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Journal of the Medical Sciences (Berkala Ilmu Kedokteran)
Published by Universitas Gadjah Mada
ISSN : 01261312     EISSN : 23563931     DOI : http://dx.doi.org/10.19106/JMedSci005303202101
Core Subject : Science,
Immunology & microbiology Neuroscience
Journal of the Medical Sciences (JMedSci) or Berkala Ilmu Kedokteran (BIK) is an international, open-access, and double-blind peer-reviewed journal, published by Faculty of Medicine, Public Health and Nursing, Universitas Gadjah Mada Yogyakarta Indonesia. JMedSci aiming to communicate high-quality articles in the areas of biomedical science from basic to clinical sciences.The journal welcomes papers from original articles, case reports, reviews, and book reviews. All papers published in JMedSci are freely available as downloadable pdf files. The journal began its publication on March 1973 and published quarterly (January, April, July, and October). JMedSci is abstracted and indexed in DOAJ, Crossref, Google Scholar, Sinta, Indonesia One Search. JMedSci is accredited by Directorate of General Higher Education, the Ministry of Research, Technology, and Higher Education, Indonesia
Arjuna Subject : Kedokteran - Kedokteran (Lain-Lain)
Articles 2,170 Documents
 80   81   82   83   84 
Formazan ring method: a simple test for screening of glucoce-6-phosphate dehydrogenase (G-6-PD) deficiency in the neonates Purnomo Suryantoro, Purnomo Suryantoro
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 28, No 02 (1996)
Publisher : Journal of the Medical Sciences (Berkala Ilmu Kedokteran)

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Abstract

Glucose-6-phosphate dehydrogenase deficiency, is an x-linked inherited disease which is responsible for episodic hemolytic anemia. The incidence correlates with the malarial epidemic in many area like Mediterranean, Africa and Asia including South East Asia.This paper reports a simple, cheap, traumatic, and reliable method screening test called formazan ring developed by Nishiyama. Among 145 new-born infants in this report the low enzyme activity was as high as 9(6.2%) Including 2(1.4%) G-6-PD deficiency.Key words: glucose-6-phosphate dehydrogenase deficiency - x-linked inherited disease - formazan ring test - neonatal screening
Effects of amodiaquine on Brugia pahangi infection in Aedes togoi Sugeng Juwono Mardihusodo., Sugeng Juwono Mardihusodo.
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 19, No 02 (1987)
Publisher : Universitas Gadjah Mada

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Abstract

A primary screening of amodiaquine as a filaricide using Brugia pahangi .Aedes togoi model infection was carried out. The drug dissolved in 10% sucrose water solution in the concentrations of 100 mg%, 200 mg% and 400 mg% fed ad libitum to the uninfected and infected mosquitoes for 4, 8 and 12 days showed definite insecticidal and filaricidal activities. Key words: primary filaricide screening - amodiaquine - Brugia pahangi - Aedes togoi model infection - filariasis - parasitology
Colour vision and the genetics of colour vision defect Hartono, Hartono
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 22, No 03 (1990)
Publisher : Journal of the Medical Sciences (Berkala Ilmu Kedokteran)

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Abstract

Colour vision is one of the visual functions which is very important in daily life. The phenomenon of normal colour vision has been explained briefly in order to understand colour vision defects.The theory of colour vision has been proposed for a long time by Young and Helmholtz. According to this theory there are three kinds of cones in the retina, namely red, green, and blue cones. Each cone has maximum absorbance for red, green, and blue colours respectively, but their absorbance curves overlap each other.The absence of the function of red, green, and blue cones will cause red blindness (pro tanopia) , green blindness (deuteroanopia), and blue blindness (tritanopia) respectively. On the other hand, partial disturbances of red, green, and blue cones give rise to red weakness (protanomaly), green weakness (dcuteroanomaly), and blue weakness (tritoanomaly).The great majority of colour defects are hereditary or genetic diseases. Congenital red and green blindness are X-linked recessive diseases. The location of red and green colour vision genes and their alleles (genes for red and green colour vision defects) are in the end of the long arm of chromosome X. The two genes arc very close to each other. Congenital blue colour defect, on the other hand, is an autosomal recessive disease, and the location of the gene is at the end of the long arm of chromosome 7.Key Words: ophthalmology - colour vision defects - X-linked recessive diseases - autosomal recessive diseases - gene locations
Teknik dermatoglyphi Yang diterapkan dalam Kedokteran Adi Soekarto, Adi Soekarto
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 10, No 03 (1978)
Publisher : Journal of the Medical Sciences (Berkala Ilmu Kedokteran)

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Abstract

Keyword : Teknik dermatoglyphi
Correlation between plasma zinc (Zn) level and nutritional status of children under five years Dedy Koesnadi, Dedy Koesnadi
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 27, No 02 (1995)
Publisher : Journal of the Medical Sciences (Berkala Ilmu Kedokteran)

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Abstract

The aim of this study is to determine Zn plasma level of under five year old children (age 12-48 months) in various nutritional status according to NCEIS - WHO (1983).There were 130 under-five-year old children involved in this study. The subjects were recruited from 10 Posyandu. Clinical data such as general health, weight/age (W/A), height/age (H/A), and clinical history as well as food behaviour were collected from Posyandu. Blood sampling for determination of Zn plasma level, total protein, albumin, hemoglobin, leucocyte count, hematocrit, IgM and IgG level were collected at Puskesmas.The food consumption consisted of 16 different kind of vegetables, 10 from animals, and 3 from fruits. The interviews were done by nutritionists from Dr. Sardjito General Hospital, Yogyakarta. Zn plasma level determination was done by Atomic Absorption Spectrophotometer (AAS), Hitachi polarized. Blood examination was done at the Department of Clinical Pathology, Faculty of Medicine, Gadjah Mada University/Dr. Sardjito General Hospital, Yogyakarta.The results of the study showed that the nutritional status of the children, age 12-48 months was as follows: According to W/A nutrient index 60.3% good, 35.6% moderate, 4.1% poor, and none was bad. According to H/A nutrient index 73.0% good, 19.1% moderate, 6.4% poor, and 1.5% bad. By w/h nutrient index 71.9% good, and 28,1% poor. According to Percentile Index those were lower than Percentile 3 are W/A 41.3%, H/A 57.7%, and W/H 15% between (3-5) percentile W/A 14.2%, H/A 7.1%, and W/H 12.9%, between (5-10) percentile W/A 19.0%, H/A 7.1%, and W/H 12.9%.The mean Zn plasma level was 43.4 (SD=30.4) mcg/dL, mean Zn consumption per day was 5.2 mg. According to the health indicator 1989 under-five-year old children with good nutrient status were 53.9%. By this study according to W/A index, 60.3% was good, H/A index, 73.0%, and by W/H index, 71.9% was good.There was an increasing correlation of coefficient, r in fail nutritional status categories (r=0.134) according to W/A, r = 0.197, according to H/A, and r = 1.000 according to H/W index in the poor category, but no correlation at all between good and moderate nutritional status categories.Key words : plasma zinc level - nutritional status category - children under five year - Zn dietary consumption.
Sebab Kematian Bayi Prematur Ditinjau dari Hasil Biopsi Post-Mortem Organ Tubuh Soeripto, Soeripto
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 17, No 01 (1985)
Publisher : Universitas Gadjah Mada

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Abstract

The mortality of the premature babies in Indonesia is still high. The exact cause of death is still unknown, because necropsy and pathological examination of the whole placenta are very rarely carried out. Needle biopsy of the dead premature babies organ is expected to help in searching the cause of death. A retrospective study was performed to reevaluate the biopsied organs of the dead prema tures from October 1970 through September 1971 in the Department of Pathology, Gadjah Mada University. Yogyakarta. The result of this study shows that early neonatal death of premature babies is 80,6% of all premature death. Based on histopathological examination of lung biopsies of those dead premature babies, it is clear that atelectasis is the primary cause, congestion the second, and Hyaline Membrane Disease (HMD) the third. Biopsies of other organs could not determine the exact cause of premature deaths as yet. Key Words: premature baby - early neonatal death - needle biopsy - atelectasis - Hyaline Membrane Disease
Mucosal IgA response in the colon against pneinnococcal polysaccharide antigen Marsetyawan HNE Soesatyo, Marsetyawan HNE Soesatyo
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 26, No 02 (1994)
Publisher : Journal of the Medical Sciences (Berkala Ilmu Kedokteran)

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Abstract

The humoral IgA response to bacterial polysaccharide antigen in rats was investigated in vivo. Rats were immunized with pneumococcal polysaccharide type-3 (PPS-3) via different routes, i.e. in the Peyers patch (iPP), in the colon (I.C.), peritoneal cavity (LP.), and intravenous (I.V.). The development of specific antibody-forming cells (AFC) and their isotypes in the intestinal mucosa, gut-associated lymphoid tissue. (GALT), mesenteric lymph nodes (MLN) and spleen were studied by immunohistochemistry. The serum antibody levels were measured by enzyme-linked " lll unosorbent assay (ELISA).The results showed that iPP immunization evoked high numbers of anti-PPS-3 AFC of the IgA isotype in the mucosa of the small intestine and in the Peyers patch. On the contrary, the I.C. route did not elicit a mucosal response, though a few AFC were found in the MLN and spleen. Following I.P. priming, a specific IgA response was found, especially in the MLN and spleen, and low response was detected in the villi. A high response was found in the parathymic lymph nodes (PTLN). The I.V. immunization gave rise to the development of AFC in the spleen. particularly of the 1gM isotype. There was no mucosal responses to PPS-3 antigen in the colon arise irrespective of the route of immunization.Key-words : IgA-colon-antibody-forming cells- pneumococcal polysaccharide antigen -immunohistochemistry
Genetic heterogeneity of retinitis pigmentosa Hartono, Hartono
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 21, No 04 (1989)
Publisher : Journal of the Medical Sciences (Berkala Ilmu Kedokteran)

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Abstract

Genetic heterogeneity is a phenomenon in which a genetic disease can be transmitted by several modes of inheritance. The understanding of genetic heterogeneity is important in giving genetic counselling.The presence of genetic heterogeneity can be explained by the existence of:1.different mutant alleles at a single locus, and2.mutant alleles at different loci affecting the same enzyme or protein, or affecting different enzymes or proteins.To have an overall understanding of genetic heterogeneity, the heterogeneities of chronic hemolytic anemia caused by beta-globin abnormality, chronic hemolytic anemia due to thalassemias, and mucopolysaccharidoses have been chosen as examples.The article is focused on the genetic heterogeneity of retinitis pigmentosa, a kind of retinal hereditary disease. The genetic heterogeneity of retinitis pigmentosa can be known by the facts thata.Retinitis pigmentosa can be either an isolated disease or a part of a certain syndrome.b.Retinitis pigmentosa as isolated disease can be genetic or non-genetic in nature.c. Retinitis pigmentosa as a genetic disease can be transmitted either by autosomal dominant, autosomal recessive, or X-linked recessive genes.The frequency of isolated hereditary retinitis pigmentosa varies between 30% to 50%. Clinically the dominant form is milder than the recessive form. Certain syndromes associated with retinitis pigmentosa which need to be mentioned are Usher syndrome, Laurence-Moon-Bardet-Biedl syndrome, and Kearn-Sayre syndrome.Key Words: genetic heterogeneity - retinitis pigmentosa - genetic disease - hemolytic anemia - Usher syndrome
Study of medical report on justice examination result at Dr. Sardjito Hospital Beta Ahlam Gizela, Beta Ahlam Gizela
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 36, No 2 (2004)
Publisher : Journal of the Medical Sciences (Berkala Ilmu Kedokteran)

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Abstract

Background: Every human violation or crime case, in which there is human as a victim needs medical proofing, namely the Medical Report for justice or Visum et Repertum in Indonesia. We also have an obligation in keeping medical confidentiality in medical practice. It is therefore, necessary to study relevance regulation of examination and disclosing medical confidentiality for justice.Objectives: The purpose of this study is to analyse management of Visum et Repertum of life person at Sardjito Hospital Yogyakarta.Methods: Visum et Repertum of life person at Medical Record Department of Sardjito Hospital was reviewed. The MEG protocol for examination and reporting from International Amnesty used as a standard. Results: The study shows that management of Visum et Repertum of life person at Sardjito Hospital was administratively inappropiate. There were some discrepancies between regulation and practice. There is no agreement between each doctors in managing cases. Generally, Visum et Repertum was accepted in content, but for the future an improvement is needed as International Amnesty.Conclusions: Management of life person cases for Visum et Repertum at Sardjito Hospital do not international standard.Key words: visum et repertum - living person - medical confidentiality - informed consent
Nonspecific Urethritis(NSU) or Non-Gonococcal Urethritis(NGU),The Biggest Problem In Sexually Transmitted Diseases(STD) A Literature Study Soedarmadi, Soedarmadi
Journal of the Medical Sciences (Berkala Ilmu Kedokteran) Vol 7, No 04 (1975)
Publisher : Universitas Gadjah Mada

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Abstract

Not adequately realized by the practicing physician is that at least half of the cases of urethritis seen in practice are not due to gonococcus and will not respond to penicillin, but will respond to tetracyclin.The etiology of NSU is not clear yet, but the role of infective agent (or agents) is clear. The diagnosis is made by exclusion.Despite the lack of agreement concerning the aetiology, the treatment is relavely simple, and tetracycline is the drug of choice.

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