Articles
<![CDATA[Masalah Perilaku pada Anak Sindroma Nefrotik ]]>
<![CDATA[Barus, Ratna Sari]]>;
<![CDATA[Ramayani, Oke Rina]]>;
<![CDATA[Siregar,, Rosmayanti]]>;
<![CDATA[Siregar, Beatrix]]>
<![CDATA[ Majalah Kedokteran Nusantara The Journal Of Medical School Vol 50, No 3 (2017): The Journal of Medical School ]]>
Publisher : <![CDATA[Fakultas Kedokteran USU ]]>
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<![CDATA[Nephrotic syndrome (NS) is the most common chronic kidney disease in children.As one of chronic diseases having remission and relapse periods and requiring longterm corticosteroid theraphy, NS has impacts on childrenâs biologic, social, and behavior functions which will affect psychosocial developmen of the children and their families. Untill now there were difficulties in determining behavioural problem in children with NS and to determine whether it was caused by the disease itself or by chronic condition or by the side effect of steroid theraphy. High-dose steroid usage, mainly in the early treatment phase of NS is proven to correlate with internalization behavioural problem, manifesting with anxiety, depression, aggressiveness, and retraction from social life.It is suggested to pay more attention to psychosocial problems in children with NS especially in the early phase of steroid theraphy.Keywords : nephritic syndrome, children, behavioral problem, steroidTinjauan Pustaka]]>
<![CDATA[ACTIVATED PARTIAL THROMBOPLASTIN TIME AND FIBRINOGEN IN PEDIATRIC NEPHROTIC SYNDROME DURING RELAPSE AND REMISSION ]]>
<![CDATA[Tarigan, Trianita]]>;
<![CDATA[Aman, Adi Koesoema]]>;
<![CDATA[Ramayani, Oke Rina]]>
<![CDATA[ INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 24, No 3 (2018) ]]>
Publisher : <![CDATA[Indonesian Association of Clinical Pathologist and Medical laboratory ]]>
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DOI: 10.24293/ijcpml.v24i3.1410
<![CDATA[ Nephrotic Syndrome (NS) is a complicated kidney disease disorder, one of the most important complications is thromboembolism which can affect the circulation, either arterial or venous in both pediatric and adult patients. Patients at risk of thromboembolism should have an angiography examination for diagnosis. There have been several studies conducted on patients with a nephrotic syndrome showing the risk of thromboembolism. This study included twelve patient of pediatric nephrotic syndrome consisting of males and females. The patient experiences a period of relapse and became a remission. Patients participating in the study were 3 to 17 years old. There were significant differences in fibrinogen in which the fibrinogen content of NS patients in children at relapse was higher compared with the time of remission (390.08 ± 164.87 vs. 273.17 ± 150.56; p=0.042). There was no significant difference in Activated Partial Thromboplastin Time (APTT) results in SN patients in relapse compared to remission (34.17 ± 5.65 vs. 30.08 ± 8.49; p=0.236). The high levels of fibrinogen in the relapse period indicate the presence of hypercoagulable state, along with other examinations such as high cholesterol and low albumin. In this study, there was no significant difference in APTT among SN patients during relapse compared with remission while in the fibrinogen examination a significant difference was found. Therefore, fibrinogen examination is important to be analyzed in order to avoid SN complications. ]]>
<![CDATA[Peningkatan Laju Endap Darah sebagai Skrining Trombosis Pasien Sindrom Nefrotik ]]>
<![CDATA[Zuriana, Dini]]>;
<![CDATA[Ramayani, Oke Rina]]>
<![CDATA[ Cermin Dunia Kedokteran Vol 45, No 10 (2018): Muskuloskeletal ]]>
Publisher : <![CDATA[PT. Kalbe Farma Tbk. ]]>
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DOI: 10.55175/cdk.v45i10.590
<![CDATA[Sindrom Nefrotik (SN) merupakan penyakit ginjal anak yang paling sering ditemukan. Trombosis merupakan komplikasi serius SN; merupakan hasil koagulasi intravaskular darah menuju bentuk trombus yang menyumbat aliran darah. Diagnosis trombosis sulit dan sering terlewatkan karena banyak pasien asimptomatik. Pemeriksaan laju endap darah diharapkan menjadi skrining diagnosis trombosis.Nephrotic syndrome (SN) is the most common kidney disease in children. Thrombosis is a serious complication in patients with SN. Diagnosis of thrombosis is difficult beacuse many patients are asymptomatic. Erythrocyte sedimentation rate examination is expected to be a diagnositic screening for thrombosis.]]>
<![CDATA[Hubungan Disomnia dan Tekanan Darah pada Remaja ]]>
<![CDATA[Sembiring, Krisnarta]]>;
<![CDATA[Ramayani, Oke Rina]]>;
<![CDATA[Lubis, Munar]]>;
<![CDATA[Siregar, Rosmayanti]]>;
<![CDATA[Siregar, Beatrix]]>
<![CDATA[ Cermin Dunia Kedokteran Vol 44, No 12 (2017): Neurologi ]]>
Publisher : <![CDATA[PT. Kalbe Farma Tbk. ]]>
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DOI: 10.55175/cdk.v44i12.690
<![CDATA[Disomnia merupakan gangguan tidur yang sering dijumpai pada remaja, disebabkan oleh faktor internal dan eksternal, serta faktor medis dan nonmedis. Disomnia dapat berdampak buruk pada kesehatan remaja. Disomnia dapat didiagnosis secara objektif maupun subjektif. Salah satu komplikasi disomnia adalah peningkatan tekanan darah. Peningkatan tekanan darah pada remaja akan menyebabkan hipertensi saat dewasa serta berbagai masalah kardiovaskuler lainnya.Dyssomnia is a common sleep disturbance in adolescents; it is caused by internal and external factors along with medical and nonmedical factors. Dyssomnia may have negative impact on adolescent’s health. One of it’s complications is increased blood pressure. Increased blood pressure in adolescent will lead to hypertension in adult together with other cardiovascular problems.]]>
<![CDATA[Peningkatan Tekanan Intraokular pada Sindrom Nefrotik ]]>
<![CDATA[Sinaga, Harida Panduwita]]>;
<![CDATA[-, Rusdidjas]]>;
<![CDATA[Ramayati, Rafita]]>;
<![CDATA[Rina Ramayani, Oke]]>;
<![CDATA[Siregar, Rosmayanti]]>;
<![CDATA[Siregar, Beatrix]]>
<![CDATA[ Cermin Dunia Kedokteran Vol 45, No 8 (2018): Alopesia ]]>
Publisher : <![CDATA[PT. Kalbe Farma Tbk. ]]>
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DOI: 10.55175/cdk.v45i8.626
<![CDATA[Sindrom nefrotik (SN) merupakan penyakit ginjal terbanyak pada anak. Tatalaksana medikamantosa utama SN adalah menggunakan kortikosteroid; penggunaan dosis tinggi dan jangka lama dapat memiliki efek samping, salah satunya peningkatan tekanan intraokular. Mekanisme pasti peningkatan tekanan intraokular diinduksi steroid belum diketahui pasti, steroid dapat meningkatkan resistensi pengeluaran aqueous humor.Nephrotic syndrome (NS) is the most common kidney disease in children. Corticosteroid is still preferred as primary treatment. High-dose and long term use of corticosteroids can increase intraocular pressure. The mechanism of steroid-induced increased intraocular pressure is not clearly understood; steroids can increase aqueous humor outflow resistance.]]>
<![CDATA[Hubungan ASI Eksklusif dan Enuresis Primer pada Anak ]]>
<![CDATA[Hasibuan, Syahreza]]>;
<![CDATA[Ramayani, Oke Rina]]>;
<![CDATA[Lubis, Munar]]>;
<![CDATA[Siregar, Rosmayanti]]>;
<![CDATA[Siregar, Beatrix]]>
<![CDATA[ Cermin Dunia Kedokteran Vol 47, No 8 (2020): Kardiologi ]]>
Publisher : <![CDATA[PT. Kalbe Farma Tbk. ]]>
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DOI: 10.55175/cdk.v47i8.782
<![CDATA[Enuresis adalah keluarnya air kemih yang tidak disadari dan berulang pada anak berusia 5 tahun atau lebih yang diharapkan sudah dapat mengendalikan proses berkemihnya. Istilah enuresis primer dipakai untuk anak yang belum pernah berhenti mengompol sejak bayi. Salah satu penyebab enuresis primer adalah keterlambatan maturasi sistem saraf pusat. ASI merupakan nutrisi yang ideal untuk bagi perkembangan sistem saraf. Pemberian ASI eksklusif menunjukkan manfaat besar dalam perkembangan visual, kognitif, dan neurologis anak sehingga berperan dalam mencegah enuresis pada anak. Hal ini didukung oleh beberapa penelitian yang melaporkan hubungan antara pemberian ASI eksklusif dengan enuresis pada anak.Enuresis is repeated involuntary discharge of urine in children 5 years of age or older expected to be able to control the micturition. Primary enuresis is when children 5 years of age or older still bed-wetting. One of the causes of primary enuresis is delayed maturation of the central nervous system. Exclusive breastfeeding has beneficial effect in visual, cognitive, and neurological development in children; it may have a role in preventing enuresis. Several studies report the correlation between exclusive breastfeeding and enuresis prevention in children. ]]>
<![CDATA[Childhood Hyperuricemia as Risk Factor of Hypertension in Adulthood ]]>
<![CDATA[Oke Rina Ramayani]]>
<![CDATA[ The Indonesian Biomedical Journal Vol 4, No 1 (2012) ]]>
Publisher : <![CDATA[The Prodia Education and Research Institute (PERI) ]]>
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DOI: 10.18585/inabj.v4i1.156
<![CDATA[BACKGROUND: Uric acid is the end product of purine metabolism. Hyperuricemia can occur because of decreased excretion, increased production and/or a combination of both mechanisms. Elevation of uric acid in the blood (>5.5 mg/dL) in children is associated with the occurrence of essential hypertension. The relevance of pediatric hyperuricemia into adult hypertension have been widely studied.CONTENT: The high percentage of children and adolescents with metabolic syndrome who had an elevated concentration of uric acid could be of great concern if it were concluded that uric acid was an independent risk factor for cardiovascular disease. The minimum age that has shown blood pressure is significantly associated with adult life is unknown. There are a number of possible explanations for the phenomenon of blood pressure tracking, including hyperuricemia. Several pathophysiological mechanisms increase uric acid with cardiovascular damage through proliferation of vascular smooth muscle cells, stimulate inflammatory path, and then prothrombotic effects triggered by the activation of platelets. Once vascular lesion has appeared, then arises the sodium-sensitive hypertension, although uric acid levels have returned to normal. Persistant mechanism of sodium sensitivity is caused by renal ischemia that leads to activation of the renin-angiotensin system, renal vasoconstriction and increased reabsorption of salt. This supports better understanding of the link between childhood hyperuricemia and adulthood hypertension.SUMMARY: Childhood hyperuricemia is an independent risk factor of hypertension and is ‘linked to’ adult blood pressure.KEYWORDS: uric acid, hyperuricemia, primary hypertension, children, adult]]>
<![CDATA[Luaran Pasien Anak dengan Gagal Ginjal Terminal ]]>
<![CDATA[Oke Rina Ramayani]]>;
<![CDATA[Rosmayanti Rosmayanti]]>;
<![CDATA[Rafita Ramayati]]>;
<![CDATA[Rusdidjas Rusdidjas]]>
<![CDATA[ Sari Pediatri Vol 14, No 5 (2013) ]]>
Publisher : <![CDATA[Badan Penerbit Ikatan Dokter Anak Indonesia (BP-IDAI) ]]>
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DOI: 10.14238/sp14.5.2013.277-82
<![CDATA[Latar belakang. Gagal ginjal terminal merupakan kondisi yang tidak reversibel dan berlanjut pada kematian. Hal tersebut masih merupakan penyebab penting morbiditas dan mortalitas pada anak.Tujuan. Penelitian bertujuan untuk mengetahui etiologi dan modalitas terapi pasien gagal ginjal terminal.Metode. Penelitian retrospektif dengan melakukan telaah rekam medis terhadap 38 anak yang didiagnosis gagal ginjal terminal antara tahun 2005-2010 di RS H.Adam Malik, Medan.Hasil. Selama penelitian, 38 anak (38/355) terdiagnosis gagal ginjal terminal di antara keseluruhan anak penyakit ginjal di RS H. Adam Malik, Medan. Selama kurun waktu tersebut 41.130 anak berobat di Bagian Anak atau lebih kurang 0,1%. Dua puluh lima anak di antaranya (25/38) adalah laki-laki. Rerata umur saat datang pertama kali 9,8 tahun. Penyebab utama gagal ginjal terminal adalah glomerulonefritis kronik (24/38), pielonefritis kronik (5/38), hidronefrosis (2/38) dan penyebab lain (7/38). Modalitas terapi untuk pasien tersebut adalah hemodialisis (17/38), dialisis peritoneal mandiri berkesinambungan (DPMB) (4/38), hemodialisis diikuti DPMB (1/38) dan terapi konservatif /menolak dialisis (16/38). Dua puluh empat anak (24/38) meninggal, 7 anak (7/38) tidak dapat dipantau, dan 7 anak lagi bertahan hidup.Kesimpulan. Glomerulonefritis kronik merupakan penyebab utama gagal ginjal terminal pada anak di RS H Adam Malik, Medan.Angka mortalitas pasien anak dengan gagal ginjal terminal masih tinggi.]]>
<![CDATA[Relationship between childhood blood pressure and birth weight ]]>
<![CDATA[Sri Rahayu]]>;
<![CDATA[Rusdidjas Rusdidjas]]>;
<![CDATA[Rafita Ramayati]]>;
<![CDATA[Oke Rina Ramayani]]>;
<![CDATA[Rosmayanti Siregar]]>
<![CDATA[ Paediatrica Indonesiana Vol 55 No 2 (2015): March 2015 ]]>
Publisher : <![CDATA[Indonesian Pediatric Society ]]>
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DOI: 10.14238/pi55.2.2015.117-20
<![CDATA[cardiovascular mortality and morbidity. Some studies havereported a significant relationship between elevated blood pressurein children with low birth weight.Objective To assess blood pressure differences in primary schoolstudents who had low and normal birth weights.Methods This cross-sectional study was conducted in 170children aged 6 to 12 years in March 2011 at a Medan primaryschool, North Sumatera. Blood pressure was measured with astandard mercury sphygmomanometer. A parental questionnairewas used to collect information on birth weight. Data wereanalyzed by student’s T-test for numerical data and Spearman’scorrelation test for a relationship between blood pressure andbirth weight.Results The subjects consisted of 85 children with low birth weightand 85 children with normal birth weight. The mean systolic (SBP)and diastolic blood pressures (DBP) were significantly higherin children with low birth weight than those with normal birthweight [SBP: 106.7 vs. 99.8 mmHg, respectively, (P=0.0001); andDBP: 69.2 vs. 63.5 mmHg, respectively, (P=0.0001)]. There wererelationships between elevated SBP and DBP and low birth weight,as indicated by correlation coefficient [r=-0.365 and r=-0.425,respectively, (P=0.0001)].Conclusion Blood pressure is significantly higher in children withlow birth weight than in those with normal birth weight. Birthweight was inversely related both to systolic and diastolic bloodpressure.]]>
<![CDATA[Renal manifestations in tuberous sclerosis patients: two case reports ]]>
<![CDATA[Oke Rina Ramayani]]>;
<![CDATA[Rosmayanti Rosmayanti]]>;
<![CDATA[Rafita Ramayati]]>;
<![CDATA[Rusdidjas Rusdidjas]]>
<![CDATA[ Paediatrica Indonesiana Vol 53 No 1 (2013): January 2013 ]]>
Publisher : <![CDATA[Indonesian Pediatric Society ]]>
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DOI: 10.14238/pi53.1.2013.56-8
<![CDATA[Tuberous sclerosis (TS) is a neurocutaneous disorder, characterized bymental retardation, epilepsy, and facialangiofibromas. The incidence has beenestimated to be 1 case per 6000 live births.Although rare, this disorder is a cause of mentalretardation with severe epilepsy.1 Scientists havefound manifestations of this disorder not only inbrain and skin, but also in the eyes, heart, lungs, andkidneys, so it has been renamed tuberous sclerosiscomplex (TSC).]]>
