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Raymond Pranata
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Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
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INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 712 Documents
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The incremental value of combination copeptin and troponin T for early diagnosis of acute myocardial infarction Daniel R Mulyono; Arfenda P Mustikawati; Galuh A Tyagitha; Anthony Wijaya; Irna C Wyrahardja; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 32, No. 2 April - Juni 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i2.108

Abstract

Background: Copeptin is a protein that is released when the body experiences endogenic stress. The level of copeptin elevated very early and slowly decreases in myocardial infarction. Aim: to determine the incremental value of combination copeptin and troponin T for early diagnosis of Acute Myocardial Infarction.Methods: A search was conducted on PubMed and Cochrane. After screening title and abstract using inclusion and exclusion criteria, three articles were available as full text, but only 2 articles were considered useful by authors and were appraised based on its validity, importance and applicability. The other one article was excluded because the outcome of the study basically differentiates ACS and cardiomyopathy. Results: Both articles showthat the diagnostic value results are not so different. From the first article, a combination of copeptin and troponin T gives the results of sensitivity 87.4%, specificity 66.2%, PPV 46.9%, NPV 93.9%, pre-test probability 21.5%, post-test probability 40.8%, and AUC 0.91. The second article showed a combination of both gave sensitivity values of 98.8%, specificity 77.1%, PPV 46.2%, NPV 99.7%, pre-test probability 16.67%, and post-test probability 46.29%, and AUC 0,97. Conclusion: the combination of troponin T increase the sensitivity of diagnosis of AMI among patients suspected with acute coronary syndrome compared to troponin T alone. The combination of both can be considered as a test to diagnose AMI at early hours.
Anestesi Dalam Persalinan Pada Wanita Dengan Kelainan Jantung Cindy E Boom
Jurnal Kardiologi Indonesia Vol. 32, No. 2 April - Juni 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i2.109

Abstract

Insidensi penyakit jantung selama kehamilan hampir tidak berubah selama puluhan tahun dengan penyebab terseringnya adalah kehamilan pada ibu dengan penyakit jantung kongenital, diikuti oleh kehamilan pada ibu dengan penyakit jantung rematik. Angka kematian maternal menurun dari sebesar 6% pada tahun 1930-an hingga kini menjadi 0.5%-2.7%. Kehamilan akan meningkatkan resiko mortalitas maternal pada pasien dengan kelainan jantung dibandingkan dengan populasi normal. Kelainan jantung selama kehamilan juga akan meningkatkan morbiditas akibat kejadian gagal jantung, aritmia, dan stroke. Perkembangan sirkulasi uteroplasenter menginduksi perubahan besar dalam fisiologi kardiovaskular wanita hamil. Pada wanita hamil terdapat peningkatan volume intravaskular sampai 60%, penurunan tekanan darah, peningkatan denyut jantung sebesar 10%-20%, peningkatan curah jantung hingga 40%-50%, dan kejadian supine hypotensive syndrome10%-20%. Perubahan-perubahan ini dapat menyebabkan deteriorasi hemodinamik pada wanita hamil dengan kelainan jantung. Manajemen anestesi pada persalinan pasien dengan kelainan jantung bergantung pada kelainan jantung yang dideritanya. Pemberian anestesi pada sebagian besar wanita hamil dengan kelainan jantung umumnya dapat ditoleransi dengan baik. Persalinan pervaginam direkomendasikan pada sebagian besar kasus, kecuali pada beberapa keadaan khusus.
Peran Remodeling Atrium pada Fibrilasi Atrium Ignatius Yansen; Muhammad Munawar
Jurnal Kardiologi Indonesia Vol. 32, No. 2 April - Juni 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i2.110

Abstract

Atrial fibrillation (AF) is the most common arrhytmia. AF usually found at older age. AF means greater complication with higher morbidity and mor-talitiy. AF tends to be more frequent and severe. Patients with AF usually very difficult to maintain sinus rhytm after cardioversion. This phenom-enon shows that AF tends to be more severe after time. Atrial fibrillation can influence the condition in atrium so the atrium tends the withold the arryhtmia condition. Changes in atrium induced by atrial fibrillation that starts atrial fibrillation called atrial remodelling. This phenomenon is called AF begets AF. Respons can be miocyt growth, hyperthrophy, necrosis, and apoptosis, the disturbance of extracellular matrix composition, changes in the expression of ion channel, atrial hormon and return to fetal gen programe. These changes causing cascade of reaction that makes atrial remodelling with structural, functional and electrical effect.
Pemeriksaan Trans-Esofageal Ekokardiografi Agnes Lucia Panda; Amiliana M Soesanto
Jurnal Kardiologi Indonesia Vol. 32, No. 2 April - Juni 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i2.111

Abstract

Trans-Esofageal Ekokardiografi (TEE) merupakan cara pendekatan pencitraan jantung dengan menggunakan transduser khusus berfrekuensi 5-7,5 MHz yang diletakkan pada esofagus.Pada edisi sebelumnya telah dibahas mengenai indikasi, kontraindikasi, komplikasi serta prosedur pemeriksaan TEE. Pada edisi 2 ini akan dibahas mengenai beberapa view standar yang sering digunakan dalam pemeriksaan TEE. Akuisisi Gambar dan Manipulasi TransduserTerdapat 4posisi standar utama berdasar kedalaman probe; yaitu: upper-esophageal(20-30cm), mid-esophageal(30-40cm), transgastric (40-45cm), dandeeptransgastric(45-50cm). Untuk memperoleh gambar yang diinginkan, transduser (probe) dapat dimanupulasi sebagai berikut: ditarik/didorong (advanced/withdrawn); diputar ke kanan atau ke kiri (clockwise//counter clockwise); diantefleksi/retrofleksi; maupun dirotasi dari 0-1800 (Gambar 1). Penting diingat bahwa manuver harus dilakukan secara perlahan dan jika gambar yang terlihat lebih jelek, lakukan manuver kearah sebaliknya.
Apa yang Terbaik untuk Pasien Ini? Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 32, No. 2 April - Juni 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i2.112

Abstract

Seorang laki-laki67tahun, normotensi, non-diabetik dengan riwayat infarkmiokard anterior datang dengan takikardia kompleks QRS lebar (Gambar 1) yang me-merlukan defibrilasi karena hemodinamik yang tidak stabil. Pemeriksaan ekokardiografi memperlihatkan penurunan fungsi sistolik dengan fraksi ejeksi 20-25% dan gambaran aneurisma di apeksventrikel kiri. Fungsi ginjal dan paru dalam batas normal.
Manfaat Suplemen Antioksidan: Fakta atau Fiksi ? Faisal Baraas
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.113

Abstract

Peranan suplemen antioksidan masih tetap kontroversial sampai saat ini. Boleh dikatakan, belum ada rilis hasil penelitian tentang manfaat antioksidan bagi  penderita penyakit jantung koroner dalam jurnal-jurnal terbaru beberapa tahun belakangan ini sebagai hasil penelitian klinis mutakhir yang bisa dianggap konklusif. Manfaat antioksidan masih tetap kontroversial dan memerlukan penelitian lebih lanjut dengan disain penelitian yang lebih scrutinized dan analisis yang lebih kritis.Tidak ada isu lain di bidang terapi kardiovaskuler yang lebih kontroversial saat ini, melainkan isu tentang peranan antioksidan itu. Seorang pejabat FDA menyebutkan bahwa efektivitas antioksidan dalam pengobatan penyakit jantung memang masih tetap belum konklusif, walau pun sudah banyak penelitian yang dilakukan. Sebagian penelitian itu memang sudah menunjukkan hasil yang positif, tapi sebagian lagi masih tetap menjadi bahan perdebatan yang panjang.Maka pertanyaan, “perlukah suplemen antioksidan diberikan secara rutin pada pasien dengan kelainan koroner – karena manfaatnya dan keamanannya?”, belum bisa dijawab dengan pasti dan masih tetap mengambang.
Hubungan Matriks Metaloproteinase-9 (MMP-9) Dengan Troponin-I (cTn-I) pada Infark Miokard dengan ST-Elevasi (STEMI) dan Sindrom Koroner Akut Tanpa ST-Elevasi (NSTEACS) Budi Yuli Setianto; Indwiani Astuti; Bambang Irawan; Sofia Mubarika
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.114

Abstract

Background: Acute coronary syndrome (ACS) is due to plaque rupture or erosion. Plaque rupture or erosion occurs because of the extra-cellular matrix destruction by an MMP (matrix metalloproteinase). Troponin I (cTn-I) is a biomarker that will increase in ACS with myocardial necrosis.Objective: To determine levels of MMP-9 difference between STEMI and NSTEACS and the relationship between levels of MMP-9 and cTn-I levels between the two groups.Methods: The sample examination performed in 80 patients with ACS (39 STEMI and 41 with NSTE-ACS) prior to the act of intravenous thrombolysis or coronary intervention.Analysis of MMP-9 levels relationship and cTn-I using Spearman test, and analysis of the cut-off relationship MMP-9 in the STEMI and NSTEACS groups were tested by Chi square.Results: Group STEMI had higher levels of MMP-9 and significantly higher compared with NSTE-ACS group (p = 0.002). Spearman correlation test showed a significant and positive correlation between MMP-9 and troponin-I between the two groups (p = 0.003 and r = 0.33).Conclusion: Increased levels of MMP-9 were significantly higher in STEMI compared with NSTE-ACS and its association with elevated levels of tro-ponin-I, provide information about the role of MMP-9 against the severity of heart muscle damage that occurred.
Dapatkah Matrix Metalloproteinase Memprediksi Luasnya Kerusakan Miokard? Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.115

Abstract

Trombosis arteri merupakan dasar terjadinya sindrom koroner akut. Sebagian besar trombosis terjadi pada robekan plak pada bagian  fibrous cap yang tipis. Plak aterom yang memiliki fibrous cap yang tipis dan mudah mengalami robekan sering disebut sebagai vulnerable plaque. Penting dicatat bahwa derajat stenosis plak yang mengalami robekan seringkali hanya  ringan atau sedang saja.3 Para peneliti berhipotesis bahwa kehilangan komponen matriks khususnya serabut kolagen pada fibrous cap menyebabkan penipisan cap. Hal ini akan memudahkan terjadinya robekan baik secara spontan maupun sebagai respon terhadap trigger hemodinamik ata yang lainya.Matrix metalloproteinases (MMPs) agaknya memainkan peran penting dalam remodeling plak aterosklerotik, walaupun berbagai bukti menunjukkan hubungan peningkatan MMP dengan terjadinya instabilitas plak masih kontroversial.
Pengaruh Komposisi Asupan Makan terhadap Komponen Sindrom Metabolik pada Remaja Djanggan Sargowo; Sri Andarini
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.116

Abstract

Obesity as the component of metabolic syndrome and its associated disor-ders are a growing epidemic across the world. Several genetic, behavioral, and physiological factors play a role in etiology of obesity. Behavior factor such as high-carbohydrate and high-fat diet is important as this factor, but not the sedentary factor, is still having possibility to be altered. This study is performed to explain the influence of food intake to the components of metabolic syndrome. The method used is case-control analysis of population-based, epidemiological surveys using the metabolic syndrome definition of International Diabetes Federation. The results suggest that the total cholesterol and waist circumference components have higher pathway co-efficiency than other components. The compositions of food intake causing the metabolic syndrome are total  calories followed by fat and carbohydrate, consecutively. From these results, reduction of total calorie and fat can be recommended as dietary modification that reduces the risk of developing metabolic syndrome.
Pengaruh Makanan Pada Sindrom Metabolik Sutomo Kasiman
Jurnal Kardiologi Indonesia Vol. 32, No. 1 Januari - Maret 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i1.117

Abstract

Sejak 15 sampai 20 tahun terakhir ini sejumlah perubahan yang berhubungan dengan resistensi insulin termasuk hipertensi, obesitas, hiperinsulinemia, hipertrigliseridemia dan HDL yang rendah sudah dipahami dengan baik. Reaven yang menyatakan bahwa perubahan itu disebut sebagai sindrom metabolik yang bukan suatu penyakit tetapi merupakan sekumpulan kelainan metabolisme dimana penyebab utama sindrom ini saling berinteraksi, yaitu obesitas dan kerentanan metabolisme endogen. The National Cholesterol Education Program (NCEP) Adult Treatment Panel III (ATP III) menyatakan bahwa diagnosis sindrom metabolik harus memenuhi 3 atau lebih faktor risiko yaitu obesitas abdomen, trigliserida, kadar HDL, tekanan darah dan kadar gula darah puasa.

Page 24 of 72 | Total Record : 712

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