cover
Article Per Year (5 Year)
All
Home Page
OAI Link
Editorial Team
Contact
Reviewer
Google Scholar
Contact Name
Raymond Pranata
Contact Email
raymond_pranata@hotmail.com
Phone
+6282112918892
Journal Mail Official
ijc@inaheart.org
Editorial Address
Editorial Office: Heart House, Jalan Katalia Raya No. 5, Kota Bambu Utara West Jakarta, 11430 - Indonesia Telephone: +62 21 5681149, Fax: +62 21 5684220 Email: ijc@inaheart.org
Location
Kota adm. jakarta barat,
Dki jakarta
INDONESIA
Indonesian Journal of Cardiology
Published by Indonesian Heart Association
ISSN : 28303105     EISSN : 29647304     DOI : -
Core Subject : Health,
Health Professions Medicine & Pharmacology
Indonesian Journal of Cardiology (IJC) is a peer-reviewed and open-access journal established by Indonesian Heart Association (IHA)/Perhimpunan Dokter Spesialis Kardiovaskular Indonesia (PERKI) [www.inaheart.org] on the year 1979. This journal is published to meet the needs of physicians and other health professionals for scientific articles in the cardiovascular field. All articles (research, case report, review article, and others) should be original and has never been published in any magazine/journal. Prior to publication, every manuscript will be subjected to double-blind review by peer-reviewers. We consider articles on all aspects of the cardiovascular system including clinical, translational, epidemiological, and basic studies. Subjects suitable for publication include but are not limited to the following fields: Acute Cardiovascular Care Arrhythmia / Cardiac Electrophysiology Cardiovascular Imaging Cardiovascular Pharmacotherapy Cardiovascular Public Health Policy Cardiovascular Rehabilitation Cardiovascular Research General Cardiology Heart Failure Hypertension Interventional Cardiology Pediatric Cardiology Preventive Cardiology Vascular Medicine
Arjuna Subject : Kedokteran - Cardiology and Cardiovascular Medicine
Articles 712 Documents
 51   52   53   54   55 
Peran Ekokardiografi sebagai Modalitas Diagnostik Pendukung pada Diseksi Aorta Stanford A Terlokalisir Haris Munirwan; Amiliana Mardiani Soesanto; Rina Ariani; Taofan Taofan; Suko Adiarto; Ismoyo Sunu; Bagus Herlambang; Dicky Aligheri; Retno Dwi Astuti
Jurnal Kardiologi Indonesia Vol. 35, No. 4 Oktober - Desember 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i4.497

Abstract

Ascending Stanford type A aortic dissection carries a high morbidity and mortality. Proper identification of the proximal origin of the dissection and determination of concomitant aortic valve involvement significantly facilitate surgical repair which may improve survival.2Rapid imaging is necessary for the timely diagnosis of a potentially life-threatening condition. Transthoracic Echocardiography is highly accurate for the detection of acute aortic syndromes especially identify ascending aortic pathology such as type A aortic dissection.3 We report the unusual case of 52-years old male who present atypical presentation of aortic dissection with unclear view of dissection by CT Angiography aorta, and diagnosed as Localized Stanford A Aortic dissection with supported data by echocardiography modality.
Vena Varikosa Putri Septiani; Heru Sulastomo
Jurnal Kardiologi Indonesia Vol. 35, No. 4 Oktober - Desember 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i4.498

Abstract

Varicose vein is a common problem in vein. It is a dilated, elongated, and tortuous vein. It used to be a cosmetic problem, but nowadays it has become a more serious problem, since it can affect quality of life. It usually occurs in the inferior extremities. The venous system is divided into superficial vein, deep vein, perforator vein, and intramuscular sinus venosus. Pathological conditions that usually occur in the vein system are obstruction and valve incompetence. The risk factors of varicose vein are: elderly, history of family with varicose vein, pregnancy, occupation that requires long standing, sedentary life style, and obesity. Ultrasonography (USG) duplex is the most recommended modality to diagnose varicose vein. The classification of this disease is based on chronic venous disease classification, the CEAP (Clinical, Etiology, Anatomy, Pathophysiology) classification. The management for varicose vein includes intervention (sclerotherapy, endovenous ablation, and surgery) and non-intervention (education, compression stocking, and pharmacology) therapy.
What is The Pacing Mode? Beny Hartono
Jurnal Kardiologi Indonesia Vol. 35, No. 4 Oktober - Desember 2014
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v35i4.499

Abstract

Seorang wanita 56 tahun datang ke poliklinik dengan keluhan dada rasa tidak nyaman dan sensasi seperti ada hentakan di dada sampai ke leher yang dirasakan sejak 3 bulan terakhir yang makin sering. Kadang pasien merasakan sesak nafas dan nyeri kepala. Pasien dengan riwayat pemasangan pacu jantung kurang lebih 8 tahun yang lalu. Pemeriksaan fisik dalam batas normal. Rekaman EKG terlihat seperti gambar di bawah ini,Pada panel A, tampak rekaman EKG yang memperlihatkan adanya pacing spike sebelum timbulnya gelombang QRS, yang menandakan pemacuan dari ventrikel. Namun bila kita lihat lebih memperlihatkan adanya pacing spike sebelum timbulnya gelombang QRS, yang menandakan pemacuan dari ventrikel. Namun bila kita lihat lebihPanel A. Rekaman 12 sadapan EKGJurnal Kardiologi Indonesia304Jurnal Kardiologi Indonesia • Vol. 35, No. 4 • Oktober - Desember 2014teliti, pada beat pertama dan beat kedua terlihat adanya gelombang P yang mendahului (terutama pada sadapan III, gelombang P terlihat paling jelas) sebelum timbulnya gelombang QRS. Tidak ada pacing spike yang mendahului timbulnya gelombang P menandakan tidak adanya pemacuan dari atrium, hanya sensing dari gelombang atrium. Pada beat ke-3 dan ke-4 tidak terlihat adanya gelombang P yang mendahului pemacuan ventrikel, dan terlihat interval yang memanjang antara beat ke-2 dan ke-3 serta beat ke-4. Dari EKG diatas, apa mode pacu jantung yang dipasang pada pasien tersebut dan apakah terdapat disfungsi alat pacu jantung sehingga terdapat keluhan tidak nyaman pada pasien ini dalam 3 bulan terakhir?
Kontroversi peran studi elektrofisiologi pada sindrom brugada Yoga Yuniadi
Jurnal Kardiologi Indonesia Vol. 36, No. 4 Oktober - Desember 2015
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v36i4.500

Abstract

Sindrom Brugada adalah suatu abnormalitas sistem listrik jantung yang merupakan predisposisi terjadinya takikardia ventrikel dan hilang kesadaran. Takikardia ventrikel dapat berhenti spontan dan pasien pulih dari sinkop lalu berobat dengan keluhan sinkop, atau takikardia berdegenerasi menjadi fibrilasi ventrikel (FV)dan menyebabkan kematian jantung mendadak. Oleh karena itu sangat penting mengenal gambaran EKG sindrom Brugada lalu melakukan stratifikasi risiko yang cermat.Salah satu yang menjadi perdebatan hangat adalah stratifikasi risiko sindrom Brugada melalui tindakan studi elektrofisiologi (SEF). Nilai prediktif induksi aritmia ventrikel saat SEF masih kontroversial. Studi yang melibatkan 408 pasien sindrom Brugada tanpa riwayat henti jantung menunjukkan bahwa pasien dengan indusibilitas FV memiliki risiko kematian jantung mendadak karena FV enam kali lipat dalam pengamatan 2 tahun.1 Akan tetapi studi multisenter dari Eropa,2, 3 Jepang4, 5 dan beberapa metaanalisis6, 7 tidak menunjukkan hasil yang positif sehingga indikasi SEF untuk stratifikasi risiko sindrom Brugada hanya IIb pada tahun 2013 (Gambar 1). Metaanalisis8 yang lain memperlihatkan hasil yang positif yaitu ketika presentasi klinis pasien sindrome Brugada dipisahkan antara sinkop dan asimtomatik dalam analisanya.
Peranan Mitral Leaflet Separation Index (Mlsi ) dalam Menentukan Severitas Stenosis Mitral I Made Junior Rina Artha; Amiliana M Soesanto; Indriwanto Sakidjan; Ganesja M Harimurti
Jurnal Kardiologi Indonesia Vol. 36, No. 4 Oktober - Desember 2015
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v36i4.501

Abstract

Objective. To Correlate MLSI with 3-D mitral valve area (MVA) planimetry in determining mitral stenosis (MS) severity.Background. Mitral Stenosis (MS) is still a major problem in cardiology, and causes of morbidity dan mortality worldwide. Echocardiogrphy plays an important role in assessing mitral stenosis severity. Mitral leaflet separation index (MLSI) is one of simple method that can be used in peripheral by using common ultrasound to assess the severity mitral stenosis.Methods. We employed a cross sectional study. Mitral stenosis patients who referred for evaluation echocardiography in National Cardiac Center Harapan Kita from April to September 2011. MLSI was obtained by averaging the maximal leaflet separation distance at the tips in diastole in parasternal long-axis and apical four- chamber views. 3-Dimensional (3-D) mitral valve area (MVA) planimetry as a reference. The only exclusion criteria was severe calcification and poor echo window. Echocardiography examination using Philips E33i.Results. Seventy six consecutive patients were enrolled, 5 subjects were excluded from study because of severe calcification and poor echo window. Proportion of woman is 73.2 % and mostly in age group < 40 years old (43.7 %). Severe mitral stenosis was dominate the subject, 47 subject (66.2 %), moderate was 19 subject (26.8 %), and mild only 5 subjects (7.0 %). Analysis with Spearman correlations obtained a good correlation with r = 0.70, p < 0.001, good correlation was found in sinus rhythm with r = 0.78, p < 0.001 and atrial fibrillation with r = 0.79, p < 0.001. MLSI less than 0.69 cm predicted severe MS with 85 % sensitivity and 82.4 % specificity.Conclusions. Mitral leaflet separation index (MLSI) has a good correlation with 3-D MVA planimetry. MLSI less than 0.69 cm can estimate severe SM.
Pemanfaatan Indole-3-Carbinol Sebagai Inhibitor Flavin Monooxygenase 3 (Fmo 3) Dalam Upaya Prevensi Aterosklerosis Bagus Indra Kusuma; Brenda Desy Romadhon; Silvi Ahmada Chasya; Hazmi Dwinanda Nurqistan; Lusi Padma Sulistianingsih Mata
Jurnal Kardiologi Indonesia Vol. 36, No. 4 Oktober - Desember 2015
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v36i4.502

Abstract

Cardiovascular disease is the leading cause of death in the world, reaching 30% of all mortality. The most common cause of cardiovascular disease is the formation of atherosclerotic plaque in blood vessels. Treatment has been done to overcome atherosclerosisonly curative and still no preventive. Processes that play a role in the formation of atherosclerotic plaque is very complex and one of the causes deposition of plaques is the formation of the compound trimethylamine oxide (TMAO) in the body. TMAO that has produced can increase the accumulation of cholesterol in macrophages so that increasing the formation of foam cells in the arterial wall. These compounds are derived from trimethylamine (TMA), which is converted into TMAO by enzyme flavin monooxygenase (FMO). FMO enzyme that is able to make an impact in the formation of TMAO is flavin monooxygenase 1 (FMO1) and flavin monooxygenase 3 (FMO3). However, FMO3 showed activity ten times greater than FMO1 in turning TMA into TMAO. Indole-3-carbinol can be a role for this enzyme inhibitor so that the therapeutic use of indole-3-carbinol is expected to inhibit TMAO. Therefore, the authors propose the use of research in the form of indole-3-carbinol as an inhibitor of flavin monooxygenase 3 (FMO3) in atherosclerosis prevention efforts. The study design used was pure experimental research design (true experimental design) with post test only randomized control group design. Mice (Mus musculus) males were treated indole-3-carbinol and then is given atherogenic diet for the provision of intravenous adrenaline 0.00084 mg / 20 gBW and egg yolks 0.2 cc / day. The treatment group consisted of a positive control, negative control, treatment A (10 mg / kg BW of indole-3-carbinol), B (200 mg / kg BW I3C), and C (500 mg / kg BW I3C). The data observed in the form of cholesterol, foam cell histopathological picture of the aorta and the density of the band FMO3 activity. Blood cholesterol levels showed a decrease in accordance with increase in dose. Cholesterol control of negative group, positive, A, B and C respectively of 119.4 ± 28.94 mg / dL, 246 ± 8.52 mg / dL, 224 ± 15.30 mg / dL, 170.6 ± 12.54 mg / dL, and 154.8 ± 14.46 mg / dL (p <0.05). Histopathologic features foam cell in the aorta of mice showed an improvement with the increase in dose. FMO3 enzyme activity also showed a decrease when compared to the positive control in the optical density relative scale along with rising doses of indole-3-carbinol given. This shows that the use of indole-3-carbinol is very effective in atherosclerosis prevention efforts.
Pengaruh Trimetazidine Terhadap Jumlah Netrofil Pasca IKPP Taka Mehi; Yoga Yuniadi; Nur Haryono
Jurnal Kardiologi Indonesia Vol. 36, No. 4 Oktober - Desember 2015
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v36i4.503

Abstract

Background: Reperfusion strategy, either with thrombolytic or Primary Percutaneous Coronary Intervention (PPCI), is the core treatment for Acute ST-Segment Elevation Myocardial Infarct (STEMI). The goal of PPCI is to restore the patency of infarcted epicardial artery and establish microvascular reperfusion as soon as possible so that necrotic myocardial area can be reduced. However, successful restoration of infarcted epicardial artery is not always followed by enough reperfusion to the microvascular part. Trimetazidine can reduce neutrophil which was mediated by tissue trauma during ischemic heart condition. Unfortunately, its influence over neutrophil accumulation in acute STEMI patients which undergo PPCI is not well understood.Method: There were 68 consecutive-selected acute STEMI patients which undergo PPCI since January 2015 until Juni 2015. They were admitted in emergency department. Peripheral vein blood sampling was taken to measure neutrophil before PPCI was performed. Six hour after PPCI was conducted, another peripheral vein blood sampling was taken for another neutrophil measurement. Neutrophil measurement was performed with Sysmex 2000i. Statistical analysis was performed by using SPSS 17.Result: Among 68 patients, divided in two groups, trimetazidine 28 patients and plasebo 40 patients. There were no differences amount of neutrophils in trimetazidine or plasebo group, before or after PPCI. Neutrophil pre PPCI in trimetazidine vs plasebo group 10.71 ± 3.263 vs 10.99 ± 3.083, p:0,341. Neutrophil post PPCI in trimetazidine vs plasebo group 9.49 ± 3.135 vs 9.92 ± 3.463, p:0,664.Conclusion: There were no reducing amount of neutrophils after trimetazidine was given in patients STEMI which underwent PPCI
Fistula Arteri Koroner, Penyebab Nyeri Dada yang Jarang Ditemukan pada Dewasa : Peran Computed Tomography Angiografi Prima Almazini; Andy Rahman; Dian Yaniarti; Elen Sahara; Celly Anantaria; Manoefris Kasim
Jurnal Kardiologi Indonesia Vol. 36, No. 4 Oktober - Desember 2015
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v36i4.504

Abstract

Coronary artery fistula, usually congenital in origin, is an abnormal communication between a coronary artery and a cardiac chamber or great vessel. A coronary artery fistula can produce myocardial ischemia from coronary steal phenomenon. First case, a 54-year-old man was found to have a fistula from the left anterior descending coronary artery and right coronary artery to the main pulmonary artery, a rare anomaly. This patient developed chest pain due to myocardial ischemia in the left anterior descending coronary artery distribution for several months before evaluation. The patient was suggested to close the fistula but rejected. Second case, a-57 year-old woman was found to have fistula from left anterior descending coronary artery to main pulmonary artery. Patient presented with chest pain since 6 years ago. The patient was planned to close the fistula transcutaneously. Coronary artery fistulas, though rare, should be considered in the differential diagnosis when an adult patient presents with chest pain. Although coronary angiography is the gold standar diagnostic test for detection of coronary artery fistula, computed tomography angiography may be an alternative test through its good spatial resolution.
Farmakoterapi pada Upaya Berhenti Merokok Diyan Ekawati; Basuni Radi; Feni Fitriani Taufik
Jurnal Kardiologi Indonesia Vol. 36, No. 4 Oktober - Desember 2015
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v36i4.505

Abstract

Nicotine dependence is a main role in tobacco dependence. Its physiological and psychological effect make smoker difficult to quit, making low success rate in smoking cessation. Combination of different method and continuity will increase smoking cessation success rate. Evidence shown that pharmacotherapy will increase success rate. Guideline’s for smoking cessation endorse physician to use first line pharmacotherapy, but it’s clinical application still limited. Physician should be aware about possibility for side effect. Some randomized controlled trial report major cardiovascular event in first line pharmacotherapy. Recent systematic review report no correlation between first line pharmacotherapy in smoking cessation and major cardiovascular event.
Terapi Resinkronisasi Jantung pada Penanganan Gagal Jantung Hilman Zulkifli Amin; Siska Suridanda Danny
Jurnal Kardiologi Indonesia Vol. 36, No. 4 Oktober - Desember 2015
Publisher : The Indonesian Heart Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.30701/ijc.v36i4.506

Abstract

Heart failure (HF) is a worldwide health problem with high prevalence rate. The prevalence is over 23 million worldwide. It is a chronic disease characterized by the inability of the heart to pump an adequate amount of blood to achieve the demand of the different organ systems and/or doing so at increased filling pressures. Despite many recent advances in medication, the rate of people with HF is rising. This health challenges need to be answered properly. One of the new important treatment for HF is cardiac resynchronization therapy (CRT). Many patients with HF also have an abnormality of the heart’s electrical system resulting in asynchronous contraction pattern of heart muscle.The ultimate goal of CRT is to restore synchrony of the heart rhythm in HF patients. CRT implantation in heart failure patients with proper indications like wide QRS complexes, low left ventricular ejection fraction (LVEF), and left bundle branch block (LBBB) has been proved to reduce morbidity, mortality, and also improve symptoms and quality of life (QoL).

Page 53 of 72 | Total Record : 712

 51   52   53   54   55 

Filter by Year

2002 2025


Reset
Filter By Issues
All Issue Vol 46 No 4 (2025): October - December, 2025 Vol 46 No 3 (2025): July - September, 2025 Vol 46 No 2 (2025): April - June, 2025 Vol 46 No 1 (2025): January - March, 2025 Vol 45 No Suppl_A (2024): Vol 45 No Suppl_A (2024): Abstracts of the 6th Indonesian Intensive & Acut Vol 45 No 4 (2024): Online First - Indonesian Journal of Cardiology April-June 2021 Vol 45 No 3 (2024): July - September, 2024 Vol 45 No 2 (2024): April - June, 2024 Vol 45 No 1 (2024): January - March, 2024 Vol 45 No Suppl_C (2024): Abstracts of the 33rd Annual Scientific Meeting of the Indonesian Heart As Vol 45 No Suppl_B (2024): Abstracts of the 11th Annual Scientific Meeting of the Indonesian Heart Rh Vol 44 No 4 (2023): Indonesian Journal of Cardiology: October - December 2023 Vol 44 No 3 (2023): Indonesian Journal of Cardiology: July - September 2023 Vol 44 No 2 (2023): Indonesian Journal of Cardiology: April - June 2023 Vol 44 No 1 (2023): Indonesian Journal of Cardiology: January - March 2023 Vol 44 No Suppl_A (2023): Abstracts of the 32nd Annual Scientific Meeting of the Indonesian Heart As Vol 44 No Suppl_B (2023): Abstracts of the 10th Annual Scientific Meeting of the Indonesian Heart Rh Vol 43 No 4 (2022): Indonesian Journal of Cardiology: October - December 2022 Vol 43 No 3 (2022): Indonesian Journal of Cardiology: July - September 2022 Vol 43 No 2 (2022): Indonesian Journal of Cardiology: April - June 2022 Vol 43 No Suppl_B (2022): Abstracts of the 6th InaPrevent (2022) Vol 43 No 1 (2022): Indonesian Journal of Cardiology: January - March 2022 Vol 43 No Suppl_C (2022): Abstracts of the 31st Annual Scientific Meeting of the Indonesian Heart As Vol 43 No Suppl_D (2022): Abstracts of the 9th Annual Scientific Meeting of the Indonesian Heart Rhy Vol 43 No Supplement (2022): Abstracts of the 5th Indonesian Intensive and Acute Cardiovascular Care Vol 42 No 4 (2021): Indonesian Journal of Cardiology: October - December 2021 Vol 42 No 3 (2021): Indonesian Journal of Cardiology: July - September 2021 Vol 42 No 2 (2021): Indonesian Journal of Cardiology: April - June 2021 Vol 42 No 1 (2021): Indonesian Journal of Cardiology: January - March 2021 Vol 42 No Supplement (2021): Abstracts of the 5th InaPrevent in Conjunction with the 1st InTension S Vol 42 No Supplement (2021): Abstracts of the 13th Indonesian Society of Interventional Cardiology A Vol 42 No Supplement (2021): Abstracts of the 8th Annual Scientific Meeting of the Indonesian Heart Vol 41 No 4 (2020): Indonesian Journal of Cardiology: October - December 2020 Vol 41 No 3 (2020): Indonesian Journal of Cardiology: July - September 2020 Vol 41 No 2 (2020): Indonesian Journal of Cardiology: April - June 2020 Vol 41 No 1 (2020): Indonesian Journal of Cardiology: Januari - Maret 2020 Vol 41 No Suppl_A (2020): Abstracts of the 29th Annual Scientific Meeting of the Indonesian Heart As Vol 41 No Suppl_B (2020): Abstracts of the 12th Indonesian Society of Interventional Cardiology Annu Vol 40 No 4 (2019): Indonesian Journal of Cardiology: October-December 2019 Vol 40 No 3 (2019): Indonesian Journal of Cardiology: July-September 2019 Vol 40 No 2 (2019): Indonesian Journal of Cardiology: April-June 2019 Vol 40 No 1 (2019): Indonesian Journal of Cardiology: January-March 2019 Vol 39 No 4 (2018): Indonesian Journal of Cardiology: October-December 2018 Vol 39 No 3 (2018): Indonesian Journal of Cardiology: July-September 2018 Vol 39 No 2 (2018): Indonesian Journal of Cardiology: April-June 2018 Vol 39 No 1 (2018): January - March 2018 Vol 39 No 1 (2018): January - March 2018 Vol 39 No Suppl_B (2018): Indonesian Journal of Cardiology Supplement_B Vol 39 No Suppl_B (2018): Indonesian Journal of Cardiology Supplement_B Vol 39 No Suppl_A (2018): Indonesian Journal of Cardiology Supplement_A Vol 39 No Suppl_A (2018): Indonesian Journal of Cardiology Supplement_A Vol 38 No 4 (2017): October - December 2017 Vol 38 No 4 (2017): October - December 2017 Vol 38 No 3 (2017): July - September 2017 Vol 38 No 3 (2017): July - September 2017 Vol. 38, No. 2 April-June 2017 Vol. 38, No. 2 April-June 2017 Vol. 38, No. 1 Januari-Maret 2017 Vol. 37, No. 4 Oktober - Desember 2016 Vol. 37, No. 3 Juli - September 2016 Vol. 37, No. 2 April - Juni 2016 Vol. 37, No. 1 Januari - Maret 2016 Vol. 36, No. 4 Oktober - Desember 2015 Vol. 36, No. 3 Juli - September 2015 Vol. 36, No. 2 April - Juni 2015 Vol. 36, No. 1 Januari - Maret 2015 Vol. 35, No. 4 Oktober - Desember 2014 Vol. 35, No. 3 Juli - September 2014 Vol. 35, No. 2 April - Juni 2014 Vol. 35, No. 1 Januari - Maret 2014 Vol. 34, No. 4 Oktober - Desember 2013 Vol. 34, No. 3 Juli - September 2013 Vol. 34, No. 2 April - Juni 2013 Vol. 34, No. 1 Januari - Maret 2013 Vol. 33, No. 4 Oktober - Desember 2012 Vol. 33, No. 3 Juli - September 2012 Vol. 33, No. 2 April - Juni 2012 Vol. 33, No. 1 Januari - Maret 2012 Vol. 32, No. 4 Oktober - Desember 2011 Vol. 32, No. 3 Juli - September 2011 Vol. 32, No. 2 April - Juni 2011 Vol. 32, No. 1 Januari - Maret 2011 Vol. 31, No. 3 September - Desember 2010 Vol. 31, No. 2 Mei - Agustus 2010 Vol. 31, No. 1 Januari - April 2010 Vol. 30, No. 3 September - Desember 2009 Vol. 30, No. 2 Mei - Agustus 2009 Vol. 30, No. 1 Januari - April 2009 Vol. 29, No. 3 September - Desember 2008 Vol. 29, No. 2 Mei - Agustus 2008 Vol. 29, No. 1 Januari - April 2008 Vol. 28, No. 6 November 2007 Vol. 28, No. 5 September 2007 Vol. 28, No. 4 Juli 2007 Vol. 28, No. 3 Mei 2007 Vol. 28, No. 2 Maret 2007 Vol. 28, No. 1 Januari 2007 Vol. 26, No. 1 Januari - Maret 2002 More Issue