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Departement of Neurology, Faculty of Medicine Universitas Sebelas Maret, Surakarta, Indonesia Jl. Kol. Sutarto No. 132 Surakarta, Indonesia
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Magna Neurologica
Published by Universitas Sebelas Maret
ISSN : 29636027     EISSN : 29853729     DOI : https://doi.org/10.20961/magnaneurologica.v1i2.647
Core Subject : Health, Science,
Health Professions Immunology & microbiology Medicine & Pharmacology Neuroscience Public Health
Magna Neurologica is a peer-reviewed and open access journal that focuses on promoting neurological sciences generated from basic neurosciences and clinical neurology. This journal publishes original articles, reviews, and also interesting case reports. Brief communications containing short features of medicine, latest developments in diagnostic procedures of neurology disease, treatment, or other health issues related to neurology that is important also acceptable. Letters and commentaries of our published articles are welcomed.
Arjuna Subject : Kedokteran - Neurologi Klinis
Articles 52 Documents
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Guillain-Barre Syndrome Associated with Disseminated Tuberculosis: A Case Report Sulfana Putri, Elsa Primadona; Mardhiyah, Idzni; Evianti, Sri Wahyu
Magna Neurologica Vol. 3 No. 2 (2025): July
Publisher : Department of Neurology Faculty of Medicine Universitas Sebelas Maret

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20961/magnaneurologica.v3i2.2270

Abstract

Background: Guillain-Barre syndrome (GBS) is a life-threatening, acute, immune-mediated polyneuropathy associated with preceding infections. Tuberculosis (TB), although it has a high incidence rate, is rarely reported to be associated with GBS. Case: We report the case of a 20-year-old female admitted to our hospital with a progressive symmetrical paraparesis, which further developed to paraplegia, along with a month-long history of constitutional symptoms. After investigation, the patient was diagnosed with an acute motor and sensory axonal neuropathy (AMSAN) variant of GBS and disseminated TB. The patient was treated with plasmapheresis, and first-line anti-tuberculosis therapy was initiated. The patient demonstrated significant improvement in muscle strength in response to the treatment. The pathogenesis of GBS in TB is believed to be due to molecular mimicry, leading to nerve damage or direct invasion of the nerve root by tubercular bacilli. Discussion: Guillain-Barre syndrome could be induced by tuberculosis, and treatment for both could improve the outcome. Thus, early diagnosis is critical. Further investigations must be conducted to understand the association of GBS and TB. Conclusion: Guillain-Barre syndrome could be induced by tuberculosis, and treatment for both could improve the outcome. Thus, early diagnosis is critical. Further investigations must be conducted to understand the association of GBS and TB.
Acute Hemiballismus Due to Vascular or Diabetic Striatopathy: A Rare Case Report from KHZ Musthafa General Hospital, Tasikmalaya Nurul, Nasyifa; Ramdani, Billy Muchamad; Affandi, Indra Gunawan
Magna Neurologica Vol. 3 No. 2 (2025): July
Publisher : Department of Neurology Faculty of Medicine Universitas Sebelas Maret

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20961/magnaneurologica.v3i2.2276

Abstract

Background: Hemiballismus is a hyperkinetic movement disorder characterized by sudden, involuntary, high-amplitude ballistic movements of the arm and leg on one side of the body, often due to contralateral central nervous system dysfunction. The most common causes are ischemic stroke and hyperosmolar hyperglycemic syndrome. Stroke-related movement disorders are rare, with an incidence of 0.4%–0.54%, while the prevalence of hyperglycemia-induced, known as diabetic striatopathy (DS), affects approximately 1 in 100,000 individuals, predominantly older women. Case: A 63-year-old male presented with sudden, involuntary left arm movements for one week, starting with throwing-like motions, followed by stiffness, tremors, rapid arm drop, and facial grimacing on the left side. The condition was accompanied by left leg weakness. He had a history of uncontrolled type 2 diabetes mellitus. Physical examination showed left hemiballismus with motor strength 5/5/3/2 and a positive Babinski reflex on the left. Blood tests revealed a random glucose of 686 mg/dL, HbA1C of 15%, and 126 mmol/L sodium. A non-contrast CT scan showed multiple bilateral lacunar infarcts in the basal ganglia region. Discussion: Hemiballismus occurs sporadically and is caused by multiple bilateral lacunar infarcts in the basal ganglia, with risk factors including type 2 diabetes mellitus and advanced age. Other causes, such as stroke and genetic factors, may still be considered. Conclusion: Hemiballismus is a rare form of movement disorder. Vascular and metabolic disorders are the most common causes of hemiballismus. In this case, acute hemiballismus occurred in an elderly stroke patient with type 2 diabetes mellitus as a risk factor.

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