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Jurnal Neuroanestesi Indonesia

jni
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Published by Indonesian Society of Neuroanesthesia & Critical Care

ISSN : 20889674 EISSN : 24602302 DOI : https://doi.org/10.24244/jni

Core Subject : Health, Science, Education,

Biochemistry, Genetics & Molecular Biology Education Medicine & Pharmacology Neuroscience Public Health

Editor of the magazine Journal of Neuroanestesi Indonesia receives neuroscientific articles in the form of research reports, case reports, literature review, either clinically or to the biomolecular level, as well as letters to the editor. Manuscript under consideration that may be uploaded is a full text of article which has not been published in other national magazines. The manuscript which has been published in proceedings of scientific meetings is acceptable with written permission from the organizers. Our motto as written in orphanet: www.orpha.net is that medicine in progress, perhaps new knowledge, every patient is unique, perhaps the diagnostic is wrong, so that by reading JNI we will be faced with appropriate knowledge of the above motto. This journal is published every 4 months with 8-10 articles (February, June, October) by Indonesian Society of Neuroanesthesia & Critical Care (INA-SNACC). INA-SNACC is associtation of Neuroanesthesia Consultant Anesthesiology and Critical Care (SpAnKNA) and trainees who are following the NACC education. After becoming a Specialist Anesthesiology (SpAn), a SpAn will take another (two) years for NACC education and training in addition to learning from teachers in Indonesia KNA trainee receive education of teachers/ experts in the field of NACC from Singapore.

Arjuna Subject : Kedokteran - Anesthesiology and Pain Medicine

Articles 8 Documents

Search results for , issue "Vol 3, No 2 (2014)" : 8 Documents clear

Penurunan Kadar Glutamat pada Cedera Otak Traumatik Pascapemberian Agonis Adrenoseptor Alpha-2 Dexmedetomidin sebagai Indikator Proteksi Otak Prihatno, MM Rudi; Harahap, M. Sofyan; Akbar, Ieva B; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 3, No 2 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Latar Belakang dan Tujuan: Dexmedetomidin untuk kasus-kasus neurotrauma masih kontroversi, antara yang setuju dan menolak. Dexmedetomidin sebagai agonis adrenoseptor ?2 memiliki beberapa keuntungan dalam kaitannya dengan kemampuannya sebagai neuroprotektan. Penelitian ini bertujuan untuk mengkaji efek neuroproteksi dari dexmedetomidin yang dilihat dari pengaruhnya terhadap penurunan kadar glutamat.Subjek dan Metode Penelitian single blind randomized controlled trial dilakukan pada 16 orang yang datang ke IGD RSUD Prof. Dr. Margono Soekarjo dengan cedera otak traumatik dengan GCS ?8 pada MeiDesember 2013. Subjek dibagi dalam 2 kelompok yaitu kelompok dexmedetomidin dan NaCl 0,9%. Pembedahan dilakukan dalam rentang waktu 9 jam pascatrauma. Pemeriksaan kadar glutamat dengan menggunakan metode ELISA. Analisis data menggunakan uji-t dan uji Mann-Whitney.Hasil: Kelompok yang mendapatkan dexmedetomidin menunjukkan bahwa pemberian dexmedetomidin 0,4 ?g/kgBB/jam secara kontinyu, menunjukkan penurunan kadar glutamat yang diukur mulai dari awal perlakuan hingga jam ke-24 sebanyak 27,9% (p=0,025), dari jam ke-24 hingga jam-72 sebanyak 9,6% (p=0,208), serta dari awal perlakuan hingga jam ke-72 sebanyak 57,1% (p=0,036). Kelompok yang tidak mendapatkan dexmedetomidin mengalami peningkatan kadar glutamat.Simpulan: Pemberian dexmedetomidin 0,4 ?g/kgBB/jam dapat menurunkan kadar glutamat pada pasien cedera otak traumatik dengan GCS ? 8.Decreased Level of Glutamate after Administration of Dexmedetomidine (Alpha-2 Adrenoreceptor Agonist) as Neuroprotective Indicator in Traumatic Brain InjuryBackground and Objective: The usage of Dexmedetomidine in neurotrauma cases is still controversial, between the pros and cons. Dexmedetomidine as ?2-adrenoceptor agonist has several benefits in concomitant with its properties as neuroprotector. This study aims to evaluate the neuroprotection effect of dexmedetomidine based on the decline in glutamate level.Subject and Method: This single blind randomized controlled trial was done in 16 TBI patients with GCS ? 8, recruited from May-December 2013. Subjects were equally divided into 2 groups: dexmedetomidine and 0.9% NaCl group. Surgery was performed within 9 hours post TBI. Glutamate level was examined using ELISA method. Data were analyzed using t-test and Mann-Whitney test.Result: This study showed that glutamate levels in patient who received continuous intravenous dexmedetomidine 0.4 mcg / kg / h were decreased, starting from baseline to 24 h (27.9%, p=0.025), 24 to 72 h (9.6%, p= 0.208) and baseline to 72 h (57.1%, p= 0.036). All patients in NaCl 0.9% group experienced an increase in glutamate level.Conclusion: Administration of dexmedetomidine 0.4 mcg/kg/h in TBI patient with GCS ? 8 could decrease glutamate level.

Penatalaksanaan Perioperatif Perdarahan Intraserebral Sandhi Christanto; Nazaruddin Umar; A Himendra Wrgahadibrata
Jurnal Neuroanestesi Indonesia Vol 3, No 2 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Perdarahan intraserebral spontan nontraumatik didefinisikan sebagai ekstravasasi spontan darah ke dalam parenkim otak yang dapat meluas ke ventrikel otak atau pada kasus yang jarang dapat sampai ke ruang subarachnoid. Perdarahan intraserebral merupakan penyakit yang sering dijumpai, di Amerika Serikat tiap tahunnya terdapat sekitar 37 ribu sampai 52 ribu orang mengalami perdarahan intraserebral.1,2 Tercatat sekitar 10–30% dari semua kasus stroke di rumah sakit merupakan akibat perdarahan intraserebral, angka mortalitas mencapai 30–50% pada 30 hari pertama perawatan dan hanya sekitar 20% pasien yang mendapatkan kembali kemampuan dan kemandirian fungsionalnya dalam jangka waktu 6 bulan.2,3,4Faktor resiko paling penting dan paling sering untuk PIS adalah hipertensi, yang rata-rata mencapai 60–70% dari semua kasus PIS.1,3 Seorang wanita, 41 tahun berat badan 60 kg datang dengan kesadaran menurun sejak 1 hari sebelum masuk rumah sakit disertai bagian tubuh sebelah kanan yang terasa lemas. Keluhan tersebut dirasa semakin lama semakin berat sampai keesokan harinya kesadaran makin menurun dan bagian tubuh kanan tidak bisa digerakkan. Pada pemeriksaan didapatkan jalan napas bebas, laju napas 18–20 x/menit, tekanan darah 200/100 mmHg, laju nadi 70x/menit, skor GCS E2M5V–, Hasil Ctscan menunjukkan adanya perdarahan intraserebral di basal ganglia kiri volume 52 ml dengan midLine shift ke kanan sejauh 1,1 cm, skor PIS 2. Keputusan kraniotomi evakuasi hematoma dilakukan untuk keselamatan pasien. Penatalaksanaan berkesinambungan dengan memperhatikan prinsip neuroresusitasi, neuroanestesia, neurointensive care serta neuroproteksi sangat penting dilakukan dalam menangani pasien dengan perdarahan intraserebral. Perioperative Management of Intracerebral HemorrhageSpontaneous non traumatic intracerebral hemorrhage is devined as an extravasation of blood into the brain parenchym that may extend into the ventricles and, in a rare case, to the subarachnoid space. Each year, approximately 37,000 to 52,000 people in the United States are suffered from an intracerebral hemorrhage. Intracerebral hemorrhage accounts for 10 to 30 percent of all cases of stroke with the 30-days mortality rate, ranges from 30%–50% and only 20% of survivors expected to have full functional recovery within 6 months. Hypertension is by far the most important and prevalent risk factor, directly accounted for about 60–70% of cases. A 41-year old woman weighted 60 kgs was admitted to the hospital with decreased level of conciousness and weak right side of her body, which became worsen in the next morning. On examination, airway was clear, respiratory rate was 18–20 x/min, blood pressure was 200/100 mmHg, heart rate was 70 bpm, GCS score was E2M5V–, CT-scan examination showed a 52 cc of intracerebral hemorrhage in left basal ganglia, mid line shifted 1,1 cm to the right and ICH score was 2. The decision of emergency hematoma evacuation was immediately made for life saving. Continuous and comprehensive management with neuro-resuscitation, neuroanestesia, neuro intensive care and brain protectio.

Manajemen Anestesi pada Pasien dengan Cedera Medula Spinalis Segmen Cervicalis Kurniawan Komala, Tomas Ari; Suarjaya, I Putu Pramana; Sinardja, I Ketut
Jurnal Neuroanestesi Indonesia Vol 3, No 2 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Manajemen medula spinalis, terutama bagian cervical selama operasi dan resusitasi pasien dengan cedera spinal, memiliki banyak pertimbangan penting untuk ahli anestesi, antara lain dengan memperhitungkan hal-hal yang berpotensi menyebabkan cedera berat irreversibel selama dilakukan intubasi trakeal. Pasien laki-laki usia 57 tahun, datang ke Rumah Sakit Sanglah Denpasar dalam kondisi sadar mengeluh nyeri pada leher dan tidak bisa menggerakkan ke empat anggota geraknya segera setelah kecelakaan. Pengelolaan anestesi untuk membantu tindakan operasi ini dilakukan dengan anestesi umum inhalasi dengan pemasangan pipa nasotrakheal non kinking, nafas kendali. Untuk premedikasi diberikan midazolam intravena, induksi dengan propofol dan fentanyl intravena, dan fasilitasi intubasi dengan menggunakan vekuronium intravena. Intubasi dikerjakan dengan bantuan glidescope untuk meminimalisasi ekstensi kepala. Pemeliharaan anestesi dengan menggunakan N2O, O2, sevofluran dan vekuronium intermitten. Monitoring tanda vital tekanan darah, laju nadi, EKG, SaO2, dan ET CO2. Operasi dikerjakan dengan posisi telungkup, pendekatan dari posterior. Selama operasi hemodinamik pasien relatif stabil. Hari I pascaoperasi dimulai program diet enteral, hari II pascaoperasi penderita dipindahkan ke ruangan biasa. Penilaian nyeri dengan Numeric Rating Scale (NRS) dengan hasil 12. Fungsi motorik pasien meningkat 1 point dibandingkan pre op, hari IX pascaoperasi penderita diprogram rawat jalan oleh sejawat Bedah Saraf. Cedera pada medulla spinalis segmen cervical memerlukan penanganan yang cermat. Penanganan jalan nafas definitif dengan melakukan intubasi trakheal harus sangat berhatihati, dan harus dijaga agar tidak terjadi cedera lebih jauh akibat tindakan laryngoscopy. Anesthetic Management for Patient with Cervicalis Spinal Cord InjuryManagement for spinal cord injury, especially the cervical part during surgery and also resuscitation of patients with spinal injuries, has many important considerations for anesthesiologists,which is also have potential to cause severe irreversible injury during tracheal intubation. Patient male, 57 years old, came to Sanglah Hospital with chief complain neck pain and could not move all extremities immediately after an accident. Anesthesia performed by general anesthesia inhalation with insertion nasotracheal tube. For premedication was given IV midazolam. Induction with IV propofol and fentanyl, and vecuronium used as muscle relaxant. Intubation performed with glidescope guidance to minimize the extension of the head. Maintenance of anesthesia with N2O, O2, sevoflurane and intermittent IV vecuronium. Monitoring during anesthesia and surgery such as blood pressure, pulse rate, ECG, SaO2, and ET CO2. The surgery was done with prone position and posterior approach. During surgery the patients hemodynamic relative stable. Day I post operation, patient start to have enteral diet, and the next day patient was transferred to regular ward. Pain assesment was done with Numeric Rating Scale (NRS) with score 12. Motor function of the patients increased 1 point compared to preoperation. Day IX post operation, patient was discharged from the hospital. Cervical spinal cord injury requires careful handling. Definitive airway by endotracheal intubation should be done with extreme careful, and shall not cause further injury due to laryngoscopy.

Efek Anestesia Aliran Rendah Sevofluran terhadap Respon Inflamasi pada Susunan Saraf Pusat Harimin, Kusuma; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 3, No 2 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Anestesi aliran rendah adalah teknik anestesi yang menggunakan aliran gas 1L/menit. Oleh karena adanya rebreathing, maka pada anestesi aliran rendah yang menggunakan sevofluran akan terjadi produk degradasi dengan CO2 absorber sehingga terbentuk senyawa A dan senyawa B. Senyawa A bersifat neprotoksik pada ginjal tikus, karena enzim ? liase 30 kali lebih aktif pada tikus dari pada manusia, sedangkan pada manusia tidak terbukti senyawa A berefek neprotoksik. Anestesia sevofluran dapat menimbulkan respons inflamasi yang diawali dengan pelepasan interleukin (IL)1 dan TNF?, kemudian menstimulasi IL6 yang sangat berperan pada respons fase akut. Akan terjadi interaksi antara sistem imun dengan sistem neuroendokrin, yang mana IL1 dan IL6 dapat menstimulasi adrenocorticotrophic hormone (ACTH) sehingga terjadi peningkatan pelepasan kortisol. Metabolit sevofluran dan senyawa A tidak dapat menembus sawar darah otak sehingga pengaruh negatif dari metabolit dan produk degradasi sevofluran terhadap otak tidak ada. Bahkan, melalui penelitian lebih lanjut, sevofluran diketahui mempunyai efek neuroproteksi.The Effect of Sevoflurane Low Flow Anesthesia to Inflammatory Response on Central Nervous SystemLow flow anesthetic is an anesthesia technique using gas flow less than 1 L/ min. Due to the rebreathing system, a low flow anaesthesia using sevoflurane will produce degradation products through reaction with the CO2 absorber which will form compound A and compound B. Compound A is nephrotoxic to rat kidney because the ? -lyase enzyme in rat is 30-fold more active than in human, and this compound has been proven to be not nephrotoxic in human. Sevoflurane can cause inflammatory response which started with the release of interleukin (IL)-1 and TNF-? followed by stimulation of IL-6, which plays important part in the acute phase. Interaction between the neuroendocrine and immune systems will occur where IL-1 and IL-6 cytokines will stimulate the production of adrenocorticotrophic hormone (ACTH), which in turn will increase the production of cortisol. Sevoflurane metabolites and compound A can not penetrate blood brain barrier, therefore, the negative effects of sevoflurane metabolites and degradation products to the brain does not happen. Further advanced studies even showed that sevoflurane has a neuroprotective effect.

Penatalaksanaan Anestesi pada Pasien Stroke Hemoragik Rebecca Sidhapramudita Mangastuti; Bambang J. Oetoro; Sudadi Sudadi
Jurnal Neuroanestesi Indonesia Vol 3, No 2 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Stroke terjadi akibat terganggunya aliran darah ke otak secara tiba-tiba. Penyebab terbanyak stroke adalah berkurangnya pasokan darah ke otak (stroke iskemik). Penyebab stroke lainnya adalah perdarahan (stroke hemoragik). Perdarahan intraserebral (ICH) terjadi akibatnya pecahnya pembuluh darah otak. Lokasi terjadinya stroke dapat di basal ganglia, cerebelum, batang otak atau kortek serebri. Penyebab perdarahan intraserebral adalah hipertensi, trauma, infeksi, tumor, defisiensi faktor pembekuan darah, terapi antikoagulan, malformasi arterivena (AVM). Laki-laki, 67 tahun dengan GCS 5 (E1M3V1) dengan terapi rutin antikoagulan menderita serangan stroke hemoragik. CT scan memperlihatkan adanya perdarahan intraparenkim lobus parieto-temporo-oksipital kanan 53,3 ml, perifokal edema, herniasi subflacin kiri 13,9 mm dan herniasi central downward. Pasien dilakukan kraniotomi evakuasi hematom dan dekompresi dengan anestesi umum. Pasien dalam kondisi umum stabil saat operasi berlangsung. Postoperasi, pasien dirawat di Intensive Care Unit. Pasien dinyatakan mati batang otak pada hari kedua pasca operasi dan meninggal pada hari keempat. Anesthetic Management in Patients with Hemorrhagic StrokeStroke is triggered by a sudden interruption of blood supply to the brain. The most frequent etiology of stroke is decrease blood supply to the brain (ischemic stroke). Another stroke is caused by rupture of blood brain vessel (hemorrhagic stroke). Intracerebral hemorrhage (ICH) occurs when a blood vessel within the brain bursts. Stroke locates mainly in basal ganglia, cerebellum, brain stem or cerebral cortex. The common cause of intracerebral hemorrhage are hypertension, trauma, infection, tumors, blood coagulation factor deficiencies, anticoagulant therapy, or arteriovenous malformations. We reported a 67-years old, man with, GCS 5 (E1M3V1) on routine anticoagulant therapy who experienced hemorrhagic stroke. Brain CT-scan examination showed bleeding in intra parenchimal right parieto-temporo-occipital lobe about 53,3 mL, perifokal edema, subflacin sinistra 13,9 mm and central downward herniation. Patient was performed craniotomy to evacuate the hematome and decompresion with general anesthesia. During surgery patient had a relatively stable condition. After surgery, the patient was treated in intensive care unit but declared brain stem dead on day-2 post surgery an died day-4.

Gangguan Koagulasi dan Hubungannya dengan Luaran Pascacedera Otak Traumatik Kenanga Marwan; Nazaruddin Umar; Siti Chasnak Saleh; A Himendra Wargahadibrata
Jurnal Neuroanestesi Indonesia Vol 3, No 2 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Gangguan koagulasi umum terjadi pada cedera otak traumatik (COT) dan seringkali memperburuk luaran. Gangguan ini diawali dengan lepasnya faktor jaringan dan trombin secara berlebihan pada bagian otak yang rusak, yang selanjutnya mengaktifkan jalur koagulasi. Gangguan koagulasi pada COT bersifat kompleks dan berupa kombinasi koagulopati dan hiperkoagulabilitas. Hiperkoagulabilitas dikaitkan dengan terjadinya disseminated intravascular coagulation (DIC) secara sistemik dan secara lokal dengan terbentuknya mikrotrombi di area penumbra daerah yang mengalami kontusi. Nilai abnormal fungsi koagulasi dapat terjadi karena kerusakan endotel saat cedera langsung dan tidak langsung oleh proses inflamasi, toksin dan iskemia. Koagulopati terjadi akibat turunnya jumlah platelet dan faktor pembekuan akibat kehilangan darah atau karena kosumsi oleh DIC, dan dipercepat dengan dilusi, asidosis dan hipotermi. DIC merupakan prediktor penting luaran COT yang buruk. Coagulation Disorders Post Traumatic Brain Injury Related OutcomeTraumatic disorder of hemocoagulation is common in traumatic brain injury (TBI) and frequently related to poor outcome. It begins with the massive release of thrombin or tissue factor from damage brain cells, following the activation of coagulation pathway. Coagulation disorder in TBI are complex and characterised by combination of coagulopathy and hypercoagulability. A hypercoagulable state may be generalised in the case of disseminated intravascular coagulation (DIC) or local with the development of microthrombi in the penumbra of a contusion. The presence of abnormal coagulation test in traumatic brain injury may be caused by cerebral vascular endothelial damage after direct injury, as well as indirect damage through inflammation, toxins, and ischaemia. Coagulopathy may result from depletion of platelets and clotting factors following blood loss or comsumption due to DIC, and may further be enhanced by dilution, acidosis and hypothermia. DIC is an important predictor of poor outcome in TBI.

Penatalaksanaan Anestesi pada Pasien dengan Tumor Supratentorial Berukuran Besar Suspek Konveksitas Meningioma Wullur, Caroline; Bisri, Dewi Yulianti
Jurnal Neuroanestesi Indonesia Vol 3, No 2 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Penatalaksanaan anestesi untuk kasus meningioma memiliki beberapa hal khusus yang penting untuk dilaksanakan. Jaringan otak tertutup oleh tulang kranium. Karena hubungan kontinu dari aliran darah dan volume jaringan otak, maka resiko perdarahan dan edema sangat tinggi. Tanpa pendekatan anestesi yang tepat, maka dapat meningkatkan resiko edema dan perdarahan otak karena manipulasi operasi. Pada kasus ini dilaporkan pasien berusia 35 tahun dengan keluhan nyeri kepala di daerah frontal disertai dengan penurunan penglihatan sejak 1 tahun sebelum masuk rumah sakit. Pasien tidak pernah mengalami kejang ataupun penurunan kesadaran. Pasien didiagnosa dengan tumor supratentorial ec suspek conveksitas meningioma yang direncanakan dilakukan pembedahan kraniotomi untuk pengangkatan tumor. Status fisik ASA 2 dengan defisit neurologis. Pasien dilakukan dengan anestesi umum dengan intubasi. Induksi dengan fentanil, propofol dan vecuronium. Operasi berlangsung selama 7,5 jam. Pascabedah, pasien dirawat di Unit Perawatan Intensif selama 2 hari sebelum pindah ke ruangan. Perlakuan anestesi dan pengaturan faktor fisiologi mempunyai dampak yang besar terhadap jaringan otak. Dokter anestesi harus mempunyai pengetahuan mengenai efek obat dan manipulasi lainnya untuk mencapai hasil operasi yang baik.Anaesthetic Management of a Patient with Large Supratentorial Brain Tumor Suspected Convexity MeningiomaAnesthesia for meningioma cases has several specific important considerations. The brain is enclosed in a rigid skull. Brain tissue is highly vascularized therefore the risk of bleeding and edema are very high. Without the correct anaesthetic approach, the risk of bleeding and edema due to surgical manipulation may be increased. This phenomenon may have negative impact since the visual of surgical field will be limited. In this case, we reported a 35-year old female patient with severe headache at the frontal region accompanied with visual impairment since 1 year prior to hospital admittance. This patient was never experienced any seizures or inconsiousness. Patient was diagnosed with supratentorial tumor caused by suspect of convexity meningioma and was planned tumor removal craniotomy. ASA II physical status with neurological deficit. The patient was on general anaesthesia with intubation. Induction was performed using fentanyl, propofol and vecuronium while continuous propofol and vecuronium were used for maintenance. The surgery lasted for 7.5 hours. After surgery, the patient was treated in the Intensive Care Unit for 2 days prior to inpatient ward transfer. Anaesthetic management and physiological factors control have a positive impact on the brain tissue. Anaesthesiologist must have the comprehensive knowledge on drug effects and other manipulations to achieve positive result of a surgery.

Penatalaksanaan Anestesi pada Kehamilan dengan Tumor Medula Spinalis Supradnyawati, Ni Made; Suarjaya, I Putu Pramana; Sinardja, I Ketut
Jurnal Neuroanestesi Indonesia Vol 3, No 2 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Anestesi pada pembedahan nonobstetri dalam kehamilan merupakan tantangan khusus bagi ahli anestesi. Sekitar 0,75%2% pembedahan nonobstetri dilakukan selama masa kehamilan. Setiap tahunnya di AS diperkirakan sekitar 75.000 wanita hamil menjalani anestesi dan pembedahan. Penatalaksanaan anestesi optimal memerlukan pemahaman mengenai perubahan fisiologi maternal, pertimbangan terhadap fetus akibat pembedahan dan anestesi, dan upaya mempertahankan perfusi uteroplasenta dan oksigenasi maternal-fetus. Tujuan yang ingin dicapai adalah anestesi yang aman kepada ibu dan memelihara kesejahteraan janin. Kami melaporkan kasus wanita berusia 29 tahun dengan G4P1A21 25?26 minggu janin tunggal hidup yang mengalami kelemahan motorik akut pada kedua tungkai bawah, gangguan sensibilitas semua kualitas setinggi Th6, serta inkontinensia urine dan alvi. Hasil pemeriksaan penunjang magnetic resonance imaging thorakolumbal menunjukkan suatu massa di daerah epidural setinggi C7Th1 sisi kanan dan hambatan aliran likuor serebrospinal. Pasien dilakukan anestesi umum dengan intubasi endotrakeal. Induksi menggunakan propofol dan fentanyl, diikuti dengan penekanan krikoid. Fasilitas intubasi menggunakan vecuronium. Pemeliharaan menggunakan isofluran, oksigen, compressed air, bolus fentanyl dan vecuronium intravena intermitten. Posisi operasi adalah posisi prone. Intraoperatif ditemukan tumor ekstradura setinggi level C7Th1, dilakukan laminektomi total dan stabilisasi dengan pemasangan pedicle screw. Pascabedah pasien menunjukkan perbaikan status neurologis dan kehamilan dapat dipertahankan sampai aterm.Anesthesia Management for Spinal Cord Tumor in PregnancyAnesthesia management for non-obstetric surgery in pregnancy was considered a specific challenge for anesthesiologist. About 0,752% of non-obstetric surgery is performed during pregnancy. Annually in the US, about 75.000 pregnant women are exposed to anesthesia and surgery. Optimal anesthetic management requires comprehensive understanding on maternal physiologic changes, fetal consideration due to effect of surgery and anesthesia, and maintaining uteroplacental perfusion and maternal-fetal oxygenation. The endpoint is to provide safe anesthesia for both the mother and fetal well being. We reported a case of a 29-year old pregnant woman G4P1021 single fetus with 2526 weeks of gestation, acute weakness of lower limbs, and sensibility impairment on all qualities at Th 6 level, as well as urine and alvi incontinence. Thoraco lumbal MRI examination showed epidural mass at C 7Th 1 level of the right side vertebrae, and cerebrospinal fluid flow obstruction. The patient underwent general anesthesia with endotracheal intubation. Induction with propofol and fentanyl, followed by cricoid pressure. Intubation was facilitated with vecuronium. Maintenance with isoflurane, oxygen, compressed air, intermittent IV bolus of fentanyl and vecuronium. Surgery was performed on prone position. Extradural tumor at C7Th1 vertebrae level was found intraoperatively and total laminectomy and stabilization with pedicle-screw were performed. Patient showed improvement in neurological status after the surgery, and the pregnancy was survived until aterm period.

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Editorial Address
Jl. Prof. Eijkman No. 38 Bandung 40161, Indonesia Lt 4 Ruang JNI

Location
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INDONESIA