Garuda - Garba Rujukan Digital

Article Per Year (5 Year)

All

Dental Journal (Majalah Kedokteran Gigi)

MKG
Website

Published by Universitas Airlangga

ISSN : 19783728 EISSN : 24429740 DOI : -

Core Subject : Health,

Dentistry

The Dental Journal (Majalah Kedokteran Gigi) (e-ISSN:2442-9740; p-ISSN:1978-3728) is published by the Faculty of Dental Medicine, Universitas Airlangga. Its diciplinary focus is dental science and dental hygiene. The Dental Journal (Majalah Kedokteran Gigi) is published in English on a quarterly basis with each 50-60 page edition containing between nine and eleven scientific articles on research, study literature and case studies. Contributors to the Dental Journal (Majalah Kedokteran Gigi) included: dental researchers, dental practitioners, lecturers, and students drawn from Indonesia and a wide range of other countries.

Arjuna Subject : -

Articles 950 Documents

1 2 3 4 5

Optimum dose of 2-hydroxyethyl methacrylate based bonding material on pulp cells toxicity Widya Saraswati
Dental Journal (Majalah Kedokteran Gigi) Vol. 43 No. 2 (2010): June 2010
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: 2-hydroxyethyl methacrylate (HEMA), one type of resins commonly used as bonding base material, is commonly used due to its advantageous chemical characteristics. Several preliminary studies indicated that resin is a material capable to induce damage in dentin-pulp complex. It is necessary to perform further investigation related with its biological safety for hard and soft tissues in oral cavity. Purpose: The author performed an in vitro test to find optimum dose of HEMA resin monomer that may induce toxicity in pulp fibroblast cells. Method: The method of this study was experimental laboratory with post test control group design. Primary cell culture was made from dental pulp fibroblast cells, and was given with HEMA resin bonding material in various concentrations (5 µg/ml–2560 µg/ml), and then subjected to toxicity test (MTT assay). Result: HEMA optimum concentration was 320 µg/ml to induce cytotoxicity in pulp fibroblast cells. Conclusion: The used of HEMA - base bonding material with the concentration of 200 µg/ml may induced pulp fibroblas cell toxicity.Latar belakang: Keberhasilan suatu bahan bonding secara klinis tergantung pada kandungan fisik, kimia dan keamanan secara biologis. HEMA (2-hydroxyethyl methacrylate) adalah bahan resin yang paling banyak digunakan karena memiliki sifat fisik-kimia yang baik. Beberapa penelitian pendahuluan menyebutkan bahwa resin merupakan bahan yang mampu menyebabkan gangguan pada kompleks dentin pulpa sehingga perlu dilakukan penelitian lebih lanjut menyangkut segi keamanan secara biologis bagi jaringan keras dan jaringan lunak di rongga mulut. Tujuan: Penelitian ini akan menguji secara in vitro (pada kultur sel fibroblas pulpa gigi) untuk mengetahui dosis optimal monomer resin HEMA yang dapat menyebabkan toksisitas pada sel fibroblas pulpa. Metode: Metode penelitian ini adalah eksperimental laboratoris dengan rancangan penelitian post test control group design. Kultur sel primer dibuat dari sel fibroblas pulpa gigi, dan diberi bahan bonding resin HEMA dengan berbagai konsentrasi (5 µg/ml–2560 µg/ml) kemudian dilakukan uji toksisitas (MTT assay) Hasil: Didapatkan konsentrasi optimal HEMA adalah 320µg/ml untuk dapat menginduksi terjadinya sitotoksisitas pada sel fibroblas pulpa. Kesimpulan: Penggunaan bahan dasar bonding HEMA dengan konsentrasi mulai 320 µg/ml dapat menyebabkan toksisitas pada sel fibrosis pulpa.

Changes in setting time of alginate impression material with different water temperature Decky J. Indrani; Niti Matram
Dental Journal (Majalah Kedokteran Gigi) Vol. 46 No. 1 (2013): March 2013
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: Previous studies showed that setting process of alginates can be influenced by temperature. Purpose: To determine the changes in setting time due to differences in water temperature and to determine the correlation between water temperature and the setting time. Methods: Seven groups of dough alginate were prepared by mixing alginate powder and water, each using a temperature between 13° C–28° C with a interval of 2.5° C. A sample mold (Θ = 30 mm, t = 16 mm) was placed on a flat plate and filled with doug alginate. Immediately the flat end of a polished acrylic rod was placed in contact with the surface of dough alginate. Setting time of alginat was measured from the starting of the mix to the time when the alginate does not adhere to the end of the rod. Setting time alginate data were analyzed using one way ANOVA, LSD and Pearson. Results: Setting time of alginate with water temperature between 13° C–28° C were 87 to 119.4 seconds and were significantly different (p < 0.01). The setting time between group were also significantly different (p<0.01). There was an inverse correlation between water temperature and the setting time (r = -0.968). Conclusion: Water temperature between 13° C–28°C with a difference of 2.5° C produced significant differences in alginate setting time; the lower the water temperature being used the longer the setting time was produced.Latar belakang: Penelitian-penelitian sebelumnya menunjukkan bahwa proses pengerasan alginat dapat dipengaruhi oleh suhu. Tujuan: Mengetahui perubahan waktu pengerasan alginat akibat perbedaan suhu air serta mengetahui hubungan antara suhu air dan waktu pengerasan. Metode: Tujuh kelompok adonan alginat yang dipersiapkan dengan mencampur bubuk alginat dan air, masingmasing menggunakan suhu antara 13°C–28° C dengan interval 2,5° C. Pengukuran waktu pengerasan alginat dilakukan sesuai dengan spesifikasi ADA no.18. Sebuah cetakan sampel terbuat dari pralon berbentuk cincin (Θ = 30 mm, t = 16 mm) ditempatkan di atas plat datar dan dipenuhi dengan adonan alginat. Pengukuran waktu pengerasan dilakukan segera dengan menyentuhkan ujung datar batang akrilik yang telah dipoles di permukaan adonan alginat. Waktu pengerasn alginat diukur dari awal pencampuran bubuk alginat dan air sampai dengan waktu awal ketika alginat tidak melekat di ujung batang. Data waktu pengerasan yang diperoleh dianalisis menggunakan uji statistik One-way Anova, LSD dan Pearson. Hasil: Suhu air antara 13° C–28° C telah menghasilkan waktu-waktu pengerasan alginat 87 detik hingga 119,4 detik yang berbeda signifikan (p < 0,01). Waktu pengerasan antar grup juga menunjukkan perbedaan signifikan (p<0,01). Antara suhu air dan waktu pengerasan alginat terdapat hubungan terbalik (r=-0,968). Kesimpulan. Suhu air antara 13°C–28°C dengan interval 2,5° C menghasilkan perbedaan waktu pengerasan alginat; makin rendah suhu air yang digunakan untuk mencampur makin panjang waktu pengerasan alginat.

Management of idiopathic alveolar bone necrosis associated with oroantral fistula after upper left first molar extraction Ni Putu Mira Sumarta
Dental Journal (Majalah Kedokteran Gigi) Vol. 43 No. 3 (2010): September 2010
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: Complications such as alveolar osteonecrosis and oroantral fistula can occure in maxillary molar extraction. The management of such complication is done by treating to treat any persisting maxillary sinusitis if present, prevent further antral contamination, wound bed preparation, and oroantral fistula closure with appropriate method. Purpose: This case report presents a treatment stage of an idiopathic upper alveolar bone necrosis and oroantral fistula that occurred 4 months after left upper first molar extraction. Case: A case of an idiopathic upper alveolar bone necrosis associated with oroantral fistula that occurred 4 months after left upper first molar extraction is presented. Patient suffered from pain and swelling at left upper jaw since 2 month before admission. There was a history of complicated tooth extraction 4 months earlier. Patient also complained pus and blood discharge from post extraction socket. Patient occasionally choked when drinking and fluids escaped through the nostril. There was a diffuse swelling in the left maxillary region; there was no hyperemia, with soft consistency and no pain on palpation. In the 26, 27 region there was a

Inhibition effect of cashew stem bark extract (Anacardium Occidentale L.) on biofilm formation of Streptococcus sanguinis Rizni Amaliah; Sri larnani; Ivan Arie Wahyudi
Dental Journal (Majalah Kedokteran Gigi) Vol. 45 No. 4 (2012): December 2012
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: Biofilm is communities of microorganisms attached to solid surface and enclosed in extracellular matrix that protected microorganisms from antibacterial agents and host defense. One of bacteria might have a role in initial colonization of biofilm formation is Streptococcus sanguinis (S. sanguinis). Previous studies showed that cashew stem bark extract (Anacardium occidentale L.) can inhibit the growth of Streptococcus strains. Purpose: The purpose of this study was to determine the inhibition effect of cashew (Anacardium occidentale L.) stem bark ethanol extract on biofilm formation of S. sanguinis. Methods: Streptococcus sanguinis grown in Brain Heart Infusion (BHI) + 2% sucrose medium by using microplate polystyrene 96 wells. The samples were divided into 3 groups, 5% polyethyleneglycol (PEG) as negative control, cashew stem bark extract (concentration 3.125 mg/ml, 6.25 mg/ml, 9.375 mg/ml, and 12.5 mg/ml), and 0.12% chlorhexidine (as positive control). Biofilm was stained by 1% crystal violet. Afterwards, optical density (OD) of samples were measured by microplate reader λ 595 nm. The data of biofilm formation inhibition percentage were analyzed by one way ANOVA and then continued by Least Significant Difference (LSD) test. Results: The result of one way ANOVA showed that there were significant differences in inhibition of S. sanguinis biofilm formation (p<0.05). LSD test showed that concentration extract 3.125 mg/ml had significant difference with concentration 9.375 mg/ml and 12.5 mg/ml. Reciprocally, concentration 6.25 mg/ml had significant difference with concentration 9.375 mg/ml and 12.5 mg/ml. Conclusion: Cashew stem bark extract was able to inhibit biofilm formation of S. sanguinis.Latar belakang: Biofilm merupakan sekumpulan mikroorganisme yang melekat pada permukaan solid dan diselubungi oleh matriks ekstraseluler yang melindungi mikroorganisme dari bahan-bahan antibakteri dan sel-sel pertahanan tubuh. Salah satu bakteri yang berperan pada awal pembentukan biofilm adalah Streptococcus sanguinis (S. sanguinis). Beberapa penelitian menunjukkan bahwa ekstrak kulit batang jambu mete (Anacardium occidentale L.) dapat menghambat pertumbuhan bakteri strain Streptococcus. Tujuan: Penelitian ini bertujuan untuk mengetahui pengaruh ekstrak etanol kulit batang jambu mete (Anacardium occidentale L.) terhadap pembentukan biofilm S. sanguinis. Metode: Media pertumbuhan S. sanguinis menggunakan Brain Heart Infusion (BHI) + 2% sukrosa yang ditumbuhkan pada microplate polystyrene 96 wells. Kelompok perlakuan dibagi menjadi tiga kelompok yaitu PEG 5% (kontrol negatif), ekstrak kulit batang jambu mete (konsentrasi 3,125 mg/ml, 6,25 mg/ml, 9,375 mg/ml, dan 12,5 mg/ml), dan klorheksidin 0,12% (kontrol positif). Biofilm yang terbentuk diwarnai dengan crystal violet 1%. Kemudian optical density (OD) sampel diukur menggunakan microplate reader λ 595 nm. Data berupa persentase penghambatan pembentukan biofilm dianalisis menggunakan uji one way ANOVA dan dilanjutkan dengan uji Least Significant Difference (LSD). Hasil: Uji one way ANOVA menunjukkan terdapat perbedaan daya hambat pembentukan biofilm S. sanguinis yang signifikan (p<0,05). Hasil uji LSD menunjukkan konsentrasi 3,125 mg/ml memiliki perbedaan yang signifikan dengan konsentrasi 9,375 mg/ml dan konsentrasi 12,5 mg/ml. Begitu juga dengan konsentrasi 6,25 mg/ml memiliki perbedaan yang signifikan dengan konsentrasi 9,375 mg/ml dan konsentrasi 12,5 mg/ml. Kesimpulan: Ekstrak kulit batang jambu mete dapat menghambat pembentukan biofilm S. sanguinis.

The importance of masticatory functional analysis in the diagnostic finding and treatment planning for prosthodontic rehabilitation Harry Laksono; Agus Dahlan; Sonya Harwasih
Dental Journal (Majalah Kedokteran Gigi) Vol. 45 No. 2 (2012): June 2012
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: The masticatory system as a biologic system is subjected to harmful influences of varying severity. Almost half of routine patients requesting prosthodontic treatment indicated at least one sign or symptom of temporomandibular disorders. Analysis of the masticatory system often neglected by dentist. Untreated temporomandibular disorders may significantly implicated in the perpetuation of the disorder and may interfere with routine prosthodontic clinical procedures. It would be resulted unsuccessful long term goal of prosthodontic rehabilitation because of the uncompleted diagnoses and treatment plan. Purpose: The purpose of this case report was to give the information of the importance of masticatory functional analysis in the diagnostic finding for treatment planning in the prosthodontic rehabilitation. Case: A 45 year - old male patient, partial dentate with reduced chewing efficiency, mild pain in right preauricular region in function, left click in opening mouth, severe attrition on all anterior lower teeth with vertical dimension of occlusion decreased due to loss of posterior support. He wanted to make a new denture. Case management: Record and analyze of active and passive mandibular movement, opening pathway, muscle and temporomandibular joints palpation, load testing, and vertical dimension of occlusion with manual functional analysis (MFA), occlusal condition and radiographic examination. Treatment plan was formulated into 3 phases: stabilization of the masticatory system, definitive treatment and periodical control. The result of this treatment excellent for 1 year evaluation after permanent cementation. Conclusion: Masticatory functional analysis is very important and must be done in the diagnosis finding for treatment planning in every case of prosthodontic rehabilitation.Latar belakang: Sistem pengunyahan sebagai sistem biologis sewaktu-waktu dapat terjadi gangguan dengan berbagai derajat keparahan. Hampir setengah dari jumlah pasien yang memerlukan perawatan prostodontik minimal menunjukkan satu tanda atau keluhan dari gangguan temporomandibular. Analisis fungsional sistem pengunyahan masih sering dilupakan oleh dokter gigi. Gangguan temporomandibular yang tidak dirawat akan terus ada dan mungkin dapat mengganggu prosedur klinis perawatan prostodonsia. Hal tersebut akan menyebabkan keberhasilan klinis jangka panjang perawatan prostodonsia tidak dapat tercapai karena diagnosis dan rencana perawatan yang kurang lengkap. Tujuan: Tujuan dari laporan kasus ini adalah untuk memberikan informasi tentang pentingnya analisis fungsional sistem pengunyahan untuk menegakkan diagnosis dan rencana perawatan pada perawatan prostodonsia. Kasus: Pasien laki-laki usia 45 tahun, bergigi sebagian merasa sulit untuk mengunyah makanan, nyeri ringan di daerah depan telinga kanan saat fungsi, keletuk sendi kiri saat membuka mulut, atrisi pada seluruh gigi depan rahang bawah disertai penurunan dimensi vertikal oklusi akibat kehilangan dukungan gigi belakang. Dia ingin membuat gigi tiruan yang baru. Tatalaksana kasus: Mencatat dan menganalisis pergerakan aktif dan pasif rahang bawah, arah pergerakan rahang bawah saat membuka mulut, palpasi otot-otot pengunyahan dan sendi temporomandibula, uji beban, dimensi vertikal oklusi dengan metode analisis fungsional secara manual, keadaan oklusal dan radiologis. Rencana perawatan dibagi menjadi 3 tahap berupa stabilisasi sistem pengunyahan, perawatan tetap dan kontrol secara periodik. Hasil perawatan menunjukkan keberhasilan klinis yang baik setelah dilakukan evaluasi selama 1 tahun setelah penyemenan tetap. Kesimpulan: Analisis fungsional sistem pengunyahan sangat penting dan harus dilakukan untuk menegakkan diagnosis dan rencana perawatan pada setiap perawatan prostodonsia.

The effect of 1,25-dihydroxyvitamin D3 on MSX2 gene expression during tooth and alveolar bone development Intan Ruspita
Dental Journal (Majalah Kedokteran Gigi) Vol. 48 No. 1 (2015): March 2015
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: 1,25-dihydroxyvitamin D3 has been proven to be able to control the formation and biomineralization of tissue through a regulatory gene. A previous research even showed that a cell responsible for the formation of the enamel (ameloblasts), dentin (odontoblasts) and bone (osteoblasts, osteoclasts) was the target of 1,25-dihydroxivitamin D3. Purpose: This research was aimed to determine the role of 1,25- dihydroxyvitamin D3 in vivo in the development of teeth and alveolar bone tissue by analyzing MSX2 gene expression as a gene marker responsible for the growth and development of enamel, dentin, tooth root and alveolar bone. Methods: Samples used for RT-PCR analysis were total RNA of insisivus teeth and alveolar bone derived from mice. RT-PCR analysis was conducted by using primer-specific gene, MSX2. Primer gene, GAPDH, was also used as an internal control. Five hundred nanograms of total RNA were used as a template for PCR. Semi quantitative results of PCR were quantified by using ImageJ software. Results: RT-PCR analysis showed that the expression level of MSX2 was enhanced in the samples of teeth and alveolar bone treated with 1,25 dihydroxyvitamin D3. The increasing of MSX2 expression significantly occurred in alveolar bone samples. Conclusion: It can be concluded that 1,25 dihydroxyvitamin D3 could enhance MSX2 expression as a marker of the development of teeth and alveolar bone tissue. Therefore, 1,25-D3 dihydroxyvitamin is expected to be used as an agent to help the regeneration of teeth and bone tissue.

Uji toksisitas ekstrak bawang putih (Allium Sativum) terhadap kultur sel fibroblast (Garlic (Allium Sativum) extract toxicity test on fibroblast cell culture) Yulie Emilda; Els Budipramana; Satiti Kuntari
Dental Journal (Majalah Kedokteran Gigi) Vol. 47 No. 4 (2014): December 2014
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: Previous studies have found antimicrobial effect of garlic (allium sativum). Garlic has potential as sterilization material for root canal treatment. Nevertheless, such material has to be non toxic and has to have adequate biocompatibility. Purpose: The study was aimed to examine the toxicity of garlic (allium sativum) on fibroblast cell culture. Method: Toxicity test was conducted using 50%, 75%, 100% of garlic extract, and Chlorphenol Kamfer Menthol (ChKM) as control. BHK-21 cell-culture was put into microplate 96 wells with 2x105 densities and incubated in a 37oC. The garlic extracts in various concentration and ChKM were then placed into the wells. MTT assay test was then use to analyze toxicity, a 50% percentage of living culture-cell was set as a parameter whether the extract is toxic or not. Results: The results showed that in 50%, 75%, and 100% garlic concentration indicates a non toxic characteristic on fibroblast cell culture. The non toxic property was consistent in 72, 96, and 120 hours of observation point. Conclusion: The study revealed that garlic on consentration of 50,%, 75%, 100% did not show toxic effect on fibroblast culture cell, but it needs further research for preparing it as an alternative medicament of root canal treatment.Latar belakang: Penelitian sebelumnya telah menemukan efek antimikroba bawang putih (allium sativum). Bawang putih memiliki potensi sebagai bahan sterilisasi pada perawatan saluran akar. Namun bahan tersebut harus tidak toksik dan memiliki biokompatibilitas yang memadai. Tujuan: Penelitian ini bertujuan menguji toksisitas bawang putih (allium sativum) terhadap kultur sel fibroblast. Metode: Uji toksisitas dilakukan dengan menggunakan konsentrasi 50%, 75%, 100% dari ekstrak bawang putih, dan Chlorphenol Kamfer Menthol (ChKM) sebagai kontrol. BHK-21 kultur sel dimasukkan ke dalam microplate 96 sumuran dengan kepadatan 2x105 dan diinkubasi di suhu 37°C. Ekstrak bawang putih dalam berbagai konsentrasi dan ChKM kemudian ditempatkan pada sumur dipersiapkan sebelumnya. MTT assay test kemudian digunakan untuk menganalisis toksisitas, persentase 50% dari kultur sel hidup digunakan sebagai parameter apakah ekstrak beracun atau tidak. Hasil: Hasil penelitian ini menunjukkan pada konsentrasi bawang putih 50%, 75%, 100% menunjukkan karakteristik non toksis terhadap kultur sel fibroblast. Kondisi non toksik konsisten di 72, 96, dan 120 jam pengamatan. Simpulan: Penelitian ini menunjukkan bahwa ekstrak bawang putih dengan konsentrasi 50,%, 75%, 100% tidak bersifat toksik terhadap kultur sel fibroblast, namun masih diperlukan pengujian lebih lanjut untuk dapat digunakan sebagai alternatif bahan sterilisasi saluran akar.

The effect of spirulina gel on fibroblast cell number after wound healing Fitria Rahmitasari; Wisnu Setyari J; Ester Arijani Rachmat
Dental Journal (Majalah Kedokteran Gigi) Vol. 44 No. 4 (2011): December 2011
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: Wound healing treatment after tooth extraction should be an important consideration due to mouth discomfort and pain. Spirulina (blue green algae) consists of C-phycocyanin, b–carotenoids, vitamin E, zinc, some other trace elements and natural phytochemical which are believed to act as antioxidant and takes part in wound healing process. Purpose: The purpose of this study was to examine the effect of spirulina gel on fibroblast cell number after wound healing process. Methods: Twenty eight males guinea pig are devided into four group, 7 guinea pig each. They are control group and treatment group which is given 0%, 3%, 6%, and 12% spirulina gel. After tooth extraction, histopathological evaluation was done to count fibroblast cell. The data was analyzed by One-Way ANOVA and Tukey HSD. Results: The research has proven the relation between the increased growth of fibroblast cell and spirulina gel application. The higher the doses, the more cell growth. Hence, there has been significant different (p < 0.05) among groups. Conclusion: Spirulina gel increases the number of fibroblast in wound after tooth extraction and 12% spirulina gel has the most potential ability.Latar Belakang: Proses penyembuhan luka pasca pencabutan gigi merupakan salah satu hal yang penting karena akan menimbulkan rasa nyeri dan tidak nyaman dalam rongga mulut. Spirulina (Blue green Algae) mengandung C-phycocyanin, b-carotenoids, vitamin E, seng, beberapa trace elemen lainnya, dan phytochemical alami yang terbukti dapat berperan sebagai antioksidan dalam proses penyembuhan luka. Tujuan: Tujuan dari penelitian ini adalah untuk mengetahui efek pemberian gel spirulina terhadap jumlah sel fibroblas pada proses penyembuhan luka pasca pencabutan gigi. Metode: Dua puluh delapan ekor guinea pig jantan dibagi dalam 7 kelompok, masing-masing terdiri dari 4 ekor. Kelompok tersebut adalah kelompok kontrol dan kelompok perlakuan yang diberikan gel spirulina dengan konsentrasi 0%, 3%, 6%, dan 12%. Setelah pencabutan gigi, dilakukan penghitungan sel fibroblas dengan metode histopatologi. Hasil: Penelitian ini menunjukkan adanya hubungan antara pemberian gel spirulina terhadap peningkatan jumlah sel fibroblas. Semakin tinggi dosis gel spirulina akan semakin meningkatkan jumlah sel fibroblas pula. Didapatkan perbedaan yang signifikan antar kelompok (p < 0,05). Kesimpulan: Gel spirulina meningkatkan jumlah sel fibroblas pada luka bekas pencabutan dan gel spirulina dengan konsentrasi 12% mempunyai kemampuan yang paling potensial.

Surgical exposure dan perawatan ortodontik pada impaksi gigi insisif sentral rahang atas (Surgical exposure and orthodontic treatment on labially impacted maxillary central incisor) Bingah Fitri Melati; Teguh Budi Wibowo; Betadion Rizki
Dental Journal (Majalah Kedokteran Gigi) Vol. 47 No. 2 (2014): June 2014
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: As a clinician we have to concern for an unerupted teeth especially in mixed dentition. Eruption failure can also be caused by early loss of deciduous teeth. Purpose: To report a case of unerupted maxillary central incisor caused by early loss of deciduous teeth due to trauma and the combination of excisional and orthodontic treatment. Case: A 8-years-old girl in mixed dentition phase came to Universitas Airlangga Dental Hospital with chief complaint of unerupted right maxillary central incisor while the left central incisor and both lateral incisor had erupted already. She had trauma when she was 1 year old and loss mostly her primary maxillary central incisors. An intraoral examination revealed lack of space in #11 region with root retained of #51, bulge was palpated in vestibulum and periapical radiograph showed that a delayed eruption upper central incisor without presence of disturbance. Case management: The exposure of the tooth was under local anesthesia a year after the orthodontic performed to make enough space for traction the tooth. A button was placed at palatal and used elastic strait to traction the tooth. After 3 months, bracket placed at labial to positioning until leveled and aligned with adjacent teeth. Conclusion: A simple excisional and orthodontic treatment were succesfully treated the labially impacted teeth.Latar belakang: Sebagai seorang klinisi kita harus memperhatikan apabila terdapat gigi yang belum erupsi terutama pada fase gigi pergantian. Kegagalan erupsi gigi juga dapat disebabkan karena tanggal premature gigi sulung. Tujuan: Melaporkan kasus impaksi gigi insisif sentral rahang atas yang disebabkan kehilangan premature gigi sulung karena trauma dengan kombinasi eksisi sederhana dan perawatan ortodontik. Kasus: Anak perempuan usia 8 tahun pada fase gigi pergantian datang ke Rumah Sakit Gigi dan Mulut Universitas Airlangga dengan keluhan gigi insisif sentral kanan rahang atasnya (#11) belum erupsi meskipun gigi insisif sentral kiri #21 dan kedua insisif lateralnya #22 sudah erupsi. Pasien tersebut pernah terjatuh saat masih usia 1 tahun dan hampir kehilangan seluruh gigi sulung insisif sentral rahang atasnya. Pada pemeriksaan klinis tampak ruang yang sempit pada region #11 dan terdapat sisa akar gigi #51, jaringan keras teraba pada palpasi daerah vestibulum dan pemeriksaan radiografi periapikal tampak impaksi gigi insisif sentral rahang atas tanpa adanya penghalang. Tatalaksana kasus: Exposure gigi dilakukan dibawah anestesi lokal 1 tahun setelah perawatan ortodontik untuk membuka space bagi #11. Button diletakkan di palatal gigi 11 dan digunakan elastic strait untuk traksi gigi tersebut. Setelah 4 bulan bracket dipasang untuk memposisikan gigi pada lengkung yang benar. Simpulan: Teknik eksisi sederhana dan perawatan ortodontik berhasil merawat gigi impaksi yang terletak di labial.

Odontoblast layer structure alteration as a response to carious lesions Tetiana Haniastuti
Dental Journal (Majalah Kedokteran Gigi) Vol. 44 No. 3 (2011): September 2011
Publisher : Faculty of Dental Medicine, Universitas Airlangga https://fkg.unair.ac.id/en

Background: Dental caries is a bacterial disease affecting the hard tissue of the teeth as well as the pulp. The human dental pulp consists of odontoblast which are organized as a densely packed cell layer. Odontoblasts is located at the periphery of the pulp; therefore, they are the first cells encountered by cariogenic bacteria and their products that are represented in the carious lesion. Purpose: This study aimed to elucidate the effect of cariogenic bacteria to odontoblasts of human teeth. Methods: Five intact third molars and 15 third molars with occlusal caries at various stages of decay were extracted because of orthodontic or therapeutic reasons. The tooth specimens were fixed, decalcified with 10% EDTA solution (pH 7.4), and embedded in paraffin. Serial sections of 5 μm thickness were cut and stained with haematoxylin eosin and Gram’s, in addition to nestin immunohistochemistry. The specimens were then examined under light microscopy. Results: In normal teeth, odontoblast layer were aligned along the pulp chamber showing normal morphology of the cells. Slight disorganization of odontoblast layer was seen in the cases of carious lesions confined to enamel. In the cases of carious lesions confined to dentin, odontoblast layer was not observed in the areas subjacent to the lesions, only single cells showing flattened cell morphology were found. Odontoblasts beneath the lesion suffered severe damage and diminished nestin immunoreaction were observed in all cases of carious lesions with pulp exposure. Conclusion: Cariogenic bacteria invasion may damage the odontoblasts by affecting the morphology and vitality of the cells. The severity of the damage of the odontoblasts may increase as the bacterial invasion progresses toward the pulp.Latar belakang: Karies merupakan penyakit yang disebabkan oleh bakteri, yang dapat memengaruhi jaringan keras gigi maupun pulpa. Pada pulpa gigi manusia terdapat sel odontoblas yang tersusun atas lapisan sel. Odontoblas terletak pada tepi kamar pulpa, sehingga sel ini merupakan sel yang pertama kali bertemu dengan bakteri kariogenik dan produk-produknya yang terdapat dalam lesi karies. Tujuan: Penelitian ini bertujuan untuk mengetahui pengaruh invasi bakteri kariogenik terhadap sel odontoblas gigi manusia. Metode: Lima buah gigi molar ketiga utuh dan 15 gigi molar ketiga yang mengalami karies pada permukaan oklusal dengan berbagai tingkat keparahan diekstraksi karena akan menjalani perawatan ortodontik atau perawatan lainnya. Gigi-geligi tersebut kemudian difiksasi, didekalsifikasi dengan larutan EDTA 10% (pH 7,4), dan ditanam dalam parafin. Spesimen gigi tersebut kemudian dipotong dengan ketebalan 5 μm dan diwarnai dengan hematoksilin eosin dan Gram, serta immunohistokimia dengan nestin. Spesimen kemudian diamati di bawah mikroskop cahaya. Hasil: Pada gigi normal, lapisan odontoblas terdapat di sepanjang tepi kamar pulpa dengan morfologi sel normal. Disorganisasi ringan pada lapisan odontoblas tampak pada kasus-kasus karies dengan kedalaman enamel. Pada kasus-kasus lesi karies dengan kedalaman dentin, lapisan odontoblas tidak tampak pada daerah di bawah lesi, hanya ditemukan sel odontoblas tunggal dengan dengan morfologi sel yang pipih. Odontoblas di bawah lesi mengalami kerusakan yang parah dan tidak menunjukkan nestin immunopositif merupakan gambaran dari kasus-kasus karies dengan pulpa terbuka. Kesimpulan: Invasi bakteri kariogenik dapat menyebabkan kerusakan sel odontoblas dengan menyebabkan perubahan morfologi dan memengaruhi vitalitas selnya. Kerusakan sel akan semakin parah dengan semakin dalam invasi bakteri ke arah pulpa.

Page 2 of 95 | Total Record : 950

1 2 3 4 5

Filter by Year

2005 2025

Filter By Issues

All Issue Vol. 58 No. 4 (2025): December Vol. 58 No. 3 (2025): September Vol. 58 No. 2 (2025): June Vol. 58 No. 1 (2025): March Vol. 57 No. 4 (2024): December Vol. 57 No. 3 (2024): September Vol. 57 No. 2 (2024): June Vol. 57 No. 1 (2024): March Vol. 56 No. 2 (2023): June 2023 Vol. 56 No. 1 (2023): March 2023 Vol. 56 No. 4 (2023): December Vol. 56 No. 3 (2023): September Vol. 55 No. 4 (2022): December 2022 Vol. 55 No. 3 (2022): September 2022 Vol. 55 No. 2 (2022): June 2022 Vol. 55 No. 1 (2022): March 2022 Vol. 54 No. 4 (2021): December 2021 Vol. 54 No. 3 (2021): September 2021 Vol. 54 No. 2 (2021): June 2021 Vol. 54 No. 1 (2021): March 2021 Vol. 53 No. 4 (2020): December 2020 Vol. 53 No. 3 (2020): September 2020 Vol. 53 No. 2 (2020): June 2020 Vol. 53 No. 1 (2020): March 2020 Vol. 52 No. 4 (2019): December 2019 Vol. 52 No. 3 (2019): September 2019 Vol. 52 No. 2 (2019): June 2019 Vol. 52 No. 1 (2019): March 2019 Vol. 51 No. 4 (2018): December 2018 Vol. 51 No. 3 (2018): September 2018 Vol. 51 No. 2 (2018): June 2018 Vol. 51 No. 1 (2018): March 2018 Vol. 50 No. 4 (2017): December 2017 Vol. 50 No. 3 (2017): September 2017 Vol. 50 No. 2 (2017): June 2017 Vol. 50 No. 1 (2017): March 2017 Vol. 49 No. 4 (2016): December 2016 Vol. 49 No. 3 (2016): September 2016 Vol. 49 No. 2 (2016): June 2016 Vol. 49 No. 1 (2016): March 2016 Vol. 48 No. 4 (2015): December 2015 Vol. 48 No. 3 (2015): September 2015 Vol. 48 No. 2 (2015): June 2015 Vol. 48 No. 1 (2015): March 2015 Vol. 47 No. 4 (2014): December 2014 Vol. 47 No. 3 (2014): September 2014 Vol. 47 No. 2 (2014): June 2014 Vol. 47 No. 1 (2014): March 2014 Vol. 46 No. 4 (2013): December 2013 Vol. 46 No. 3 (2013): September 2013 Vol. 46 No. 2 (2013): June 2013 Vol. 46 No. 1 (2013): March 2013 Vol. 45 No. 4 (2012): December 2012 Vol. 45 No. 3 (2012): September 2012 Vol. 45 No. 2 (2012): June 2012 Vol. 45 No. 1 (2012): March 2012 Vol. 44 No. 4 (2011): December 2011 Vol. 44 No. 3 (2011): September 2011 Vol. 44 No. 2 (2011): June 2011 Vol. 44 No. 1 (2011): March 2011 Vol. 43 No. 4 (2010): December 2010 Vol. 43 No. 3 (2010): September 2010 Vol. 43 No. 2 (2010): June 2010 Vol. 43 No. 1 (2010): March 2010 Vol. 42 No. 4 (2009): December 2009 Vol. 42 No. 3 (2009): September 2009 Vol. 42 No. 2 (2009): June 2009 Vol. 42 No. 1 (2009): March 2009 Vol. 41 No. 4 (2008): December 2008 Vol. 41 No. 3 (2008): September 2008 Vol. 41 No. 2 (2008): June 2008 Vol. 41 No. 1 (2008): March 2008 Vol. 40 No. 4 (2007): December 2007 Vol. 40 No. 3 (2007): September 2007 Vol. 40 No. 2 (2007): June 2007 Vol. 40 No. 1 (2007): March 2007 Vol. 39 No. 4 (2006): December 2006 Vol. 39 No. 3 (2006): September 2006 Vol. 39 No. 2 (2006): June 2006 Vol. 39 No. 1 (2006): March 2006 Vol. 38 No. 4 (2005): December 2005 Vol. 38 No. 3 (2005): September 2005 Vol. 38 No. 2 (2005): June 2005 Vol. 38 No. 1 (2005): March 2005 More Issue

Home Page

OAI Link

Editorial Team

Contact

Reviewer

Google Scholar

Contact Name
Saka Winias

Contact Email
saka.winias@gmail.com

Phone
-

Journal Mail Official
dental_journal@yahoo.com

Editorial Address
-

Location
Kota surabaya,

Jawa timur

INDONESIA

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Home Page

OAI Link

Editorial Team

Contact

Reviewer

Google Scholar

Contact Name Saka Winias

Contact Email saka.winias@gmail.com

Phone -

Journal Mail Official dental_journal@yahoo.com

Editorial Address -

Location Kota surabaya, Jawa timur INDONESIA

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Contact Name
Saka Winias

Contact Email
saka.winias@gmail.com

Phone
-

Journal Mail Official
dental_journal@yahoo.com

Editorial Address
-

Location
Kota surabaya,

Jawa timur

INDONESIA