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All Journal Jurnal Ilmu Kedokteran (Journal of Medical Science)
Hotber Pasaribu
KSM Ilmu Kesehatan Anak Fakultas Kedokteran Universitas Riau
Biochemistry, Genetics & Molecular Biology Dentistry Health Professions Medicine & Pharmacology Public Health

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Mikrosirkulasi Pada Resusitasi Cairan Hotber Pasaribu
Jurnal Ilmu Kedokteran Vol 12, No 2 (2018): Jurnal Ilmu Kedokteran
Publisher : Fakultas Kedokteran Universitas Riau

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (111.99 KB) | DOI: 10.26891/JIK.v12i2.2018.63-71

Abstract

The aim of fluid therapy during hemodynamic resuscitation is to increase global blood flow and increase flow to the microcirculation and therby, and thus oxygen availability for cellular respiration. Effect of volume expansion on microvascular perfusion might not always be in coherence with its macrocirculatory. It should be stressed that the total amount of volume administered during fluid resuscitation is believed to be a major determinant of outcome in critically ill shock patients. The purpose of this review is to evaluate the boundaries of macrohemodynamic approach to fluid administration and the integrated with microcirculatory.
Peningkatan Kadar Troponin-I Paska Resusitasi Cairan pada Sus Scrofa Sebagai Model Hewan Coba Renjatan Hotber E. R. Pasaribu; Antonius H Pudjiadi; Rismala Dewi
Jurnal Ilmu Kedokteran Vol 12, No 1 (2018): Jurnal Ilmu Kedokteran
Publisher : Fakultas Kedokteran Universitas Riau

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (75.355 KB) | DOI: 10.26891/JIK.v12i1.2018.7-12

Abstract

Provision large amounts of fluids in a short period is known can cause hypervolemia. Therefore, an examination is needed to find out that the fluid resuscitation being administered does not cause hypervolemia. The purpose of this study is to assess the effect of hypervolemic resuscitation on cardiac contractility. The study was conducted on 10 male Sus Scrofa aged 6-8 weeks, as shocked animal models. There are 3 types of resuscitation treatments : normovolemic, hypervolemic-1, and hypervolemic-2. Cardiac contractility was assessed using DPmax and troponin-i levels. There was an increase in troponin-i levels after hypervolemic fluid resuscitation (p = 0.05). There is a decrease in cardiac contractility after hypervolemic resuscitation. Decreased cardiac contractility is associated with increased troponin-i (r = 0.720; p = 0.020). Based on the results, we conclude hypervolemic resuscitation causes changes in troponin-i levels, which reflect changes in cardiac contractility.
Co-Authors Antonius H. Pudjiadi Rismala Dewi