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Murdani Abdullah
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ina.jghe@gmail.com
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Divisi Gastroenterologi, Departemen Ilmu Penyakit Dalam, FKUI/RSUPN Dr. Cipto Mangunkusumo, Jl. Diponegoro No. 71 Jakarta 10430 Indonesia
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INDONESIA
The Indonesian Journal of Gastroenterology, Hepatology and Digestive Endoscopy
Published by The Indonesian Society for Digestive Endoscopy
ISSN : 14114801     EISSN : 23028181     DOI : -
Core Subject : Health,
Medicine & Pharmacology
The Indonesian Journal of Gastroenterology, Hepatology and Digestive Endoscopy is an academic journal which has been published since 2000 and owned by 3 Societies: The Indonesian Society of Gastroenterology; Indonesian Association for the Study of the Liver; The Indonesian Society for Digestive Endoscopy. The aim of our journal is to advance knowledge in Gastroenterology, Hepatology, and Digestive Endoscopy fields. We welcome authors for original articles, review articles, and case reports in the fields of Gastroenterology, Hepatology, and Digestive Endoscopy.
Arjuna Subject : Kedokteran - Ilmu Pencernaan
Articles 6 Documents
 1 
Search results for , issue "VOLUME 3, NUMBER 1, April 2002" : 6 Documents clear
Esophageal Varices Bleeding in Portal Hypertension due to Portal Vein and Splenic Vein Obstruction David R.S. Samosir; Loli Simanjuntak; Bambang Setiyohadi; L. A. Lesmana; Aru W. Sudoyo; Marcellus Simandibrata
The Indonesian Journal of Gastroenterology, Hepatology, and Digestive Endoscopy VOLUME 3, NUMBER 1, April 2002
Publisher : The Indonesian Society for Digestive Endoscopy

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24871/31200224-27

Abstract

Based on its relation to the liver sinusoid, increased pressure of portal vein can occur at three levels: presinusoid, sinusoid ,and postsinusoid. Obstruction of the presinusoid veins can be caused by extra- hepatic condition such as venous thrombosis. We reported a case of portal hypertension with esophageal varices bleeding was a result of obstruction due to thrombosis of the splenic vein and portal vein under hypercoagulant conditions due to thrombocytosis. The management of esophageal varices was sclerotherapy while for overcome the thrombosis the patient was given hydroxy urea.    Keywords: esophageal varices bleeding, portal hypertension, vein obstruction
Non Helicobacter pylori - Duodenal Ulcer in a Liver Cirrhosis Patient Bayu Sukresno; Haryono Achmad
The Indonesian Journal of Gastroenterology, Hepatology, and Digestive Endoscopy VOLUME 3, NUMBER 1, April 2002
Publisher : The Indonesian Society for Digestive Endoscopy

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24871/31200228-30

Abstract

Peptic ulcer is an clearly marginated ulceration in mucosal membran that can penetrate until muscularis layer and resulted from imbalance between aggressive factor (gastric acid and pepsin) and defensive factor (gastric mucous, bicarbonate and prostaglandin, mucosal blood flow, and cell replacement). Factors that can act as aggressive factor include H. pylori, NSAID, and smoking. Duodenal ulcer is frequently associated with H. pylori, in which Helicobacter pylori is found in 95 - 100% of duodenal ulcer patients.. It was reported, a 39 years old female patient with cirrhosis hepatis who suffered from melena in which endoscopic examination revealed duodenal ulcer as a source of bleeding.There was no H. pylori, based on serologic examination (IgG antiHP) and culture. The ulcer is suspected caused by  NSAID based on history of using traditional medicine that may contain NSAID. Treatment with proton pump inhibitor and sucralfate can heal the ulcer after two week treatment.    Keywords: H. pylori, duodenal ulcer – liver cirrhosis
Risk Factors for Spontaneous Bacterial Peritonitis in Patients with Liver Cirrhosis and Ascites Fachrul Razy; Nanang Sukmana; Dharmika Djojoningrat; Sjaifoellah Noer
The Indonesian Journal of Gastroenterology, Hepatology, and Digestive Endoscopy VOLUME 3, NUMBER 1, April 2002
Publisher : The Indonesian Society for Digestive Endoscopy

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24871/31200212-16

Abstract

Spontaneous bacterial peritonitis (SBP) is one of serious complication of liver cirrhosis. Most of the patient with SBP have severe reduced liver function that clasified as Child Plugh class C. There are other risk factors for SBP such as poor nutritional status, GI bleeding, intravascular catheter insertion, ascites fluid protein concentration of less than 1 g/L, large volume paracentesis, urinary tract infection and respiratory tract infection. The management of SBP is mainly the administration of proper antibiotics. The antibiotic of choice for the emperial treatment is cefotaxim.    Keywords: sbp,liver cirrhosis, risk factor, ascites
The Relationship between Gastric Mucosa Mucous Thickness and Gastroscopic Findings in Patients Receiving Non-Steroidal Anti-Inflammatory Drugs Dono Antono; Chudahman Manan; Harry Isbagio; Vera D. Joewono
The Indonesian Journal of Gastroenterology, Hepatology, and Digestive Endoscopy VOLUME 3, NUMBER 1, April 2002
Publisher : The Indonesian Society for Digestive Endoscopy

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24871/3120021-8

Abstract

Non-steroid anti-inflammatory drugs (NSAID) can cause gastropathy and gastric mucosa, especially the mucous may play an important prevention role. This cross-sectional, single group study was conducted to know the difference in mucous thickness in antrum or corpus mucosa and the correlation of gastric mucous thickness to gastropathy. Patients who received NSAID from the rheumatology clinic were studied. Healthy subjects of 14 – 65 years old who never received NSAID were included as normal controls. Piroxicam 20 mg daily was given to the patients for 7 days, then gastroscopy and gastric mucosa biopsy with frozen section were performed. Specimens were stained with haematoxyline eosin and thickness of the mucous layer was measured using ocular micrometer. Thirty-two out of 70 patients participated in the study. All cases had hyperemia on gastroscopy with erosions and ulcer in 32 and 9 cases, respectively. The mean thickness of mucosa in distal antrum, proximal antrum and corpus was 28.5 ± 9, 37.4 ± 13.1 and 43.3 ± 13.1 microns, respectively. There was significant relationship between gastric mucosa mucous thickness with gastroscopic findings. In conclusion, this study confirmed that thickness of gastric mucosa mucous has an important role in  preventing NSAID gastropathy and dyspeptic complaints in this kind of patients does not suggest abnormalities of gastric mucosa.    Key Words: Gastric mucosa mucous thickness, NSAID, gastroscopy
Clinical Manifestation and Management of Extra-Esophageal Gastroesophageal Reflux Disease Juwanto Juwanto; Chudahman Manan
The Indonesian Journal of Gastroenterology, Hepatology, and Digestive Endoscopy VOLUME 3, NUMBER 1, April 2002
Publisher : The Indonesian Society for Digestive Endoscopy

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24871/31200217-23

Abstract

GERD is a condition that gastric content go back into the esophagus. This condition could came disturbances in the respiratory tract, heart and otolaryng. Those extra-esophageal clinical manifestation are common but often miss our attention. So it is important to explorate it further. The management of extra-esophageal GERD is similar with management of GERD. It is included life style modification and reducing refluxate with controlling pH with aggressiveness.    Keywords: extra gerd, non cardiac chest pain, respiratory disturbance, omeprazole
Fluid Sucralphate in Post Esophageal Varices Ligation Esophageal Ulcer Marcellus Simadibrata; Syarif Hidayat; Chudahman Manan
The Indonesian Journal of Gastroenterology, Hepatology, and Digestive Endoscopy VOLUME 3, NUMBER 1, April 2002
Publisher : The Indonesian Society for Digestive Endoscopy

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24871/3120029-11

Abstract

Background: One of the complications of esophageal varices ligation is deep ulcer. Sucralphate has been shown to prevent and heal post ligation esophageal ulcer. Materials and method: All patients with liver cirrhosis and portal hypertension undergoing esophageal varices ligation in the year 2000/2001 were included in this study. Using “double-blind random sampling”, patients were divided into 2 groups, those receiving sucralphate and those receiving placebo for 2 weeks. Endoscopy of the upper gastrointestinal tract was conducted regularly on the 7th, 10th, and 14th day to evaluate the diameter of the esophageal ulcer based on the diameter of forceps biopsy. The data were evaluated using analysis of variance (ANOVA) or kruskal wallis. Results: Statistically, there was no significant difference in the characteristics of group A patients receiving sucralphate and group B patients receiving placebo. Only 17 patients from group A and 20 from group B were allowed to participate in this study. On the 10th day, the average diameter of the esophageal ulcer in the sucralphate group was significantly smaller than that from the placebo group. (4.74 + 2.02 mm vs 5.42 + 2.22 mm; p=0.04). On the 14th day, the average diameter of esophageal ulcer in the sucralphate group was also significantly less than that of the placebo group (0.88 + 1.58 vs 2.99 + 2.04 mm; p < 0.01). Conclusion:Sucralphate is able to accelerate post esophageal varices ligation esophageal ulcer. Keywords: sucralphate, esophageal ulcer, esophageal varices ligation

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