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Raymond Pranata
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ijc@inaheart.org
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Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
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INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 8 Documents
 1 
Search results for , issue "Vol. 29, No. 3 September - Desember 2008" : 8 Documents clear
Seks dan Penyakit Jantung Dede Kusmana
Jurnal Kardiologi Indonesia Vol. 29, No. 3 September - Desember 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i3.179

Abstract

Hubungan seks adalah fitrah manusia yang berkeluarga dalam membentuk ikatan pernikahan yang diridhoi Allah SWT agar tercipta keluarga yang sakinah (tentram), mawaddah- warahmah (kasih sayang).Kebutuhan manusia sebagai mahluk sosial disamping makanan, pakaian, perumahan juga berkeluarga atau dalam istilah populer kebutuhan seks, tidak terkecuali penderita penyakit jantung. Penelusuran pustaka yang mengungkap aktifitas seksual terbatas pada penderita penyakit jantung koroner, sehingga untuk penderita lainnya belum ada. Penderita penyakit jantung akan mengalami berbagai kendala berkaitan dengan tingkat berat ringannya penyakit, kondisi psikologis (perasaan takut) penderita maupun pasangannya. Semua kendala tersebut akan mempengaruhi kehidupan seksual, sehingga tidak jarang mengalami kemunduran atau malahan tidak melakukan hubungan sama sekali.
Disfungsi Autonom pada Pasien Penyakit Jantung Hipertensi Asimptomatik: Hasil evaluasi pemulihan laju jantung Yasmin Tadjoedin; Ismoyo Suni; Basuni Radi
Jurnal Kardiologi Indonesia Vol. 29, No. 3 September - Desember 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i3.180

Abstract

Background.Imbalance of the autonomic nervous system has been known to have an important role in the pathophysiology of chronic heart failure (CHF). How early this imbalance occurs in CHF patients has not much been studied. We hypothesized that imbalance of the autonomic function evaluated with heart rate recovery (HRR) already occur in asymptomatic hypertensive patients with left ventricular hypertrophy (LVH) as subset of an early stage of CHF patients . Methods.Sixty two hypertensive patients (31 with LVH by echocardio-graphic criteria and 31 patients without LVH ) underwent treadmill exercise testing using the Bruce protocol and immediate supine on the recovery phase. Heart rate recovery was evaluated on minute 1 and 2 of the recov-ery phase and considered abnormal if = 18x/min in minute-1(HRR1) and = 42x/min in minute-2 (HRR2).Results.The mean HRR was significantly lower in hypertensive patients with LVH then the ones without LVH (19,48 ± 7,21 vs 26,45 ± 8,03) in minute-1, and (38,35 ± 11,19 vs 45,35 ± 9,97) in minute-2. An abnormal-ity in HRR1 percentage was found in 48.4% in patients with HHD and in 12.9% in patients without HHD. An inverse weak correlation (r = 0.329, p = 0.009) was found between LV mass index and HRR1.Conclusions.Abnormality of the HRR has already been found in hyper-tensive patients with LVH without signs or symptom of CHF suggesting an early occurrence of imbalance of the autonomic nervous system in early stage of CHF.
Pemulihan Laju Jantung pada Penyakit Jantung Hipertensi Budhi Setianto
Jurnal Kardiologi Indonesia Vol. 29, No. 3 September - Desember 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i3.181

Abstract

Penelitian disfungsi autonom menggunakan tes treadmil pada fase pemulihan laju jantungnya pada fenomena menurunnya heart rate recovery memang lazim.Kizilbash MA tahun 2006, telah menelitinya pada sindroma metabolik, menyimpulkan terjadinya disfungsi autonom pada komponen metabolik sindrom: tekanan darah, trigliserid, lingkar perut dan LDL-C.Dengan metode lainnya, yaitu standard deviation of RR intervals (SDNN), Wu JS telah melaporkan penelitiannya bahwa disfungsi parasimpatis telah ada pada normotensi dengan riwayat keluarga hipertensi, juga tampak pada pre-hipertensi dan hipertensi. Perubahan peranan syaraf otonom dengan penguatan simpatis lebih nyata pada kasus pre-hipertensinya.
Hipertensi Pulmonal Persisten Segera Setelah Balloon Mitral Valvuloplasty Irwan H Siahaan; Manoefris Kasim; Amiliana M Soesanto; Ganesja M Harimurti
Jurnal Kardiologi Indonesia Vol. 29, No. 3 September - Desember 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i3.182

Abstract

Background.Pulmonary hypertension frequently complicates mitral stenosis. Increased pulmonary artery pressure results from raised left atrial pressure, pulmonary arteriolar vasoconstriction, and obliterative changes in the pulmonary vascular bed, and usually responds to relief of mitral stenosis. However, severe pulmonary hypertension may persist after mechanical relief of mitral stenosis.Methods.A case cross sectional study was conducted in mitral stenosis patients to study the immediate effect of BMV for severe pulmonary hypertension. Pulmonary hypertension was assessed by echocardiography before procedur and 24 – 72 hours after procedure. Analysis was done to assess the relation of age, gender, left atrial size, gradient across mitral valve, mitral valve area and systolic PA pressure before procedure on the persistency of severe pulmonary hypertension.Results.There were 183 patients with mitral stenosis were found in January 2007 – December 2008. Seventy three were severe pulmonary hypertension (systolic PAP = 80 mmHg) and 61 patients were mild – moderate pulmonary hypertension ( systolic PAP < 80 mmHg). Only 54 patients were fulfilling study criteria. Of 54 cases included to analysis, 66.7% were female, 70.4% were in sinus rhytm and had age of 35.44 ± 10.55 yo. The mean mitral valve gradient (MVG) decreased from 18.53 ± 6.40 to 8.50 ± 4.84 mm Hg (p< 0.001). Mitral valve area (MVA) increased from 0.76 ± 0.26 to 1.36 ± 0.36 cm 2 (p < 0.02). LA diameter decreased from 52.87 ± 7.14 to 49.13 ± 7.35 mm (p = 0.01). The pulmonary artery systolic pressure decreased from 100.48 ± 15.77 to 79.83 ± 17.49 mm Hg (p< 0.001). Systolic PAP > 95 mmHg was associated with persistency of PH in mitral stenosis patients with 58% sensitivity and 30% specificity.Conclusions.The degree of Systolic PAP before BMV is associated with persistency of severe pulmonary hypertension. Systolic PAP > 95 mmHg was associated with persistency of PH in mitral stenosis patients with 58% sensitivity and 70% specificity.
Hipertensi Pulmonalis Resisten Pasca Peniupan Balon pada Stenosis Mitral Budi Susetyo Pikir
Jurnal Kardiologi Indonesia Vol. 29, No. 3 September - Desember 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i3.183

Abstract

Hipertensi Pulmonalis (Arterial) yang terjadi pada Stenosis Mitral termasuk kelompok Hipertensi Pulmonalis Arterial Sekunder akibat adanya Hipertensi Pulmonalis Venous Pasif yang dapat disebabkan berbagai penyakit yaitu antara lain trombosis vena pulmonalis dan bermacam kelainan Jantung Kiri termasuk Penyakit Katub Mitral dan katub Aorta.Dengan koreksi Stenosis Mitral (dengan peniupan balon atau pembedahan) diharapkan terjadi pe-nurunan Hipertensi Pulmonalis Arterial tersebut. Tetapi kenyataannya tidak semua penderita mengalami penurunan tekanan darah arteri pulmonalis. Pada Penelitian Irwan dkk. sekitar 21.1 % penderita masih terdapat Hipertensi Pulmonalis berat. Hal ini mudah dipahami bahwa Hipertensi Pulmonalis apapun sebabnya akhirnya akan terjadi perubahan patologi arteria pulmonalis.Pada Penyakit Jantung Bawaan (PDA, VSD besar, ASD Besar) dapat terjadi pada dekade 1 sampai ke-3 yang akhirnya menjadi Sindroma Eisenmenger. Heath dan Edwards membuat klasifikasi perubahan patologi menjadi 6 derajat. Pada Hipertensi Pulmonalis Arterial karena penyakit Mitral pun dapat mengalami perubahan patologi.
Penilaian Fungsi Diastolik Ventrikel Kiri Dengan Ekokardiografi Ario Soeryo Kuncoro
Jurnal Kardiologi Indonesia Vol. 29, No. 3 September - Desember 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i3.184

Abstract

Diastol merupakan proses yang kompleks yang dipengaruhi oleh beberapa faktor meliputi iskemi, frekuensi denyut jantung, kecepatan relaksasi, complianceventrikel kiri ( terdiri dari elastic recoildan stiffness), pengisian ventrikel kiri, complianceatrium kiri, gradien antara atrium kiri dan ventrikel kiri dan segmental wall coordination of the heart muscle.
POBA pada Stenosis Sinus Koronarius Saat Implantasi CRT Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 29, No. 3 September - Desember 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i3.185

Abstract

Seorang laki-laki 54 tahun dengan PJK dan gagal jantung refrakter sekalipun diberikan terapi yang optimal. EKG menunjukkan gambaran irama sinus dengan RBBB, interval PR 240 mdet dan durasi QRS 160 mdet. Pemeriksaan ekokardiografi menunjukkan fungsi sistolik yang rendah dengan EF 15%, dilatasi LV dan terdapat bukti disinkroni intraventrikular.Terapi rutin pasien meliputi dosis optimal ACE inhibitor, ARB, beta blocker, lasix, spironolacton, nitrat, dan aspirin. Akan tetapi pasien masih sangat simtomatik dengan kelas fungsional NYHA kelas IV. Diputuskan untuk dilakukan pemasangan CRT.
Gelombang P yang Berubah-ubah: Apa mekanismenya? Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 29, No. 3 September - Desember 2008
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v29i3.186

Abstract

Seorang anak laki-laki, 13 tahun diketahui menderita ASD sekundum besar dengan pirau kiri ke kanan. Flow ratio 2,5.Terdapat hipertensi pulmonal ringan dengan MPAP 50 mmHg. Keluhan mudah cape. Pasien mendapat terapi amiodaron dan bisoprolol karena ada episode VPC bigemini, selain mendapat captopril. EKG strip menunjukkan perubahan dinamis yang tidak berkaitan dengan pola aktivitas pasien.

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