basalamah; Muhammad Munawar
Jurnal Kardiologi Indonesia Vol. 29, No. 2 Mei - Agustus 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i2.192

Abstract

We report a case of a 48-year old female with secundum atrial septal defect (ASD), already undergone device closure with an Amplatzar Septal Occluder (ASO). The patient subsequently underwent radiofrequency catheter ablation for typical atrial flutter, three months after ASD closure with good results. During follow up the patient remained in sinus rhythm and the complaints of dyspnoe and palpitation subsided.
Pengukuran Fungsi Sistolik Global Ventrikel Kiri Amiliana M Soesanto
Jurnal Kardiologi Indonesia Vol. 29, No. 2 Mei - Agustus 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i2.193

Abstract

Pengukuran fungsi sistolik jantung dengan pemeriksaan ekokardiografi dapat dilakukan dengan beberapa cara. Pada kesempatan kali ini akan diuraikan cara pengukuran fungsi sistolik ventrikel kiri yang dapat digunakan.
Complex VPC: R on T Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 29, No. 2 Mei - Agustus 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i2.194

Abstract

Seorang wanita 36 tahun dirujuk dari RS Pasar Rebo dengan keluhan utama pingsan. Keluhan ini baru pertama kali dirasakan. Pemeriksaan ekokardiografi menunjukkan suatu VSD perimembran dengan pirau kiri ke kanan dan EF 53%.Gambaran EKG di UGD terlihat pada gambar 1. Sepintas tampak suatu irama sinus dengan Ventricular Premature Contraction(VPC) bigemini. Akan tetapi bila diperhatikan lebih jauh terlihat irama atrial yang diikuti VPC couplet. VPC yang pertama mempunyai amplitudo yang lebih rendah dibandingkan yang kedua.
Dampak Pasar Bebas ASEAN Terhadap Praktek Kardiologi di Negara – Negara ASEAN Anna Ulfah Rahajoe
Jurnal Kardiologi Indonesia Vol. 29, No. 1 Januari - April 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i1.195

Abstract

Tanggal 19 Oktober 2008 yang lalu Dr. Dar-Ching Wu, Ahli Bedah Senior dari Singapura telah menyampaikan Sukaman Lecture,segera setelah The 17th Asean Congress of Cardiology dibuka. Acara Ilmiah Kardiologi yang diselenggarakan di kota Hanoi, Vietnam ini sangat meriah, sejalan dengan tumbuhnya perekonomian negara tersebut.Menurut dokter yang sudah banyak makan garam ini, kedokteran adalah praktek dari pengobatan dan pencegahan suatu penyakit. Dokter disanjung sebagai profesi yang sangat terhormat, karena dengan segala kemampuan yang ia miliki dapat menolong orang sakit yang seringkali sangat parah kondisinya. Terdapat hubungan langsung antara dokter dengan pasien, dan pasien sangat menghargai pertolongan dokternya. Dengan berkembangnya ilmu dan teknologi kedokteran, karakter layanan kesehatan pun berubah. Banyak pelaku lain yang ikut berperan, yakni tenaga kesehatan selain dokter, manajemen rumah sakit, dan penyandang dana. Kedokteran akhirnya berubah menjadi industri jasa pelayanan kesehatan.
Trigliseridemia Postprandial Sebagai Faktor Prediksi Kejadian Kardiak Sindroma Koroner Akut Berulang Muh A Sungkar; Harmani Kalim; Sjukri Karim
Jurnal Kardiologi Indonesia Vol. 29, No. 1 Januari - April 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i1.196

Abstract

Background. Plasminogen Activator Inhibitor-1 (PAI-1) is an important hemostatic factor of thrombosis that inhibits fibrinolysis mechanism by tissue-Plasminogen Activator (t-PA). PAI-1 is a predictor for recurrentacute coronary syndrome (ACS). PAI-1 is also associated with Insulin Resistance syndrome that manifest as increase level of Triglyceride (Tg), decrease of HDL cholesterol, hypertension and glucose intolerance. Acute or chronic hypertriglyceridemia is often associated with high PAI-1 plasma activity. Objective. To know the relationship between postprandial Tg (ppTg) and increasing PAI-1 plasma levels in patients with ACS that may cause recur-rent cardiac events.MethodsThis is a case-control study, that included 54 ACS patients (aged 54-63 years), with or without ST segment elevation, without diabetes mel-litus, who were admitted to Emergency Department of National Cardiac Center Harapan Kita, Jakarta. All patients were followed up during hos-pitalization and one month after discharged. Subjects were divided into 2 groups based on ppTg level and compared it to age group, sex, body mass index, smoking habit, systolic blood pressure, pulse, ST segment deviation, CKMB, total cholesterol, low density lipoprotein (LDL), HDL and PAI-1 levels, as haemostatic factor. Patients with ppTg level < 153.5 mg/dL is a control case. Results.There was an increasing PAI-1 levels (mean 28.9 ± 25.81 ng/dL) as predictor of post ACS events in the study subjects. The level of PAI-1 plasma increased in patients with hypertriglyceridemia (p=0.004). Despite no significant association between pp hipertriglicerydemia and MACE, the overall results showed a significant association between postprandial hipertriglicerydemia and obesity (p=0.037) as well as HDL cholesterol as components of metabolic syndrome. Conclusions. The postprandial hipertriglicerydemia is a condition that can be found in metabolic disturbances and may influence coagulation status. There are association between pp hipertriglicerydemia and obesity. PAI-1 level is predictor for recurrent cardiac events post ACS.
Hubungan Antara Job Strain Dengan Terjadinya Infark Miokard Pada Pasien Pusat Jantung Nasional Rima Melati; Endang Basuki; Budhi Setianto
Jurnal Kardiologi Indonesia Vol. 29, No. 1 Januari - April 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i1.197

Abstract

Background. Coronary heart disease is the most frightening disease and still become a problem in the developed and developing countries. The prevalence of myocardial infarction is also increasing from year to year. Beside the conventional risk factors, it is also influenced by occupational factors. Although job strain can cause stress which would have impact on the occurence of myocardial infarction, the prevention strategies being implemented are just for conventional risk factors. There is still no concern for occupational factors which can also cause job strain. This study was aimed to assess the relationship between job strain and other risk factors with myocardial infarction among workers.Methods. The study design was matched case – control 1:1 for age. Data were collected by using general questionnaire which covered demography characteristics, conventional risk factors, job characteristics, and demand – control questionnaire (JCQ) to assess job strain.Result. Job strain, smoking and dyslipidemia were risk factors which had relationship with myocardial infarction. Job strain increased myocardial infarction risk by 6.8 times (Adj OR 6.80, 95% CI: 2.72 ; 16.98, p = 0.000). Light smokers increased myocardial infarction risk by 15 times (Adj OR 14.97, 95% CI: 3.17 ; 70.74, p = 0.001), medium smokers increased myocardial infarction risk by 7.7 times (Adj OR 7.72, 95% CI: 2.73 ; 21.84, p = 0.000), and heavy smokers increased myocardial infarction risk by 26 times (Adj OR 25.61, 95% CI: 5.25 ; 124.88, p = 0.000). Dyslipidemia increased myocardial infarction risk by 2.8 times (Adj OR 2.82, 95% CI: 1.07 ; 7.44, p = 0.035). Job strain component which increased myocardial infarction risk was high job demand (Adj OR 2.44, 95% CI: 1.02 ; 5.85, p = 0.046).Conclusion. Job strain, smoking and dyslipidemia simultaneously had relationship with myocardial infarction.

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