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The Relationship Between Peer Support and the Incidence and Dimensions of Burnout Among Medical Students Naf'an, Nabila Utiya; Prathita, Yana Aurora; Lestari , Yani Dwi
Jurnal Medis Islam Internasional Vol 7 No 2 (2025): December
Publisher : UNUSA Press

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.33086/iimj.v7i2.7332

Abstract

Introduction: Burnout is a psychological condition characterized by chronic stress that persists over a long period as a response to ongoing stressors. Burnout can lead to decreased academic performance, increased risk of dropout, and even suicide. Peer support may enhance self-confidence, reduce social pressure, and provide emotional stability, ultimately helping reduce the risk of burnout. Objective: This study examines the relationship between peer support and burnout among medical students at Sultan Ageng Tirtayasa University (Untirta). Methods: The research design employed was a cross-sectional design, utilizing the simple random sampling technique for data collection. The subjects were preclinical medical students from the 2021, 2022, 2023, and 2024 cohorts. Sampling was conducted using a simple random sampling technique. Data on peer support were collected using the Social Provision Scale (SPS), while burnout data were obtained through the Maslach Burnout Inventory–Student Survey (MBI-SS). The data were analyzed using univariate and bivariate analyses. Results: The results showed that the prevalence of burnout among students was 14.6%. Based on the dimensions of burnout, the majority of students reported high levels of exhaustion (58.4%), moderate levels of cynicism (47.2%), and low efficacy (100%). Regarding peer support, 96.6% of students reported high levels of peer support. Fisher's exact test yielded a p-value of 0.381. Conclusions: This study concludes that there is no significant relationship between peer support and burnout among medical students at the University
Impact on the Kidney of Pancreas Damage due to Streptozotocin-Induced Hyperglycemia Prathita, Yana Aurora; Jusuf, Ahmad Aulia; Simadibrata, Christina; Djaali, Wahyuningsih; Viventius, Yoshua
Folia Medica Indonesiana Vol. 59, No. 2
Publisher : Folia Medica Indonesiana

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Abstract

Highlights: • This study observed the histology of pancreatic β-cell damage without any intervention to the kidneys of the animal models. • The histological analysis of the kidneys shows that STZ-induced animal models can be used for assessing kidney abnormalities due to hyperglycemia. • A scoring system for the histological analysis was developed to evaluate the changes in the kidney cells. Abstract The kidneys are one of the organs affected by microvascular complications due to diabetes mellitus. Hyperglycemia plays an important role in glomerular, mesangial cell, and tubular damage in the kidneys. Metabolic dysregulation, including hyperglycemia, initiates cellular damage in the kidneys. Streptozotocin (STZ) is a chemical compound that is known to damage pancreatic cells and cause hyperglycemia. This study aimed to examine the effects of hyperglycemia on the morphology of the kidneys. Kidney tissues were observed histologically using a light microscope. Samples were taken from the kidneys of experimental animals administered with STZ to induce hyperglycemia. Observation was performed afterwards to investigate any damage to pancreatic cells. A total of 12 kidney samples were divided into two groups: the control group and the STZ-induced group. The samples were prepared before staining with hematoxylin-eosin and Masson's trichrome. The endothelium, podocytes, mesangial cells, and basement membrane of the glomerulus were examined. The tubules of the kidneys were also examined, and the presence or absence of connective tissue formation in both groups was statistically tested. The results suggested a significant difference in tubular damage (p<0.05) and an insignificant difference in an increase in the damage of other components of the kidneys (p>0.05) in the STZ-induced group. Significant morphological changes were observed in the hyperglycemic renal tubules due to the administration of STZ. In conclusion, STZ-induced hyperglycemia caused damage to the kidney components but overall had no significant impact on the kidney.