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Raymond Pranata
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Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
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INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 712 Documents
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Stenosis Mitral Kongenital: Parachute Mitral Valve Laporan 2 kasus NIR Asnil; Ganesja M Harimurti; Poppy S Roebiono; Anna Ulfah Rahajoe; J Rachmat
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.268

Abstract

Mitral stenosis kongenital merupakan penyakit jantung bawaan (PJB) yang jarang terjadi. Laporan-laporan terdahulu menyebutkan angka insiden sekitar 0,4-0,8% dari seluruh pasien PJB. Keadaan ini dapat terjadi tersendiri (isolated), atau disertai dengan PJB lainnya, seperti pada  Shone’s complex,atau sebagai bagian dari Hypoplastic Left Heart Syndrome.
Tatalaksana sindroma Wolf-Parkinson-White terselubung pada pasien dengan Alat Pacu Jantung Daf Juzar; Yoga Yuniadi; Muhammad Munawar
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.269

Abstract

Wolf-Parkinson-White syndrome is part of the supraventricular tachycardias, which are relatively common, often repetitive, occasionally persistent and rarely life threat-ening. Management of atrioventricular reciprocating tachycardia (AVRT) in patients with permanent pacemaker (PPM) is rarely being discussed. We would like to report our experience in the management of two cases with AVRT, one on VVIR and another one on AAI. In the first case catheter ablation of the atrioven-tricular juction was performed, while in the second case arrhythmia was termi-nated using burst pacing.
Tatalaksana Gagal Jantung Akut pada pasien dengan Gangguan Fungsi Ginjal Muhammad A Basalamah
Jurnal Kardiologi Indonesia Vol. 28, No. 1 Januari 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i1.270

Abstract

Gagal jantung (GJ) merupakan penyebab utama mortalitas dan morbiditas kardiovaskular. Dalam kurun waktu 20 tahun terakhir jumlah pasien GJ terus meningkat hingga mencapai 155%. Hal ini terjadi karena populasi usia lanjut terus bertambah dan tatalaksana infark miokard akut semakin berkembang.Gagal jantung akut (GJA) merupakan salah satu penyakit yang menghabiskan biaya perawatan kesehatan terbesar. Sekitar 47% pasien GJA yang pulang dari perawatan rumah sakit, akan menjalani rawat ulang dalam kurun waktu 90 hari. Yang menjadi alasan utama adalah, perawatan medis yang kurang agresif, terapi sub-optimal dan tingkat kepatuhan pasien yang rendah.
Efek Enhanced External Counterpulsation Terhadap Perbaikan Kontraktilitas Miokard yang masih viabel pada penderita penyakit jantung koroner Utojo Lubiantoro; Fadilah Supari; Lies Dina Liastuti
Jurnal Kardiologi Indonesia Vol. 26, No. 1 Januari - Maret 2002
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v26i1.271

Abstract

Objective: To obtain data about the effect of EECP on dysfunctional but viable myocardial contractility in patient with coronary heart disease.Methods: This is an experimental study which include twenty male coronary heart disease but viable myocardium. Before EECP treatment, all subjects under 2D echocardiographic examination and myocardial contractility was measured as WMSI. Patients is given EECP 1 hour daily (5-6 times a week) for a total of 36 hours. Immediately after EECP treatment, WMSI is measured again for each subject. The value between before and after EECP treatment is analyzed with Wilcoxon signed rank test. Significant result if p < 0,05.Results: WMSI decreased after EECP treatment from 1,58+0,39 to 1,53+0,40, but the difference was not significant.Conclusion: EECP does not improve dysfunctional but viable myocardial contractility in patients with coronary heart disease
Perubahan Nilai Aggregasi Platelet Pada Penderita Sindrom Koroner Akut dengan Terapi Enoxaparine dibandingkan dengan Terapi Heparin Tak Terfraksinasi Ridwan Sofyansyah; Otte J Rachman
Jurnal Kardiologi Indonesia Vol. 26, No. 1 Januari - Maret 2002
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v26i1.272

Abstract

Tujuan dan Latar Belakang: Pengaruh heparin tak terfraksinasi (UFH) terhadap platelet dapat menyebabkan platelet inhabitasi atau ativasi. Menginhibsi platelet melalui ikatan heparin - AT III dapat mengurangi produksi tromblin. Pengaruh heparin pada fungsi platelet dapat meningkatkan produksi tomboxan A2 dan inhibitasi produksi dari prostacyclin I2. Percobaan ini didesain untuk meneliti pengaruh UFH atau enoxaparine pada aggregasi platelet pada pasien dengan sindrom koroner akut.Metoda: Penelitian ini melibatkan 33 pasien dengan sindrom koroner, 17 pasien menerima UFH dan 16 pasien menerima enoxaparine. Penelitian ini merupakan penelitian acak yang terbuka. Penelitian aggregasi platelet dilakukan terhadap semua pasien sebelum dan setelah 5 hari pemberian UFH atau enoxaparine. Peneliti menggunakan program komputer SPSS versi 10\0 untuk menganalisa data.Hasil: Aggregasi platelet menurun pada kelompok enoxaparun dari 29,5+11,8% ke 21,6+10,3%, p=0,04 (dengan 5 um ADP) dan dari 44,1+16% ke 33,6+10,8%, p=0,04 (dengan 10 um ADP). Dalam kelompok UFH dari 25,7+13,5% ke 22,6+12,5%, p=0,46 (dengan 5 um ADP) dan 39,2%+14,4% ke 37,4+14,3%, p=0,45 (dengan 10 um ADP).Kesimpulan: Terjadi penurunan aggregasi platelet pada pasien dengan sindroma koroner akut yang mendapatkan terapi enoxaparine tapi tidak pada pasien yang mendapatkan terapi UFH.
Pengaruh Angioplasti Koroner Terhadap perubahan Dispersi QT Pasca Infark Miokard Pudjo Harasto; Rossana Barack; Anasthasia Sari; Otte J Rachman
Jurnal Kardiologi Indonesia Vol. 26, No. 1 Januari - Maret 2002
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v26i1.273

Abstract

Latar Belakang: Artimia ventrikular adalah penyebab yang paling umum dari kematian jantung mendadak dalam pasca infark miokard (IM). Banyak tanda-tanda dari kematian jantung mendadak pasca infark miokard yang telah dikenali. Salah satu dari tanda-tanda ini adalah dispersi QT yang meningkat pada kejadian iskhemia. Penelitian terdahulu mengungkapkan korelasi antara dispersi QT dan artimia ventrikular. Kami mengevaluasi pengaruh dari angioplasti koroner pada dispersi QT pada penderita pasca-IM.Metoda dan Hasil: Kami mengukur QT dan mengoreksi dispersi QT (QTc) sebelum angiopati, 24 jam dan 33 hari setelah prosedur. 37 Pasien diikutsertakan dalam penelitian ini, tetapi hanya 24 pasien yang dapat dianalisa. Garis acuan dispersi QT adalah 101,67+38,75 (115,02+36,82) ms. Berlainan halnya dengan dispersi QT, dispersi QT yang terkoreksi menurun secara bermakna menjadi 94,50+36,05 ms (p=0,018 dibanding garis acuan) setelah 24 jam. Evaluasi selanjutnya ditemukan bahwa dispersi QT tidak berubah terhadap garis acuan setelah 33 hari.Kesimpulan: Disimpulkan bahwa dispersi QT (QTc) pada pasien pasca-IM tidak menurun setelah angioplasti dengan berhasil.
Kematian Jantung Mendadak di Indonesia Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 30, No. 3 September - Desember 2009
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v30i3.275

Abstract

Kematian jantung mendadak (KJM) adalah kematian tidak diduga dan bersifat mendadak yang terjadi dalam waktu kurang dari satu jam setelah timbul gejala dan disebabkan oleh penyakit jantung. KJM terjadi akibat suatu hentu jantung yang tidak tertolong. Terdapat beberapa istilah yang saling tumpang tindih yaitu KJM, henti jantung dan kolaps kardiovaskular. KJM merupakan suatu keadaan berhentinya fungsi biologis yang menetap, terjadi secara tiba-tiba dan tidak terduga. Mekanisme KJM umumnya akibat hilangnya kemampuan mekanik jantung yang luas yang terjadi akibat henti jantung.
Volum Atrium Kiri Pra-Bedah Sebagai Prediktor Fibrilasi Atrium Pasca Bedah Pintas Arteri Koroner Juzni Alkatiri; Muhammad Munawar; Bambang B Siswanto
Jurnal Kardiologi Indonesia Vol. 30, No. 3 September - Desember 2009
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v30i3.276

Abstract

Background. Atrial fibrillation is the most common arrhythmia complication following coronary artery bypass graft (CABG). The incidence is raging from 10% to 50%. Previous studies have shown that an increasing left atrial (LA) volume is one of the risk factor in the occurence of atrial fibrillation following CABG.Aim. To investigate whether LA volume before CABG is an independent predictor for the occurence of atrial fibrillation following CABG.Methods. This is an observational cross-sectional study on patients underwent CABG in National Cardiac Center Harapan Kita. A thorough echocardiography examination, including the LA volume, was done and other clinical parameters were collected. The occurence of atrial fibrillation was observed during hospitalization.Result. One hundred patients were included in this study and using logistic regression analysis, we found that age (OR 1, 532 (CI:1,264 - 1,857); p < 0,001) and LA volume (OR 1,113(CI:1,042-1,189) are significantly correlated with the occurence of atrial fibrillation post-CABG. One mililiter increase in LA volume will increase 11% of the risk of atrial fibrillation following CABG.Conclusions. Left atrial volume age were independent predictors of atrial fibrillation following CABG.
Pembesaran Volum Atrium Kiri Sebagai Prediktor Fibrilasi Atrium Pasca Bedah Pintas Koroner: Pentingnya Koreksi Terhadap Luas Permukaan Tubuh Hamed Oemar
Jurnal Kardiologi Indonesia Vol. 30, No. 3 September - Desember 2009
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v30i3.277

Abstract

Pada populasi umum atrium kiri yang membesar sering ditemui dan merupakan tanda dari suatu penyakit kardiovaskular. Banyak penelitian yang sudah membuktikan adanya korelasi yang kuat antara atrium kiri yang membesar (diameter atau volum) dengan terjadinya fibrilasi atrium (FA). Meningkatnya volum atrium kiri (VAKI) merupakan marka peningkatan tekanan pengisian ventrikel kiri yang kronis dan merupakan prediktor FA setelah suatu bedah pintas arteri koroner (BPAK). Diameter atrium kiri diukur dengan M-mode pada pandangan parasternal short axis pada posisi basal jantung sesuai dengan rekomendasi ASE. Pengukuran atrium kiri seharusnya dikoreksi terhadap luar permukaan tubuh.
Hubungan Intimal Media Thickness Karotis Dengan Faktor Risiko Kardiovaskular pada Pasien Gagal Ginjal Kronik Pra-Dialisis Vicky Wahidji; Ismoyo Sunu; Dede Kusmana
Jurnal Kardiologi Indonesia Vol. 30, No. 3 September - Desember 2009
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v30i3.278

Abstract

Background. The incidence of cardiovascular diseases is markedly higher in patients with Chronic Kidney Disease (CKD). Both conventional and non-conventional cardiovascular risk factors play a role in this observed phenomenon. In general population, Intima Media Thickness (IMT) has been found to correlate with the incidence of cardiovascular disease and it also serves as an independent predictor of cardiovascular mortality in CKD patients undergoing chronic hemodialysis.Objectives. To investigate the relationship between carotid IMT with cardiovascular risk factors in a population of CKD patients prior to chronic hemodyalisis.Methods. This was a cross sectional study to measure carotid IMT in CKD patients using B-mode ultrasonography. Glomerular Filtration Rate of subjects was calculated by Modification of Diet in Renal Disease (MDRD) formula. The cardiovascular risk factors assessed included plasma blood sugar, lipid profile, renal function, blood pressure, body weight, sex and age.Results. We recruited 76 subjects, mostly male, over 65 years of age and also obese (mean body mass index of 26,3 kg/m2). Most of the subjects had uncontrolled systolic blood pressure, plasma sugar level and lipid profile. The mean systolic blood pressure was 134.21 mmHg, fasting blood sugar 110mg/dl, high and low density lipoprotein were 41.83 mg/dl and LDL 117.36 mg/dl, respectively. There was also a trend of CKD resulted in mean of IMT with the fall in GFR; stadium 3, 4 and 5 of CKD resulted in mean IMT of 0.9 and 1.3 mm, respectively.Conclusion. We did not find any correlation between IMT and sex, age, hypertension, diabetes mellitus, smoking nor obesity. The decline in renal function was associated with a significant increase in IMT (p < 0.005).

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