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Sukri Rahman
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Departemen THT-KL FK Universitas Andalas/ RSUP. Dr. M. Djamil Padang Lt.3 Gedung Administrasi dan Poliklinik RSUP. Dr. M. Djamil Jl. P. Kemerdekaan Padang, 25127
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INDONESIA
Jurnal Otorinolaringologi Kepala dan Leher Indonesia (JOKLI)
Published by Universitas Andalas
ISSN : -     EISSN : 29617480     DOI : https://doi.org/10.25077/jokli
Core Subject : Health,
Medicine & Pharmacology
Jurnal Otorinolaringologi Kepala dan Leher Indonesia (JOKLI), The Indonesian Journal of Otorhinolaryngology Head and Neck, merupakan Jurnal Ilmiah di bidang Telinga Hidung Tenggorok Kepala dan Leher yang melalui peer review yang menerbitkan artikel ilmiah dalam bentuk Penelitian, Tinjauan Pustaka dan Laporan Kasus dalam bahasa Indonesia dan Bahasa Inggris
Arjuna Subject : Kedokteran - Otorhinolaryngology
Articles 55 Documents
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Quality of Life Profile of NPC Patients at ENT-HNS Department of RSUP Dr. M. Djamil Padang Iffah Nisrina Reyatri; Sukri Rahman; Lili Irawati
Jurnal Otorinolaringologi Kepala dan Leher Indonesia Vol. 5 No. 1 (2026): June 2026
Publisher : Fakultas Kedokteran Universitas Andalas

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.25077/jokli.v5i1.106

Abstract

Quality of Life Profile of NPC Patients at ENT-HNS Department of RSUP Dr. M. Djamil Padang Background: Nasopharyngeal carcinoma (NPC) and its treatment modalities, such as chemotherapy and radiotherapy, can impact various aspects of life, thereby affecting the patient's quality of life (QoL). This study aims to determine the QoL profile of NPC patients at the ENT-HNS Department of Dr. M. Djamil General Hospital, Padang. Methods: The study design used a quantitative descriptive approach. Primary data collection was conducted subjectively using questionnaires from the European Organization for Research and Treatment of Cancer (EORTC): the general QoL instrument (QLQ-C30) and the head and neck cancer-specific module (QLQH&N43), administered through interviews with NPC patients. Results: The majority of patients were male (67.4%), aged 25–64 years (89.1%), and at an advanced stage (80.4%). The majority had undergone or were undergoing concurrent chemoradiotherapy (43.5%). The worst QoL scores from the QLQ-C30 questionnaire were observed advanced-stage patients (QoL 63.96±16.44; functional 62.86±22.98; symptom 41.07±25.64) and patients before therapy (QoL 59.37±17.50; functional 58.29±62.85; symptoms 51.47±18.61). The worst QoL scores from the QLQ-H&N43 questionnaire were found in advanced-stage patients (multi-item 37.06±20.35; single item 36.42±24.18) and patients who had undergone and/or were undergoing concurrent chemoradiotherapy (multi-item 41.80±16.60; single item 39.76±22.08). Conclusion: Disease stage and treatment status significantly influencing QoL in NPC patients. General QoL was lower among advanced-stage and patients before therapy, whereas head and neck-specific complaints were more severe in advancedstage patients and those who had completed or were undergoing therapy.
Diagnosis and Management Options for Achalasia Fajar Dirgantara Edward; Ade Asyari
Jurnal Otorinolaringologi Kepala dan Leher Indonesia Vol. 5 No. 1 (2026): June 2026
Publisher : Fakultas Kedokteran Universitas Andalas

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.25077/jokli.v5i1.107

Abstract

Diagnosis and Management Options for Achalasia Background: Achalasia is a rare esophageal motility disorder characterized by increased lower esophageal sphincter tone and loss of esophageal peristalsis. The main symptoms include difficulty swallowing, vomiting, chest pain, weight loss, heartburn, and respiratory complications due to regurgitation. Objective: To know and understand the diagnosis and management options for achalasia.  Literature Review: All treatment methods aim to reduce outflow resistance and improve dysphagia. It is widely recognized that medical management without endoscopic or surgical intervention plays a minor role in the treatment of achalasia. Standard endoscopic methods include pneumatic dilation, botulinum toxin injection, and, more recently, the increasing availability and acceptance of peroral endoscopic myotomy. Time-tested surgical therapies include Heller myotomy with or without anti-reflux procedures, as well as esophagectomy for advanced-stage achalasia or failure of other surgical methods. Conclusion: Achalasia is a chronic and progressive esophageal motility disorder characterized by failure of lower esophageal sphincter relaxation and loss of esophageal peristalsis. Available management methods include pharmacological and non-pharmacological therapy or surgery, with each intervention selected based on indications and carrying its own side effects and complications.
Diagnosis and Management of Superior Canal Dehiscence Syndrome Rio Mulya Riharta; Rossy Rosalinda; Muhammad Dilga Caesario
Jurnal Otorinolaringologi Kepala dan Leher Indonesia Vol. 5 No. 1 (2026): June 2026
Publisher : Fakultas Kedokteran Universitas Andalas

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.25077/jokli.v5i1.108

Abstract

Diagnosis and Management of Superior Canal Dehiscence Syndrome Background: Superior Canal Dehiscence Syndrome (SCDS) is a vestibulocochlear disorder caused by a bony defect in the superior semicircular canal, creating a pathological third window in the labyrinth. This condition produces a variety of vestibular and auditory manifestations that significantly impair quality of life. Objective: To know and understand the diagnosis and management of superior canal dehiscence syndrome. Literature Review: The etiology of SCDS may be congenital or acquired. The primary pathophysiology relates to the third window mechanism that disrupts acoustic energy transmission and generates abnormal pressure pathways within the labyrinth. Characteristic vestibular symptoms include the Tullio phenomenon, Hennebert sign, oscillopsia, and disequilibrium, while cochlear symptoms encompass autophony, bone-conduction hyperacusis, pulsatile tinnitus, and low-frequency conductive hearing loss. Diagnosis is established based on the Barany Society triadic criteria: clinical symptoms, neurophysiological testing (VEMP, audiometry), and High-Resolution Computed Tomography (HRCT) imaging with multiplanar reconstruction. Conservative management is indicated for mild cases, while surgical treatment, via the middle cranial fossa (MCF) or transmastoid (TM) approach, is recommended for patients with severe, debilitating symptoms. Conclusion: Superior canal dehiscence syndrome (SCDS) is a third mobile window disorder caused by a bony defect of the superior semicircular canal, resulting in vestibular and auditory symptoms. Diagnosis is established through an integrative approach based on the Bárány Society criteria, incorporating clinical correlation, VEMP and/or audiometry, and high-resolution CT imaging. Management is stepwise, with surgical intervention (plugging or resurfacing) serving as the definitive treatment in severely symptomatic cases to eliminate the pathological pathway and improve clinical outcomes.
Diagnosis and Management of Autoimmune Inner Ear Disease Muhammad Arifudin; Rossy Rosalinda
Jurnal Otorinolaringologi Kepala dan Leher Indonesia Vol. 5 No. 1 (2026): June 2026
Publisher : Fakultas Kedokteran Universitas Andalas

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.25077/jokli.v5i1.109

Abstract

Diagnosis and Management of Autoimmune Inner Ear Disease Background: Autoimmune Inner Ear Disease is an autoimmune disease of sensorineural hearing loss, with an incidence of less than 1% worldwide. Symptoms of Autoimmune Inner Ear Disease often resemble the symptoms of other types of sensorineural hearing loss, making the diagnosis difficult. Objective: To understand all aspects of Autoimmune Inner Ear Disease. Literature Review: Autoimmune Inner Ear Disease is a rare autoimmune disease characterized by progressive and fluctuating sensorineural hearing loss. Autoimmune Inner Ear Disease is more common in women, aged between 20 and 50 years. Autoimmune Inner Ear Disease can be primary or secondary. The mechanism of Autoimmune Inner Ear Disease can be damage mediated by autoantibodies, immune complex deposition, T-cell inflammation. Typical symptoms and signs include bilateral sensorineural hearing loss, tinnitus, sometimes vestibular disorders and response to corticosteroid treatment. Complete blood count, erythrocyte sedimentation rate, antinuclear antibodies, and other immunological profiles can confirm the diagnosis. Magnetic resonance imaging was performed to rule out retrocochlear pathology and demyelination. High-dose corticosteroids are the first-line treatment. Conclusion: Autoimmune Inner Ear Disease is an autoimmune disease characterized by progressive sensorineural hearing loss, which may be accompanied by tinnitus and vestibular disorders. Corticosteroids are the first-line treatment.
Canal Wall Down Tympanomastoidectomy and Facial Nerve Decompression in Patients with Facial Nerve Palsy Caused By Cholesteatoma-Type Chronic Suppurative Otitis Media Leon Gaya; Jacky Munilson
Jurnal Otorinolaringologi Kepala dan Leher Indonesia Vol. 5 No. 1 (2026): June 2026
Publisher : Fakultas Kedokteran Universitas Andalas

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.25077/jokli.v5i1.110

Abstract

Canal Wall Down Tympanomastoidectomy and Facial Nerve Decompression in Patients with Facial Nerve Palsy Caused By Cholesteatoma-Type Chronic Suppurative Otitis Media Background: Facial nerve palsy is a neurological complication that can occur in patients with chronic suppurative otitis media (CSOM) of the cholesteatoma type. Cholesteatoma can damage the facial nerve through mechanisms such as compression, erosion of the facial nerve canal, and chronic inflammation. Management of peripheral facial nerve palsy caused by cholesteatoma-type CSOM can be effectively achieved through surgical intervention. Case Report: A 36-year-old male presented with a 10-year history of discharge from the right ear, who developed right-sided facial paralysis classified as House-Brackmann grade V, with a lesion level at the geniculate ganglion due to cholesteatoma-type CSOM. The patient underwent Canal Wall Down (CWD) tympanomastoidectomy, including cholesteatoma excision, facial nerve decompression, underlay graft placement, and reconstruction of the ear canal wall. Conclusion: Facial nerve palsy is a complication of cholesteatoma-type chronic suppurative otitis media (CSOM), which can be caused by direct or indirect compression of the facial nerve. Surgical intervention with Canal Wall Down (CWD) tympanomastoidectomy, including complete cholesteatoma excision and indirect facial nerve decompression, provides a good recovery of nerve function.

Page 6 of 6 | Total Record : 55

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