Garuda - Garba Rujukan Digital

Article Per Year (5 Year)

All

Jurnal Neuroanestesi Indonesia

jni
Website

Published by Indonesian Society of Neuroanesthesia & Critical Care

ISSN : 20889674 EISSN : 24602302 DOI : https://doi.org/10.24244/jni

Core Subject : Health, Science, Education,

Biochemistry, Genetics & Molecular Biology Education Medicine & Pharmacology Neuroscience Public Health

Editor of the magazine Journal of Neuroanestesi Indonesia receives neuroscientific articles in the form of research reports, case reports, literature review, either clinically or to the biomolecular level, as well as letters to the editor. Manuscript under consideration that may be uploaded is a full text of article which has not been published in other national magazines. The manuscript which has been published in proceedings of scientific meetings is acceptable with written permission from the organizers. Our motto as written in orphanet: www.orpha.net is that medicine in progress, perhaps new knowledge, every patient is unique, perhaps the diagnostic is wrong, so that by reading JNI we will be faced with appropriate knowledge of the above motto. This journal is published every 4 months with 8-10 articles (February, June, October) by Indonesian Society of Neuroanesthesia & Critical Care (INA-SNACC). INA-SNACC is associtation of Neuroanesthesia Consultant Anesthesiology and Critical Care (SpAnKNA) and trainees who are following the NACC education. After becoming a Specialist Anesthesiology (SpAn), a SpAn will take another (two) years for NACC education and training in addition to learning from teachers in Indonesia KNA trainee receive education of teachers/ experts in the field of NACC from Singapore.

Arjuna Subject : Kedokteran - Anesthesiology and Pain Medicine

Articles 8 Documents

Search results for , issue "Vol 3, No 3 (2014)" : 8 Documents clear

Awake Craniotomy pada Biopsi Steriotaktik Tumor Supratentorial di daerah Thalamus Dextra et causa Suspect Thalamic Glioma Satriyanto, M. Dwi; Saleh, Siti Chasnak
Jurnal Neuroanestesi Indonesia Vol 3, No 3 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Awake craniotomy merupakan suatu prosedur yang banyak digunakan pada kasus-kasus intrakranial dengan berbagai tujuan, yang memungkinkan dapat menentukan lokasi kelainan di otak yang akurat dan meminimalkan risiko cedera neurologis selama tindakan. Peran anaesthesiologist adalah untuk memberikan analgesia dan sedasi yang memadai sambil mempertahankan ventilasi dan stabilitas hemodinamik pada pasien yang sadar dan harus kooperatif selama tindakan berlangsung. Seorang wanita berusia 32 tahun dengan tumor supratentorial at region thalamus dextra et causa suspek thalamic glioma untuk dilakukan tindakan steriotaktik biopsi dengan Awake craniotomy. Pada pemeriksaan ditemukan keluhan sulit berjalan sejak 4 tahun karena sisi tubuh bagian kiri lemah, bicara cedal, mulut mencong ke kanan, kejang pada kepala dan mata sebelah kiri. Pasien dirujuk karena muntah hebat dan sakit kepala hebat 1 minggu terakhir, kesadaran komposmentis, GCS E4M6V5. Paresenerves VI kanan-kiri, parese nerves VII sinistra sentral. Pemeriksaan laboratorium, ECG dan foto thorak tidak didapatkan kelainan, sedangkan pada MSCT kepala didapat kan adanya massa berbatas tidak tegas, dinding tidak teratur dengan kalsifikasi minimal di thalamus kanan disertai edema perifokal kemungkinan suatu low grade astrocytoma dan hydrocephalus obstruksi. Tindakan biopsi steriotaktik terhadap tumor supratentorial ini dilakukan dengan tehnik anestesi awake craniotomy dengan obat dexmedetomidin, propofol dan fentanyl. Pengawasan pasien di ruang pemulihan selama 4 jam.Setelah Modified Aldrete score 910, pasien dipindahkan ke ruangan.Awake Craniotomy in Stereotactic Biopsy for Supratentorial Tumorsat Thalamus Dextra Region et causa Suspect Thalamic GliomaAwake craniotomy is a procedure that is widely used in intracranial procedures with a variety purposes, which also allows an accurate localization of abnormalities in the brain, and to minimize the risk of neurological injury. Anaesthesiologist role is to provide adequateanalgesia and sedation while maintaining ventilation and hemodynamic stability in patients that still conscious and cooperative during the surgery. A 32years old woman with supratentorial tumor at theright thalamus with suspected thalamic glioma. Stereotactic biopsy was performed under awake craniotomy.She was sufferedwith difficulty in walking for 4 years due to weakness of the left side of the body,slurred talking, and lopsided mouth to the right, withspastic on the head and left eye. She was referred because of severe vomiting and headaches since 1 week, but still fully alert withGCS E4M6V5. She had bilateral nerve VI and central of left nerve VIIpareses. Her laboratory examinations, ECG and thoracic images were normal, whereas MSCT showeda mass with not firm verge, irregular wall with minimal calcification in the right thalamus and perifocaledema, suggested as a low grade astrocytoma and hydrocephalus obstruction. Stereotactic biopsy of supratentorial tumors was performed under awake craniotomy with dexmedetomidine, propofol and fentanyl. The patient was observed at the PACU for 4 hours, and after Modified Aldrete score reached 910, the patient was transferred to the ward.

Korelasi antara Rentang Waktu Cedera Otak Traumatik dengan Dimulainya Terapi Pembedahan Kraniotomi terhadap Kejadian dan Beratnya Post Traumatic Headache (PTH) Halimi, Radian Ahmad; Fuadi, Iwan; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 3, No 3 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Latar Belakang dan Tujuan: Keluhan nyeri kepala setelah terjadinya Cedera Otak Traumatik (COT) dikenal sebagai Post Traumatic Headache (PTH) yang dapat terjadi setelah cedera kepala ringan, sedang, atau berat. Tujuan penelitian ini adalah mencari apakah ada korelasi antara rentang waktu kejadian COT hingga dilakukannya terapi pembedahan kraniotomi terhadap angka kejadian dan beratnya PTH. Subjek dan Metode: Penelitian observasional kohort prospektif pada 33 orang pasien COT derajat ringan atau sedang dengan pengambilan data secara consequetif sampling. Parameter yang dicatat dalam penelitian ini antara lain usia, jenis kelamin, berat badan, GCS, rentang waktu dari kejadian COT hingga dilakukannya terapi pembedahan kraniotomi, angka kejadian PTH, derajat berat nyeri dengan menggunakan sistem penilaian Numeric Rating Scale (NRS). Analisis korelasi linear dua variabel dihitung berdasarkan analisis korelasi Spearman. Hubungan korelasi bermakna bila koefisien korelasi (R) 0,4 dan nilai p0,05. Hasil: Hasil penelitian menunjukkan adanya korelasi yang kuat antara rentang waktu terhadap kejadian PTH (r = 0,75) dengan korelasi searah dan bermakna (p0,05). Terdapat korelasi yang kuat antara rentang waktu terhadap derajat beratnya PTH (r = 0,82) dengan korelasi searah dan bermakna (p0,05). Simpulan: semakin lama rentang waktu dari kejadian COT hingga dilakukannya terapi pembedahan kraniotomi maka akan semakin banyak angka kejadian dan semakin berat PTH.The Correlation between The Interval of Traumatic Brain Injury with Craniotomy Surgery Start on The Incidence and Severity of Post Traumatic Headache (PTH)Background and Objective: Complaints of headache in the aftermath of Traumatic Brain Injury (TBI) is known as Post Traumatic Headache (PTH), which can occur after mild, moderate, or severe head injury. The purpose of this study is to find a correlation between the time span from the TBI events until the craniotomy surgical therapy was performed with the incidence and severity of PTH.Subject and Method: Prospective observational cohort study in 33 patients with mild or moderate TBI with data retrieval consequetif sampling. The parameters recorded in this study including age, gender, weight, GCS, time interval between the events of TBI until the craniotomy surgical therapy was performed, the incidence of PTH, severity of pain using NRS score. Analysis of linear correlation of two variables calculated by Spearman correlation analysis. Significant correlation when the correlation coefficient (R) 0.4 and p 0.05.Result: The results showed a strong correlation between the interval of the incidence with the incidence of PTH (r = 0.75) with unidirectional and significant correlation (p 0.05). There is a strong correlation between the time span from TBI events until the craniotomy surgical therapy with the severity of PTH (r = 0.82) with unidirectional and significant correlation ( p 0.05).Conclusions: the longer of interval between the TBI events to craniotomy surgical treatment, the more of the incidence and severity of PTH.

Pemantauan Neurofisiologis Intraoperatif selama Anestesia untuk Operasi Meningioma Foramen Magnum Firdaus, Riyadh; Suryono, Bambang; Saleh, Siti Chasnak
Jurnal Neuroanestesi Indonesia Vol 3, No 3 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Pemantauan neurofisiologis intraoperatif (Intraoperative neurophysiological monitoring/IONM) pada operasi yang rentan mencederai saraf sangat penting untuk menunjang proses keputusan medis intraoperatif dan pada akhirnya mengurangi angka morbiditas. Operasi meningioma foramen magnum sangat berisiko cedera saraf dan morbiditas sehingga menjadi kandidat yang cocok untuk penggunaan IONM. Cakupan manajemen anesthesia pada operasi yang menggunakan IONM adalah pertimbangan tentang pilihan dan dosis obat anestesia yang digunakan serta perhatian terhadap kestabilan homeostasis pasien. Pemahaman yang baik oleh dokter bedah, anestesi dan neurologi akan membuat tindakan operasi berjalan dengan lancar dan mencegah terjadinya komplikasi intra dan pascaoperasi. Seorang wanita umur 39 tahun dengan keluhan utama nyeri kepala belakang sejak 2 bulan yang lalu. Berdasarkan anamnesis, pemeriksaan fisik, dan pemeriksaan penunjang pasien di diagnosis tumor meningioma pada regio foramen magnum. Pasien dilakukan operasi kraniotomi removal tumor dengan panduan IONM dalam posisi park bench. Lama operasi kurang lebih 14 jam. Pascaoperasi pasien tidak dilakukan ekstubasi dan dirawat di ICU sehari.Intraoperative Neurophysiological Monitoring (IONM) during Anesthesia for Meningioma Foramen Magnum SurgeryIntraoperative neurophysiological monitoring (IONM) in a surgery that is prone to neuronal injury is very useful to guide intraoperative decision makings and to reduce morbidity. Foramen magnum tumor surgerycarries a very high risk for neuronal injury, and thereforeapplication of IONM would be advantageous. The termsof anesthetic management in IONM-guided-surgery are the selection of anesthetic agents with limitation of the dosageswhileremain focusingon stability of patients homeostasis. A thorough understanding and communication among surgeon, neurologist and anesthesiologist are important to createan uneventful procedure and to reduce intra and postoperative complications.A 39 years old female with severe headache for 2 months was diagnosed with meningioma at foramen magnum based on history, physical examination, and advanced examination procedures. The patient was underwent tumor removal guided by IONM on park bench position. The duration of surgery was 14 hours. The patient was not extubatedpostoperatively and admitted to ICU for a day.

Penatalaksanaan Anestesi dengan TIVA Propofol-Dexmedetomidine-Fentanyl untuk Operasi Meningioma Frontalis Sinistra Rebecca Sidhapramudita Mangastuti; A. Himendra Wargahadibrata; Nazaruddin Umar
Jurnal Neuroanestesi Indonesia Vol 3, No 3 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Meningioma merupakan tumor intrakranial jinak yang sering ditemukan. Berasal dari jaringan meningen dan medulla spinalis, tidak tumbuh dari jaringan otak. Pada kasus ini, pasien laki-laki, 46 tahun, 80 kg, datang ke rumah sakit dengan keluhan kejang berulang dan sakit kepala yang hilang timbul sejak 5 bulan yang lalu. Kesadaran composmentis, GCS 15, pupil isokor bilateral 2 mm, hemodinamik stabil, jantung dan paru tidak ada kelainan dan tidak ada kelumpuhan atau kelemahan pada ke empat ekstremitas. Magnetic Resonance Imaging (MRI) brain ditemukan masa hipointens yang melekat dengan meningen di frontal kiri ukuran 52x48x43 mm, kesan convexitas meningioma disertai perifokal edema dengan midline shift ke kanan sekitar 7 mm. Disimpulkan meningioma frontal sinistra dan dianjurkan kraniotomi pengangkatan tumor. Operasi dilakukan dengan anestesi umum. Tehnik anestesi menggunakan Total Intra Venous Anesthesia (TIVA) dengan syringe pump. Operasi berlangsung selama 7 jam dan tumor dapat terangkat semua. Jumlah perdarahan 1000 mL. Pasien mendapat 300 ml Fresh Frozen Plasma (FFP) dan 500 ml Packed Red Cell (PRC) intraoperasi. Untuk mengurangi tekanan intrakranial, diberikan manitol 0,5 gram/kgBB dan drainase cairan serebrospinal 10–20 mL langsung ke ventrikel lateral oleh operator. Pascaoperasi, pasien diekstubasi dan rawat diruang ICU. Dengan data five year survival rate untuk meningioma jinak 70%, meningioma ganas 55%, diharapkan prognosis pasien pascaoperasi adalah dubia ad bonam. Management Anesthesia with TIVA Propofol-Dexmedetomidine-Fentanyl for Meningioma Frontalis Sinistra OperationMeningiomas are the most common benign intracranial tumors. These tumors originate from the meninges and spinal cord, not from the brain tissue. A 46 year old 80 kgs male patient, was admited to the hospital with recurrent seizures and intermittent headaches that occured since five months ago. He was fully alert, GCS 15, both pupils were isokor (2 mm), with stable hemodynamic, no parese in all extremities and normal heart and lung. Magnectic Resonance Imaging (MRI) result showed a 52x48x43 mm mass attached to the meninges at the left frontal with perifocal tumour edema and midline shifted to the right about 7 mm. The patients was diagnosed with the left frontal meningioma and suggested for craniotomy tumour removal. The surgery was performed under general anesthesia using. Total Intra Venous Anesthesia (TIVA) with syringe pump. The 7 hours surgery performed uneventfully with total bleeding of 1000 mL and the patient was received 300 mL Fresh Frozen Plasma (FFP) and 500 ml Packed Red Cell (PRC) intraoperatively. To reduce intracranial pressure, a 0.5gr/kg mannitol was and a 10–20 cc of cerebrospinal liquor drainage through the lateral ventricle was performed by the operator. The patient was extubated after the operation and admitted the ICU for futher management. With the five year survival rate of 70% for benign meningioma and 55% for malignant meningiomas, the prognosis of this patient is dubia ad bonam.

Terapi Hipotermi setelah Cedera Otak Traumatik Dewi Yulianti Bisri; Tatang Bisri
Jurnal Neuroanestesi Indonesia Vol 3, No 3 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Mekanisme proteksi otak hipotermi adalah mengurangi kebutuhan metabolik, cerebral metabolic rate for oxygen (CMRO2), eksitotoksisitas, menurunkan pelepasan glutamat, menurunkan pembentukan radikal bebas, mengurangi pembentukan edema, stabilisasi membran, memelihara adenosine triphosphate (ATP), menurunkan influx Ca, dan tekanan intrakranial. Sedangkan komplikasi hipotermi berat adalah pneumonia, sepsis, disritmia jantung, hipotensi, masalah perdarahan dan menggigil. Temperatur ideal untuk hipotermia terapeutik adalah 35 0C. Pertanyaan untuk terapi hipotermik (HT) adalah bagaimana mekanisme terapi hipotermi sebagai protektor otak? Berapa derajat C penurunan suhu tubuhnya? Bagaimana cara melakukan penurunan suhu? Berapa cepat hipotermia harus dicapai? Berapa lama hipotermi dipertahankan? Bagaimana memulihkan ke normotermi (rewarming)? Bagaimana hasilnya? Apakah ada penelitian yang sedang berlangsung? Untuk menggunakan hipotermia sebagai neuroprotektor, diperlukan mencapai keadaan hipotermi secepat mungkin setelah cedera dan pertahankan pada level aman. Metode hipotermi terapeutik adalah pendinginan permukaan tubuh, pendinginan endovaskuler, pendinginan kepala. Selama penghangatan kembali pasien dengan hipertensi intrakranial telah diketahui bisa terjadi peningkatan tekanan intrakranial selama pemanasan yang cepat. Dianjurkan pemanasan lambat lebih dari 12 jam dengan kecepatan 0,1 0C/jam. Sebagai simpulan, hipotermi terapeutik masih kontroversi, tapi dalam situasi klinik pertahankan suhu pasien 35 0C dan harus dihindari temperatur lebih dari 37 0C. Untuk mencapai suhu inti 35 0C dianjurkan digunakan metode pendinginan permukaan tubuh. Hypothermia Therapy after Traumatic Brain InjuryThe mechanism of hypothermia as neuro protector are by reducing metabolic demand of the brain, cerebral metabolic rate of oxygen (CMRO2), excitotoxicity, decrease the glutamate release, reduction of free radical formation, edema formation, membrane stabilization, maintains adenosine triphosphate (ATP), decrease in Ca influx, and intracranial pressure. In the order hand, complication of deep hypothermia are pneumonia, sepsis, cardiac dysrrhythmia, hypotension, bleeding problem and shivering. The ideal temperature for therapeutic hypothermia is 35 0C. Question arised for hypothermic therapy (HT) are what is the therapeutic mechanism of HT as neuroprotective? What is the proper degree for hypothermia? What can we do to induce hypothermia? How soon should we do the HT? How long hypothermia should be maintain? How to restore normothermia (rewarming)? What is the result? Is there any ongoing research?. For the use hypothermia as one of neuroprotective therapy, it is necessary to implement it as soon as possible after the insult and to maintain it at the lowest safe level. Methods of therapeutic hypothermia are surface cooling, endovascular cooling, as well as selective head cooling. During rewarming, patients with intracranial hypertension are known to have reflex that would increase ICP during rapid rewarming. Slow rewarming over a period of 12 hrs at the rate of 0.1 0C/hr is desirable. As conclusion, therapeutic hypothermia still controversial, but in clinical situation keep the patient 35 0C is desirable and temperature more than 37 0C should be avoided. To reach core temperature 35 0C, surface cooling is recommended.

Tehnik Proteksi Otak pada Pembedahan Non Neurosurgery (Radical Neck Dissection) dengan Premorbid Space Occupying Lesion (SOL) dan Infark Serebri Laksono, Buyung Hartiyo; Saleh, Siti Chasnak
Jurnal Neuroanestesi Indonesia Vol 3, No 3 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Insidensi kasus tumor dengan metastase otak berkisar antara 100.000 sampai 170.000 pertahun. Metastase otak bersifat multiple dengan 80% terletak pada hemis ferserebri. Pendesakan akibat lesi tersebut mengakibatkan gangguan neurologis dan peningkatan tekanan intrakranial (TIK). Seorang laki-laki, 62 tahun dengan tumor sub mandibula direncanakan radical neck dissection. Pada pasien didapatkan proses metastase pada serebri dan cerebropontine angle disertai infark serebri daerah pons dan otak tengah. Defisit neurologis berupa kelemahan ekstremitas kanan dan disartria. Preoperatif diberikan kortikosteroid untuk menurunkan edema perifokal. Tatalaksana anestesi dengan prinsip tehnik proteksi otak, dilakukan induksi kombinasi dengan midazolam, fentanyl, lidokain, propofol dan rocuronium. Kontrol ventilasi target paCO2 3035 mmHg. Pemeliharaan anestesi dengan kombinasi sevofluran dan propofol. Pembedahan berjalan 7 jam, temperature selama pembedahan 3536 C dan MAP dijaga 70 mmHg. Dilakukan ekstubasi, setelah menilai status neurologis dan hemodinamik, difasilitasi dengan lidokain. Pascabedah tidak didapatkan perburukan defisit neurologis. Pasien dirawat di ICU selama 2 hari kemudian ke ruangan dengan perbaikan status neurologis. Tehnik proteksi otak bertujuan mencegah cedera sekunder dari SOL dan iskemia. Tindakan anestesi dan pembedahan dapat menambah perburukan cedera sekunder. Penatalaksanaan anestesi yang baik dengan prinsip proteksi otak akan menghasilkan outcome pembedahan sesuai yang diharapkan. Brain Protection Technique in Non Neurosurgical Procedure (Radical Neck Dissection) on a Patient with Space Occupying Lession (SOL) and Cerebral InfarctionThe incidence of tumors with brain metastases ranged from 100,000 to 170,000 per year. Brain metastases are multiple with 80% of lesion located on the cerebral hemispheres. These lesions could cause neurological disorders and increase intracranial pressure (ICP). A 62 years old male, diagnosed with sub mandibular tumour was scheduled for radical neck dissection. From preoperative evaluation he hadcerebral metastasis at the cerebrum and cerebro-pontine angle with cerebral infarction at pons and middle brain regions. Neurological deficits were weakness of the right limband dysarthria. The patient received corticosteroids pre-operatively to reduce perifocal edema. Anesthesia management was given using brain protection principles. Induction was done by using midazolam, fentanyl, lidocaine, propofol and rocuronium. Ventilation was controlled with a target PaCO2 of 3035 mmHg. Sevoflurane and propofol was given as anesthesia maintenance. Surgery was done for 7 hrs, temperature was 3536 C during surgery and MAP was maintained 70 mmHg. Extubation was done after assessing the neurologic and hemodynamic status,facilitated with lidocaine. There was no worsening of neurologic deficits post surgery. Patients was managed in the ICU for 2 days and transferred to ward with increased neurological state. The technique of brain protection aims to prevent further process of secondary injury from SOL and ischemia. Anesthesia and surgery itself could increase the progression of secondary injury. Anesthesia management usingbrain protection principles will provide better outcomes as expected.

Disseminated Intravascular Coagulation pada Cedera Otak Traumatik Suyasa, Agus Baratha; Sudadi, Sudadi; Suryono, Bambang
Jurnal Neuroanestesi Indonesia Vol 3, No 3 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Disseminated Intravascular Coagulation (DIC) merupakan konsekuensi yang sering dan penting pada cedera otak traumatik (Traumatic Brain Injury/TBI) dan menyebabkan cedera otak sekunder. Walaupun perkembangan proses ini belum dapat dijelaskan secara keseluruhan, namun abnormalitas koagulasi darah adalah bukti yang ditemukan pascatrauma. DIC adalah proses patofisiologi dan bukan merupakan suatu penyakit tersendiri. Gangguan yang terjadi meliputi ketidaktepatan, berlebihan dan aktivasi proses hemostasis yang tidak terkontrol. Karakteristik DIC adalah konsumsi faktor pembekuan darah dan trombosit dalam sirkulasi yang menimbulkan berbagai derajat obstruksi pembuluh darah mikro sehubungan dengan deposisi fibrin. Masalah dan gambaran utama akut DIC adalah perdarahan. Gangguan mekanisme hemostatik sangat penting dalam TBI. Perdarahan mikro sering terjadi di parenkim otak dan status koagulasi normal adalah penting untuk mencegah perkembangannya menjadi hematom yang lebih besar. Abnormalitas koagulasi tidak hanya hasil dari cedera, tetapi juga menyebabkan cedera sekunder. Gangguan koagulasi dalam TBI sangat kompleks dan dapat disertai dengan koagulopati dan hiperkoagulabilitas. Di temukan bukti bahwa luasnya trauma jaringan otak memiliki peran penting terhadap gangguan koagulasi dibandingkan syok traumatik maupun hipoksia. Adanya koagulopati pada TBI mengindikasikan prognosis yang buruk, sehingga pemeriksaan rutin terhadap status koagulasi harus selalu dilakukan pada semua pasien TBI. Disseminated Intravascular Coagulation on Traumatic Brain InjuryDisseminated Intravascular Coagulation (DIC) is a frequent and important consequence of traumatic brain injury and may cause secondary brain injury. Although the mechanism of this process cannot be explained as a whole, but abnormalities of blood coagulation after trauma is the evidence. DIC in brain trauma is a pathophysiological process and is not due to a disease in itself. Disturbance includes inaccuracy, excessive and activation of uncontrolled hemostasis process. Characteristic of DIC is the consumption of blood clotting factors and platelets in the circulation that cause various degrees of micro vascular obstruction in conjunction with the deposition of fibrin. The main problem features of acute DIC are bleeding. Impaired hemostatic mechanism plays an important role in traumatic brain injury (TBI). Micro bleeding often occurs in the brain parenchyma and normal coagulation status is important to prevent its development into a larger hematoma. Coagulation abnormality is not the only discouraging factor of injury, but also lead to secondary injury. Coagulation disorders in TBI are very complex and can be accompanied by coagulopathy and hypercoagulability. Found evidence ofextented trauma in the brain tissue plays more important role to coagulation disorder than traumatic shock and hypoxia. The presence of coagulopathy in TBI indicates a poor prognosis, so the routine inspection of the coagulation status should always be performed in all patients with TBI.

Anestesi untuk Malformasi Arnold Chiari Arianto, Ardana Tri; Sudjito, M.H
Jurnal Neuroanestesi Indonesia Vol 3, No 3 (2014)
Publisher : https://snacc.org/wp-content/uploads/2019/fall/Intl-news3.html

Malformasi Arnold-Chiari, merupakan suatu bentuk malformasi pada otak. Pada malformasi ini terjadi pergeseran (displasi) tonsila serebelum ke arah bawah melalui foramen magnum (lubang di basis kranii), yang terkadang menyebabkan hidrosefalus non-komunikans sebagai akibat terjadinya obstruksi aliran keluar dari cairan serebrospinal. Seorang wanita 23 tahun datang dengan keluhan sering pusing, nyeri tengkuk, serta kelemahan pada lengan kanan. CT Scan dan MRI didapatkan gambaran cerebellar tonsil yang mendukung Arnold Chiari Malformation. Dilakukan operasi osteotomi suboccipital dengan posisi prone. Rumatan anestesi dengan sevoflurane 1 vol% dan O2: udara 1,5: 1,5, analgetik fentanyl 25 mcg tiap 30 menit, pelumpuh otot vecuronium 3 mg/jam. Operasi berlangsung selama 2 jam 45 menit. Hemodinamik selama operasi stabil. Dilakukan ekstubasi segera di kamar operasi. Pascaoperasi pasien dirawat di unit intensif selama sehari. Hemodinamik selama di ICU stabil. Tidak ada keluhan selama di ICUAnesthesia for Arnold Chiari MalformationArnold-Chiari's malformation, is a brain malformation caused by the displacement of the cerebellar tonsil caudally into the foramen magnum, which in some cases will cause obstruction of the cerebrospinal fluid flow, resulting in a communicating hydrocephalus condition. A 23 years old female patient with a chief complaint of having frequent dizzines, painful neck, and weakness of the right arm. CT scan and MRI reveal cerebellar tonsil imaging that support the diagnosis of Arnold-Chiari's malformation. Surgical procedure was performed using suboccipital osteotomy approach in a prone position. Maintenance anesthesia with sevoflurane 1 vol% and O2: air 1,5: 1,5, analgetic fentanyl 25 mcg every 30 minute, muscle relaxant vecuronium 3 mg/hour. The time of surgery was 2 hours and 45 minutes. Hemodynamics were stable during the procedure. Patient was extubated early after surgery at operating room, and admitted to the ICU for 24 hours. Hemodynamics parameter were stable, without any remarkable events.

Page 1 of 1 | Total Record : 8

Filter by Year

2014 2014

Filter By Issues

All Issue Vol 14, No 3 (2025) Vol 14, No 2 (2025) Vol 14, No 1 (2025) Vol 13, No 3 (2024) Vol 13, No 2 (2024) Vol 13, No 1 (2024) Vol 12, No 3 (2023) Vol 12, No 2 (2023) Vol 12, No 1 (2023) Vol 11, No 3 (2022) Vol 11, No 2 (2022) Vol 11, No 1 (2022) Vol 10, No 3 (2021) Vol 10, No 2 (2021) Vol 10, No 1 (2021) Vol 9, No 3 (2020) Vol 9, No 2 (2020) Vol 9, No 1 (2020) Vol 8, No 3 (2019) Vol 8, No 2 (2019) Vol 8, No 1 (2019) Vol 7, No 3 (2018) Vol 7, No 2 (2018) Vol 7, No 1 (2018) Vol 6, No 3 (2017) Vol 6, No 2 (2017) Vol 6, No 1 (2017) Vol 5, No 3 (2016) Vol 5, No 2 (2016) Vol 5, No 1 (2016) Vol 4, No 3 (2015) Vol 4, No 2 (2015) Vol 4, No 1 (2015) Vol 3, No 3 (2014) Vol 3, No 2 (2014) Vol 3, No 1 (2014) Vol 2, No 3 (2013) Vol 2, No 2 (2013) Vol 2, No 1 (2013) Vol 1, No 4 (2012) Vol 1, No 3 (2012) Vol 1, No 2 (2012) Vol 1, No 1 (2012) More Issue

Home Page

OAI Link

Editorial Team

Contact

Reviewer

Google Scholar

Contact Name
Agni Susanti

Contact Email
jurnalneuroanestesi@gmail.com

Phone
+6287722631615

Journal Mail Official
jni@inasnacc.org

Editorial Address
Jl. Prof. Eijkman No. 38 Bandung 40161, Indonesia Lt 4 Ruang JNI

Location
,

INDONESIA

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Home Page

OAI Link

Editorial Team

Contact

Reviewer

Google Scholar

Contact Name Agni Susanti

Contact Email jurnalneuroanestesi@gmail.com

Phone +6287722631615

Journal Mail Official jni@inasnacc.org

Editorial Address Jl. Prof. Eijkman No. 38 Bandung 40161, Indonesia Lt 4 Ruang JNI

Location , INDONESIA

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Abstract

Contact Name
Agni Susanti

Contact Email
jurnalneuroanestesi@gmail.com

Phone
+6287722631615

Journal Mail Official
jni@inasnacc.org

Editorial Address
Jl. Prof. Eijkman No. 38 Bandung 40161, Indonesia Lt 4 Ruang JNI

Location
,

INDONESIA