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INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 712 Documents
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Hubungan Kadar Soluble Intercellular Adhesion Molecule-1 dan Soluble Vascular Cell Adhesion Molecule-1 dengan Gradasi Trombosis Atrium Kiri pada Stenosis Mitral Elen Sahara,; Yoga Yuniadi; Ismoyo Sunu
Jurnal Kardiologi Indonesia Vol. 35, No. 2 April - Juni 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i2.395

Abstract

Background. The relationship between inflammation and coagulation has been widely described, which adhesion molecules play important role in inflammation. Soluble intercellular adhesion molecule-1 (sICAM-1) and soluble vascular cell adhesion molecule-1 (sVCAM-1) seem to be related to thrombosis in few previous studies. The level of those molecules were increased in mitral stenosis (MS), however their relationship with left atrial thrombosis gradation is still unknown.Methods. Patients with moderate-severe MS (without any significant mitral regurgitation) who underwent transesophageal echocardiography were recruited consecutively in September-October 2013. They were divided into 3 categories of left atrial thrombosis gradation: non-thrombus without dense left atrial spontaneous echo contrast (LASEC) group, and non-thrombus with dense LASEC group, and thrombus group.Results. A total of 39 subjects were enrolled in the study with a mean age of 40.97±9.61 year. Moreover, 71.8% of them were female and 67.7% of them had atrial fibrillation (AF). Evaluation on left atrial thrombosis gradation as mentioned above showed that sICAM-1 levels were 284.74 (218.79-321.00) ng/mL, 346.86 (125.68-698.12) ng/mL, and 395.93 (171.44-1021.53) ng/mL, consecutively (p=0.280). While sVCAM-1 levels gradually increased based on those groups consecutively: 729.01 (543.93-967.80) ng/mL, 1066.00 (581.36-2470.60) ng/mL, and 1158.00 (668.66-2498.30) ng/mL (p=0.016). Multivariate analysis showed that AF and mitral valve area (MVA) influence thrombosis gradation.Conclusion. Difference in sVCAM-1 levels were found among left atrial thrombosis gradation groups in mitral stenosis, but its effect on thrombosis gradation was influenced by AF and MVA.
Fibrilasi Atrial pada sindrom WPW Dony Yugo; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 35, No. 2 April - Juni 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i2.396

Abstract

Background : In large-scale general population studies involving children and adults, the prevalence of WPW is estimated to be 1–3 in 1000 individuals. At present, it is estimated that approximately 65% of adolescents and 40% of individuals over 30 years with WPW pattern on a resting ECG are asymptomatic. An incidence of 4.5 episodes of sudden death, including resuscitated sudden cardiac death (SCD), per 1000 patient-years was recently reported. The mechanism of sudden cardiac death in patients with WPW is thought to be associated with atrial fibrillationCase Illustration : We reported a case of 45 year old female came with unstable irregular wide complex tachycardia in the form of pre-excited AF. Cardioversion successfully terminate the tachyaarhythmias. ECG in sinus rhythm revealed WPW pattern ECG. Patient then referred for catheter ablation. Successful ablation was done resulting a normal pattern ECGConclusion : AF in WPW syndrome could lead to devastating events such as cardiac arrest. ECG recognition at the first place is very important for early management. Early stratification and management in patients with WPW is important to diminished the risk of AF occurence and SCD.
Diagnosis Sindrom Brugada Edward Faisal
Jurnal Kardiologi Indonesia Vol. 35, No. 2 April - Juni 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i2.397

Abstract

Brugada syndrome is an inherited autosomal dominant disease that cause sudden death, which related with mutation of SCN5A gene, ? subunit of sodium channel. The risk Brugada syndrome in male is 8 times more than females. The average age is 40 years old, which can happen between age 1 to 77 years old. The Incidence is 5 to 66 per 10.000 people. The golden diagnostic tools is ECG, an abnormality QRS-T found in lead V1-V3. I report a case of Brugada syndrome with neither sign nor symptoms. The disease coincidental in routine medical examination.
Pedoman Tata Laksana Fibrilasi Atrium Perhimpunan Dokter Spesialis Kardiovaskular Indonesia 2014 Yoga Yuniadi; Dicky A Hanafy; Sunu B Rahardjo; Alexander E Tondas; Erika Maharani; Dony Y Hermanto; Muhammad Munawar
Jurnal Kardiologi Indonesia Vol. 35, No. 2 April - Juni 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i2.398

Abstract

Atrial fibrillation is the most frequent arrhythmia in clinical practice and causing huge health problem. Therefore, a national guidelines in management of AF that comprise of pathomechanism, clasifications, anticoagulants management for stroke prevention, rate and rhythm control is developed to ensure patients get best therapeutic option. The guidelines is developed in line with variable health care level in Indonesia from primary up to tertiary care.
Fibrilasi Atrial dengan Takikardia QRS Lebar Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 35, No. 2 April - Juni 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i2.399

Abstract

Seorang pria Bngladesh, 65 tahun datng ke UGD dengan keluhan mau pingsan. Pasien adalah pasien lama dengan 3VD yang sudah dilakukan revaskularisasi lengkap dengan pemasangan stent. Riwayat medis sebelumnya adalah rawat inap berulang karena ADHF. Hasil ekokardiografi menunjukkan suatu disfungsi ventrikel kiri berat, fraksi ejeksi (EF) 35%, dimensi end diastolik ventrikel kiri (EDD) 66 mm dan dimensi atrium kiri (LAD) 52 mm. Pemeriksaan fisik dalam batas normal.
Novel Bullet for Dyslipidemia and Cardiovascular Disease in the Horizon: Does Genetics Contribute to the Blueprint? Anwar Santoso
Jurnal Kardiologi Indonesia Vol. 35, No. 3 Juli - September 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i3.423

Abstract

The development of novel therapy for dyslipidemia and cardiovascular diseases (CVD) had been constrained by some challenges, and several recent approaches have failed for lack of efficacy. Progress had been made by a single, greatest contribution from statins in reducing the risk of CVD. However, the burden of CVD and residual risk remains quite high, and new pathways to prevent and treat the diseases are still needed. Despite this clear unmet need, nevertheless many research institutions have begun to withdraw their efforts in discovering ‘the new bullet’ for this prevalent diseases1.
Asosiasi Kadar Fibrinogen dengan Indeks Resistensi Mikrovaskular pada Penderita Infark Miokard Akut dengan Elevasi Segmen ST yang menjalani Intervensi Koroner Perkutan Primer Jusup Endang; Doni Firman; Iwan Dakota
Jurnal Kardiologi Indonesia Vol. 35, No. 3 Juli - September 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i3.424

Abstract

Background: Primary percutaneus coronary intervention (PPCI) is a first of choice to return patient’s blood flow and perfusion with ST-segment elevation myocardial infarction (STEMI), however reperfusion in macrocirculation level is not always accompanied by a sufficient microcirculation reflow due to Microvascular Obstruction (MVO). Previous study demonstrated thathigh fibrinogen concentration may affect rheological parameters of the blood and play an important role in the pathomechanism of myocardial non-reperfusion phenomenon following successful mechanical recanalisation of the infarct-related coronary artery. Another study show eda more compact, lysis-resistant fibrin network in no reflow group, but without significant relation to fibrinogen level. However, there is a lack of data regarding fibrinogen and MVO. The aim of this study is to evaluate association between fibrinogen and MVO by index of microcirculatory resistance (IMR).Methods. 55 STEMI patients undergoing primary PCI were consecutively included. The fibrinogen was evaluated using clauss method and IMR was done right after PPCI to evaluate MVO.Results. From fifty-five patients included in the study, there were 87,3% men, with mean age 53,1±8.9 years old, and smoker show the biggest proportion compare with risk factor for coronary artery disease. All the patient undergo primary percutaneus coronary intervention with mean door-to-ballon time of 89.04+37.114 minute and ischemia time of 458,69+170,709 minute. Mean IMR was 55,2 + 47,454 and mean fibrinogen level was 350,8+103,19. From the scaterred plot fibrinogen prone to had a weak negative correlation with IMR and statistically non-significant(r = -0,137; p=0,319).Conclusion. There is no correlation between fibrinogen level and IMR value in STEMIpatients undergoing PPCI
An effort to find biomarker for micro-vascular obstruction in patient with myocardial infarction undergoing primary revascularization: is fibrinogen the answer? Bambang Widyantoro
Jurnal Kardiologi Indonesia Vol. 35, No. 3 Juli - September 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i3.425

Abstract

Meski revaskularisasi telah menunjukkan manfaat besar dalam menurunkan morbiditas dan mortalitas pada pasien infark miokard akut dengan elevasi segmen ST (IMA-EST), manfaat dari intervensi koroner perkutan primer (IKPP) yang berhasil mengembalikan aliran koroner epikardial seringkali tidak optimal di tingkat mikro sirkulasi karena adanya obstruksi mikrovaskular.Obstruksi mikrovaskular (OMV) berperan penting dalam menentukan prognosis pasien paska tindakan,1, 2 dan telah dilaporkan bahwa keberadaan obstruksi mikro-vaskular berkorelasi dengan penurunan survival pasien paska IMA-EST, melalui pengaruhnya pada proses remodeling ventrikel.3, 4
Hubungan Kadar P-selectin dengan Fungsi Atrium Kiri pada Stenosis Mitral Rematik Prafithrie Avialita Shanti; Yoga Yuniadi; Nur Haryono
Jurnal Kardiologi Indonesia Vol. 35, No. 3 Juli - September 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i3.426

Abstract

Background. Mitral stenosis (MS) prevalence remains significant in developing countries because of the prevalence of Rheumatic Heart Disease (RHD). In moderate-severe MS patients, enormous increase in turbulent region and shear stress cause vascular endothelial dysfunction, and as the consequence, it increases the risk of thromboembolic complications. P-selectin is an adhesion molecule that play a role in the inflammation process, where it is expressed rapidly in mere minutes. Left Atrial Volume Index (LAVI) is a superior parameter compared with other two-dimensional echocardiography method to assess left atrial function.Methods. This was a cross-sectional study involving 20 MS moderate-severe patients with mitral valve area (MVA) < 1.5 cm2, to whom successful Percutaneous transvenous Balloon Mitral Valvulotomy (PBMV) was performed. Samples were taken consecutively from May-October 2013 at the National Cardiovascular Center Harapan Kita, Jakarta. Blood samples of P-selectin were collected pre and post-PBMV. The result was statistically analyzed with echocardiography data of LAVI prior PBMV to describe any association between expression of P-selectin and atrial function.Result.We found no association between LAVI and P-selectin level pre and post-PBMV in MS patients. That is described in the value of pre PBMV ?=-0.103 (95% CI -0.251-0.045) with p=0.16 and post-PBMV ?= 0.009 (95% CI -0.155-0.172) with p=0.91 We then performed linear regression test with adjusted confounding variable including sex, age, and atrial fibrillation, still we found no association between LAVI and P-selectin level, with the value of pre PBMV ?= -0.154 (95% CI -0.340-0.032) p=0.09 and post PBMV ?= -0.049 (95% CI -0.250-0.152) p=0.61.Conclusion. There is no difference in P-selectin level pre and post PBMV. There is no association between poor LAVI value and expression of P-selectin pre and post PBMV in MS.
Secreted Left Atrial P-Selectin in Mitral Stenosis after PBMV: When to Measure M. Saifur Rohman
Jurnal Kardiologi Indonesia Vol. 35, No. 3 Juli - September 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i3.428

Abstract

The glycoprotein P-selectin, a membrane component of cell storage granules, is rapidly translocated from ?-granules of platelets and the Weibel- Palade bodies of endothelial cells to the cell surface following an inflammatory process or other stimulations. P-selectin is a cell adhesion molecule of activated platelets and endothelial cells of interest because of its role in modulating interactions between blood cells and the endothelium, and also because of the possible use of its soluble form in plasma as a predictor of adverse cardiovascular events.1In endothelial cells, within minutes of its stimulation in vitro by inflammatory mediators, such as histamine, thrombin, or phorbol esters, or hypoxia, Weibel–Palade bodies are mobilized and their von Willebrand factor are degranulated. At the same time, P-selectin is also expressed at the surface as quick astwo minutes after stimulation. However, this expressionis short-lived, reaching its peak after only 10 minutes, declining back to baseline levels after 3 hours. Additional synthesis of P-selectin is brought about within 2 hours by cytokines, such as interleukin-1 (IL-1), tumor necrosis factor-? (TNF-?), and by thrombin, lipopolysaccharide or oxygen radicals. Immunofluorescence and confocal laser cytometry are usually used to measure the translocation upon this activation.1

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