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All Journal Budapest International Research and Critics Institute-Journal (BIRCI-Journal): Humanities and Social Sciences Narra J Journal of Medical Science; Jurnal Ilmu Medis Rumah Sakit Umum Daerah dr. Zainoel Abidin, Banda Aceh The Journal of Society Medicine (JSOCMED) Narra X Indonesian Journal of Case Reports Trends in Infection and Global Health
Maimun Syukri, Maimun
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Religion Arts Humanities Biochemistry, Genetics & Molecular Biology Chemistry Dentistry Economics, Econometrics & Finance Health Professions Immunology & microbiology Materials Science & Nanotechnology Medicine & Pharmacology Neuroscience Nursing Physics Public Health Social Sciences

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Journal : The Journal of Society Medicine (JSOCMED)

 1 
Role of Glucagon in The Metabolic Response: Review Tona, Azwar Iwan; Syukri, Maimun
Journal of Society Medicine Vol. 2 No. 2 (2023): February
Publisher : CoinReads Media Prima

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.47353/jsocmed.v2i2.62

Abstract

Historically, glucagon is the counter-regulatory hormone of insulin. Glucagon secretion is induced by fasting conditions or hypoglycaemia to increase glucose levels. Glucagon is the dominant product of alpha cells in the islet and was first identified in 1923 during an attempt to purify insulin, where it was identified as a contaminant hyperglycaemia factor. Further research determined that the hyperglycaemic action of glucagon is mediated by increased hepatic glycogenolysis and gluconeogenesis to increase endogenous glucose production. Insulin and glucagon as opposing hormones work together for glycaemic control. Diabetic hyperglycaemia is caused by increased impaired insulin action and inappropriately elevated glucagon levels. This review summarizes an important function of glucagon is its role as a regulator of glucose homeostasis. Increased plasma glucagon levels lead to increased hepatic glucose production. The balance between insulin and glucagon is responsible for maintaining euglycaemia conditions. In conditions of hypoglycaemia, increased glucagon secretion leads to increased hepatic glucose production through a number of cellular mechanisms including suppression of glycogenesis and glycolysis and stimulation of glycogenolysis and gluconeogenesis
Serum Procalcitonin (PCT) Level In Acute Kidney Injury (AKI) In Critical Patients: Acute Kidney Injury (AKI) In Critical Patients Iwan Tona, Azwar; Syukri, Maimun
Journal of Society Medicine Vol. 1 No. 2 (2022): November
Publisher : CoinReads Media Prima

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (427.923 KB) | DOI: 10.47353/jsocmed.v1i2.8

Abstract

Early prediction and avoidance of aggravation of AKI will be useful in identifying patients at risk of developing a higher degree of AKI. Many studies have been conducted to prevent AKI and find biomarkers to predict AKI. Many studies have identified biomarkers of AKI, such as neutrophil-associated lipocalin (NGAL), cystatin C, interleukin-18, and tissue inhibitors of metalloproteinase-2 (TIMP-2). Yet few have investigated the role of PCT as a predictor of AKI. The pathophysiological mechanisms that explain the association between serum PCT and AKI remain unclear. Various inflammatory responses are thought to play a role in the AKI development. PCT acts as a chemoattractant in the ​​inflammation area and causes more monocytes to invade the ​​inflammation. PCT is initially produced in adherent monocytes and then contributes to an increase in circulating PCT by attracting parenchymal cells as they attach directly to activated monocytes. High PCT levels ultimately act as a direct chemoattractant to monocyte counts.
Co-Authors Agung Pranata Al-Gunaid, Seba Al-Muchtari, Teuku AZ. Arief S. Kartasasmita Ayulinda, Arianisah P. Beočanin, Anđelija Darmawi . Erianza, Dava Hanifah Yusuf Harapan Harapan Hasan, Desi Salwani Hasan, Maryatun Herlina Dimiati, Herlina Ichsan Ichsan Idroes, Ghifari Maulana Iwan Tona, Azwar Jayawarsa, A.A. Ketut Kamil, Qatrunnada Kurnia F. Jamil Kurniawan , Ferry D. Liansyah, Tita M. Maelani, Imelda Mahmud , Nissa N. Marty Mawarpury Mudatsir , Mudatsir Mudatsir Mudatsir Muhammad Yani, Muhammad Nurjannah Nurjannah Oktari, Rina S. Putri, Widia Rachmad Suhanda Redwood-Campbell, Lynda Renaldi, Teuku Rima Novirianthy Rinaldi Idroes Ronny Lesmana Rudi Kurniawan Samsul Anwar Sari, Etika Soehartati Gondhowiardjo Sofyan, Sarwo E. Syamsul Rizal Teuku Rizky Noviandy Tjahjono D. Gondhowiardjo Tona, Azwar Iwan Ummu Balqis Usman, Said Yusni Yusni Yusni Yusni Zaini, Lia M.