cover
Article Per Year (5 Year)
All
Home Page
OAI Link
Editorial Team
Contact
Reviewer
Google Scholar
Contact Name
Fika Kharisyanti
Contact Email
fikakharisyanti@gmail.com
Phone
+6282232687366
Journal Mail Official
-
Editorial Address
Ruang Stem Cell, Gedung Lembaga Penyakit Tropis Lantai 2, Kampus C Universitas Airlangga
Location
Kota surabaya,
Jawa timur
INDONESIA
Journal of Stem Cell Research and Tissue Engineering
Published by Universitas Airlangga
ISSN : 26141264     EISSN : 26141256     DOI : https://dx.doi.org/10.20473/jscrte
Core Subject : Health, Science, Engineering,
Engineering Health Professions Immunology & microbiology Medicine & Pharmacology Veterinary
Journal of Stem Cell Research and Tissue Engineering (JSCRTE) is published by Stem Cell Research and Development Center, Airlangga University. Stem Cell Research is dedicated to publishing high-quality manuscripts focusing on the biology and applications of stem cell research. Submissions to Stem Cell Research, may cover all aspects of stem cells, including embryonic stem cells, tissue-specific stem cells, cancerstem cells, developmental studies, genomics and translational research. Special focus of JSCRTE is on mechanisms of pluripotency and description of newly generated pluripotent stem cell lines. Articles that go through the selection process will be review by peer reviewer or editor. The journal is published regularly twice a year in December and May. Every publication consists of 60-70 pages and 5 scientific articles in the form of research, study literature, and the case study in English. The contributors Journal of Stem Cell Research and Tissue Engineering are Stem Cell researchers, lecturers, student and practitioners that came from Indonesia and abroad.
Arjuna Subject : Kedokteran - Imonologi dan Alergi
Articles 90 Documents
 5   6   7   8   9 
MESENCHYMAL STEM CELLS FROM ADIPOSE TISSUE TO TIGHTEN FACIAL SKIN Najah, Adilla Syahsiyatun; Fadila, Aghniya Nur Rizka; Wibawa, Alifah Mustari Mukti; Sulasikin, Almaydha Naim; Salsabila, Aulia Putri; Salsabila, Anbar Rania; Luthfi, Cahya Arsya; Raflianti, Desti; Hakim, Elsya Bayduri; Haryanto
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 1 (2025): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i1.67461

Abstract

Skin elasticity is an important aspects of facial care. This study aims to explore the potential of mesenchymal stem cells (MSCs) derived from adipose tissue (adipose-derived stem cells) in facial skin tightening and assess their advantages and limitations in clinical applications. The methods used include literature searches from various officially published online sources as well as analysis of research data regarding the process of isolation, culture, differentiation and clinical applications of ADSCs. The main findings show that adipose-derived stem cells (ADSCs) are able to form new collagen, increase skin elasticity, and repair tissue damage due to aging. The culture process involving isolation of cells from adipose tissue and differentiation of the cells into fibroblasts is essential to exploit the regenerative potential of these cells. Although this technology is promising, this study also identified technical and regulatory challenges that need to be overcome, including patient health criteria, safe ADSc harvest procedures, isolation and culture processes that have a high chance of differentiation and post-application care of ADSc therapy. The significance of these findings is that a better understanding of the mechanism of action of ADSCs can make a positive contribution to the development of beauty therapies that are more effective and safer compared to conventional methods that are often unsatisfactory. Additionally, this approach is expected to provide a long-term solution for individuals who wish to maintain a youthful appearance without the risk of significant side effects. Thus, this research can be a step in integrating stem cell technology in the beauty industry, especially for anti-aging treatments.
FUNCTIONAL ROLE OF VIMENTIN'S CYSTEINE IN XIST-MEDIATED EMT INHIBITION IN BREAST CANCER Jayanti; Melisa; Arnando; Mur'ah; Noviyanti
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 1 (2025): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i1.73554

Abstract

Breast cancer is the most commonly diagnosed malignancy among women worldwide and a leading cause of cancer-related mortality, primarily due to its high metastatic potential. One mechanism underlying metastasis is the epithelial-to-mesenchymal transition (EMT), which enhances cancer cell mobility, invasiveness, and resistance to treatment. Vimentin, a type III intermediate filament protein, is a hallmark of EMT and plays a structural and regulatory role in cytoskeletal organization and cellular stress responses. Recent studies have highlighted the importance of a single cysteine residue at position 328 (C328) in vimentin, which functions as a redox-sensitive site influencing filament dynamics. However, the role of C328 in cancer progression remained largely unexplored. This literature review investigates the effect of a single amino acid substitution—C328 to serine (C328S)—on breast cancer cell behavior, focusing on findings published between 2020 and 2025, sourced from PubMed and Google Scholar. Evidence from MCF-7 breast cancer cell models reveals that expression of C328S-VIM induces morphological changes, cytoskeletal disorganization, and increased proliferation, migration, and invasion. Notably, C328S-VIM upregulates the long non-coding RNA XIST, which promotes EMT, estrogen independence, and stem-like properties. These findings indicate that the native C328 residue serves a tumor-suppressive function, partly through modulation of XIST activity. Overall, this review presents a novel insight into how a single amino acid mutation in vimentin can reprogram breast cancer cells toward a more aggressive and stem-like phenotype. The study highlights C328 as a potential therapeutic target and broadens our understanding of the molecular mechanisms driving breast cancer progression.
STEM CELL HIERARCHY AND IMMUNE TOLERANCE IN BONE MARROW MICHES: MECHANISMS AND EMERGING PERSPECTIVES Elyusaputra, Harry; Anggriawati, Moudy
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 1 (2025): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i1.73555

Abstract

Bone marrow niches play a crucial role in regulating the hierarchy of hematopoietic stem cells (HSCs) and immune tolerance. In this microenvironment, interactions between niche cells and molecular components such as Nitric Oxide (NO) and the CD200 protein influence HSC function. NO acts as a signaling molecule that supports HSC proliferation at low concentrations and promotes differentiation at higher concentrations. HSCs with high NO production are often located near CD200-rich capillaries, providing protection from immune responses. Additionally, the signaling pathway involving IFT20, CD200, eNOS, and autophagy supports cellular homeostasis and the survival of cells under stress, including in cancer. The expression of CD200 in capillaries contributes to a tolerogenic immune environment, protecting tissues from excessive inflammation. However, excessive CD200 expression can be exploited by tumor cells to evade immune detection. This review highlights the critical role of the bone marrow microenvironment in regulating stem cell behavior and immune tolerance, offering insights into how these molecular interactions govern stem cell fate and tissue homeostasis. A deeper understanding of these pathways can pave the way for novel therapeutic strategies in blood disorders, cancer, and inflammatory diseases. By unraveling the intricate molecular interactions within the bone marrow niche, this research provides new perspectives on stem cell regulation and immune tolerance, with implications for improving clinical outcomes in stem cell-based therapies.
LONG-TERM EVALUATION OF THE SAFETY AND EFFECTIVENESS OF NEURAL STEM CELL TRANSPLANTATION FOR CHRONIC THORACIC SPINAL CORD INJURY Harmin; Maharani, Hanisa Aulia
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 1 (2025): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i1.73556

Abstract

Spinal cord injury (SCI) is a debilitating neurological condition that leads to partial or complete loss of motor and sensory function, depending on the injury’s severity and location. Conventional therapies focus on surgical stabilization, prevention of secondary damage, and rehabilitation. However, these approaches often fall short in restoring long-term functionality. In recent years, cell-based therapies have emerged as promising alternatives, particularly those involving neural stem cells (NSCs). This literature review explores the long-term safety and effectiveness of NSC transplantation for chronic thoracic SCI, based on studies published between 2010 and 2025. Research shows that fetal-derived NSCs, such as HuCNS-SC, demonstrate a high safety profile and low risk of tumor formation due to their committed neural lineage. Clinical trials report early signs of motor improvement and reduced spasticity in chronic SCI patients following transplantation. Additionally, mesenchymal stem cells (MSCs) have shown the ability to migrate to injury sites and exert therapeutic effects, though these benefits tend to be short-lived. The post-injury inflammatory microenvironment poses a significant barrier to the success of NSC therapies by impairing stem cell differentiation and survival. Therefore, immunosuppressive regimens are often employed to enhance NSC efficacy by creating a more supportive environment. Overall, while both NSCs and MSCs offer promising avenues for SCI treatment, long-term recovery likely requires multimodal approaches that address both neural regeneration and immune modulation. Continued research is essential to optimize these therapies and translate them into effective clinical treatments for patients with chronic SCI.
DECODING YAP-DRIVEN MALIGNANT REPROGRAMMING IN ORAL EPITHELIAL STEM CELLS THROUGH SINGLE-CELL ANALYSIS Putri, Indah Salsabila Febriana; Harmin; Maharani, Hanisa Aulia
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 1 (2025): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i1.73558

Abstract

Oral squamous cell carcinoma (OSCC), a major subtype of head and neck squamous cell carcinoma (HNSC), is characterized by high mortality rates and cellular heterogeneity that complicates early detection and treatment. Recent advances in cancer biology suggest that tumorigenesis involves reprogramming of epithelial progenitor cells into cancer stem-like cells (CSCs), driven by oncogenic signaling such as Yes-associated protein (YAP) activation. YAP, a key effector of the Hippo pathway, regulates transcriptional programs involved in cell proliferation, dedifferentiation, and inhibition of differentiation. However, the specific mechanisms by which YAP reprograms oral epithelial stem cells remain incompletely understood. This literature review systematically explores findings from studies published between 2020 and 2025 that investigate the role of YAP in malignant reprogramming, particularly through single-cell analysis approaches. Articles were sourced from PubMed and Google Scholar using defined inclusion criteria, focusing on original studies involving in vitro, in vivo, or bioinformatic models. The review highlights that YAP activation in oral epithelial cells induces stemness-associated genes (e.g., SOX2, NANOG, OCT4), represses differentiation pathways (Notch, p63), and promotes epithelial-mesenchymal transition (EMT) markers (ZEB1, SNAI2, VIM). Single-cell RNA sequencing (scRNA-seq) has revealed dynamic and hybrid cell states, supporting the view that YAP-driven transformation is gradual and reversible. YAP also shapes the tumor microenvironment by inducing cytokines that recruit tumor-supportive immune and stromal cells. Key YAP-regulated targets such as CTGF, AXL, and ITGA6 emerge as potential therapeutic entry points, as their inhibition reduces proliferation and stemness. These findings underscore YAP’s central role in oral carcinogenesis and its promise as a molecular target for early intervention and therapy.
RECENT ADVANCES IN STEM CELLS-BASED THERAPY FOR HIV ELIMINATION Herdyan, Deta Intan; Ferni; Nugraha, Adi
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 2 (2025): Journal of Stem Cell Research and Tissue Engineering
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i2.85309

Abstract

The persistent latent reservoir of HIV remains the greatest barrier to achieving a functional cure. Stem-cell–based therapy has emerged as a promising approach to eliminate or durably suppress HIV by reconstructing the immune system with HIV-resistant cells. This review summarizes recent progress (2021–2025) in stem-cell–based therapeutic strategies for HIV elimination, including hematopoietic stem cell transplantation (HSCT), gene-edited autologous stem cells, and stem-cell–derived engineered immune effectors. A narrative literature review was conducted using PubMed and Google Scholar databases for articles published between 2021 and 2025. Keywords included “HIV cure,” “stem cell therapy,” “hematopoietic stem cell transplantation,” “CCR5 gene editing,” “CAR-T cells,” and “HSPC-based HIV therapy.” Allogeneic HSCT using CCR5Δ32/Δ32 donors provided proof-of-concept for HIV elimination. Recent reports demonstrate additional remission cases using cord blood or haploidentical grafts. Advances in CRISPR/Cas9-mediated CCR5 disruption of autologous hematopoietic stem and progenitor cells (HSPCs) achieved high editing efficiency and successful engraftment. Stem-cell–derived CAR-T and CAR-NK cells showed potent antiviral effects in preclinical and early clinical studies. However, challenges remain in eradicating viral reservoirs, preventing viral escape, and ensuring long-term safety of gene editing. Stem-cell–based therapies represent a rapidly evolving frontier toward an HIV cure. Emerging technologies in gene editing and engineered cell therapy promise scalable, safer, and potentially curative approaches, though significant translational and ethical hurdles must be addressed before widespread clinical application.
APPLICATION OF STEM CELLS AND THEIR DERIVATIVES AS DRUG DELIVERY SYSTEMS Ambarura, Elsa; Sudirman, Clarissa
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 2 (2025): Journal of Stem Cell Research and Tissue Engineering
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i2.85312

Abstract

Advances in modern drug delivery systems (DDS) are increasingly shaped by the need to improve therapeutic precision while minimizing systemic toxicity. Stem cells and their derivatives—particularly mesenchymal stem cells (MSCs)—have emerged as highly promising platforms due to their intrinsic homing capacity, immunomodulatory properties, and compatibility with molecular engineering. This review synthesizes recent progress in whole-cell, secretome-based, and extracellular vesicle (EV)/exosome-based DDS, highlighting their biological mechanisms, engineering innovations, and translational challenges. Whole-cell delivery systems exploit the ability of living stem cells to migrate toward tumors or inflamed tissues while carrying therapeutic agents such as paclitaxel or oncolytic viruses, enabling site-specific drug release with enhanced local potency. Complementarily, MSC secretome—rich in soluble factors and vesicles—demonstrates synergistic cytotoxic, anti-inflammatory, and microenvironment-modulating effects that strengthen therapeutic outcomes. Exosomes and EVs offer nanoscale, low-immunogenic carriers capable of transporting small-molecule drugs, siRNA, or immunotherapeutic combinations, with engineering strategies such as surface functionalization, electroporation, and genetic modification further improving cargo loading, targeting specificity, and stability. This review employed a structured literature search (2020–2025) across PubMed and Google Scholar, followed by rigorous screening and qualitative synthesis of eligible preclinical and clinical evidence. Key translational barriers include large-scale manufacturing, batch variability, pharmacokinetic limitations, and regulatory ambiguity surrounding stem-cell-derived therapeutics. Emerging GMP-compatible pipelines and gene-edited stem cell systems show promise in addressing these hurdles. Overall, stem cell–based DDS represent a rapidly evolving biological technology ecosystem, integrating natural cellular behaviors with engineered precision. Their versatile platforms hold substantial potential for targeted cancer therapy, immune modulation, and RNA interference–based treatments, positioning them at the forefront of next-generation therapeutic delivery strategies.
THE ROLE OF STEM CELLS IN INCURABLE DISEASES: A SYSTEMATIC REVIEW WITH META-ANALYSIS Kanagaratnam, Pakeerathan
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 2 (2025): Journal of Stem Cell Research and Tissue Engineering
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i2.85317

Abstract

Stem cells, due to their unique capacities for self-renewal and differentiation, represented a highlypromising therapeutic strategy for a range of currently incurable diseases. This systematic review andmeta-analysis aimed to assess the clinical efficacy of stem cell interventions acrossneurodegenerative, cardiovascular, diabetic, and hematological disorders. A comprehensive literaturesearch of PubMed, Embase, Web of Science, and CENTRAL databases was conducted, covering theperiod from January 1995 to July 2025. A total of seventy studies were included in the qualitativesynthesis, of which forty-five studies were incorporated into the quantitative meta-analysis. Thepooled results demonstrated that stem cell therapy significantly improved functional outcomes inpatients with neurodegenerative disorders, enhanced cardiac performance in cardiovascular disease,promoted glycemic control in diabetic patients, and facilitated hematopoietic recovery inhematological conditions. Effect sizes ranged from moderate to large, indicating clinically meaningfulbenefits. Mechanistic analyses suggested that the therapeutic effects were mediated through multiplepathways, including differentiation into target cell types, paracrine signaling that modulated the localmicroenvironment, immunomodulatory actions that reduced inflammation, and stimulation ofangiogenesis that improved tissue perfusion. Despite these encouraging findings, several challengeswere identified. Heterogeneity in study protocols, including variations in stem cell type, source, dose,and delivery route, contributed to inconsistent outcomes. Potential risks such as tumorigenicity—especially with embryonic stem cells and induced pluripotent stem cells—and immune rejection wererecognized as significant safety concerns. Additionally, most studies had limited follow-up durations,restricting the evaluation of long-term efficacy and safety. Addressing these limitations throughstandardized protocols, large multicenter randomized trials, and extended follow-up was deemedessential. Collectively, the evidence supported the therapeutic potential of stem cell interventionswhile emphasizing the need for further research.
TRANSFORMATION OF LENS EPITHELIAL CELLS INTO STEM CELLS FOR REGENERATIVE THERAPY APPLICATIONS IN OCULAR TISSUE Khadijah, Nurul; Damawati, Esti Utami
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 2 (2025): Journal of Stem Cell Research and Tissue Engineering
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i2.85321

Abstract

Visual impairment and blindness remain major global health challenges, driven primarily by cataract, corneal disorders, retinal degeneration, and glaucoma. Although current surgical and pharmacological interventions improve visual outcomes, they do not address the fundamental limitation of irreversible tissue loss. Consequently, regenerative medicine has emerged as a promising approach in ophthalmology, with stem cell–based therapies receiving considerable attention. Among ocular tissues, the lens epithelium has recently been recognized as a potential endogenous source of cells with regenerative capacity. Lens epithelial cells (LECs), which persist throughout life beneath the anterior lens capsule, retain proliferative activity and exhibit notable cellular plasticity. Increasing evidence suggests that LECs can acquire stem cell–like properties under specific biological, molecular, or microenvironmental conditions. This review systematically synthesizes current evidence on the transformation of LECs into stem cell–like or lens progenitor cells and evaluates their potential applications in ocular regenerative therapy. A structured literature search of PubMed and Google Scholar was conducted for English-language studies published between 2020 and 2025, followed by systematic screening and qualitative narrative synthesis. Experimental findings demonstrate that subsets of LECs express pluripotency-associated transcription factors, including SOX2, PAX6, OCT4, and NANOG, and can be induced to form lentoid bodies and lens-like structures in vitro. Lens capsule–based regeneration models and preclinical animal studies further support the capacity of LECs to regenerate lens tissue and partially restore optical function when guided by appropriate molecular cues. Overall, LEC-derived stem cell strategies offer compelling advantages, including reduced immunogenicity, tissue-specific compatibility, and ethical feasibility. Nevertheless, challenges related to controlled reprogramming, safety, and long-term functionality must be addressed to enable successful clinical translation.
THE IMPACT OF SINGLE AMINO ACID MUTATIONS OR ALTERATIONS IN REGULATORY PROTEINS ON THE STEMNESS PROPERTIES OF CANCER CELLS Sartika, Dewi; Laili, Irmatul; Utami, Sri
Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 2 (2025): Journal of Stem Cell Research and Tissue Engineering
Publisher : Stem Cell Research and Development Center, Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jscrte.v9i2.85323

Abstract

Cancer stem cells (CSCs) represent a highly plastic subpopulation within tumors that drives tumor initiation, progression, metastasis, therapeutic resistance, and disease relapse. Although CSC traits were initially attributed to stable genetic hierarchies, growing evidence suggests that cancer stemness is a dynamic and reversible state regulated by precise molecular events at the protein level. This literature review aims to synthesize experimental evidence regarding the impact of single amino acid mutations and alterations in regulatory proteins on the stemness properties of cancer cells. A systematic literature search was conducted using PubMed and Google Scholar to identify original research articles published between 2020 and 2025 that examined protein-level mutations, post-translational modifications, epigenetic regulators, and chromatin remodeling factors in diverse cancer models. The reviewed studies consistently demonstrate that single amino acid substitutions in oncogenic, tumor suppressor, or regulatory proteins can profoundly affect protein conformation, stability, enzymatic activity, and protein–protein interactions. These alterations frequently result in sustained activation of key stemness-associated signaling pathways, including Wnt/β-catenin, Notch, Hedgehog, and STAT3, thereby enhancing self-renewal capacity, stem cell marker expression, tumor-initiating potential, and resistance to conventional therapies. In parallel, dysregulation of post-translational modification systems, such as phosphorylation, ubiquitination, acetylation, and methylation, promotes epigenetic reprogramming and reactivation of embryonic transcriptional programs that favor cellular plasticity. Moreover, protein-level alterations strengthen cancer cell interactions with the tumor microenvironment, increasing responsiveness to hypoxia, inflammatory signals, and metabolic stress. Collectively, these findings highlight protein-level regulation as a central determinant of cancer stem cell maintenance and plasticity, and suggest that effective therapeutic strategies should target aberrant protein stability, post-translational modification machinery, and stemness-related signaling networks to achieve durable clinical responses.

Page 9 of 9 | Total Record : 90

 5   6   7   8   9 

Filter by Year

2017 2025


Reset
Filter By Issues
All Issue Vol. 9 No. 2 (2025): Journal of Stem Cell Research and Tissue Engineering Vol. 9 No. 1 (2025): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 8 No. 2 (2024): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 8 No. 1 (2024): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 7 No. 2 (2023): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 7 No. 1 (2023): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 6 No. 2 (2022): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 6 No. 1 (2022): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 5 No. 2 (2021): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 5 No. 1 (2021): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 4 No. 2 (2020): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 4 No. 1 (2020): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 3 No. 2 (2019): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol 3, No 2 (2019): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 3 No. 1 (2019): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 2 No. 2 (2018): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 2 No. 1 (2018): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING Vol. 1 No. 1 (2017): JOURNAL OF STEM CELL RESEARCH AND TISSUE ENGINEERING More Issue