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Too “Big-Headed”, to Exit, a Massive Fetal Hydricephalus Causing Maternal Uterine Rupture. : Uterine Rupture After Delivery of Fetal Hydrocephalus Jihwaprani, Muhammad Candragupta; Ismirahmadani , Tiara Dinar; Kurniawan, Hendro
Ahmad Dahlan Medical Journal Vol. 5 No. 1 (2024): May 2024
Publisher : Universitas Ahmad Dahlan

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Abstract

Uterine rupture is an obstetric emergency, defined as a complete tear of all three layers of the uterus. It carries catastrophic maternal and fetal outcomes. While most commonly associated with previous cesarean deliveries or surgical obstetric-related procedures, it may very rarely be associated with abnormally high intrauterine pressure secondary to obstructed labor. Our center experienced a referred case of a spontaneous uterine rupture occurring after augmentation of labor and instrumental delivery of a hydrocephalic intrauterine fetal demise in an unbooked 29-year-old G4P3002 woman at 33 weeks’ gestation. She presented with acute hypotension and severe postpartum hemorrhage. Uterine rupture was diagnosed on clinical grounds. Early stabilization by maternal resuscitation and bleeding control was followed by an emergency total hysterectomy due to refractory hemorrhage. Our case depicted obstructed labor as a potential cause of uterine rupture and underpins the importance of routine antenatal care awareness which is still frequently overlooked in developing countries
THE IMPACTS OF STIMULATION IN PROTRACTED LABOR TO CORTISOL LEVELS AND INCIDENCE OF POST-PARTUM BLUES Soetrisno, Soetrisno; Respati, Supriyadi Hari; Sulistyowati, Sri; Kurniawan, Hendro
Folia Medica Indonesiana Vol. 53 No. 1 (2017): JANUARY - MARCH 2017
Publisher : Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (331.722 KB) | DOI: 10.20473/fmi.v53i1.5483

Abstract

Delivery, induction, stimulation, cesarean section with the aid of a vacuum extraction, for example, can reduce maternal confidence on the smooth delivery process, as well as improve postpartum stress. These stressors stimulate the HPA axis (hypothalamus-pituitary-adrenal), so that the adrenal cortex produces more cortisol hormone, it can increase postpartum blues. The objective of this study was to analyze the effect of delivery stimulation on protracted labor on cortisol levels and the occurrence of postpartum baby blues. This was an experimental study of non-randomized post-test control group. The subject of the study 30 patients in labor taken consecutive sampling, divided into 2 groups (normal delivery and stimulation) each group consist of 15 patients. In stimulation delivery group, it is examined of cortisol serum levels after five days and then continued for postpartum blues occurrence measurement. Statistical analysis using t-test for differences in levels of cortisol and chi square for analyzing the effect on the occurrence of post-partum blues (a=0.05). Mean  level of cortisol in delivery stimulation group is 40.29 ± 5.58, in normal delivery is 33.59 ± 11.17, with p=0.047, meaning there are significant differences both study groups. Stimulation delivery increases the occurrence of post-partum blues 5.50 times compared to normal delivery (OR=5.50 and p=0.028). Mean cortisol levels on post-partum blues higher at 42.90 ± 6.97 compared to no post-partum blues 30.14 ± 6.66, p=0:00, which means there are significant differences both groups. In conclusion, there was significant relationship between stimulation in protrated labor that increases cortisol serum level and post partum blues incidence.