cover
Article Per Year (5 Year)
All
Home Page
OAI Link
Editorial Team
Contact
Reviewer
Google Scholar
Contact Name
-
Contact Email
-
Phone
-
Journal Mail Official
-
Editorial Address
-
Location
Kota adm. jakarta pusat,
Dki jakarta
INDONESIA
Paediatrica Indonesiana
Published by Ikatan Dokter Anak Indonesia
ISSN : 00309311     EISSN : 2338476X     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Paediatrica Indonesiana is a medical journal devoted to the health, in a broad sense, affecting fetuses, infants, children, and adolescents, belonged to the Indonesian Pediatric Society. Its publications are directed to pediatricians and other medical practitioners or researchers at all levels of health practice throughout the world.
Arjuna Subject : -
Articles 2,118 Documents
 16   17   18   19   20 
Effect of glucocorticoid therapy on adrenal function in children with acute lymphoblastic leukemia Gita Widyapuri; Djajadiman Gatot; Aman Bakti Pulungan; Badriul Hegar
Paediatrica Indonesiana Vol 54 No 1 (2014): January 2014
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (127.077 KB) | DOI: 10.14238/pi54.1.2014.15-21

Abstract

BackgroundGlucocorticoids play an important role in thetreatment ofacute lymphoblastic leukemia (ALL), but can causeside effects such as suppression of the hypothalamic-pituitaryadrenal (HPA) axis. Suppression of the HPA axis causes adrenal insufficiency, disturbs the cortisol response to stress, and may be a cause of morbidity and mortality in children with ALL.ObjectiveTo evaluate adrenal function in children with ALL afterinduction chemotherapy with high dose glucocorticoids.MethodsThe adrenal function of 20 children with ALL wasevaluated using a standard dose (250 μ g) adrenocorticotropinhormone (ACTH) test performed before and after a 6 week oftreatment with glucocorticoids induction phase chemotherapy,which was followed by a week period tapering off. Adrenalinsuffien cy was defined as blood cortisol level of < 18 μg/dLResultsAdrenal insufficiency was found in 14/20 subjects afterthe induction phase followed by a week period of tapering off.Median cortisol levels pre- and post-stimulation before inductionphase were 14.72 (range 2.0 1- 46. 1) μg/dL and 29.29 (range 21.65 - 55 .15) μg/dL, respectively. Median cortisol levels pre- and poststimulation after induction phase were 5.87 (range 0.2 - 20.53)μg/dL and 10.49 (range 0.33 - 28.69) μg/dL, respectively. Clinicalsigns and symptoms did not differ between those with and withoutadrenal insufficiency.ConclusionOf 20 children with ALL, 14 develop adrenalinsufficiency after a 6-week induction therapy with glucocorticoidsand followed by a week period of tapering off. No specific clinicalsigns and symptoms are identified to be related to the adrenalinsufficiency.
NPHS2 gene mutation, atopy, and gender as risk factors for steroid-resistant nephrotic syndrome in Indonesians Dedi Rachmadi; Danny Hilmanto; Ponpon Ijradinata; Abdurahman Sukadi
Paediatrica Indonesiana Vol 51 No 5 (2011): September 2011
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (333.237 KB) | DOI: 10.14238/pi51.5.2011.272-6

Abstract

Background Steroid-resistant nephrotic syndrome (SRNS) often develops into end stage renal disease. Previous studies have reported that NPHS2 gene mutation, gender, and atopic history are risk factors associated with SRNS. Interethnic, sociocultural, and environmental differences have also been suggested to affect these mutations.Objective To analyze possible risk factors for SRNS, including NPHS2 gene mutations (412C→T and 419delG), gender and atopic history, in Indonesian subjects with SRNS.Methods A case-control study with 153 subjects, consisting of 88 SRNS patients and 65 control subjects, was undertaken in 10 Indonesian teaching centre hospitals from September 2006 to December 2007. Analysis of the NPHS2 gene mutation in 412 C→T was performed by amplification refractory mutation system-polymerase chain reaction (ARMS-PCR), while that for the NPHS2 gene mutation in 419delG was performed by restriction fragment length polymorphism (RFLP). Data was analyzed by multiple logistic regression.Results In our Indonesian subjects, the significant risk factors for SRNS were male gender (OR=2.21; CI 95%:1.07-4.56, P=0.036), NPHS2 412C→T gene mutation (OR=18.07; CI 95%:6.76-48.31, P<0.001), and NPHS2 419delG gene mutation (OR=4.55; CI 95%:1.66-12.47, P=0.003). However, atopic history was not a significant risk factor for SRNS (OR=1.807; CI 95%:0.642-5.086, P=0.262).Conclusion NPHS2 412C→T and 419delG gene mutations, as well as male gender are risk factors for SRNS in Indonesian subjects. Atopic history was not significantly associated with SRNS in our subjects. [Paediatr Indones. 2011;51:272-6].
Prevalence of glucose-6-phosphate dehydrogenase (G6PD) deficiency in neonates in Bunda Women's and Children's Hospital, Jakarta, Indonesia Risma Kerina Kaban; Veronica Wijaya
Paediatrica Indonesiana Vol 51 No 1 (2011): January 2011
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.14238/pi51.1.2011.29-33

Abstract

Background Glucose-6-phosphate dehydrogenase (G6PD) deficiency is the most connnon enzyme deficiency in the world. Itis a risk factor for hyperbilirubinemia in neonates, which can cause serious complications such as bilirubin􀁗induced encephalopathy or kernicterus. WHO recommends universal neonatal screening for G6PD deficiency when the frequency exceeds 3􀁗5% of male newborns.Objective To assess the prevalence of G6PD deficiency among neonates in Bunda Women and C hildren Hospital (Bunda WCH), Jakarta, in order to detennine if there is a need for routine G6PD neonatal screening.Methods This is a cross-sectional and retrospective study; infants' data were obtained from medical records. From January 2009 to May 2010, all neonates in Bunda WCH were screened for G6PD deficiency on the yd day of life. Blood samples were collected using filter papers. We considered a result to be nonnal if it exceeded 3.6 U/g Hb.Results A total 1802 neonates were screened. We found 94 neonates (5.2%) with G6PD deficiency. Out of 943 males, 59 (6.26%) were G6PD deficient, and out of 859 females, 35 (4.07%) were G6PD deficient. We observed that prevalence of G6PD deficiency according to sex distribution was significantly higher in males than females (6.26% vs. 4.07%, P=0.037). There was no significant difference in the risk for severe hyperbilirubinemia between the G6PD deficient infants and the nonnal infants (P=0.804).Conclusions The frequencies of G6PD deficiency were 6.26% of male neonates and 4.07% of female neonates. We recommend universal neonatal screening for G6PD deficiencies in Jakarta since our findings exceed the WHO recommendation for routine testing.
Lactose intolerance among healthy adults Achmad Surjono; Teluk Sebodo; Jati Sunarto; Moenginah P. A.
Paediatrica Indonesiana Vol 13 No 2 (1973): February 1973
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.14238/pi13.2.1973.49-54

Abstract

Lactose occurring naturally only in milk, must be hydrolyzed into glucose and galactose by small-intestinal enzyme lactase in order to beutilized. The production and presence of lactase is in the brush-borderof the intestinal epithelial cells and its activity takes place within those cells (Johnson, 1967; Levine and Haft, 1970). The lactase activity is ditributed uniformly throughout the small intestines, except in the duodenum and distal ileum where low levels are found (Auricchio, et al.,1965). 
Serum 25-hydroxyvitamin-D level and atopic dermatitis severity in children Laily Munawwarah; Rita Evalina; Sri Sofyani
Paediatrica Indonesiana Vol 57 No 5 (2017): September 2017
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (1209.021 KB) | DOI: 10.14238/pi57.5.2017.234-8

Abstract

Background Vitamin D plays an important role in the immune system. It inhibits B-lymphocyte proliferation and modulates the humoral response to suppress IgE production. Studies on the relationship between serum 25-hydroxyvitamin-D level and the severity of atopic dermatitis in several countries have had varying results.Objective To assess for a possible correlation between serum 25-hydroxyvitamin-D level and atopic dermatitis severity in children.Methods A cross-sectional study was conducted in 26 children with atopic dermatitis from September to December 2015. We evaluated the severity of disease using the Scoring of Atopic Dermatitis (SCORAD) index and measured serum 25-hydroxyvitamin-D levels. Spearman’s test was used to analyze for a correlation between serum 25-hydroxyvitamin-D level and the atopic dermatitis  score in children with atopic dermatitis.Results Mean SCORAD index was 32.0 (SD 14.99) , with a range of 10.9 to 71.4. Mean serum 25-hydroxyvitamin-D level was 41.1 (SD 24.81) ng/mL, with a range of 10-137 ng/mL. There was a moderate correlation between serum 25-hydroxyvitamin-D level and the SCORAD index (r=-0.591), with higher SCORAD index associated with lower serum 25-hydroxyvitamin-D level (P=0.01).Conclusion There is a moderate correlation between serum 25-hydroxyvitamin-D level and the SCORAD index in children with atopic dermatitis.
Association between C-reactive protein levels and physical fitness in 7 to 9 year old children at poor elementary schools in Surakarta, Indonesia Lilisianawati Lilisianawati; Endang Dewi Lestari; Diana Mayasari Hadianto; Maria Galuh Kamenyangan Sari; Leilani Lestarina; Harsono Salimo
Paediatrica Indonesiana Vol 49 No 2 (2009): March 2009
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (126.27 KB) | DOI: 10.14238/pi49.2.2009.75-81

Abstract

Background  Data  regarding inflammatory factors  in  children arenot  so  well developed  as  in adults. Higher levels  of  physical fitnessmay be helpful in modifying the low-grade inflammatory state  thatis  indexed  by  C-reactive protein (CRP) levels.Objective  To  evaluate the association  of  physical fitness with  CRPlevels in children  and  to identify the possible related factors.Methods  This  cross-sectional study was  conducted  at  poorelementary schools in Surakarta, Indonesia using a group  of  217underweight children aged from 7 to 9 years old. Physical fitnesswas assessed using a modified Harvard Step Test.  CRP  levels weremeasured using a high-sensitivity  assay.  The  association betweenphysical fitness and  CRP  level was assessed using linear regressionanalysis. Multivariate analyses were used to adjust covariates,and statistical analyses were performed using SPSS for Windowssoftware version 15.0.Results  Subjects were 48% female  and  52% male, and elevenchildren (5%) had a  CRP  level  >  5 mg/L. Mean fitness level  andCRP  level did not differ  by  age and gender. Fitness level was notinversely correlated with  CRP  (r=0.10,  P=0.14).  Physical fitnesswas significantly correlated with BMI  (r=0.14;  P=0.04),  physicalactivity  [0R=3.3  (95% CI  1.7  to 6.4)], and fat intake  [0R=0.5(95%  CI  0.2 to 0.9) ].Conclusion  These findings indicate  that  physical fitness  is  notinversely correlated to  CRP  levels in children. However, ourstudy reveased  an  association between high fat intake and lowphysical fitness as well  as  a significant association  betweenhigh physical activiry and physical fitness.
Sildenafil for pulmonary hypertension due to left-toright shunt after corrective procedure Dyahris Kuntartiwi; Piprim Basarah Yanuarso; Sudigdo Sastroasmoro
Paediatrica Indonesiana Vol 55 No 5 (2015): September 2015
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (105.217 KB) | DOI: 10.14238/pi55.5.2015.257-62

Abstract

Background Pulmonary arterial hypertension (PAH) is a common complication seen in those with a left-to-right shunt congenital heart defect (CHD). Corrective procedures by surgery or catheterization are the therapies of choice for reversible PAH. Since morbidity and mortality due to PAH after correction is high, sildenafil has been used as a selective vasodilator of the pulmonary artery, in order to decrease pulmonary arterial pressure. Objectives To evaluate the effect of sildenafil on pulmonary arterial pressure and clinical outcomes after left-to-right shunt CHD corrective procedures. Methods Left-to-right shunt patients aged < 18 years scheduled for corrective treatment were randomized in a double-blind fashion, to receive either oral sildenafil or placebo, given on days 3 to 30 after the corrective procedure. Clinical and pulmonary arterial pressures were evaluated by echocardiography before, 3 days after, and 30 days after the corrective procedure. Results From July 2013 to June 2014, 36 patients were included in the study: 17 in the placebo and 19 in the sildenafil groups. There were no differences in pulmonary arterial pressure or in clinical outcomes after corrective procedure between the two groups. There were no adverse events during the treatment. Conclusion Sildenafil has little effect on decreasing pulmonary arterial pressure, as most of our subjects seem to have hyperkinetic PAH. As such, pulmonary arterial pressure returns to normal soon after corrective procedures.
Association between obesity and lipid profile in children 10--12 years of age Ruqoyatul Himah; Endy P. Prawirohartono; Madarina Julia
Paediatrica Indonesiana Vol 48 No 5 (2008): September 2008
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (100.748 KB) | DOI: 10.14238/pi48.5.2008.257-60

Abstract

Background Worldwide prevalence of obesity in children hasbeen increasing. Together with dyslipidemia and hypertension,obesity is associated with higher risk of cardiovascular morbiditylater in life.Objective The aim of this study was to assess associationbetween obesity and occurrence of dyslipidemia in 10-12 yearsold children.Methods We performed a cross sectional study on 53 obese and53 non-obese children matched for age and gender in Yogyakarta.Obesity was defined as body mass index (BMI) at or above the95th percentile of the CDC 2000 reference. The levels of totalcholesterol, LDL-cholesterol, HDL-cholesterol and triglyceridewere measured. Dyslipidemia was defined as increased level oftotal cholesterol, LDL-cholesterol, triglyceride and decreasedlevel of HDL-cholesterol.Results Obese children had higher risk of increased level oftriglyseride, i.e. RR 2.6 (95% CI 1.6 to 4.4), P<O.OOl anddecreased level ofHDL-cholesterol, i.e. RR 17.8 (95%CI 14.0 to20.1), P=0.003. Overall, relative risk for dyslipidemia in obesechildren was 5.2 (95%CI 4.2 to 5.9), P=0.002, higher than innon-obese children.Conclusion Compared to non-obese children, obese children havehigher risk of dyslipidemia, particularly hypertriglyceridemia andhypo-HDL.
The use of pediatric logistic organ dysfunction (PELOD) scoring system to determine the prognosis of patients in pediatric intensive care units Dewi Metta; Dedi Soebardja; Dadang Hudaya S
Paediatrica Indonesiana Vol 46 No 1 (2006): January 2006
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.14238/pi46.1.2006.1-6

Abstract

Background Prediction of outcome of patients admitted to pediat-ric intensive care unit (PICU) requires an objective tool for measure-ment. It is important to provide information for the patient’s familyand to explain the objectives of intensive care.Objective To evaluate whether the Pediatric Logistic Organ Dys-function (PELOD) scoring system can be used to determine the prog-nosis of patients treated in PICU.Methods A longitudinal-observational study on patients treated inthe PICU of Hasan Sadikin General Hospital was conducted in No-vember 2004-December 2004. The PELOD scoring system wasapplied to all subjects within the first 24 hours of PICU admission.The scoring system consists of physical and laboratory variables of6 organs, namely neurological, cardiovascular, renal, respiratory,hematological, and hepatic systems. The sum of scores were ana-lyzed by logistic regression method to calculate the probability ofdeath. The subjects were monitored until they passed away or weredischarged from PICU.Results There were 32 subjects who met the inclusion criteria. Themean (SD) of PELOD scores in survivors was 13.5 (8.5) and in nonsurvivors was 22.2 (10.1) (Z M-W =-2.507;P=0.012), while the meanof PELOD scores in survivors of PELOD scores validation studywas 31.0, and in non survivors was 9.4. The increase of PELODscores correlated with the increase in the probability of death(P=0.038), and a linear regression chart showed a positive correla-tion (R 2 =0.93). PELOD scores at a 50% probability of death was 20,while the mean PELOD scores validation study was 26. Based onthe probability of death of P<0.5, the death prediction and observeddeath had a sensitivity of 54.5% and a specificity of 80.9%. Amongpatients with high PELOD scores (>20) and those with low PELODscores (<20), the number of patients who survived with extendedlength of stay in PICU, were not significantly different (P=0.15).Conclusions PELOD scoring system can be used to determine thepatient’s probability of death in PICU, but can not be used to predictthe length of stay in PICU
Recurrent abdominal pain in children Pitono Soeparto; Liek Djupri; Rustiadji Rustiadji; Harjoga Sandi
Paediatrica Indonesiana Vol 21 No 7-8 (1981): July - August 1981
Publisher : Indonesian Pediatric Society

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.14238/pi21.7-8.1981.168-74

Abstract

A total of 48 children aged between 3-12% years, who had complained of at least three episodes of abdominal pain in a period of longer than 3 months, underwent investigations.Routine studies included: blood exam, stool and urine analysis and G.l. X-ray series. EEG, lVP and gall bladder study were occasionally performed.Of the 48 patients studied, 25 (52.1%) were of unknown cause, 11 (22.9%) had emotional disorders, 6 (12.5%) had allergic disorders, 3 (6.3%) had ulcers, 2 (4.2%) had epilepsy and (2.1%) had narrowing of the duodenum.Treatment consisted mostly of psychological approach supported by symptomatic therapy (antacid + fentonium bromide = ulcesium). The results of treatment were in general satisfactory.Upper G.l. studies appeared still to be of value in children with R.A.P. Besides for detecting. organic causes, it is also of importance as part of the psychological approach to convince both the parents and the child that no real organic disease actually existed.

Page 18 of 212 | Total Record : 2118

 16   17   18   19   20 

Filter by Year

1973 2025


Reset
Filter By Issues
All Issue Vol. 65 No. 5 (2025): September 2025 Vol. 65 No. 4 (2025): July 2025 Vol. 65 No. 3 (2025): May 2025 Vol. 65 No. 2 (2025): March 2025 Vol. 65 No. 1 (2025): January 2025 Vol. 64 No. 6 (2024): November 2024 Vol. 64 No. 5 (2024): September 2024 Vol. 64 No. 4 (2024): July 2024 Vol. 64 No. 3 (2024): May 2024 Vol. 64 No. 2 (2024): March 2024 Vol. 64 No. 1 (2024): January 2024 Vol. 63 No. 6 (2023): November 2023 Vol. 63 No. 5 (2023): September 2023 Vol. 63 No. 4 (2023): July 2023 Vol 63 No 4 (2023): July 2023 Vol 63 No 3 (2023): May 2023 Vol 63 No 2 (2023): March 2023 Vol 63 No 1 (2023): January 2023 Vol 63 No 1sup (2023): Supplementary Issue March 2023 Vol 62 No 6 (2022): November 2022 Vol 62 No 5 (2022): September 2022 Vol 62 No 4 (2022): July 2022 Vol 62 No 3 (2022): May 2022 Vol 62 No 2 (2022): March 2022 Vol 62 No 1 (2022): January 2022 Vol 61 No 6 (2021): November 2021 Vol 61 No 5 (2021): September 2021 Vol 61 No 4 (2021): July 2021 Vol 61 No 3 (2021): May 2021 Vol 61 No 2 (2021): March 2021 Vol 61 No 1 (2021): January 2021 Vol 60 No 6 (2020): November 2020 Vol 60 No 5 (2020): September 2020 Vol 60 No 4 (2020): July 2020 Vol 60 No 3 (2020): May 2020 Vol. 60 No. 2 (2020): March 2020 Vol 60 No 2 (2020): March 2020 Vol 60 No 1 (2020): January 2020 Vol 59 No 6 (2019): November 2019 Vol 59 No 5 (2019): September 2019 Vol 59 No 4 (2019): July 2019 Vol 59 No 3 (2019): May 2019 Vol 59 No 2 (2019): March 2019 Vol 59 No 1 (2019): January 2019 Vol 58 No 6 (2018): November 2018 Vol 58 No 5 (2018): September 2018 Vol 58 No 4 (2018): July 2018 Vol 58 No 3 (2018): May 2018 Vol 58 No 2 (2018): March 2018 Vol 58 No 1 (2018): January 2018 Vol 57 No 6 (2017): November 2017 Vol 57 No 5 (2017): September 2017 Vol 57 No 4 (2017): July 2017 Vol 57 No 3 (2017): May 2017 Vol 57 No 2 (2017): March 2017 Vol 57 No 1 (2017): January 2017 Vol 56 No 6 (2016): November 2016 Vol 56 No 5 (2016): September 2016 Vol 56 No 4 (2016): July 2016 Vol 56 No 3 (2016): May 2016 Vol 56 No 2 (2016): March 2016 Vol 56 No 1 (2016): January 2016 Vol 55 No 6 (2015): November 2015 Vol 55 No 5 (2015): September 2015 Vol 55 No 4 (2015): July 2015 Vol 55 No 3 (2015): May 2015 Vol 55 No 2 (2015): March 2015 Vol 55 No 1 (2015): January 2015 Vol 54 No 6 (2014): November 2014 Vol 54 No 5 (2014): September 2014 Vol 54 No 4 (2014): July 2014 Vol 54 No 3 (2014): May 2014 Vol 54 No 2 (2014): March 2014 Vol 54 No 1 (2014): January 2014 Vol 53 No 6 (2013): November 2013 Vol 53 No 5 (2013): September 2013 Vol 53 No 4 (2013): July 2013 Vol 53 No 3 (2013): May 2013 Vol 53 No 2 (2013): March 2013 Vol 53 No 1 (2013): January 2013 Vol 52 No 6 (2012): November 2012 Vol 52 No 5 (2012): September 2012 Vol 52 No 4 (2012): July 2012 Vol 52 No 3 (2012): May 2012 Vol 52 No 2 (2012): March 2012 Vol 52 No 1 (2012): January 2012 Vol 51 No 6 (2011): November 2011 Vol 51 No 5 (2011): September 2011 Vol 51 No 4 (2011): July 2011 Vol 51 No 3 (2011): May 2011 Vol 51 No 2 (2011): March 2011 Vol 51 No 1 (2011): January 2011 Vol 50 No 6 (2010): November 2010 Vol 50 No 5 (2010): September 2010 Vol 50 No 4 (2010): July 2010 Vol 50 No 3 (2010): May 2010 Vol 50 No 2 (2010): March 2010 Vol 50 No 1 (2010): January 2010 Vol 49 No 6 (2009): November 2009 Vol 49 No 5 (2009): September 2009 Vol 49 No 4 (2009): July 2009 Vol 49 No 3 (2009): May 2009 Vol 49 No 2 (2009): March 2009 Vol 49 No 1 (2009): January 2009 Vol 48 No 6 (2008): November 2008 Vol 48 No 5 (2008): September 2008 Vol 48 No 4 (2008): July 2008 Vol 48 No 3 (2008): May 2008 Vol 48 No 2 (2008): March 2008 Vol 48 No 1 (2008): January 2008 Vol 47 No 6 (2007): November 2007 Vol 47 No 5 (2007): September 2007 Vol 47 No 4 (2007): July 2007 Vol 47 No 3 (2007): May 2007 Vol 47 No 2 (2007): March 2007 Vol 47 No 1 (2007): January 2007 Vol 46 No 6 (2006): November 2006 Vol 46 No 5 (2006): September 2006 Vol 46 No 4 (2006): July 2006 Vol 46 No 3 (2006): May 2006 Vol 46 No 2 (2006): March 2006 Vol 46 No 1 (2006): January 2006 Vol 45 No 6 (2005): November 2005 Vol 45 No 5 (2005): September 2005 Vol 45 No 4 (2005): July 2005 Vol 45 No 3 (2005): May 2005 Vol 45 No 2 (2005): March 2005 Vol 45 No 1 (2005): January 2005 Vol 44 No 6 (2004): November 2004 Vol 44 No 5 (2004): September 2004 Vol 44 No 4 (2004): July 2004 Vol 44 No 3 (2004): May 2004 Vol 44 No 2 (2004): March 2004 Vol 44 No 1 (2004): January 2004 Vol 43 No 6 (2003): November 2003 Vol 43 No 5 (2003): September 2003 Vol 43 No 4 (2003): July 2003 Vol 43 No 3 (2003): May 2003 Vol 43 No 2 (2003): March 2003 Vol 43 No 1 (2003): January 2003 Vol 42 No 11-12 (2002): November 2002 Vol 42 No 9-10 (2002): September 2002 Vol 42 No 5-6 (2002): May 2002 Vol 41 No 11-12 (2001): November 2001 Vol 41 No 9-10 (2001): September 2001 Vol 41 No 7-8 (2001): July 2001 Vol 41 No 5-6 (2001): May 2001 Vol 41 No 3-4 (2001): March 2001 Vol 41 No 1-2 (2001): January 2001 Vol 39 No 11-12 (1999): November - December 1999 Vol 39 No 9-10 (1999): September - October 1999 Vol 39 No 7-8 (1999): July - August 1999 Vol 39 No 5-6 (1999): May - June 1999 Vol 39 No 3-4 (1999): March - April 1999 Vol 39 No 1-2 (1999): January - February 1999 Vol 38 No 11-12 (1998): November - December 1998 Vol 38 No 9-10 (1998): September - October 1998 Vol 38 No 3-4 (1998): March - April 1998 Vol 38 No 1-2 (1998): January - February 1998 Vol 37 No 9-10 (1997): September - October 1997 Vol 37 No 5-6 (1997): May - June 1997 Vol 37 No 3-4 (1997): March - April 1997 Vol 37 No 1-2 (1997): January - February 1997 Vol 36 No 11-12 (1996): November - December 1996 Vol 36 No 7-8 (1996): July - August 1996 Vol 36 No 5-6 (1996): May - June 1996 Vol 35 No 9-10 (1995): September - October 1995 Vol 35 No 7-8 (1995): July - August 1995 Vol 35 No 5-6 (1995): May - June 1995 Vol 35 No 3-4 (1995): March - April 1995 Vol 35 No 1-2 (1995): January - February 1995 Vol 34 No 7-8 (1994): July - August 1994 Vol 34 No 5-6 (1994): May - June 1994 Vol 34 No 3-4 (1994): March - April 1994 Vol 34 No 1-2 (1994): January - February 1994 Vol 33 No 7-8 (1993): July - August 1993 Vol 33 No 5-6 (1993): May - June 1993 Vol 33 No 3-4 (1993): March - April 1993 Vol 33 No 1-2 (1993): January - February 1993 Vol 32 No 11-12 (1992): November - December 1992 Vol 32 No 7-8 (1992): July - August 1992 Vol 32 No 5-6 (1992): May - June 1992 Vol 32 No 3-4 (1992): March - April 1992 Vol 31 No 11-12 (1991): November - December 1991 Vol 31 No 9-10 (1991): September - October 1991 Vol 31 No 7-8 (1991): July - August 1991 Vol 31 No 5-6 (1991): May - June 1991 Vol 31 No 3-4 (1991): March - April 1991 Vol 30 No 11-12 (1990): November - December 1990 Vol. 30 No. 9-10 (1990): September-October 1990 Vol. 30 No. 5-6 (1990): May-June 1990 Vol 30 No 1-2 (1990): January - February 1990 Vol 29 No 11-12 (1989): November - December 1989 Vol 29 No 9-10 (1989): September - October 1989 Vol 29 No 7-8 (1989): July - August 1989 Vol 29 No 5-6 (1989): May - June 1989 Vol 29 No 3-4 (1989): March - April 1989 Vol 29 No 1-2 (1989): January - February 1989 Vol 28 No 11-12 (1988): November - December 1988 Vol 28 No 9-10 (1988): September - October 1988 Vol 28 No 7-8 (1988): July - August 1988 Vol 28 No 5-6 (1988): May - June 1988 Vol 28 No 3-4 (1988): March - April 1988 Vol 28 No 1-2 (1988): January - February 1988 Vol 26 No 7-8 (1986): July - August 1986 Vol 25 No 5-6 (1985): May - June 1985 Vol 24 No 11-12 (1984): November - December 1984 Vol 24 No 9-10 (1984): September - October 1984 Vol 24 No 7-8 (1984): July - August 1984 Vol 24 No 5-6 (1984): May - June 1984 Vol 24 No 3-4 (1984): March - April 1984 Vol 24 No 1-2 (1984): January - February 1984 Vol 23 No 9-10 (1983): September - October 1983 Vol 23 No 5-6 (1983): May - June 1983 Vol 23 No 3-4 (1983): March - April 1983 Vol 23 No 1-2 (1983): January - February 1983 Vol 22 No 11-12 (1982): November - December 1982 Vol 22 No 9-10 (1982): September - October 1982 Vol 22 No 7-8 (1982): July - August 1982 Vol 22 No 5-6 (1982): May - June 1982 Vol 22 No 3-4 (1982): March - April 1982 Vol 22 No 1-2 (1982): January - February 1982 Vol 21 No 11-12 (1981): November - December 1981 Vol 21 No 9-10 (1981): September - October 1981 Vol 21 No 7-8 (1981): July - August 1981 Vol 21 No 5-6 (1981): May - June 1981 Vol 21 No 3-4 (1981): March - April 1981 Vol 21 No 1-2 (1981): January - February 1981 Vol 20 No 11-12 (1980): November - December 1980 Vol 20 No 9-10 (1980): September - October 1980 Vol 20 No 7-8 (1980): July - August 1980 Vol 20 No 5-6 (1980): May - June 1980 Vol 20 No 3-4 (1980): March - April 1980 Vol 20 No 1-2 (1980): January - February 1980 Vol 19 No 11-12 (1979): November - December 1979 Vol 19 No 9-10 (1979): September - October 1979 Vol 19 No 3-4 (1979): March - April 1979 Vol 19 No 1-2 (1979): January - February 1979 Vol 18 No 11-12 (1978): November - December 1978 Vol 18 No 9-10 (1978): September - October 1978 Vol 18 No 5-6 (1978): May - June 1978 Vol 18 No 3-4 (1978): March - April 1978 Vol 18 No 1-2 (1978): January - February 1978 Vol 16 No 9-10 (1976): September - October 1976 Vol 16 No 3-4 (1976): March - April 1976 Vol 16 No 1-2 (1976): January - February 1976 Vol 15 No 11-12 (1975): November - December 1975 Vol 15 No 9-10 (1975): September - October 1975 Vol 15 No 7-8 (1975): July - August 1975 Vol 15 No 3-4 (1975): March - April 1975 Vol 15 No 1-2 (1975): January - February 1975 Vol 14 No 11-12 (1974): November - December 1974 Vol 14 No 9-10 (1974): September - October 1974 Vol 14 No 7-8 (1974): July - August 1974 Vol 14 No 5-6 (1974): May - June 1974 Vol 14 No 3-4 (1974): March - April 1974 Vol 14 No 1-2 (1974): January - February 1974 Vol 13 No 4 (1973): April 1973 Vol 13 No 3 (1973): March 1973 Vol 13 No 2 (1973): February 1973 Vol 13 No 1 (1973): January 1973 More Issue