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Fatikha Rudia Ahda
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Biochemistry, Genetics & Molecular Biology Health Professions Immunology & microbiology Mechanical Engineering Public Health

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Graves Disease: Diagnosis dan Tatalaksana Lestary, Ayundha Rizky; Fatikha Rudia Ahda; I Gede Aditya Satrya Bhuwana Cakra; Ni Made Sri Padma Puspita; Eva Triani
Lombok Medical Journal Vol. 2 No. 3 (2023): Lombok Medical Journal Volume 2 Nomor 3
Publisher : Faculty of Medicine, Universitas Mataram

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29303/lmj.v2i2.2754

Abstract

Abstract: Hyperthyroidism is an increase in the amount of thyroid hormone synthesis and secretion by the thyroid gland. Graves Disease (GD) is the most common cause of hyperthyroidism. Graves Disease (GD) is more common in women than men and in patients with a family history of GD. Graves Disease can also be triggered by environmental factors such as stress, smoking, infection, and exposure to iodine. Graves Disease manifests in the form of the Merseburger triad consisting of thyrotoxicosis, diffuse goiter, and ophthalmopathy (orbitopathy) with symptoms of agitation, palpitations, and muscle weakness. In making the diagnosis of GD, 2 tests are carried out, namely a thyroid function test which consists of examinations to diagnose hypothyroidism, namely Wayne index scoring, TSH test, and FT4 as well as tests to distinguish GD from other causes of hyperthyroidism, namely TRab, radioactive iodine uptake scan with I-123 or I-131, thyroid ultrasound with doppler, T3/T4 ratio values, and CT or MRI. The goal of treatment for GD is to control and improve the condition based on the pathophysiology of GD, namely the antigen-antibody reaction in the thyroid gland. Modalities for the treatment of GD consist of anti-thyroid drugs, surgery, and radioactive iodine (RAI) treatment with sodium-131(131I). The choice of treatment is based on the severity of thyrotoxicosis, age, thyroid size, availability of modalities, response to treatment, and other comorbidities.
The Article Review Penggunaan Aspirin sebagai Faktor Pemicu Sindrom Reye Radwa, Ghaniyyah Atifah; Fatikha Rudia Ahda; Indri Setiawati; Khaula Karima; Ni Made Ayu Shinta Dewi; Arif Zuhan
Lombok Medical Journal Vol. 2 No. 3 (2023): Lombok Medical Journal Volume 2 Nomor 3
Publisher : Faculty of Medicine, Universitas Mataram

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29303/lmj.v2i2.3174

Abstract

Reye syndrome (RS) is a rare metabolic disorder which primarily affects children. The disease is generally characterized by acute encephalopathy and fatty degeneration of several parenchymes, especially the liver. Aspirin or acetylsalicylic acid is an over-the-counter drug as a solution to viral infection that would be the trigger for developing this syndrome. Reye syndrome is most commonly caused by viral pathogens like influenza A and B and varicella. Reye syndrome presents signs and symptoms of continued vomiting with or without dehydration, encephalopathy and, in 50% of patients, hepatomegaly. According to the clinical stage developed and modified by Hurwitz, the clinical stage of Reye syndrome is divided in 0-5, including the non-clinical stage (stage 0). The pathophysiological profile of Reye syndrome is unknown. However, it appears to be associated with mitochondrial dysfunction caused by the use of aspirin resulting in impaired fatty acid metabolism, reduced excretion, and impaired hepatic metabolism. A case-control study suggested a strong association between aspirin-taking behavior and the development of Reye's syndrome. A case-control study suggested a strong association between aspirin-taking behavior and the development of Reye's syndrome. This literature will present a discussion of the relationship of aspirin as a trigger for Reye syndrome based on the etiology, pathophysiology, and pharmacodynamics of aspirin.
Graves Disease: Diagnosis dan Tatalaksana Lestary, Ayundha Rizky; Fatikha Rudia Ahda; I Gede Aditya Satrya Bhuwana Cakra; Ni Made Sri Padma Puspita; Eva Triani
Lombok Medical Journal Vol. 2 No. 3 (2023): Lombok Medical Journal Volume 2 Nomor 3
Publisher : Faculty of Medicine, Universitas Mataram

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29303/lmj.v2i2.2754

Abstract

Abstract: Hyperthyroidism is an increase in the amount of thyroid hormone synthesis and secretion by the thyroid gland. Graves Disease (GD) is the most common cause of hyperthyroidism. Graves Disease (GD) is more common in women than men and in patients with a family history of GD. Graves Disease can also be triggered by environmental factors such as stress, smoking, infection, and exposure to iodine. Graves Disease manifests in the form of the Merseburger triad consisting of thyrotoxicosis, diffuse goiter, and ophthalmopathy (orbitopathy) with symptoms of agitation, palpitations, and muscle weakness. In making the diagnosis of GD, 2 tests are carried out, namely a thyroid function test which consists of examinations to diagnose hypothyroidism, namely Wayne index scoring, TSH test, and FT4 as well as tests to distinguish GD from other causes of hyperthyroidism, namely TRab, radioactive iodine uptake scan with I-123 or I-131, thyroid ultrasound with doppler, T3/T4 ratio values, and CT or MRI. The goal of treatment for GD is to control and improve the condition based on the pathophysiology of GD, namely the antigen-antibody reaction in the thyroid gland. Modalities for the treatment of GD consist of anti-thyroid drugs, surgery, and radioactive iodine (RAI) treatment with sodium-131(131I). The choice of treatment is based on the severity of thyrotoxicosis, age, thyroid size, availability of modalities, response to treatment, and other comorbidities.
The Article Review Penggunaan Aspirin sebagai Faktor Pemicu Sindrom Reye Radwa, Ghaniyyah Atifah; Fatikha Rudia Ahda; Indri Setiawati; Khaula Karima; Ni Made Ayu Shinta Dewi; Arif Zuhan
Lombok Medical Journal Vol. 2 No. 3 (2023): Lombok Medical Journal Volume 2 Nomor 3
Publisher : Faculty of Medicine, Universitas Mataram

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29303/lmj.v2i2.3174

Abstract

Reye syndrome (RS) is a rare metabolic disorder which primarily affects children. The disease is generally characterized by acute encephalopathy and fatty degeneration of several parenchymes, especially the liver. Aspirin or acetylsalicylic acid is an over-the-counter drug as a solution to viral infection that would be the trigger for developing this syndrome. Reye syndrome is most commonly caused by viral pathogens like influenza A and B and varicella. Reye syndrome presents signs and symptoms of continued vomiting with or without dehydration, encephalopathy and, in 50% of patients, hepatomegaly. According to the clinical stage developed and modified by Hurwitz, the clinical stage of Reye syndrome is divided in 0-5, including the non-clinical stage (stage 0). The pathophysiological profile of Reye syndrome is unknown. However, it appears to be associated with mitochondrial dysfunction caused by the use of aspirin resulting in impaired fatty acid metabolism, reduced excretion, and impaired hepatic metabolism. A case-control study suggested a strong association between aspirin-taking behavior and the development of Reye's syndrome. A case-control study suggested a strong association between aspirin-taking behavior and the development of Reye's syndrome. This literature will present a discussion of the relationship of aspirin as a trigger for Reye syndrome based on the etiology, pathophysiology, and pharmacodynamics of aspirin.
Co-Authors Arif Zuhan Eva Triani I Gede Aditya Satrya Bhuwana Cakra Indri Setiawati Khaula Karima Lestary, Ayundha Rizky Ni Made Ayu Shinta Dewi Ni Made Sri Padma Puspita Radwa, Ghaniyyah Atifah