cover
Article Per Year (5 Year)
All
Home Page
OAI Link
Editorial Team
Contact
Reviewer
Google Scholar
Contact Name
-
Contact Email
-
Phone
-
Journal Mail Official
-
Editorial Address
Indonesian Center for Animal Science Research and Development Jl. Raya Pajajaran Kav E-59 Bogor 16151 Bogor Indonesia
Location
Kota bogor,
Jawa barat
INDONESIA
WARTAZOA Indonesian Bulletin of Animal and Veterinary Sciences
Published by Indonesian Center for Animal Research and Development
ISSN : 02166461     EISSN : 23546832     DOI : 10.14334
Core Subject : Health,
Veterinary
WARTAZOA. Indonesian Bulletin of Animal and Veterinary Sciences ISSN: 0216-6461 E-ISSN: 2354-6832 is a peer-reviewed, scientific journal published by Indonesian Center for Animal Research and Development (ICARD). The aim of this journal is to publish high-quality articles dedicated to all aspects of the latest outstanding developments in the field of animal and veterinary science. It was first published in 1983. The journal has been registered in the CrossRef system with Digital Object Identifier (DOI) prefix 10.14334.
Arjuna Subject : -
Articles 5 Documents
 1 
Search results for , issue "Vol 29, No 2 (2019): June 2019" : 5 Documents clear
Efficacy, Mechanism and Antiviral Resistance of Neuraminidase Inhibitors and Adamantane against Avian Influenza Dyah Ayu Hewajuli; NLPI Dharmayanti
WARTAZOA, Indonesian Bulletin of Animal and Veterinary Sciences Vol 29, No 2 (2019): June 2019
Publisher : Indonesian Center for Animal Research and Development

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (393.982 KB) | DOI: 10.14334/wartazoa.v29i2.1951

Abstract

Vaccination and antiviral drug are often used to control influenza. However, the effectiveness of vaccine was impaired due to the emergence of new variant of virus strain. Antiviral drug consists of prophylactic and curative substances, namely M2 ion channel inhibitors (adamantane; amantadine and rimantadine) and neuraminidase (NA) inhibitors (NAIs; oseltamivir, zanamivir, peramivir, laninamivir). The synthesis and modification of antiviral neuraminidase (NA) inhibitors (NAIs) and adamantanes increased the antiviral effectiveness. The mechanism of the neuraminidase inhibitor is to prevent influenza infection by inhibiting the release of the virus from internal cells. Adamantane is antiviral drug that selectively inhibits the flow of H+ ions through M2 protein to prevent the uncoating virus particles getting into the endosome. The substitution of (H275Y, S247N, I223L, K150N, R292K, I222T, R152K, R118K, E119V) on NA protein caused resistance of avian influenza virus against the neuraminidase inhibitor. The combination of mutations (S247N, I223L, K150N) increased the resistance of influenza A (H5N1) virus. The diffusion of adamantane resistance varies among HA subtypes, the species of host, the period of isolation, and region. Mutations at residues of 26, 27, 30, 31 or 34 transmembrane M2 protein caused adamantane resistance. The unique substitution (V27I) of M2 protein of clade 2.3.2 H5N1 subtype isolated in Indonesia in 2016 has been contributed to the amantadine resistance. Antiviral combination of M2 ion channel inhibitors and neuraminidase (NA) inhibitors is effective treatments for the resistance.
Melanocortin-4 Receptor (MC4R) Gene as the Main Gene for Rapid Growth Selection in Beef Cattle Peni Wahyu Prihandini; D Maharani
WARTAZOA, Indonesian Bulletin of Animal and Veterinary Sciences Vol 29, No 2 (2019): June 2019
Publisher : Indonesian Center for Animal Research and Development

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (277.736 KB) | DOI: 10.14334/wartazoa.v29i2.1955

Abstract

Genetic improvement for economic traits such as growth is important in beef cattle selection program. This paper discusses melanocortin-4 receptor (MC4R) gene as a candidate gene for rapid growth based on its function, location and pathway, as well as its polymorphism and identified genotypes associated with economic traits, and its application as a marker-assisted selection. MC4R is a prominent gene encoding growth traits and has a critical role in mediating the effect of leptin in regulating food consumption and energy balance. Many identified MC4R gene polymorphisms are associated with growth traits. The MC4R gene, therefore, is considered as a functional candidate gene for growth traits and can be used as a marker in selection program based on molecular genetics. The use of molecular markers such as MC4R gene, therefore, can be applied in genetic improvement program for growth traits in cattle. By using MC4R gene, the efforts to build breeding system in small populations can be proposed. For instance, with an effective population size (Ne) of about 40 heads, the first generation (G1) will be obtained with a composition of GG 61.1%, CG 33.3% and CC 5.6% and male and female ratio of 178:11 and generates ideal Ne of 41 heads. In the third generation, therefore, GG composition will be 100%. The results of molecular analysis can be further used as a guideline in the development and genetic improvement strategies of beef cattle.
The Use of Enrofloxacin Antibiotic as a Veterinary Drug and Its Residual Hazards on Public Health Prima Mei Widiyanti; Mirnawati Bachrum Sudarwanto; Etih Sudarnika; raphaella widiastuti
WARTAZOA, Indonesian Bulletin of Animal and Veterinary Sciences Vol 29, No 2 (2019): June 2019
Publisher : Indonesian Center for Animal Research and Development

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (208.754 KB) | DOI: 10.14334/wartazoa.v29i2.2015

Abstract

The livestock sector can improve the community's economy and has a role in fulfilling food needs, especially animal protein. One of the problems in the livestock sector is the presence of infectious diseases that consequently need treatment using veterinary drugs. This paper describes the use of enrofloxacin antibiotics as veterinary drug and their residual hazards on public health. Enrofloxacin is an antibiotic from the family of fluoroquinolones (second generation of quinolone). Enrofloxacin is a broad-spectrum antibiotic that is effective to kill Gram positive and negative bacteria, so it was used for the treatment of various diseases in animals. Pharmacokinetically, enrofloxacin will be metabolized into ciprofloxacin and other metabolites. The improper use of enrofloxacin antibiotics caused residues in food products of animal origin, microbial resistance and toxicity, therefore the use of enrofloxacin needs to be monitored and evaluated for the sake of animal health and society.
Utilization of Synthetic Antibody for Fumonisin Determination in Feed and Food Hasim Munawar; Kal Karim; Sergey A Piletsky
WARTAZOA, Indonesian Bulletin of Animal and Veterinary Sciences Vol 29, No 2 (2019): June 2019
Publisher : Indonesian Center for Animal Research and Development

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (458.422 KB) | DOI: 10.14334/wartazoa.v29i2.1999

Abstract

Fumonisin contamination in food is limited around 2 – 4 ppm and in feed for different animals varies from 5 to 100 ppm. This regulation is to prevent animal and human from carcinogenic effect from fumonisins. Measurement of fumonisins frequently uses chromatography methods such as High-Performance Liquid Chromatography (HPLC) and Liquid chromatography tandem-mass spectrometry (LCMS/MS); however, the sample preparation and analysis process for these methods are costly and time consuming. Immunoassays have also been employed for detecting fumonisins in food or feed. Unfortunately, the instability of antibody to harsh condition such as high temperature and pH becomes the drawback for immunoassay method. Currently, the technology based on molecularly imprinting, which is called synthetic antibody, has been established for replacing antibody functions. Therefore, the aim of this review is to describe development of molecularly imprinted polymer (MIP) in fumonisin analysis in feed and food. Herein, the composition and production of MIP were described comprehensively. Bulk polymerization and solid phase synthesis were methods for production of MIP in micro and nano sizes. The application of MIP was reported for sample preparation as solid phase extraction measured continuously by HPLC showing the high recovery (> 60%). Then, MIP replaced antibody in direct competitive enzyme-linked immunosorbent assay (ELISA) for quantifying fumonisins in maize with high recovery (>90%) and limit detection (2 – 6 pM). Lastly, MIP was also employed in electrochemical sensor application as receptor for recognizing fumonisin in milk and maize. In conclusion, the performance of MIP has been applied successfully for fumonisin analysis comprehensively from sample preparation and quantification. The MIP would be developed for wider application for other toxins in feed or food such as veterinary drug, heavy metals, or pesticides.
Candidate Gene of Milk Protein for Genetic Improvement of Dairy Cattle Santiananda Arta Asmarasari; C Sumantri; A Gunawan; E Taufik; A Anggraeni
WARTAZOA, Indonesian Bulletin of Animal and Veterinary Sciences Vol 29, No 2 (2019): June 2019
Publisher : Indonesian Center for Animal Research and Development

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (488.399 KB) | DOI: 10.14334/wartazoa.v29i2.1890

Abstract

The objective of this paper is to explore some efforts to increase milk protein of dairy cows through a milk protein control gene selection approach. Improving the quality of cow's milk has shifted to increase milk protein levels, due to nutritional and economic interest. The breeding process in producing dairy cattle with the advantage of having high milk protein content is more effectively carried out by molecular biotechnology approaches. The content of cow's milk protein is controlled by cow's milk protein control genes. In the process to produce dairy cattle with the advantage of having high milk protein content, it can be done with a selection approach based on milk protein control genes, namely CSN1S1, CSN2, CSN1S2, and CSN3. Once known, the main controller gene that causes high levels of cow's milk protein, it will be easy to identify dairy cattle that have these advantages, so that the selection of dairy cattle can be done since at early age.

Page 1 of 1 | Total Record : 5

 1 

Filter by Year

2019 2019


Reset
Filter By Issues
All Issue Vol 32, No 4 (2022): December 2022 (In Press) Vol 32, No 3 (2022): September 2022 Vol 32, No 2 (2022): June 2022 Vol 32, No 1 (2022): March 2022 Vol 31, No 4 (2021): December 2021 Vol 31, No 3 (2021): September 2021 Vol 31, No 2 (2021): June 2021 Vol 31, No 1 (2021): March 2021 Vol 30, No 4 (2020): December 2020 Vol 30, No 3 (2020): September 2020 Vol 30, No 2 (2020): June 2020 Vol 30, No 1 (2020): March 2020 Vol 29, No 4 (2019): December 2019 Vol 29, No 3 (2019): September 2019 Vol 29, No 2 (2019): June 2019 Vol 29, No 1 (2019): March2019 Vol 28, No 4 (2018): December 2018 Vol 28, No 3 (2018): September 2018 Vol 28, No 2 (2018): June 2018 Vol 28, No 1 (2018): March 2018 Vol 27, No 4 (2017): December 2017 Vol 27, No 3 (2017): September 2017 Vol 27, No 2 (2017): June 2017 Vol 27, No 1 (2017): March 2017 Vol 26, No 4 (2016): DECEMBER 2016 Vol 26, No 3 (2016): SEPTEMBER 2016 Vol 26, No 2 (2016): JUNE 2016 Vol 26, No 2 (2016): JUNE 2016 Vol 26, No 1 (2016): MARCH 2016 Vol 25, No 4 (2015): DECEMBER 2015 Vol 25, No 3 (2015): SEPTEMBER 2015 Vol 25, No 3 (2015): SEPTEMBER 2015 Vol 25, No 2 (2015): JUNE 2015 Vol 25, No 2 (2015): JUNE 2015 Vol 25, No 1 (2015): MARCH 2015 Vol 25, No 1 (2015) Vol 24, No 4 (2014): DECEMBER 2014 Vol 24, No 3 (2014): SEPTEMBER 2014 Vol 24, No 2 (2014): JUNE 2014 Vol 24, No 1 (2014): MARCH 2014 Vol 24, No 4 (2014) Vol 24, No 3 (2014) Vol 24, No 2 (2014) Vol 24, No 1 (2014) Vol 23, No 4 (2013): DECEMBER 2013 Vol 23, No 3 (2013): SEPTEMBER 2013 Vol 23, No 2 (2013): JUNE 2013 Vol 23, No 1 (2013): MARCH 2013 Vol 23, No 4 (2013) Vol 23, No 3 (2013) Vol 23, No 2 (2013) Vol 23, No 1 (2013) Vol 22, No 4 (2012): DECEMBER 2012 Vol 22, No 3 (2012): SEPTEMBER 2012 Vol 22, No 2 (2012): JUNE 2012 Vol 22, No 1 (2012): MARCH 2012 Vol 22, No 4 (2012) Vol 22, No 3 (2012) Vol 22, No 2 (2012) Vol 22, No 1 (2012) Vol 21, No 4 (2011): DECEMBER 2011 Vol 21, No 3 (2011): SEPTEMBER 2011 Vol 21, No 2 (2011): JUNE 2011 Vol 21, No 1 (2011): MARCH 2011 Vol 21, No 4 (2011) Vol 21, No 3 (2011) Vol 21, No 2 (2011) Vol 21, No 1 (2011) Vol 20, No 3 (2010): SEPTEMBER 2010 Vol 20, No 2 (2010): JUNE 2010 Vol 20, No 1 (2010): MARCH 2010 Vol 20, No 3 (2010) Vol 20, No 2 (2010) Vol 20, No 1 (2010) Vol 19, No 4 (2009): DECEMBER 2009 Vol 19, No 3 (2009): SEPTEMBER 2009 Vol 19, No 2 (2009): JUNE 2009 Vol 19, No 1 (2009): MARCH 2009 Vol 19, No 4 (2009) Vol 19, No 3 (2009) Vol 19, No 2 (2009) Vol 19, No 1 (2009) Vol 18, No 4 (2008): DECEMBER 2008 Vol 18, No 3 (2008): SEPTEMBER 2008 Vol 18, No 2 (2008): JUNE 2008 Vol 18, No 1 (2008): MARCH 2008 Vol 18, No 4 (2008) Vol 18, No 3 (2008) Vol 18, No 2 (2008) Vol 18, No 1 (2008) Vol 17, No 4 (2007): DECEMBER 2007 Vol 17, No 3 (2007): SEPTEMBER 2007 Vol 17, No 2 (2007): JUNE 2007 Vol 17, No 1 (2007): MARCH 2007 Vol 17, No 4 (2007) Vol 17, No 3 (2007) Vol 17, No 2 (2007) Vol 17, No 1 (2007) Vol 16, No 4 (2006): DECEMBER 2006 Vol 16, No 3 (2006): SEPTEMBER 2006 Vol 16, No 2 (2006): JUNE 2006 Vol 16, No 1 (2006): MARCH 2006 Vol 16, No 4 (2006) Vol 16, No 3 (2006) Vol 16, No 2 (2006) Vol 16, No 1 (2006) Vol 15, No 4 (2005): DECEMBER 2005 Vol 15, No 3 (2005): SEPTEMBER 2005 Vol 15, No 2 (2005): JUNE 2005 Vol 15, No 1 (2005): MARCH 2005 Vol 15, No 4 (2005) Vol 15, No 3 (2005) Vol 15, No 2 (2005) Vol 15, No 1 (2005) Vol 14, No 4 (2004): DECEMBER 2004 Vol 14, No 3 (2004): SEPTEMBER 2004 Vol 14, No 2 (2004): JUNE 2004 Vol 14, No 1 (2004): MARCH 2004 Vol 14, No 4 (2004) Vol 14, No 3 (2004) Vol 14, No 2 (2004) Vol 14, No 1 (2004) Vol 13, No 4 (2003): DECEMBER 2003 Vol 13, No 3 (2003): SEPTEMBER 2003 Vol 13, No 2 (2003): JUNE 2003 Vol 13, No 1 (2003): MARCH 2003 Vol 13, No 4 (2003) Vol 13, No 3 (2003) Vol 13, No 2 (2003) Vol 13, No 1 (2003) Vol 12, No 3 (2002) Vol 12, No 3 (2002) Vol 12, No 2 (2002) Vol 12, No 2 (2002) Vol 12, No 1 (2002) Vol 12, No 1 (2002) Vol 11, No 2 (2001) Vol 11, No 2 (2001) Vol 11, No 1 (2001) Vol 11, No 1 (2001) Vol 10, No 2 (2000) Vol 10, No 2 (2000) Vol 10, No 1 (2000) Vol 10, No 1 (2000) Vol 9, No 2 (1999) Vol 9, No 2 (1999) Vol 9, No 1 (1999) Vol 9, No 1 (1999) Vol 8, No 2 (1999) Vol 8, No 2 (1999) Vol 8, No 1 (1999) Vol 8, No 1 (1999) More Issue