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Journal : Jurnal Neuroanestesi Indonesia

Hipotensi Berat Intraoperatif Tiba-Tiba saat Kraniotomi Pengangkatan Meningioma Bisri, Dewi Yulianti; Habibi, Muhammad; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 12, No 2 (2023)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24244/jni.v12i2.550

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Tekanan darah adalah perkalian cardiac output (CO) dengan systemic vascular resistance (SVR) dan CO ditentukan oleh stroke volume dan frekuensi denyut jantung. Seorang wanita usia 61 tahun, berat badan 49 kg, tinggi badan 154 cm, datang di rumah sakit Santosa Bandung Central dengan keluhan benjolan pada bagian belakang kepala sejak 3 tahun lalu dan semakin lama semakin bertambah besar. Tidak terdapat tanda-tanda peningkatan tekanan intrakranial dan defisit neurologis lainnya, di diagnosa meningioma dengan hipertensi, dilakukan pengangkatan tumor dalam posisi telungkup. Induksi dengan propofol, fentanyl, vecuronium bromida, lidokain, rumatan anestesi dengan sevofluran-oksigen/udara dan propofol serta vecuronium kontinyu. Intraoperatif terjadi 2 kali penurunan tekanan darah yang terjadi tiba-tiba, bradikardia dan desaturasi. Diberikan cairan dengan ringerfundin, gelofusin 1 L, darah Pack Red Cell (PRC) 2 unit, sulfas atropin, efedrin dan dilanjutkan dengan norepinephrine. Pascabedah dirawat 1 hari di Intensive Care Unit (ICU) dan kemudian 1 hari lagi di High Care Unit (HCU), kemudian pindah ke ruang perawatan biasa dan dirawat selama 3 hari sebelum dipulangkan dari rumahsakit. Penurunan tekanan darah hebat disertai bradikardia berat dan desaturasi tidak mungkin disebabkan karena perdarahan, tapi lebih mungkin dihubungkan dengan gangguan pada jantung. Terapi yang dilakukan dengan mengembalikan parameter tersebut ke nilai fisiologis sesegera mungkin. Sebagai simpulan, penurunan tekanan darah tiba-tiba disertai bradikardia dan desaturasi kemungkinan disebabkan karena terjadinya Bezold-Jarisch Reflexes (BJR).Sudden Intraoperative Severe Hypotension during Craniotomy of Meningioma RemovalAbstractBlood pressure is the multiplication of cardiac output (CO) with systemic vascular resistance (SVR) and CO determined by stroke volume and heart rate frequency. A 61-year-old woman, weight 49 kg, height 154 cm, came to Santosa Hospital Bandung Central in with complaints of a lump on the back of the head that has gotten bigger since 3 years ago and the longer it gets bigger. There were no signs of increased intracranial pressure and other neurological deficits, diagnosed with meningioma with hypertension, tumor removal was carried out in a prone position. Induction with propofol, fentanyl, vecuronium bromide, lidocaine, anesthetic treatment with sevoflurane-oxygen/air and propofol and continuous vecuronium. Intraoperative suddenly occurs 2 times decrease in blood pressure, bradycardia and desaturation. Given liquid with ringerfundin, gelofusin 1 L, blood pack red cells (PRC) 2 units, sulfas atropine, ephedrine and continued with norepinephrine. Post-dissected treated 1 day in the Intensive Care Unit (ICU) and then another 1 day in the High Care Unit (HCU), then moved to the ward and was treated for 3 days before being discharged from the hospital. Severe drops in blood pressure accompanied by severe bradycardia and desaturation are unlikely to be caused by bleeding, but are more likely to be associated with heart disorders. Therapy is carried out by returning these parameters to physiological values as soon as possible. As conclusion, a sudden drop in blood pressure accompanied by bradycardia and desaturation may be due to the occurrence of Bezold-Jarisch Reflexes (BJR).
Perioperative Management Patients with Meningioma C1-2 Bisri, Dewi Yulianti; Indrayani, Ratih Rizki; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 13, No 1 (2024)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24244/jni.v13i1.587

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Removal of spinal meningiomas in cervical 1 and 2 has several problems, especially regarding the respiratory and cardiovascular systems. A woman, 33 years old, admit Santosa Bandung Central Hospital with complaints of weakness in her left hand and both legs since 4 months ago. Weight 50 kg, height 155 cm, blood pressure 146/102 mmHg, pulse rate 105 x/min, temperature 36.50C, SpO2 98% with room air. At diagnosis of cervical myelopathy due to space occupying lesion (SOL) intradural meningioma suspect. Induction of anesthesia with fentanyl 100 mcg, propofol 60 mg, rocuronium 40 mg, ventilated with 100% oxygen and sevoflurane 3 vol% (1.5 MAC), before laryngoscopy-intubation repeated half the initial dose of propofol. The patient is intubated in an in-line position. Anesthesia maintenance with sevoflurane 1 vol%, oxygen: air 50%, dexmedetomidine continuous 0.4 mcg/kg per hour, and continuous rocuronium 10 mcg/kgBW/min. Ventilation is controlled with a tidal volume of 360 ml, frequency 14 times/min. Then the patient is positioned in the prone position. Post-surgery is admitted to the ICU and day 5 the patient can be discharged from the hospital. The effects of C12 spinal cord tumors can affect the respiratory and cardiovascular systems. Surgical trauma can aggravate the injury before recovery occurs, so it is necessary to do ventilation assistance and cardiovascular support before recovery.
Apa yang Baru dalam Neuroanestesi untuk Cedera Otak Traumatik? Bisri, Dewi Yulianti; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 11, No 1 (2022)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (2422.671 KB) | DOI: 10.24244/jni.v11i1.447

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Konsep dasar Neuroanestesi Neuro Critical Care disebut sebagai ABCDE neuroanestesi. Early Brain Injury (EBI) dahulu dikenal sebagai cedera otak primer. Pada EBI terjadi hilangnya autoregulasi, hilangnya integritas barier darah otak. Adanya Trias Cushing menunjukkan adanya hipertensi intrakranial. Target tekanan darah pada cedera otak traumatik (traumatic brain injury/TBI) adalah hindari tekanan darah sistolik 110 mmHg, pertahankan tekanan perfusi otak (cerebral perfusion pressure/CPP) 60-70 mmHg, target pengaturan PaCO2 adalah normokarbia, PaCO2 3540 mmHg, penggunaan profilaksis phenytoin atau valproate tidak direkomendasikan untuk mencegah late post traumatic seizure (late PTS). Masih perlu menganalisa terapi decompressive craniectomy (DECRA) dibandingkan dengan terapi medikal kontinyu untuk peningkatan tekanan intrakranial (intracranial pressure/ICP) yang refrakter setelah TBI. Anestesi umum untuk pasien dengan TBI berat lebih baik dengan total intravenous anesthesia (TIVA), pemberian cairan harus mempertimbangkan osmolaritas cairan tersebut. Pada konsep yang baru, pada pasien dengan peningkatan ICP, konsentrasi anestetika volatil harus dibatasi sampai 0,5 MAC. Target gula darah adalah normoglikemia. Hipotermi profilaksis atau terapeutik tampaknya tidak memiliki tempat dalam pengelolaan cedera otak berat.What is New in Neuroanesthesia for Traumatic Brain Injury?AbstractThe basic concept of Neuroanesthesia and Neuro Critical Care is referred to as ABCDE neuroanesthesia. Early Brain Injury (EBI) was formerly know as primary brain injury. In EBI, there is loss of autoregulation, loss of integrity of the blood-brain barriere. The presence of Cushings triad indicates the presence of intracranial hypertension. Blood pressure target in traumatic brain injury is to avoid systolic blood pressure less than 110 mmHg, maintain cerebral perfusion pressure (CPP) 60-70 mmHg, target PaCO2 regulation is normocarbia, PaCO2 35-40 mmHg, prophylactic use of phenytoin or valproate is not recommended to prevent late post traumatic seizure (late PTS). Still need to analyse decompressive craniectomy (DECRA) compare with continuous medical therapy for refractory increase in intracranial pressure (ICP) after TBI. General anesthesia for patient with severe TBI is better with total intravenous anesthesia (TIVA), administration of fluids must consider the osmolarity of the fluid. In a new concept in patient with elevated ICP, volatile anesthetic concentaratiom should be limited to 0.5 MAC.Blood glucose target is normoglycemia. Prophylactic and therapeutic hypothermia not recommended for severe traumatic brain injury management.
Tantangan dalam Menjaga Cerebral Perfusion Pressure (CPP) yang Aman pada Cedera Otak Traumatik Bisri, Dewi Yulianti; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 12, No 1 (2023)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24244/jni.v12i1.531

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Tekanan perfusi serebral (cerebral perfusion pressure/CPP) adalah gradien tekanan yang mendorong pengiriman oksigen ke jaringan serebral, perbedaan antara tekanan arteri rata-rata (MAP) dan tekanan intrakranial (ICP), CPP = MAP-CVP atau CPP = MAP ICP jika ICPCVP. Tekanan perfusi serebral harus dipertahankan dalam rentang yang sempit karena tekanan yang terlalu rendah dapat menyebabkan jaringan otak menjadi iskemik, dan bila terlalu tinggi dapat meningkatkan tekanan intrakranial. Tekanan perfusi serebral yang aman adalah antara 60-80 mmHg, tetapi nilai-nilai ini dapat bergeser ke kiri atau kanan tergantung pada fisiologi individu pasien. Karena CPP adalah ukuran yang dihitung, MAP dan ICP harus diukur secara bersamaan, paling sering dengan cara invasif. Ketika terjadi cedera otak, kapiler serebral bisa menjadi "bocor" atau lebih permeabel terhadap air. Selain itu, pembuluh darah serebral dapat melebar dalam respon terhadap cedera jaringan otak, hipoksemia, hiperkarbia, asidosis, atau hipotensi. Jika tekanan darah meningkat, peningkatan CPP dapat menyebabkan peningkatan aliran darah serebral. Tujuan yang disarankan dari CPP berdasarkan pedoman dari Brain Trauma Foundation adalah 50-70 mmHg. Menargetkan CPP tinggi 70 mmHg belum terbukti bermanfaat pada pasien dengan cedera otak traumatik dan dikaitkan dengan peningkatan risiko sindrom gangguan pernapasan akut (ARDS).Challenges in Maintaining Safe Cerebral Perfusion Pressure (CPP) in Traumatic Brain InjuryAbstractCerebral perfusion pressure (CPP) is the net pressure gradient that drives oxygen delivery to cerebral tissue. It is the difference between the mean arterial pressure (MAP) and the intracranial pressure (ICP), CPP = MAP-CVP or CPP =MAP ICP if ICPCVP. Cerebral perfusion pressure must be maintained within narrow limits because too litle pressure could cause brain tissue become ischemic, and too much could raise intracranial pressure. The normal range lies between 60 and 80 mmHg, but these values can shift to the left or right depending on individual patient physiology. As CPP is a calculated measure, MAP and ICP must be measured simultaneously, most commonly by invasive means. When brain injury occurs, cerebral capillaries can become leaky or more permeable to water. In addition, cerebral blood vessels may dilate in respons to brain tissue injury, hypoxemia, hypercarbia, acidosis, or hypotension. If blood pressure becomes elevated, the increased CPP can lead to increased cerebral blood flow. The recommended goal of CPP per the Brain Trauma Foundation (BTF) guideline is 50-70 mmHg. Targeting high CPP 70 mmHg has not been shown to be beneficial in patient with traumatic brain injury and is associated with an increased risk of acute respiratory distress syndrome (ARDS).
Konsep GHOST- CAP untuk Proteksi Otak Perioperatif pada Cedera Otak Traumatik Bisri, Dewi Yulianti; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 11, No 2 (2022)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (463.3 KB) | DOI: 10.24244/jni.v11i2.472

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Kerusakan otak adalah cedera yang menyebabkan rusak atau memburuknya sel otak yang disebabkan oleh berbagai kondisi seperti trauma kepala, pasokan oksigen yang tidak memadai, infeksi atau perdarahan intrakranial. Proteksi otak adalah intervensi terapeutik preemptif untuk memperbaiki outcome neurologik pada pasien yang berisiko terjadinya iskemi serebral, sedangkan resusitasi otak merujuk ke intervensi terapeutik yang dimulai setelah terjadinya iskemi. Targetnya adalah terapi iskemi dan mengurangi cedera neuron. Kerusakan otak perioperatif adalah salah satu komplikasi merugikan yang paling serius dari operasi dan anestesi, mengakibatkan defisit neurologis baru pasca operasi. Konsep GHOST-CAP, yang merupakan akronim dari Glycemia, Hemoglobin, Oxygen, Sodium, Temperature, Comfort, Arterial Pressure dan PaCO2, digunakan pada periode pascaoperatif. G: target level glukosa antara 80 dan 180?mg/dL. H: hemoglobin, ambang batas 79g/dL. O: oksigen, targetkan SpO2 antara 94 dan 97%. S: konsentrasi sodium mempengaruhi volume otak, kadar sodium hingga 155?mEq/L dapat ditoleransi. T: temperatur diatur untuk mengoptimalkan fungsi seluler, tetapi suhu inti ?38,0C harus dihindari. C: kenyamanan pasien (comfort), termasuk kontrol nyeri, agitasi, kecemasan, dan menggigil. A: tekanan darah arteri adalah penentu utama aliran darah otak (CBF), pertahankan tekanan arteri rata-rata (MAP) 80?mmHg dan tekanan perfusi otak (CPP) 60?mmHg. P: perubahan akut PaCO2 menyebabkan perubahan CBF, maka PaCO2 tidak boleh kurang dari 35 mmHg. Tulisan ini mengkaji konsep GHOST-CAP untuk proteksi otak perioperatif, apakah cukup memadai atau harus ditambah.GHOST-CAP Concept for Perioperative Brain Protection in Traumatic Brain InjuryAbstractBrain damage is an injury that causes damage or worsening of brain cells caused by various conditions such as head trauma, inadequate oxygen supply, infection or intracranial hemorrhage. Brain protection is a preemptive measure of therapeutic interventions to improve neurological outcomes in patients at risk of cerebral ischemic, while brain resuscitation refers to therapeutic interventions that begin after the occurrence of ischemic. The target is ischemic therapy and reducing neuronal injury. Perioperative brain damage is one of the most serious adverse complications of surgery and anesthesia, resulting in new postoperative neurological deficits. The concept of GHOST-CAP, an acronym for Glycemia, Hemoglobin, Oxygen, Sodium, Temperature, Comfort, Arterial Pressure and PaCO2, can be used in the postoperative period. G: The target level of glucose is between 80 and 180 mg/dL. H: hemoglobin threshold is 7-9 g/dL. O: oxygen, target SpO2 between 94 and 97%. S: Sodium concentration affects brain volume, sodium levels up to 155 mEq/L are tolerable. T: temperatures regulated to optimize cellular function, but core temperatures 38.0C should be avoided. C: patient comfort, including pain control, agitation, anxiety, and chills. A: Arterial blood pressure is the main determinant of cerebral blood flow (CBF), maintaining an mean arterial pressure (MAP) of 80 mmHg and cerebral perfusion pressure (CPP) of 60 mmHg. P: Acute changes in PaCO2 cause CBF changes, PaCO2 not to be less than 35 mmHg. This paper examines the GHOST-CAP concept for perioperative brain protection, whether adequate or should be supplemented
Thiopental-Dexmedetomidine as Adjuvant Anesthesia for Craniotomy Tumor Removal: A Case Report Bisri, Dewi Yulianti; Nuryanda, Dian; Alifahna, Muhammad Rezanda; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 13, No 2 (2024)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24244/jni.v13i2.595

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Brain tumor surgery requires special anesthesia to get a slack brain and perform perioperative brain protection. The selected anesthetic drugs and adjuvants have the ability of anesthesia sparring effect and have a brain protective effect. Not many have done the combination of thiopental adjuvant with dexmedetomidine. The purpose of this case report is to see the effect of the combination of thiopental with dexmedetomidine as an adjuvant anesthesia on hemodynamics and slack brain and successful removal of brain tumors. A woman, 32 years old, with meningiomas had surgery to remove a brain tumor at Santosa Bandung Central Hospital. Preoperative examination showed blood lab results within normal limits, the presence of large meningioma and midline shift. Induction of anesthesia with thiopental 5 mg/kgBW, rocuronium bromide 0.9 mg/kgBW, fentanyl 3 mcg/kg and anesthetic maintenance with sevoflurane below 1.5 MAC, oxygen/air, continuous rocuronium 0.5 mg/kgBW/hour, thiopental and continuous dexmedetomidine. The anesthetic adjuvant used was thiopental 1-3 mg/kg/hour and continuous dexmedetomidine 0.40.7 mcg/kg/hour. A slack brain is obtained, and 90% of the tumor could be removed, and transfused during surgery 4 units pack red cells (PRC), crystalloid liquid as much as 2,500 cc, and colloidal fluid as much as 2,000 cc. The length of surgery is 11 hours. Post-surgery was treated in the ICU for 5 days, then moved to the ward for 2 days then the patient could be discharged from the hospital. The use of thiopental and dexmedetomidine continuously can produce slack brain and almost the entire tumor can be removed.
Kehilangan Penglihatan Pascabedah Laminektomi dalam Posisi Prone Sumardi, Fitri Sepviyanti; Bisri, Dewi Yulianti; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 1, No 4 (2012)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (238.038 KB) | DOI: 10.24244/jni.vol1i4.193

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Kehilangan penglihatan pascabedah nonocular sangatlah jarang, akan tetapi, harus dipertimbangkan sebagai komplikasi pascabedah yang tidak diinginkan. Kehilangan penglihatan unilateral atau bilateral secara tiba-tiba yang terjadi pascaanestesia umum telah dilaporkan dan dihubungkan dengan berbagai sebab diantaranya syok haemorhagik, hipotensi, hipotermia, kelainan koagulopathi, trauma langsung, emboli, dan penekanan pada bola mata yang berlangsung lama. Kasus: seorang laki-laki berusia 60 tahun dengan diagnosis radiculopathi setinggi L4 e.c protunded disc L4-5 dan L5-S1 yang menjalani laminektomi dalam anestesi umum. Terjadi komplikasi pascabedah berupa kehilangan penglihatan yang disertai opthalmoplegi total akibat oklusi arteri retina sentralis, sindroma kompartemen orbita akut, dan pseudotumor tipe miositis.Visual Loss after Prone Lumbar Spine SurgeryVisual loss after nonocular surgery is a rare but devastating postoperative complication. Sudden unilateral or bilateral visual loss occurring after general anesthesia has been reported and attributed to various causes including haemorrhagic shock, hypotension, hypothermia, coagulopathic disorders, direct trauma, embolism, and prolonged compression of the eyes. Case: a man, 60 years-old with diagnosis radiculopathy in level L4 e.c. protunded disc L4-5 and L5-S1 who required laminectomy in general anesthesia. An unusual complication of visual loss with total opthalmoplegy was caused by central retina artery occlusion, acute ischemic orbital compartement syndrome, and pseudotumor type myositis.
Pressure Reactivity Index (PRx): A Concept to Optimize Cerebral Perfusion Pressure in Traumatic Brain Injury Uhud, Akhyar Nur; Bisri, Dewi Yulianti; Jasa, Zafrullah Khany; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 14, No 2 (2025)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.24244/jni.v14i2.693

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Two common factors contributing to poorer outcomes in TBI patients are high intracranial pressure (ICP) and low cerebral perfusion pressure (CPP). These two factors constitute a vicious circle that will have a negative impact on TBI patients. An increase in ICP will cause a decrease in CPP, while a reduction in CPP will cause ischemia, which will worsen the high ICP. However, increasing the CPP by increasing MAP will not help the situation; in fact, it may worsen the impact due to impairment of cerebral autoregulation (CA). Therefore, it is critical to manage TBI patients with an ideal CPP. Pressure reactivity index (PRx) is a measurement of the linear relationship between the mean arterial pressure (MAP) and ICP. A positive correlation between ICP and MAP indicates an impairment of CA, which suggests a suboptimal CPP value. The basis of PRx theory is that the rise, because of the presence of CA, an increase in MAP should not be followed by the rise in ICP because there is a compensatory effect in the form of a decrease in cerebral blood volume, so that ICP does not increase. That being said, this mechanism will not work when the limit of autoregulation is exceeded. Based on PRx and CPP, an optimal CPP could be obtained by using a U-shaped curve. The outcomes of TBI patients can be enhanced by treating them according to their optimal CPP (CPPopt).
Tatalaksana Anestesi pada Pasien Anak dengan Cystic Craniopharyngioma yang Menjalani Gamma Knife Radiosurgery Widiastuti, Monika -; Halimi, Radian Ahmad; Fuadi, Iwan; Rahardjo, Sri; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 10, No 2 (2021)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (3082.611 KB) | DOI: 10.24244/jni.v10i2.353

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Kraniofaringioma merupakan tumor otak jinak dengan karakteristik kistik dan kalsifikasi, yang letaknya dikeliingi oleh stuktur vital sehingga sulit untuk dilakukan reseksi total. Terapi kombinasi dengan Gamma knife radiosurgery (GKRS) merupakan pilihan terapi paling tepat. Prosedur GKRS yang kompleks meliputi banyak tahap dengan durasi 6-10 jam, memerlukan pemberian anestesi pada pasien yang tidak kooperatif. Kasus ini mengenai pasien anak perempuan berusia 4 tahun dengan cystic craniopharyngioma. Pasien dengan keluhan pandangan mata buram, dari pemeriksaan fisik didapatkan papil atrofi bilateral. Hasil magnetic resonance imaging (MRI) menunjukkan massa tumor yang menekan kelenjar hipofise inferior. Pasien menjalani prosedur GKRS selama 6 jam dengan anestesi sedasi sedang menggunakan Propofol 75 mcg/kg/menit. Hemodinamika selama prosedur stabil, tidak terjadi komplikasi. Pemilihan teknik anestesi dapat berupa anestesi umum atau sedasi, tergantung pada kondisi pasien, dokter anestesi, operator, dan fasilitas. Pertimbangan anestesi pada GKRS antara lain prosedur dilakukan di luar kamar operasi, durasi panjang, transportasi ke beberapa tempat seperti radiologi dan cathlab, imobilisasi kepala untuk mencegah pergeseran frame stereotaktik, pasien sendiri di dalam ruang radiasi, prinsip neuroanestesi pediatrik.Anesthetic Management of Pediatric Patient with Cystic Craniopharyngioma Underwent Gamma Knife RadiosurgeryAbstractCraniopharyngioma is a benign tumor characterized by cystic and calcification, surrounded by vital structures therefor it is difficult to perform total tumor resection. Combination with Gamma knife radiosurgery (GKRS) is the best treatment option. The complexities of GKRS consisting of several phases lasts for 6-10 hours. Anesthesia is needed for uncooperative patients. This is a case of a 4-year-old girl with cystic craniopharyngioma. The patient had chief complaint of blurry vision, physical examinations revealed bilateral papil atrophy. Result of MRI showed tumor mass compressing inferior hypophyse. Patient underwent the procedure under moderate sedation with Propofol at 75 mcg/kg/min for 6 hours. Intraoperative hemodynamic condition was stable without adverse events. Choice of anesthesia either general anesthesia or sedation, depends on the condition of patient, considerations from anesthesiologist dan neurosurgeon, dan availability of facilities. Unique considerations for GKRS are; a non-operating room anesthesia, long duration, transportation to other units such as radiology and cathlab, head of the patients need to be immobilized to prevent frame displacement, the patient will be alone in the treatment room, and principles of pediatric anesthesia and neuroanesthesia.
Pertimbangan Anestesi Perioperatif untuk Pasien Bedah Saraf dengan Covid-19 Bisri, Dewi Yulianti; Bisri, Tatang
Jurnal Neuroanestesi Indonesia Vol 10, No 1 (2021)
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Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (2788.911 KB) | DOI: 10.24244/jni.v10i1.324

Abstract

Coronavirus yang baru, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) pertamakali timbul di Wuhan, Provinsi Hubei Cina, pada bulan Desember 2019, dan menyebar dengan cepat ke seluruh dunia ke lebih dari 190 negara. Pasien harus ditapis untuk covid-19 menggunakan kombinasi riwayat penyakit, computed tomography (CT) dada, dan pemeriksaan real time quantitative polymerase chain reaction (RT-qPCR) bergantung kebijakan institusinya. Keluhan neurologis seperti dizziness, headache, hypogeusia dan hyposmia, sering (36%) pada pasien covid-19. Encefalopati dan perubahan status mental juga terjadi pada pasien yang telah terinfeksi dengan virus SARS-CoV-2. Penyakit serebrovaskuler lebih sering pada covid-19 yang berat; acute ischemic stroke telah dilaporkan pada 5,7% dan gangguan kesadaran pada 15% pasien. Tindakan pembedahan rutin kranial dan spinal aman untuk dilakukan. Operasi endoscopic endonasal tidak aman dan harus dihindari. Ekstubasi setelah anestesi umum bila memungkinkan dilakukan di ruangan tekanan negatif, personil tetap memakai alat pelindung diri (APD) level 3. Harus dihindari pasien batuk saat ekstubasi. Setelah ekstubasi, pasang oksigen binasal, dan pasien harus memakai masker bedah dan aliran oksigen tinggi harus dihindari (berikan 6L/menit) untuk menghindari terjadinya aerosolisasiPerioperative Anesthesia Consideration for Neurosurgical patients with Covid-19AbstractThe novel coronavirus, severe acute respiratory syndrome coronavirus 2 (SARS-CoV-2) emerged in Wuhan, Hubei province China, in December 2019, and spread fast to all the world more than 190 countries. Patients should be screened for covid-19 using a combination of history, computed tomography (CT) chest, and real time quantitative polymerase chain reaction (RT-qPCR) testing depending on institutional policies. Neurological symptom as dizziness, headache, hypogeusia and hyposmia, common (36%) at covid-19 patient. Encephalopaty and changed mental status exist in patient infected by SARS-CoV-2 virus. Cerebrovascular diseases more in severe covid-19; acute ischemic stroke had reported in 5.7% and altered level of consciousnes in 15% patient. Surgical measuremet cranial and spinal rutine is safe, endoscopic endonasal surgery not safe and must be avoided. Extubation after general anesthesia if possible do air negative pressure room, and personil still use personal protection equipment (PPE) level 3. Must be avoid patient cough during extubation. After extubation, give oxygen nasal canule, surgical mask, and high flow oxygen (give 6 L/min) avoided given the risk of aerosolization
Co-Authors , Rizki , Suwarman - Irwan, - A. Himendra Wargahadibrata A. Muthalib Nawawi Agus Junaidi Aini, Quratul Akbar, Ieva B Alifahna, Muhammad Rezanda Andie Muhari Barzah, Andie Muhari Ardi Zulfariansyah Arief Kurniawan Bambang Suryono, Bambang Christanto, Sandhi Christanto, Sandhi christiana, monica Dedi Fitri Yadi Dewi Yulianti Bisri Diana C. Lalenoh, Diana C. Diana Lalenoh Erias, Muhammad Erwin Pradian Ezra Oktaliansah Firdaus, Riyadh Firdaus, Riyadh Fithrah, Bona Akhmad Fithrah, Bona Akhmad Fitri Sepviyanti Sumardi Giovanni, Cindy Giovanni, Cindy Hamzah, Hanzah Hermawanto, Agung Hindun Saadah, Hindun Ida Bagus Krisna Jaya Sutawan Ieva B. Akbar Ike Sri Redjeki Indrayani, Ratih Rizki Iwan Abdul Rachman Iwan Fuadi Jasa, Zafrullah Khany Kusuma Harimin, Kusuma Laksono, Buyung Hartiyo Lalenoh, Diana Christine Lalenoh, Diana Christine Limawan, Michaela Arshanty M. Dwi Satriyanto M. Erias Erlangga, M. Erias M. Sofyan Harahap Mariko Gunadi Martinus, Fardian Martinus, Fardian MM Rudi Prihatno, MM Rudi Muh. Rumli Ahmad Muhamad Adli Boesoirie, Muhamad Adli Muhammad Habibi Ningsih, Diana Fitria Ningsih, Diana Fitria Noer Rochmah, Elly Nugraha, Ade Aria Nugraha, Ade Aria Nuryanda, Dian Oetoro, Bambang J. Oetoro, Bambang J. Okatria, Ahmado Pontjosudargo, Fransiska Ambarukmi Priyadi, Hendri Putri, Andika C. Putu Pramana Suarjaya Radian Ahmad Halimi Rahmadsyah, Teuku Rahordjo, Sri Rasman, Marsudi Rasman, Marsudi Reza Widianto Sudjud Rose Mafiana Rovina Ruslami, Rovina Ruby Satria Nugraha Ruli Herman Sitanggang Saleh, Siti Chasnak Saleh, Siti Chasnak Saputra, Tengku Addi Saputra, Tengku Addi SATRIYAS ILYAS Septiani, Gusti Ayu Pitria Soefviana, Stefi Berlian Sri Rahardjo Stella, Angela Subekti, Bambang Eko Subekti, Bambang Eko Suryaningrat, IGB Susanto, Bahtiar Sutanto, Sigit Sutanto, Sigit Suwarman Suwarman, Suwarman Suwarman, S Suwarman, S Syafruddin Gaus Thayeb, Srilina Theresia C. Sipahutar Theresia Monica Rahardjo Uhud, Akhyar Nur Widiastuti, Monika - Wirawijaya, Dear Mohtar Wirawijaya, Dear Mohtar Wirawijaya Wullur, Caroline Wullur, Caroline Yunita Susanto Putri Zaka Anwary, Army