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INDONESIA
Indonesian Journal of Clinical Pathology and Medical Laboratory (IJCPML)
Published by Perhimpunan Dokter Spesialis Patologi Klinik Indonesia
ISSN : 08544263     EISSN : 24774685     DOI : https://dx.doi.org/10.24293
Core Subject : Health, Science,
Biochemistry, Genetics & Molecular Biology Health Professions Medicine & Pharmacology Neuroscience
Indonesian Journal of Clinical Pathology and Medical Laboratory (IJCPML) is a journal published by “Association of Clinical Pathologist” professional association. This journal displays articles in the Clinical Pathology and Medical Laboratory scope. Clinical Pathology has a couple of subdivisions, namely: Clinical Chemistry, Hematology, Immunology and Serology, Microbiology and Infectious Disease, Hepatology, Cardiovascular, Endocrinology, Blood Transfusion, Nephrology, and Molecular Biology. Scientific articles of these topics, mainly emphasize on the laboratory examinations, pathophysiology, and pathogenesis in a disease.
Arjuna Subject : Kedokteran - Kedokteran (Lain-Lain)
Articles 1,328 Documents
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ANTIBIOGRAM (Antibiogram) Jeine Stela Akualing; IGAA Putri Sri Rejeki
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 23, No 1 (2016)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v23i1.1191

Abstract

Resistensi antibiotika telah menjadi masalah kesehatan global. Upaya yang dilakukan untuk mengatasi kegentingan resistensiantibiotika adalah melalui penggunaan antibiotika secara bijak. Salah satu strategi penggunaan antibiotika secara bijak adalah denganmenyusun dan menggunakan antibiogram. Antibiogram menuntun peklinik dalam memilih pengobatan antibiotika empiris terbaiksementara menunggu hasil kultur dan uji kepekaan antibiotika. Antibiogram dapat dijadikan dasar dalam penyusunan pedomanpengobatan antibiotika empiris dan dapat digunakan dalam mendeteksi serta memantau arah resistensi antibiotika. LaboratoriumMikrobiologi Klinik di setiap lembaga pelayanan kesehatan bertanggung jawab dalam menyusun antibiogram, menyebarkannya kepadapeklinik, serta melakukan perbaikan setiap tahun. Telaah pustaka ini bertujuan untuk membahas antibiogram, termasuk cara menyusundan menyajikan antibiogram, sehingga diharapkan dapat membantu setiap lembaga dalam membuatnya.
CORRELATION PERCENTAGE OF S AND G2/M WITH PERCENTAGE OF LYMPHOBLASTS IN PEDIATRIC ACUTE LYMPHOBLASTIC LEUKEMIA Erawati Armayani; Yetti Hernaningsih; Endang Retnowati; Suprapto Ma'at Ma'at; I Dewa Gede Ugrasena
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 24, No 1 (2017)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v24i1.1155

Abstract

Leukemia Limfoblastik Akut (ALL) merupakan keganasan klonal di sumsum tulang/Bone Marrow (BM). Angka bertahan hidup5 tahun saat ini >85%, tetapi 15-20% relaps sehingga perjalanan penyakit jelek. Perjalan penyakit jelek jika setelah tahap induksilimfoblas menetap di Darah Tepi (DT) dan BM >5% serta tahap S BM >6%. Tahap G2/M merupakan petunjuk prognosis ALL anak,selain itu sebagai target pengobatan. Tujuan penelitian menganalisis kenasaban persentase tahap S dan G2/M dengan persentaselimfoblas DT pasien ALL anak sebelum dan sesudah kemoterapi induksi. Jenis penelitian analitik observasional longitudinal (kohor)di ALL anak kasus baru diperiksa sebelum dan sesudah induksi. Persentase limfoblas secara mikroskopis. Persentase fase S dan G2/MflowcytometryBD Facs Callibur. Kenasaban bermakna hanya persentase tahap S dan limfoblas sebelum induksi (r=0,449; p=0,007).Kelainan gen ALL pada ekspresi cyclins dan CDK sehingga hilang kendali checkpoint siklus sel, merangsang transisi tahap G1 menjaditahap S. Persentase tahap S tidak berbeda pada remisi dan meninggal (p=0,138). Persentase tahap G2/M berbeda antara remisi danmeninggal (p=0,006) dan bernasab dengan luaran kemoterapi induksi (koefisien Eta= 0,744), G2/M ≥1,26% meramalkan remisi.Terdapat kenasaban antara persentase siklus sel tahap S dengan persentase limfobas sebelum kemoterapi induksi. Persentase siklus seltahap S memberikan gambaran siklus sel pada sel limfoblas. Terdapat kenasaban antara persentase siklus sel tahap G2/M dengan luarankemoterapi induksi tahap G2/M menjadi faktor peramal luaran kemoterapi induksi ALL. Perlu penelitian lanjutan dengan sampel BM,subtipe dan pengamatan semua tahap kemoterapi.
IDENTIFIKASI MUTASI H63D GEN HFE PADA KELAINAN HBE Yanuarita Tursinawati; Nyoman Suci Widyastiti; Moedrik Tamam
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 22, No 2 (2016)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v22i2.1123

Abstract

The H63D HFE mutation has been reported to be responsible for primary haemochromatosis. The allele frequency in Indonesianpopulation is about 2.8%. Co inheritance between H63D mutation and hemoglobin disorders such as Thalassemia may increase theseverity of iron overload. Nevertheless, the coinheritance of this mutation with HbE disorder is the most common hemoglobin disorderin Indonesia and the gene frequency have not been reported especially in Javanese ethnic. To identify the presence and the frequency ofH63D HFE mutation in HbE disorder among Javanese ethnic. A cross sectional study involved 24 Javanese individuals who consist of21 HbE heterozygotes (HbAE) and 3 HbE homozygotes (HbEE) subjects. The subjects were screened for H63D mutation by digestion ofPCR products with MbO I restriction endonuclease. The genotype frequency for wt/wt was 95.24% in HbAE, 100% in HbEE and for wt/H63D was 4.76% in HbAE. The allele frequency for H63D HFE mutation was 2.08% in total sample of HbE. The allele frequencies inHbAE and HbEE individual were 2.38% and 0%, respectively. H63D HFE mutation is found in 24 Javanese ethnic individual with HbEdisorder. However, the allele frequency of H63D HFE mutation is low and almost similar to the allele frequency of H63D HFE mutationin Indonesian population.
QUALITY OF STORED RED BLOOD Anak Agung Wiradewi Lestari; Teguh Triyono; Usi Sukorini
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 23, No 3 (2017)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v23i3.1209

Abstract

Telah diketahui bahwa selama penyimpanan, sel darah merah mengalami sejumlah perubahan yang mempengaruhi viabilitas dankemampuannya untuk membawa oksigen ke jaringan. Perubahan tersebut digolongkan menjadi perubahan biomekanik dan biokimia.Perubahan biomekanik yang terjadi adalah perubahan membran sel. Selama penyimpanan, sel darah merah mengalami perubahanmorfologi secara pesat, dari bikonkaf menjadi echinocytes dengan tonjolan dan akhirnya menjadi spheroechinocytes. Hilangnya kesatuansel darah merah tersebut menyebabkan pelepasan hemoglobin (hemolisis) dan pembentukan mikropartikel yang dapat menyebabkankomplikasi transfusi. Pelepasan hemoglobin (Hb) dan mikropartikel bebas menyebabkan peningkatan penggunaan Nitric Oxide (NO),sebuah molekul sinyal penting yang berperan dalam aliran darah dan dapat merangsang terjadinya inflamasi. Perubahan kimia lainnyayang dapat terjadi adalah penurunan glukosa dan penumpukan asam laktat, penurunan kalium, kepekatan adenosine triphosphate (ATP)dan 2,3-diphosphoglycerate (DPG). Tidak semua kerusakan sel akibat penyimpanan ini bersifat eryptotic. Penurunan kalium bersifatpasif (suhu yang dingin menyebabkan pompa pertukaran natrium/kalium menjadi tidak aktif). Penurunan DPG juga bersifat pasif,terkait dengan perubahan kekhasan enzim diphosphoglycerate mutase/diphosphoglyceratephosphatase dan penurunan pH. PenurunanNO terjadi karena larutnya NO bersama dengan Hb yang dilepaskan saat hemolisis. Hb plasma lebih cepat bereaksi dengan NO,dibandingkan dengan Hb dalam sel darah merah. Berkurangnya NO ini berperan dalam keadaan patologis yang terjadi sehubungandengan pemberian darah simpan termasuk dalam hal pengangkutan oksigen oleh Hb. Perubahan akibat penyimpanan ini reversibel bilasel darah merah tersebut ditransfusikan kembali ke dalam peredaran. Tolok ukur utama yang dikontrol secara rutin untuk penyimpananRBC adalah 0,8–1% hemolisis, 75% in-vivo survival dalam waktu 24 jam setelah transfusi, volume dan kadar hemoglobin sel darahmerah. Tolok ukur tersebut memang sangat berguna, namun, perubahan biokimia yang berhubungan dengan fungsi vaskular jugaharus dipertimbangkan. Perubahan yang terjadi selama penyimpanan tersebut akan reversibel melalui upaya peningkatan kualitaspenyimpanan, atau menambahkan larutan additive.
ERYTHROLEUKEMIA Ailinda Theodora Tedja; Riadi Wirawan
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 23, No 2 (2017)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v23i2.1146

Abstract

Leukemia eritroid akut dibagi menjadi dua (2) subtipe berdasarkan penggolongan World Health Organization (WHO) 2008,yaitu eritroleukemia dan pure erythroid leukemia. Penggolongan WHO 2008 menganjurkan diagnosis leukemia eritroid akutcukup berdasarkan hasil menilai sumsum tulang, berdasarkan jumlah eritroblas dan sel blas, keberadaan diseritropoiesis, sertadisgranulopoiesis. Eritroleukemia merupakan bentuk leukemia mieloid akut yang jarang terjadi, yaitu <5% kasus leukemia mieloidakut. Eritroleukemia terutama terjadi di orang dewasa dan lebih sering terjadi di laki-laki. Satu kasus dilaporkan perempuan berusia42 tahun dengan hasil memeriksa pansitopenia di laboratorium, kemudian dinilai sumsum tulangnya dan didapatkan eritroblas 67%All Nucleated Cells (ANC), disertai diseritropoiesis yang jelas dan blas 25% Non-Erythroid Cells (NEC) dengan disgranulopoiesis 35%.Hasil ini menetapkan diagnosis eritroleukemia (AML-M6) berdasarkan penggolongan WHO 2008. Hasil memeriksa imunofenotip flowcytometry didapatkan komponen eritroid dan mieloid yang mendukung diagnosis eritroleukemia. Pemeriksaan imunofenotip denganflow cytometry tersebut sebenarnya tidak diperlukan untuk menetapkan diagnosis eritroleukemia. Pemeriksaan sitogenetika disarankanuntuk menentukan peramalan perjalanan penyakit.
DIAGNOSTIC CONCORDANCE BETWEEN NEXT-GENERATION AND HIGH SENSITIVE TROPONIN-I IN ANGINA PECTORIS PATIENTS Erna R Tobing; Jusak Nugraha; Muhammad Amminuddin
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 24, No 1 (2017)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v24i1.1158

Abstract

Angina pectoris merupakan gejala klinis Sindrom Koroner Akut (SKA) yang mengarah pada penyakit jantung koroner. Sindromkoroner akut terdiri dari Unstable Angina dan Infark Miokard Akut (IMA). Kadar Troponin I (TnI) dapat mendukung penegakkandiagnosis IMA di pasien angina pectoris. Beberapa metode pemeriksaan TnI semakin berkembang diantaranya TnI high sensitive (TnI hs)dan TnI next-generation (TnI ng). Tujuan penelitian ini adalah menganalisis kesesuaian diagnostik antara kadar TnI ng yang diperiksamenggunakan metode Fluorescent Enzyme Transfer Latex (FETL) [Alere Triage MeterPro®] dan TnI hs dengan metode ChemiluminescentImmunoassay (CLEIA) [Mitsubishi PathFast®] di pasien angina pectoris. Penelitian dilaksanakan di RSUD Dr.Soetomo Surabaya masawaktu Maret-Juli 2016 dengan rancangan penelitian potong lintang. Sebanyak 82 subjek penelitian dengan gejala angina pectorisdiperiksakan kadar Troponin-I menggunakan kedua metode. Subjek penelitian sebanyak 44% didiagnosis SKA, dan 56% non SKA. Nilaikesesuaian koefisien kappa antara TnI ng dan TnI hs di pasien angina pectoris adalah 0,738 (p<0,01). Kepekaan dan kekhasan TnI ngterhadap TnI hs untuk diagnosis IMA dengan cut off 0,02 ng/mL adalah 94% dan 78%. Analisis kenasaban antara kadar TnI ng danTnI hs dengan koefisien kenasaban Spearman rho (ρ) adalah 0,826 (p<0,01). Terdapat kesesuaian diagnostik antara TnI ng dan TnI hsdi pasien angina pectoris. Kedua metode pemeriksaan TnI dapat digunakan untuk membantu menegakkan diagnosis di pasien anginapectoris. Penelitian lebih lanjut diperlukan untuk mengetahui nilai prognosis TnI.
MALDI-TOF DAN SELDI-TOF MASS SPECTROMETRY DENGAN THROUGHPUT TINGGI UNTUK ANALISIS PROTEOMIK PROFIL PROTEIN DARI PETANDA BIOLOGIS Trinovia Andayaningsih; Siti Muchayat P.
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 22, No 2 (2016)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v22i2.1126

Abstract

In recent years, proteomic approach has been widely used for diagnosing the diseases and matrix assisted laser desorption /ionization- time of the flight (MALDI-TOF) mass spectrometry and its modification, surface enhanced laser desorption /ionization-timeof flight (SELDI-TOF) mass spectrometry have became very promising diagnostic tools. High throughputs and relative simplicity of thesetechnologies attracted the researchers to know the biomarkers of specific diseases by analyzing specimens of serum/plasma and otherbody fluids. Analyzing plasma specimens by MALDI/SELDI TOF mass spectrometry provides new information especially about smallprotein and peptides in high abundance. Protein profilings, resulting by these technologies provide higher sensitivity and specificity valuesthan current biomarkers. Knowing how these principle tools work and its promising application to early detection of specific diseases isthe aim of writing this paper.
INTERLEUKIN-4 DAN INTERFERON GAMMA DI NEFRITIS LUPUS: HUBUNGAN AKTIVITAS PENYAKIT SERTA KEKAMBUHAN Torajasa Achamar; Dany Farida; Hani Susianti; Kusworini Handono; Ati Rastini; R.I R.I; I Putu A.S; Atma Gunawan; Handono Kalim
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 22, No 2 (2016)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v22i2.1117

Abstract

Sampling for urinalysis to see the activity and the degree of recurrence of Lupus Nephritis (LN) is very difficult. New biomarkersthat are more simple, sensitive, specific and non-invasive in assessing the activity of the LN need to be investigated. Interleukin-4 (IL-4)and interferon gamma (IFN-γ) were implicated to LN process. Urine samples from 17 LN patients were taken every month for 6 (six)months to examine the level of uIL-4, uIFN-γ, activity and recurrence of LN. Significant differences were observed in the uIFN-γ levelsbetween the active and inactive LN groups (p=0.012), but not in uIL-4 levels (p=0.187). Correlations between each biomarker andrenal domain score were weak (r=0.201, p=0.042 for uIL-4; r=0.268, p=0.006 for uIFN-γ). Significant differences were also found inthe uIL-4 and uIFN-γ levels against LN recurrence (p=0.033; p=0.017). The best cut off values to assess recurrences and activity of LNwere 8.17 pg/mL for uIL-4 showed a sensitivity of 74%, specificity 71%, NPV 90%, PPV 42% to assess recurrences and to assess activityof LN showed sensitivity 46%, specificity 75%, NPV 48%, PPV 78%. The cut off 18.58 pg/mL for uIFN-γ to predict recurrent and assessthe activity of LN showed sensitivity 68%%, specificity 70%, NPV 88%, PPV 40% to predict the recurrent and to assess the activity of LNshowed sensitivity 57%, specificity 64%, NPV 49%, PPV 73%. Based on the research, uIL-4 and uIFN-γ are not good enough to predictrecurrence and activity of LN
THE ASSOCIATION OF INSULIN RESISTANCE AND LIPID PROFILE RATIO IN METABOLIC SYNDROME Rini Rahmayani; Adi Koesoema Aman; Santi Safril
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 25, No 1 (2018)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v25i1.1484

Abstract

The cause of metabolic syndrome is still not known for sure, but it is suspected that the pathophysiology of metabolic syndrome is associated with insulin resistance and central obesity. Researchers have attempted to evaluate insulin resistance using various serum lipid concentration ratio. This study was to observe the association between insulin resistance and lipid profile ratio using HOMA-IR in metabolic syndrome patients. This study was a cross-sectional that was conducted in Inpatient and Outpatient Adam Malik Hospital during March 2016 - April 2016. Subjects were patients with metabolic syndrome criteria according to the International Diabetic Federation 2005. All samples were examined for their waist circumference, weight, height, blood pressure, insulin, serum glucose, total cholesterol, HDL cholesterol, LDL cholesterol, Triglycerides. Among sixty-six patients in the study 40 (60.6%) were male and 26 (39.4%) female. In this study, there was a significant correlation between HOMA-IR with CT/HDL ratio (r: 0.244 p <0.05); and there was no correlated HOMA-IR, and TG/HDL ratio (r: 0.086 p > 0.05) and there was no correlation between HOMA-IR and LDL/HDL (r: 0.336 p > 0.05). There was a significant relationship between insulin resistance and ratio CT/HDL
THE CORRELATION OF ANEMIA AND HEPCIDIN SERUM LEVELS IN REGULAR HEMODIALYSIS PATIENTS WITH CHRONIC HEPATITIS C IN HAJI ADAM MALIK HOSPITAL MEDAN Wingsar Indrawanto; Adi Koesoema Aman; Alwi Thamrin
INDONESIAN JOURNAL OF CLINICAL PATHOLOGY AND MEDICAL LABORATORY Vol 25, No 2 (2019)
Publisher : Indonesian Association of Clinical Pathologist and Medical laboratory

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24293/ijcpml.v25i2.1464

Abstract

Background : End stage renal disease patients who undergo hemodialysis therapy are the high-risk populations who are infected by hepatitis C virus. Some studies have been reported that hepcidin levels decreased in patients with chronic hepatitis C. Hepcidin serum concentrations were also reported to increase in patients with renal failure in the line with increased severity of renal failure, which can cause the accumulation of hepcidin which culminates in anemia because iron deficiency. This Study was to analyze the correlation of anemia and hepcidin serum levels in patients end stage renal disease who undergoing regular hemodialysis with chronic hepatitis C.Methods : This study was an analytic observational with cross sectional design which was conduted on 24 patients end stage renal disease (ESRD) with chronic hepatitis C and 24 patients  with ESRD without hepatitis who are undergoing regular hemodialysis theraphy in Haji Adam Malik Hospital, Medan in July – September 2016. All study subjects were examined for full blood count and hepcidin serum levels. The result of the iron status were recorded from the patient’s medical record.Result : In this study, the mean hemoglobin was 8,15±1,44 g/dL, mean hematocrit 25,42±4,53%, median hepcidin levels 29,75 (4,92-359,49) in the patients ESRD with chronic hepatitis C  and mean hemoglobin 8,21±1,50 g/dL, mean hematocrit 25,25±4,37%, median hepcidin levels 30,33 (11,65-141,53) in the patients ESRD without hepatitis. In Spearman’s rho test  was showed a positive correlation that significant between hepcidin and hemoglobin (r = 0,439, p = 0,032), hepcidin and hematocrit (r = 0,021, p = 0,024) in patients ESRD with chronic hepatitis C.Conclusion : This study showed there was a positive correlation between anemia and hepcidin serum levels in patients ESRD who undergoing regular hemodialysis with chronic hepatitis C.

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