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Raymond Pranata
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Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
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INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 712 Documents
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Peran Lipopolisakarida Helicobacter pylori terhadap Aktivitas Neutrofil pada Penderita Infark Miokard Akut melalui Degradasi Kolagen Tipe IV Djanggan Sargowo; I Ketut Gede Muliartha; Mudyawati Kamaruddin
Jurnal Kardiologi Indonesia Vol. 28, No. 5 September 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i5.218

Abstract

Matrix metalloproteases (MMPs) are proteolytic enzymes that play a criti-cal role in decreasing the stability of atherosclerotic plaque causing Acute Myocardial Infarction (AMI). In this process, degradation of type IV col-lagen as an important component of endothelial membrane is thought to be important. Several microorganisms and their products including Li-popolysaccharide (LPS) have been suggested to be able to activate MMPs.The aim of this study is to investigate the role of Helicobacter pyloriLPS in type IV collagen degradation (in vitro). We used two stages in this study: MMP production stage and degradation of type IV collagen. MMP derived from the neutrophils of AMI and non-AMI patients that had been stimulated with LPS of Helicobacter pyloriwere used to digest type IV collagen. Analysis of type IV collagen degradation was performed with SDS-PAGE and Western blot.This study showed MMP isolated from neutrophil of AMI patients is an active MMP-9 at Molecular weight of 72 kDa. It can degrade type IV col-lagen at MW 78.6 kDa, 60 kDa, 50 kDa, 47.8 kDa and 43.8 kDa. Whereas MMP isolated from neutrophil of non-AMI have MW of 91.2 kDa and can only degrade collagen IVat merely 60 kDa.In conclusion, Helicobacter pyloriLPS is able to induce neutrophil of AMI patients to produce an active MMP-9 at MW 72 kDa and the enzyme may play strategic role in the degradation of type IV collagen.
Faktor-faktor Yang Mempengaruhi Heart Rate Recovery Pada Pasien Yang Menjalani Uji Latih Jantung Dengan Beban Henry A.P. Pakpahan; Andria Priyana; Adnil Basha; Basuni Radi
Jurnal Kardiologi Indonesia Vol. 28, No. 5 September 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i5.219

Abstract

Background. Heart rate recovery (HRR) has been recognized as an inde-pendent predictor of mortality.Objective.To identify factors that may affect HRR in patient with un-known coronary artery disease (CAD).Methods. This cross sectional study was performed in National Cardiac Center Harapan Kita. We reviewed the chart of patients with unknown CAD who underwent symptom-limited exercise stress testing for medical check up between January – December 2006. Factors that may influence HRR were recorded including current medications, peak exercise heart rate (HR), HR after 3-minute recovery, and the ischemic response to exer-cise. HRR was defined as the change in HR from peak exercise to 3-minute recovery. We further categorized patients to normal or abnormal HRR by using mean HRR as cut off value. All factors related to HRR and normal/abnormal categories were analyzed.Results. There were 188 patients aged 48 + 11 years. HRR significantly influenced by male sex (p=0.003), hypertension (p=0.028), â-blocker (p=0.03), calcium channel blocker (p=0.008), aspirin (p<0.001), and pro-voked ischemia (p=0.005). Using mean HRR (57x) as cut off, patients were classified as having normal (HRR <57x) or abnormal (HRR ? 57x). On fur-ther analysis, there was significant association between age (p=0.003), male sex (p=0.02), hypertension (p=0.02), use of â-blocker (p=0.026) and aspirin (p<0.005).Conclusion. In patients with unknown CAD who underwent symptom-limited exercise stress testing, HRR was influenced by age, male sex, hy-pertension, the presence of provoked ischemia, use of â-blocker, calcium channel blocker, and aspirin.
Korelasi Kadar Leptin Plasma Dengan Massa Ventrikel Kiri Pada Laki-Laki Normotensi Richard Indra Gunawan; Adrianus Kosasih; Oktavia Lilyasari; Andang H Joesoef; RWM Kaligis
Jurnal Kardiologi Indonesia Vol. 28, No. 5 September 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i5.220

Abstract

Background.Obesity is one of Cardiovascular risk factor. Obesity caused increase in heart mass due to cellular hypertrophic and metaplastic proses, independently of hemodynamic factors such as blood pressure. There were hyperleptinemia in obesity due to the selective Leptin resistency in central nervous system and peripheral organs. Study with cultured rat cardiomyocyte have shown hypertrophy and hyperplastic effect of Leptin to cardiomyocyte. Some clinical studies have shown correlation between circulating Leptin level with Left Ventricle Mass in hypertensive and insulin resistance men.Objective. This study aimed to elaborate the correlation between circu-lating Leptin level with Left Ventricle Mass in normotensive men.Methods. A cross sectional study was performed with normotensive men, which included 40 obese normotensive and 40 normoweight men. All patients underwent physical and laboratory assessment and examination of Left Ventricle with echocardiography. The circulating Leptin level were determined by ELISA method. The Leptin level were expressed as median (25th percentile; 75th percentile)Results. The circulating Leptin level were significantly different between the obese normotensive and the lean group. The Left Ventricle Mass in Obese increased, although have not fulfilled the criteria for Left Ventricle Hypertrophy. There were significant correlation between Left Ventricle Mass with BMI (r = 0,711; p<0,001) and waist circumference (r = 0,732; p<0,001). Respectively, there were significant correlation between Left Ventricle Mass Index with BMI (r = 0.541; p<0.001) and waist circumfer-ence (r = 0,558; p<0.001. There were significant correlation between circulating Leptin level with Left Ventricle Mass (r = 0,510; <0,001) and Left Ventricle Mass Index (r = 0,414; p<0,001) in obese men.Conclusion.Circulating Leptin level is correlated with Left Ventricle Mass in normotensive men.
Prediktor Left Main DiseasePada Pasien NSTEMI Akut Berdasarkan Elektrokardiogram Budiyanto Nagawidjaja; Hadi Purnomo; Budhi Setianto
Jurnal Kardiologi Indonesia Vol. 28, No. 5 September 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i5.221

Abstract

Background. The importance to identify Left Main (LM) disease in Coro-nary Artery Disease (CAD) patients is to define therapy strategy, progno-sis which can influence to length of hospitalization, survival rate, and the payment. Electrocardiogram (ECG) is a simple tool to diagnose patient with CAD.Objectives. To identify ECG indicators as related to LM disease predic-tors.Methods. 265 acute non ST elevation patients enrolled after having coro-nary angiography in National Cardiovascular Center Harapan Kita Jakarta. ST elevation, ST depression, T invertion of the 12 lead ECG and ST eleva-tion in aVR taller than V1 lead is further variable investigation. Chi-square and Mann-Withney test were done for statistical analysis. Logistic regres-sion was used in variable predictor test. Discrimination test was done by using Hosmer Lemeshow goodness of fit to determine validation model and area under curve test.Results. The largest populations were male (210 patients – 82.3%). The age range from 37 until 80 years old (mean 59,9 ± 9,1 years), 77 patients were more than 65 years old. There were 90 patients with LM disease. There was ST elevation in aVR in 88 patients, and 87 patients had higher ST elevation compared to ST segment in V1. The significant factors areelevation of ST segment in aVR, ST segment elevation in aVR > V1, ST depression in I, II, III, aVL, aVF, V2 - V6, ST elevation in lead aVR–V1, mul-tiple ST changes (elevation and depression) in many leads. Multivariate analysis showed the highest parameter to diagnose LM disease are ST el-evation in aVR and ST elevation aVR-V1 (mm) with area under curve 96% (sensitivity 92,22 / specificity 97,14 / PPV 94,31 / NPV 96,00 for ST eleva-tion in aVR and sensitivity 93,33 / specificity 98,29 / PPV 96,55 / NPV 96,62 for ST elevation aVR>V1).Conclusions. ECG indicators for LM disease are ST elevation in aVR and ST elevation in aVR>V1.
Tatalaksana Medis Protein-Losing Enteropathy Pasca Bedah Total Cavo Pulmonary Connection Syarief Hidayat; Anna Ulfah Rahajoe; Kurniawan Iskandarsyah
Jurnal Kardiologi Indonesia Vol. 28, No. 5 September 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i5.222

Abstract

Sebelum bedah Fontan diperkenalkan, penyakit jantung bawaan (PJB) kompleks yang tidak ideal untuk reparasi biventrikular dilakukan bedah paliatif pulmonar y ar ter y banding (PAB)atau systemic-to-pulmonary shunt. PAB adalah upaya untuk memperkecil lumen ar teri pulmonal sehingga alirannya berkurang, dengan memasang ikatan melingkari arteri pulmonal cabang utama.Sedangkan bedah systemic-to-pulmonar y shunt adalah memasang saluran penghubung antara cabang cranial aorta ke arteri pulmonal. Tetapi harapan hidup pasen-pasen ini hanya berlangsung dua hingga tiga dekade saja.
Ablasi Frekuensi Radio Pada Fibrilasi Atrium Paroksismal Yoga Yuniadi; Rahadian Adhantoro; Muhammad Munawar
Jurnal Kardiologi Indonesia Vol. 28, No. 5 September 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i5.223

Abstract

Fibrilasi atrium (Atrial Fibrilation, AF) merupakan aritmia yang paling sering ditemukan dalam praktek klinis, meliputi sepertiga dari perawatan gangguan irama jantung. Diperkirakan prevalensi AF 0,4% hingga 1% dari populasi umum, yang meningkat dengan bertambahnya umur. Selama 20 tahun terakhir, terjadi 66% peningkatan angka perawatan rumah sakit oleh karena AF yang berkaitan dengan faktor umur dan prevalensi penyakit jantung kronis. AF juga dapat terjadi pada pasien tanpa penyakit jantung struktural (lone AF). Berdasarkan studi pada populasi, kejadian lone AF berkisar antara 12 hingga 30% dari seluruh kasus AF. AF meningkatkan risiko stroke 4 – 5 kali pada seluruh kelompok umur. Secara keseluruhan AF bertanggung jawab terhadap 15% kasus stroke di Amerika Serikat.
Fibrilasi Atrium Pada Penyakit Hipertiroidisme Patogenesis dan Tatalaksana Isman Firdaus
Jurnal Kardiologi Indonesia Vol. 28, No. 5 September 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i5.224

Abstract

Fibrilasi atrium (atrial fibrilation, AF) merupakan aritmia jantung yang paling sering terjadi pada pasen usia diatas 65 tahun. Prevalensi semakin tinggi dengan bertambahnya usia, dan merupakan penyebab utama terjadinya stroke. AF sering timbul sebagai manifestasi hipertiroidisme, dan menjadi predisposisi terbentuknya trombus dan emboli. AF dan disritmia supraventrikular lainnya yang disebabkan oleh hipertiroidisme, menambah angka kematian penyakit vaskular. Pada sepuluh hingga limabelas persen pasen dengan hipertiroidisme akan berlanjut menjadi AF, dan insiden ini semakin tinggi bila disertai penyakit jantung.
Penyakit Jantung Koroner pada “Chronic Kidney Disease” Retna Dewayani
Jurnal Kardiologi Indonesia Vol. 28, No. 5 September 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i5.225

Abstract

Pada populasi normal, proses aterosklerosis terjadi selama puluhan tahun. Tetapi pada pasen-pasen usia muda dengan gagal ginjal kronik (Chronic Kidney Disease, CKD), telah ditemukan kelainan vaskular sebelum terjadi penyakit ginjal stadium lanjut (end stage renal disease, ESRD).Komplikasi kardiovaskular ini kemudian menjadi penyebab kematian utama pada pasen CKD. Maka timbullah pertanyaan: apakah pasien uremi mengalami percepatan aterosklerosis?Penelitian epidemiologi klinik melaporkan angka mortalitas penyakit kardiovaskular meningkat 20 kali lebih banyak pada pasen dialisis, dibanding populasi normal; karena keterlibatan faktor risiko tradisional (Framingham Risk Factors) dan faktor risiko terkait uremia.
Parasistol Isoritmis Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 28, No. 5 September 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i5.226

Abstract

Seorang wanita, 36 tahun mengeluh dada berdebar kuat. Perasaan itu timbul tidak menentu dan dapat berlangsung berjam-jam. Timbul tidak menentu. Tidak ada riwayat pingsan maupun nyeri dada. Pemeriksaan koroner angiografi pernah dilakukan satu tahun yang lalu di rumah sakit lain, dengan hasil normal. Pemeriksaan fisik dalam batas normal. Ekokardiografi tidak memperlihatkan kelainan struktur jantung.
Korelasi Kekakuan Arteri Dengan Kadar Endothelin-1 Plasma pada Laki-laki Obesitas Adrianus Kosasih; Oktavia Lilyasari; Richard Indra Gunawan; Nur Haryono; Ismoyo Sunu
Jurnal Kardiologi Indonesia Vol. 28, No. 4 Juli 2007
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v28i4.227

Abstract

Background.Obesity is associated with increase arterial stiffness and el-evated plasma endothelin-1 concentration. However, there is still conflict-ing data regarding the effect of obesity on arterial stiffness and plasma endothelin-1 concentration.Objective.The purpose of the current study was to assess this effect and analyze whether there was correlation between arterial stiffness indices and plasma endothelin-1 concentration.Methods.The design of the study was cross sectional study that compare and correlate arterial stiffness and plasma endothelin-1 concentration be-tween obesity group (n=40) and normoweight group (n=40). Obesity was defined as body mass index = 25 kg/m2. Arterial stiffness was as-sessed by measuring carotid-femoral pulse wave velocity (PWV) dan ß stiffness index (ß) non-invasively using ultrasound method. Endothelin-1 was measured by ELISA.Results.There was no significant difference regarding PWV between obesity and normoweight group (mean ± SD: 809.44 ± 137.77 versus 850.96 ± 211.60 cm/s, p=NS), but ßwas significantly higher in obese group (8.79 ± 3.15 versus 7.28 ± 1.96, p=0.012). PWV and ßcorre-lated significantly with age (PWV: r=0.446, p<0.001, ß: r=0.354, p=0.001), but only ßcorrelated with body mass index (r=0.282, p=0.011) and waist circumference (r=0.312, p=0.005). There was no significant difference between obesity and normoweight group regard-ing plasma endothelin-1 concentration (0.94 ± 0.26 versus 0.95 ± 0.18 pg/dl, p=NS). There are no significant correlations between PWV/ßand plasma endothelin-1 concentration.Conclusions.These findings suggest that the effect of obesity on arterial stiffness is not uniformly seen throughout all arterial region. Obesity im-pact on carotid artery is greater than its impact on aorta, and this impact on arterial stiffness is not mediated by endothelin-1.

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