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Raymond Pranata
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Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
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INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 712 Documents
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Blok AV Mobitz II atau APC Bigeminy? Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 33, No. 1 Januari - Maret 2012
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v33i5.78

Abstract

Seorang perempuan 82 tahun dengan keluhan fatigue dirujuk ke poli aritmia untuk evaluasi lebih lanjut kemungkinan pemasangan alat pacu jantung menetap. Pemeriksaan fisik dalam batas normal. Rekaman EKG sebagaimana terlihat pada gambar 1.Gambaran EKG di atas memperlihatkan jarak interval PP yang tetap dengan gelombang P yang dikonduksikan ke ventrikel membentuk kompleks QRS (conducted) berselang seling dengan gelombang P yang tidak diteruskan ke ventrikel (non-conducted). Secara singkatrekaman EKG tersebut akan membawa pembaca pada diagnosis blok AV tipe Mobitz II.Blok AV tipe Mobitz II disebut juga blok AV derajat kedua tipe 2 ditandai dengan gambaran EKG yang menunjukkan gelombang P non-conducted yang intermiten tanpa didahului oleh pemanjangan interval PR dan tidak diikuti oleh pemendekkan interval PR. Umumnya letak blok berada di bawah nodal AV (infranodal). Oleh karena itu kompleks QRS pada Mobitz II hampir selalu memperlihatkan gambaran blok berkas cabang atau blok fasikular. Sebagian ahli mengelompokkan blok 2:1 ke dalam kelompok tersendiri yang tidak termasuk Mobitz I atau II karena hilangnya sifat intermiten blok yang biasanya terjadi pada Mobitz II.
Paradox Obesitas pada Pasien Gagal Jantung Alvin Nursalim; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 32, No. 4 Oktober - Desember 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i4.79

Abstract

Obesitas sudah menjadi sebuah epidemi di negara maju. Ukuran objektif obesitas biasanya dinilai dari nilai IMT, dimana ukuran international untuk obesitas adalah IMT =30 kg/m2, sedangkan untuk ukuran orang Asia obesitas didefinisikan dengan nilai IMT=25 kg/m2.Obesitas memiliki hubungan yang erat dengan tingginya kejadian penyakit kardiovaskular. Obesitas dapat meningkatkan kadar trigliserid yang buruk untuk kesehatan jantung dan menurunkan kadar high density lipoprotein (HDL) yang bersifat kardioprotektif. Selain itu, seiring meningkatnya obesitas, meningkat juga angka hipertensi. Obesitas juga dapat menyebabkan disfungsi diastolik dan berhubungan dengan memburuknya fungsi sistolik.Walaupun obesitas merupakan faktor risiko penyakit jantung koroner, hal yang berbeda ditemukan pada kasus gagal jantung. Berdasarkan beberapa studi, pasien gagal jantung dengan Indeks Masa Tubuh (IMT) yang lebih tinggi memiliki prognosis yang lebih baik dibandingkan mereka dengan IMT yang lebih rendah. Selain itu, analisis dari beberapa studi oleh Oreopoulos et al menyimpulkan bahwa IMT yang lebih tinggi berhubungan dengan prognosis yang lebih baik pada pasien gagal jantung. Hal inilah yang disebut paradox obesitas (Obesity paradox).
Biomarker Baru Monoclonal Antibody Fragmentasi Collagen Type IV Untuk Mendeteksi Acute Myocard Infarction Terkait Infeksi Perviromonas Gingivalis Ketut Muliartha; Mulyohadi Ali; Djanggan Sargowo
Jurnal Kardiologi Indonesia Vol. 32, No. 4 Oktober - Desember 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i4.80

Abstract

Background. Index mortality of atherosclerotic very high. The research conducted by WHO in 1995 show 20% of IMA mortality caused by ath-erosclerotic. The new emerging risk factor infection is the Perviromonas gingivalis. The objective of study to detect IMA in blood sample by using biomarker monoclonal antibody collagen type IV.Method. This research was experimental in vitro with ethic clearance and carried on the biomedic Laboratory, Brawijaya University. The subject were (n=12) and healthy people are (n=4). To show expression of MMP-9 and fragmentation of collagen type IV use western blotting tecnique. The monoclonal antibody were obtain which sub cutan imunization followed by isolating lymphocytes and fusing with myeloma cell to growth of hibridoma and produce antibody. Result. Found in following MMP-9 product the band were 92 kDa and the collagen type IV fragmentation band 60-90 kDa. To test for biomarker reac-tion monoclonal antibody fragmentation collagen type IV in blood sample 1-12 was positive for AMI.Conclusion. The biomarker for monoclonal antibody fragmentation col-lagen type IV shown AMI reaction positive.
Biomarker pada Infark Miokard Akut: Diperlukan yang lebih Dini Suko Adiarto
Jurnal Kardiologi Indonesia Vol. 32, No. 4 Oktober - Desember 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i4.81

Abstract

Paling tidak dalam 4 dekade terakhir, Infark Miokard Akut (IMA) masih menempati urutan pertama dalam daftar penyakit penyebab kematian di seluruh dunia. Secara historis, terdapat kemajuan yang sangat pesat dalam penatalaksanaan penyakit ini, baik dalam segi diagnostik, terapi medikamentosa, maupun tindakan revaskularisasi. Khususnya di bidang diagnostik, penggunaan 3 kriteria WHO yang meliputi nyeri dada tipikal, perubahan EKG dan meningkatnya biomarker (enzim) yang spesifik sangat membantu meningkatkan akurasi diagnostik IMA secara signifikan. Bandingkan, pada tahun 1940­1950 hanya sekitar 15 persen penderita yang dirawat di ICCU dengan dugaan ternyata benar­benar mengalami IMA.Kemajuan di bidang diagnostik juga diikuti oleh kemajuan yang sangat pesat dalam penata laksanaan IMA, dimana fokus utama tatalaksana IMA telah bergeser dari limitasi luasnya infark dengan cara menurunkan kebutuhan oksigen miokard menjadi terapi reperfusi, baik dengan cara farmakologi (fibrinolitik) maupun secara mekanik (Primary PCI)
Fibrilasi Atrial Selama Perawatan Infark Miokard Akut Merupakan Prediktor Stroke Jangka Panjang Manoefris Kasim; Octavia Lilyasari; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 32, No. 4 Oktober - Desember 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i4.82

Abstract

Background. Atrial fibrillation (AF) is a common arrhythmia after acute myocardial infarction (AMI).Mortality in AMI patients with AF also been shown to be up to twice as high as for those without AF. Non hemorrhagic stroke occurs in 0.1% to 1.3%of patients with acute myocardial infarction who are treatedwith thrombolytic, with substantial associated mortality and morbidity. The aim of this study is toelaborate correlation between in hospital AF with long term stroke event inST-elevation myocardial infarction (STEMI) patients who treated with thrombolytic.Methods. Two hundred and thirty STEMI patients (27-72 yo) treated with thrombolytic agent were studied retrospectively. The study end point was Major Cardio-Cerebrovascular Event (MACCE) during 3 years follow up. Results. Eleven patients experience AF episode during STEMI hospitalization. During 3 years follow up 24 patients (10.4%) experienced MACCE which comprised of : cardiac death 4 (1.7%), fatal infarction 2 (0.9%), non fatal infarction 13 (5.7%) and stroke 5 (2.2%). Independent clinical variables were not significant as a predictor for the occurrence of future stroke event, except atrial fibrillation episode (HR 13.4; p<0.005) that was encountered during hospitalization in the setting of AMI.Conclusion. In-hospital AF in STEMI patients treated with thrombolytic agent is a predictor of long term stroke.
Hubungan Antara Matrix Metalloproteinase-9 (MMP-9) Dengan Komplikasi Gagal Jantung Akut Pada Infark Miokard Dengan ST-Elevasi (STEMI) Dan Sindroma Koroner Akut Tanpa ST-Elevasi (NSTEACS) Budi Yuli Setianto; Sofia Mubarika; Indwiani Astuti; Bambang Irawan
Jurnal Kardiologi Indonesia Vol. 32, No. 4 Oktober - Desember 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i4.83

Abstract

Background. Acute coronary syndrome (ACS) often leads to complications of acute heart failure. These complications will increase the morbidity and mortality of patients with ACS.Objective. To determine differences in levels of MMP-9 between STEMI and NSTEACS and the correlation between MMP-9 with acute heart failure between the two groups.Methods. Examination of the samples performed in 79 patients with ACS (38 STEMI and 41 NSTEACS) prior to the action of intravenous thrombolytic or coronary intervention. Differences in levels of MMP-9 in the ACS are experiencing acute heart failure and without heart failure, and differences in levels of MMP-9 in the STEMI and NSTEACS groups were tested with Chi-square, Fisher’s exact test or the Independent t-test.Results. STEMI groups had significantly higher levels of MMP-9 than NSTEACS group 1629.12 ± 719.60 compared to 1033.42 ± 777.12 (p = 0.001). However, STEMI groups who have acuteheart failure are higher but not significant compared with NSTEACS group 14 (36.84) and 11 (26.82) (p = 0.339). There are differences in levels of MMP-9 in ACS with acute heart failure than those who did not: 1698 ± 867.95 ng/mL and 1144.61 ± 713.60 ng/mL (p = 0.004). Background. Acute coronary syndrome (ACS) often leads to complications of acute heart failure. These complications will increase the morbidity and mortality of patients with ACS.Objective. To determine differences in levels of MMP-9 between STEMI and NSTEACS and the correlation between MMP-9 with acute heart failure between the two groups.Methods. Examination of the samples performed in 79 patients with ACS (38 STEMI and 41 NSTEACS) prior to the action of intravenous thrombolytic or coronary intervention. Differences in levels of MMP-9 in the ACS are experiencing acute heart failure and without heart failure, and differences in levels of MMP-9 in the STEMI and NSTEACS groups were tested with Chi-square, Fisher’s exact test or the Independent t-test.Results. STEMI groups had significantly higher levels of MMP-9 than NSTEACS group 1629.12 ± 719.60 compared to 1033.42 ± 777.12 (p = 0.001). However, STEMI groups who have acuteheart failure are higher but not significant compared with NSTEACS group 14 (36.84) and 11 (26.82) (p = 0.339). There are differences in levels of MMP-9 in ACS with acute heart failure than those who did not: 1698 ± 867.95 ng/mL and 1144.61 ± 713.60 ng/mL (p = 0.004). Conclusion. MMP-9 levels are significantly higher in STEMI groups compared with NSTEACS groups, and MMP-9 associated with the incidence ofacute heart failure in ACS. STEMI groups have tended to have acute heart failure are higher than NSTEACS groups. MMP-9 levels are significantly higher in STEMI groups compared with NSTEACS groups, and MMP-9 associated with the incidence ofacute heart failure in ACS. STEMI groups have tended to have acute heart failure are higher than NSTEACS groups.
Implantasi CRT pada Pasien dengan Katup Thebesian Prominen Erwin Mulia; Wenny Fitrina Dewi; Erika Maharani; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 32, No. 4 Oktober - Desember 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i4.84

Abstract

CRT has been established as standard treatment for refractory heart failure in many major clinical trials. Insertion of left ventricle lead is frequently most challenging step. Coronary sinus anatomy is of the most technical burden. A case of CRT implantation with presence of prominent Thebesian valve and stenotic posterolateral vein is presented in thic case report.
Jaras Tambahan Yang “Tidak Biasa” Yoga Yuniadi; Erika Maharani
Jurnal Kardiologi Indonesia Vol. 32, No. 4 Oktober - Desember 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i4.85

Abstract

Two cases of accessory pathway that were considered uncommon are presented. The first case shows of ECG limitation to identified manifest accessory pathway location based on delta wave morphology. The second case shows rare dual accessory pathways. Radiofrequency ablation has successfully eliminated all accessory pathway in both cases.
Dabigatran dibandingkan Warfarin untuk Pencegahan Stroke pada Pasien dengan Atrial Fibrilasi: Laporan Berbasis Bukti Alvin Nursalim; Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 32, No. 4 Oktober - Desember 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i4.86

Abstract

Background. Atrial fibrillation (AF) increases the risk of having stroke as high as five fold. Anticoagulant administration such as vitamin K antagonist has been used regularly to reduce the occurence of stroke. Despite the high efficacy, warfarin has several limitations, including a narrow therapeutic window, multiple food and drug interactions, and the need for frequent laboratory monitoring. Dabigatran, an oral thrombin inhibitor, displays some positive characteristics as the solution to warfarin’s limitations.Aim. To determine the efficacy of dabigatran compared to warfarin for stroke prevention in patients with atrial fibrillation.Methods. A search was conducted on PubMed and Google. The selection of title and abstract was done using inclusion and exclusion criteria. Five original articles were found, but only one study was used. The selected study was critically appraised for its validity, importance and applicability.Result. The administration of 150 mg of dabigatran was superior to warfarin with respect of stroke. The relative risk reduction was 36% in the 150 mg dabigatran group. The rate of stroke was 1.01% per year in the group that received 150 mg dabigatran, as compared with 1.57% per year in the warfarin group (relative risk 0.64; 95% confidence interval, 0.51 to 0.81, p<0.001). The administration of dabigatran increased the risk of gastrointestinal bleeding.Conclusion. In patients with atrial fibrillation, dabigatran given at a dose of 150 mg, as compared with warfarin, was associated with lower rate of stroke. Dabigatran administration requires closed gastrointestinal monitoring.
Pengaruh Latihan Fisik Dan Respon Molekuler Pembuluh Darah Arteri Dyana Sarvasti; Basuni Radi; Budhi Setianto; Andang H Joesoef
Jurnal Kardiologi Indonesia Vol. 32, No. 4 Oktober - Desember 2011
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v32i4.87

Abstract

Regular physical activity (exercise training, ET) has a strong positive link with cardiovascular health. The beneficial effects of ET on the endothelium arteries are believed to result from increased vascular shear stress during ET bouts. A number of mechanosensory mechanisms have been elucidated that may contribute to the effects of ET on vascular function. Exercise training also consistently improves the nitric oxide bioavailability, and the number of endothelial progenitor cells, and diminishes the level of inflammatory markers, namely pro inflammatory cytokines and C-reactive protein. This review summarizes current understanding of control of vascular adaptation by exercise and how these processes lead to improved cardiovascular health.

Page 21 of 72 | Total Record : 712

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