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Pramudya , I Nengah Andra Putra
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Agriculture, Biological Sciences & Forestry Biochemistry, Genetics & Molecular Biology Immunology & microbiology

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Anatomical and Physiological Characteristics of Neonatal and Infant Skin: Effects on Immunity and Dermatitis Prevalence Mario, Andina Nabilah; Girsang, Andrew Deardo Purba; Srigede, Baiq Kayla; Hadiyati, Fathmi; Pramudya , I Nengah Andra Putra; Saraswati, Ni Wayan Citra Ayu; Helmiana, Putri Fatimah; Pramudia, Prananda Rizki; Fildzani, Qashrina Vania; Nurhidayati; Tarigan, Rizky Agung Fatahillah
Jurnal Biologi Tropis Vol. 24 No. 1b (2024): Special Issue
Publisher : Biology Education Study Program, Faculty of Teacher Training and Education, University of Mataram, Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29303/jbt.v24i1b.7918

Abstract

The skin has an important role in protecting the human body. However, in infants, the skin has a structure and function that are not fully mature yet, making infants vulnerable to various skin disorders. This article aims to review the structure, function, and development of infant skin, as well as the common diseases that occur in infant skin, including the causes, pathophysiology, and management. A literature review was conducted using scientific articles related to these topics. The review results show that infant skin has a different structure compared to adult skin, and the maturation of its structure and function occurs gradually. Infant skin is protected by the microbiota and the primary immune system. Common skin diseases in infants, such as dermatitis, are caused by an interaction between genetic and environmental factors, and involve the activation of skin cells, the release of mediators, and an immune response. A good understanding of the structure, function, and skin diseases of infants is very important for making the right diagnosis and management, so that the quality of life of infants can be maintained.
Navigating Non-Immune Hemolytic Anemia Membranopathy: Definitions to Clinical Strategies Zahira, Lu'lu Al Fatina; Madani , Ayda Fitri; Kamilah , Nilam Nurmilatun; Amany , Zhafirah; Mario , Andina Nabilah; Amaliya , Annisa Risqi; Makbul , Ini Hidayat; Zakiyyah , Luthfi; Pramudya , I Nengah Andra Putra; Putra , Made Raditya Arhya
Jurnal Biologi Tropis Vol. 24 No. 1b (2024): Special Issue
Publisher : Biology Education Study Program, Faculty of Teacher Training and Education, University of Mataram, Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29303/jbt.v24i1b.7957

Abstract

Anemia may arise from increased destruction of erythrocytes, blood loss, or impaired erythropoiesis, as indicated by diminished levels of red blood cells (RBCs) or hemoglobin. Hemolytic anemia involves accelerated RBC lysis that surpasses the bone marrow's compensatory capacity (approximately 120 days) and is categorized into immune and non-immune types. Non-immune hemolytic anemia is primarily hereditary, comprising enzymes, hemoglobinopathies, and membranopathies. This literature review specifically examines non-immune hemolytic anemia membranopathies, detailing their definitions, classifications, etiologies, pathophysiologies, diagnostics, and management. A thorough literature search was conducted using books, reviews, and case studies. Membranopathies are divided into structural defects, such as hereditary spherocytosis (HS) and hereditary elliptocytosis (HE), and altered membrane transport functions, including hereditary stomatocytosis and hereditary xerocytosis. Common symptoms include fatigue, weakness, jaundice, and dark urine, diagnosed through hematological tests and genetic analysis. Management is primarily supportive, involving blood transfusions and folic acid supplementation, with splenectomy recommended for severe cases. A comprehensive understanding of the pathophysiology of these hereditary conditions is critical for accurate diagnosis and effective treatment, underscoring the importance of continued research into novel therapeutic approaches.
Co-Authors Amaliya , Annisa Risqi Amany , Zhafirah Fildzani, Qashrina Vania Girsang, Andrew Deardo Purba Hadiyati, Fathmi Helmiana, Putri Fatimah Kamilah , Nilam Nurmilatun Madani , Ayda Fitri Makbul , Ini Hidayat Mario , Andina Nabilah Mario, Andina Nabilah Nurhidayati Pramudia, Prananda Rizki Putra , Made Raditya Arhya Saraswati, Ni Wayan Citra Ayu Srigede, Baiq Kayla Tarigan, Rizky Agung Fatahillah Zahira, Lu'lu Al Fatina Zakiyyah , Luthfi