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Narra J
Published by PT Narra Sains Indonesia
ISSN : -     EISSN : 28072618     DOI : https://doi.org/10.52225/narraj
Core Subject : Health, Science,
Biochemistry, Genetics & Molecular Biology Health Professions Immunology & microbiology Medicine & Pharmacology Public Health
Narra J is a multidisciplinary journal and it is published three times (April, August, December) a year. The objective is to promote articles on infection, public health, global health, tropical infection, one health and diseases in tropics. Narra J publishes original research work across all disciplines of medicine and allied sciences, related to infection, public health, global health, tropical infection, one health and diseases in tropics. The journal publishes Original articles, Short Report, Review articles, and Letters to the Editor. All articles published in Narra J are peer-reviewed and published online for immediate access and citation. Narra J publishes the primary research papers, review articles, short communications and letters on topics but not limited to: Public health Global health Infection Tropical diseases One health Biomedical sciences Epidemiology and clinical epidemiology Molecular biology Environmental health Microbiology Pharmacological sciences Diseases in tropics
Arjuna Subject : Kedokteran - Kedokteran (Lain-Lain)
Articles 665 Documents
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Associations of self-efficacy, outcome expectancies, and socio-structural factors with teenage pregnancy in remote areas of Indonesia: An application of Social Cognitive Theory framework Liza Muknisa; Tgk. Puspa Dewi; Munizar Munizar; Niken A. Utami; Rusnaidi Rusnaidi; Rachmad Suhanda
Narra J Vol. 6 No. 3 (2026): December 2026 (In Press)
Publisher : Narra Sains Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.52225/narra.v6i3.3133

Abstract

Teenage pregnancy remains a major public health challenge in Indonesia, particularly in remote settings where sociocultural norms strongly influence adolescent behavior. In Gayo Lues Regency, Aceh Province, Indonesia, adolescent pregnancy continues to occur within a context of early marriage, limited reproductive health access, and strong community norms. The aim of this study was to examine the associations of self-efficacy, outcome expectancies, and socio-structural factors with teenage pregnancy within the framework of Social Cognitive Theory. A quantitative cross-sectional study was conducted among female adolescents aged 10 to 19 years registered across four community health centers (Puskesmas) in Gayo Lues. Participants were selected using cluster random sampling. Data were collected through structured face-to-face interviews using a validated questionnaire. The associations were analyzed using simple and multiple binary logistic regression. A history of teenage pregnancy was reported by 29.5% of participants. Bivariate analysis showed that self-efficacy, outcome expectancies, and socio-structural factors were significantly associated with the history of teenage pregnancy. In the multivariate model, higher outcome expectancies were most strongly associated with lower odds of teenage pregnancy history (odds ratio (OR): 0.424; p<0.001), followed by higher self-efficacy (OR: 0.628; p=0.027), whereas socio-structural factors were not independently associated (OR: 0.810; p=0.318). These findings suggest that personal cognitive factors play a stronger role than socio-structural factors in this setting. Interventions to prevent teenage pregnancy should prioritize strengthening adolescents’ self-efficacy and shaping positive outcome expectancies through comprehensive reproductive health education and supportive social environments.
Effects of intermittent fasting (time-restricted eating) for 30 days on body weight, visceral fat, and muscle mass in adult workers Ketus A. Prasetyo; Robiana Modjo; Eko Rizkianto; Desy Sulistiyorini
Narra J Vol. 6 No. 3 (2026): December 2026 (In Press)
Publisher : Narra Sains Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.52225/narra.v6i3.3120

Abstract

Obesity among workers is an important occupational health concern because of its association with cardiometabolic risk and productivity loss. Time-restricted eating (TRE) 12:12 is a mild intermittent fasting protocol that limits food intake to a 12-hour eating window and may be feasible for workplace implementation. This study aimed to evaluate the effect of a 30-day TRE 12:12 intervention on body weight and body composition among adult workers. A quasi-experimental one-group pretest–posttest study was conducted among adult workers in Semarang, Indonesia. Participants followed TRE 12:12 for 30 days without prescribed calorie restriction. Body composition was assessed before and after the intervention using bioelectrical impedance analysis. Changes in outcomes were analyzed using paired-samples t-test and Cohen’s d, while adherence and participants’ perceptions were summarized descriptively. After 30 days, body weight decreased by 2.38±1.97 kg, body mass index (BMI) by 0.90±0.70 kg/m², body fat by 1.08±1.23%, visceral fat level by 1.12±0.72, and waist circumference by 5.82±3.37 cm, with all changes statistically significant (p≤0.001). Skeletal muscle mass increased slightly by 0.36±0.55% (p=0.008), which was interpreted as muscle preservation rather than meaningful muscle gain. Overall adherence was high (98.1%), reported side effects were mild, and most participants perceived the intervention as beneficial. This study highlights that TRE 12:12 was associated with short-term improvements in body weight and body composition among adult workers, particularly reductions in visceral fat and waist circumference, while preserving skeletal muscle mass. Further studies with larger samples and longer follow-up are needed to confirm its effectiveness and sustainability in working populations.
Training community health volunteers on mosquito-repellent plants to support urban dengue prevention: A sequential explanatory mixed-methods study in Jakarta Kholis Ernawati; Rifda Wulansari; Dini Syafitri; Beben S. Bahri; Bina R. Permatasari
Narra J Vol. 6 No. 3 (2026): December 2026 (In Press)
Publisher : Narra Sains Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.52225/narra.v6i3.3135

Abstract

Dengue remains a major urban public health challenge in Jakarta, Indonesia, where community-level vector control often relies on reactive chemical-based approaches. Mosquito-repellent plants may serve as a practical educational medium to support environmentally oriented dengue prevention, but evidence on their use in community health volunteer training remains limited. This study evaluated mosquito-repellent plant training among community health volunteers in Jakarta using a sequential explanatory mixed-methods design. Knowledge was assessed before and after training, while qualitative interviews explored implementation readiness, barriers, support systems, and sustainability. Quantitative data were analyzed using the Wilcoxon signed-rank test, and qualitative data were analyzed thematically and integrated with the quantitative findings. Knowledge shifted toward higher categories after training, with the proportion of participants in the good knowledge category increasing from 26.7% to 56.7%. The median knowledge score increased from 60 to 80, and the improvement was statistically significant (Z=−2.103; p=0.036), with a moderate effect size (r=0.38). Qualitative findings indicated that volunteers perceived the training as practical and relevant to dengue prevention. However, implementation readiness was influenced by seedling availability, household participation, mentoring, and support from neighborhood leaders, village authorities, and primary health care centers. These findings suggest that mosquito-repellent plant training can improve volunteers’ knowledge and initial readiness to support community-based mosquito breeding site eradication and environmentally oriented dengue prevention, although sustained implementation requires continued mentoring, resource provision, and local institutional support.
Effects of combined epigallocatechin gallate and chlorogenic acid on metabolic parameters and pulmonary remodeling in a metabolic syndrome model: A preclinical study Gregorio S. Pinunggul; Susanthy Djajalaksana; Suryanti D. Pratiwi; Hendy S. Yudhanto
Narra J Vol. 6 No. 3 (2026): December 2026 (In Press)
Publisher : Narra Sains Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.52225/narra.v6i3.3138

Abstract

Metabolic syndrome (MetS) may contribute to pulmonary fibrotic remodeling through metabolic dysregulation, oxidative stress, and inflammatory signaling. Epigallocatechin gallate (EGCG) and chlorogenic acid (CGA) have antioxidant, anti-inflammatory, and metabolic regulatory properties, but their combined effects on systemic and pulmonary peroxisome proliferator-activated receptor gamma (PPARγ) levels and pulmonary fibrosis in MetS remain insufficiently characterized. The aim of this study was to evaluate the effects of combined EGCG and CGA administration on metabolic parameters, plasma and lung tissue PPARγ levels, and histological pulmonary fibrosis severity, as well as to examine the relationships between PPARγ levels and fibrosis severity in a rat model of MetS. This experimental study included 30 male Sprague-Dawley rats randomly allocated to healthy control, untreated MetS, and MetS treated with combined EGCG-CGA groups (n=10 each). MetS was induced using a high-fat, high-sucrose diet and intraperitoneal streptozotocin. Treated rats received EGCG 300 mg/kg/day and CGA 200 mg/kg/day by oral gavage for nine weeks. Metabolic parameters, plasma and lung tissue PPARγ levels, and pulmonary fibrosis severity assessed using the modified Ashcroft score were evaluated. Compared with controls, untreated MetS rats had higher body weight, fasting blood glucose, triglyceride levels, and Ashcroft scores, but lower high-density lipoprotein (HDL) cholesterol and plasma and lung PPARγ levels (all p<0.001). Compared with untreated MetS rats, EGCG-CGA-treated rats had a more favorable metabolic profile and lower Ashcroft scores (p=0.012). Plasma and lung PPARγ levels were higher in EGCG-CGA-treated rats than in untreated MetS rats (64.4±35.1 vs 17.0±4.9 ng/mL and 46.0±7.2 vs 27.9±4.9 ng/mg protein, respectively; both p<0.001). Plasma and lung PPARγ were positively correlated (r=0.805; p<0.001), whereas both were negatively correlated with Ashcroft score (r=-0.749 and r=-0.912, respectively; both p<0.001). In conclusion, combined EGCG-CGA was associated with improved metabolic profiles, increased systemic and pulmonary PPARγ levels, and lower histological pulmonary fibrosis severity in MetS rats.
Gut microbiome dysbiosis in traumatic brain injury: Implications for neurological recovery and microbiome-targeted interventions Jihan Ichsan; Aryandhito W. Nugroho
Narra J Vol. 6 No. 3 (2026): December 2026 (In Press)
Publisher : Narra Sains Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.52225/narra.v6i3.3141

Abstract

Traumatic brain injury (TBI) is a major cause of mortality and long-term neurological disability worldwide. In addition to primary brain damage, TBI induces complex secondary pathophysiological responses involving neuroinflammation, systemic immune dysregulation, gastrointestinal dysfunction, and alterations in the microbiota–gut–brain axis. Increasing attention has therefore been directed toward the role of gut microbiome dysbiosis in the progression of secondary brain injury and its potential as a therapeutic target. This narrative review aimed to synthesize current evidence regarding the relationship between TBI and disruption of the microbiota–gut–brain axis, with particular emphasis on the mechanisms underlying gut microbiome dysbiosis and its contribution to secondary brain injury. The review discusses neuroinflammatory responses, alterations in intestinal barrier integrity, enteric and autonomic nervous system involvement, vagal signaling, and microbiota-derived pathways implicated in brain–gut communication after TBI. In addition, the therapeutic rationale, available evidence, translational considerations, and current limitations of microbiome-targeted approaches, particularly probiotics and fecal microbiota transplantation, are critically discussed. This review provides an updated framework for understanding the microbiota–gut–brain axis in TBI and identifies areas requiring further mechanistic and clinical investigation.

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