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All Journal BALI MEDICAL JOURNAL (BMJ) Medicina E-Jurnal Medika Udayana E-Jurnal Agroekoteknologi Tropika (Journal of Tropical Agroecotechnology) Sari Pediatri JURNAL HAMA DAN PENYAKIT TUMBUHAN TROPIKA Paediatrica Indonesiana CHEESA: Chemical Engineering Research Articles ISM (Intisari Sains Medis) : Jurnal Kedokteran Distilat: Jurnal Teknologi Separasi Jurnal Rekayasa Proses
I Made Arimbawa
Department Of Child Health, Medical Faculty, Udayana University, Indonesia
Agriculture, Biological Sciences & Forestry Biochemistry, Genetics & Molecular Biology Chemical Engineering, Chemistry & Bioengineering Chemistry Health Professions Medicine & Pharmacology

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OSTEOPOROSIS IN A SEVENTH YEAR OLD BOY WITH ACUTE LYMPHOBLASTIC LEUKEMIA (Case Report) Salim, H; Ariawati, K.; Bikin-Suryawan, W.; Arimbawa, and I. M.
BALI MEDICAL JOURNAL Vol 2 No 3 (2013): Vol.2, No.3, September-December 2013
Publisher : BALI MEDICAL JOURNAL

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Abstract

Osteoporosis in children is rare and usually secondary to an underlying disease process whose diagnosis may be difficult to detect. It can be a manifestation of acute lymphoblastic leukemia (ALL). About a-quarters of children with ALL will have signs and symptoms of osteoporosis. We report the case of a seventh-year-old boy with back pain. His antero-posterior pelvic radiograph showed the osteoporotic bone. The bone age study revealed six-year-old bone. Review of peripheral blood smear showed normochromic anemia with thrombocytopenia. Immunophenotyping of peripheral blood revealed no dominant marker was seen, but the bone marrow aspiration confirms the diagnosis of ALL (L2).
SECKEL SYNDROME IN A - 2 YEAR OLD GIRL Yulius Malino, Imanuel; Arimbawa, Made; Suryawan, Bikin
Medicina Vol 44 No 1 (2013): Januari 2013
Publisher : Medicina

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Abstract

Seckel syndrome is a frequent autosomal recessive that cause microcephalic osteodysplastic dwarfisms. It characterized with proportionate dwarfism of prenatal onset, dysmorphic features including severe microcephaly and “bird-headed” like appearance, mental retardation and autosomal recessive inheritance, becausedefect on chromosome 3q22.1-q24 (SCKL1), chromosome 18p11.31-q11.2 (SCKL2) and chromosome 14q23 (SCKL3). We reported, 2 years, 8 months female with intrauterine growth restriction, severe proportionately short stature, a “bird-headed” profile with receding forehead, large eyes, breaks like protrusion of the nose, narrow face, receding lower jaw and micrognathia and from bone survey we found a retarded bone age on which was appropriate for 6 months of age.There was no other systems dissorder have been found and no specific medication has been given. Patient was hospitalized to establish diagnosis and was dischargedafter ten days of hospitalization
MIXED GONADAL DYSGENESIS IN A SEVEN MONTH OLD BABY Krishna Levina, Made Ayu; Arimbawa, I Made
Medicina Vol 45 No 1 (2014): Januari 2014
Publisher : Medicina

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Abstract

Mixed gonadal dysgenesis is a very rare case with genital ambiguity as a clinical manifestation.Diagnosis of this condition is emerging due to proper gender assignment and  prompt treatment toachieve optimal physical and psychologic development. We reported a genital ambigous in a 7 month old baby, who was referred with enlargement of clitoris, an unpalpable testis, but with a highconcentration testosteron serum level, an uterus from genitography, and a mosaic karyotype 45,X/46,XY. The working diagnosis of this baby is mixed gonadal dysgenesis. Patients is being evaluated by a multidisciplinary team and planned having laparoscopy.  [MEDICINA 2014;45:52-57]
HIPERPLASIA ADRENAL KONGENITAL (HAK) KLASIK SIMPLE VIRILIZING PADA ANAK UMUR 3 TAHUN Indradjaja, Alice; Bikin Suryawan, I Wayan; Arimbawa, I Made
Medicina Vol 45 No 1 (2014): Januari 2014
Publisher : Medicina

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Abstract

Hiperplasia adrenal kongenital merupakan salah satu dari kelompok kelainan genetik akibat defisiensi enzim yang diperlukan untuk biosintesis steroid di korteks kelenjar adrenal. Bentuk kelainan hiperplasia adrenal kongenital yang tersering adalah defisiensi enzim 21-hidroksilase (21OHD) hingga mencapai 90% kasus. Kelainan utama pada pasien dengan defisiensi enzim 21-hidroksilase adalah kegagalan sintesis kortisol secara adekuat. Defisiensi 21-hidroksilase klasik tipe virilisasi sederhana menyebabkan genitalia ambigu pada bayi perempuan. Dilaporkan sebuah kasus hiperplasia adrenal kongenital klasik tipe virilisasi sederhana pada anak perempuan usia tiga tahun. Pasien dirujuk ke Poliklinik anak RSUP Sanglah Denpasar dengan keluhan utama pembesaran dan pemanjangan klitoris yang progresif disertai tumbuhnya bulu pubis.  Pasien lahir dengan genitalia ambigu. Pasien didiagnosis defisiensi 21-hidroksilase berdasarkan hasil pemeriksaan kadar progesteron 17-OH >1.200ng/dl dan pemeriksaan fisik didapatkan prader derajat III. Pada pemeriksaan usia tulang menunjukkan usia tulang yang melebihi umurnya, USG abdomen dalam batas normal dengan hasil analisis kromosom 46,XX. Pasien tidak pernah mengalami krisis adrenal selama 3 tahun dan menjalani tindakan pembedahan pada usia 3 tahun. Keluarga pasien diberikan konseling, dilakukan monitor  berkala pada pasien dan terapi hidrokortison. Prognosis pada pasien ini baik. [MEDICINA 2014;45:58-64].
PREVALENCE AND ASSOCIATION OF GLYCOSURIA WITH SEVERAL RISK FACTORS FOR DIABETES MELLITUS IN PRIMARY SCHOOL CHILDREN IN BALI Sutawan, Ida Bagus Ramajaya; Suryawan, I Wayan Bikin; Arimbawa, I Made
Medicina Vol 45 No 3 (2014): September 2014
Publisher : Medicina

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Abstract

Nowadays, diabetes mellitus (DM) is often found in children. One of important screening for DM isurine examination to determine glycosuria. The current data on the prevalence of glycosuria in childrenin Indonesia, including Bali is very limited.The purpose of this study is to determine the prevalence ofglycosuria in primary school children in Bali and the association of several risk factors for DM on theoccurrence of glycosuria.A cross sectional study of children aged 6-12 years in several primary schoolsin Bali was done. Samples were taken using multistage random sampling technique. Glycosuria wasdetermined by urine reagent stripsand determination of risk factors for DM was by questionnaire andphysical examination. Total of 1020 children were examined in this study. Fiftyone point five percentsof samples were male, obesity was found in 36.8% of samples. Family history of DM was found in 2.6%of samples. This study found 17 cases of glycosuria with a prevalence of 1.7%. Several factors werefound associated with glycosuria, including obese with OR 5.32 (95% CI 1.65 to 17.16; P=0.005), familyhistory of DM with OR 12.98 (95% CI 3.11 to 54.27; P<0.001), and male gender with OR 6.05 (CI 95%1.34 to 27.33; P=0.019).Based on this study, it could be concluded thatthe prevalence of glycosuria wasquite high at primary school children in Bali. Therefore glycosuria screening in children needs to bedone,  especially  in  those with  risk  factors  for DM  to  detect  this  disease  earlier.  [MEDICINA2014;45:156-160].
CASE REPORT KALLMANN SYNDROME IN A 14-YEAR-OLD BOY Anggraeni, Melisa; Arimbawa, Made; Suryawan, IW Bikin
Medicina Vol 44 No 1 (2013): Januari 2013
Publisher : Medicina

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Abstract

Kallmann syndrome is a combination of hypogonadotropic–hypogonadism due to gonadotropine releasing hormone (GnRH) deficiency with anosmia or hyposmia. Magnetic resonance imaging (MRI) reveals hypoplasia or aplasia of the olfactory bulbs. The incidence is estimated at 1 in 10.000 and 50.000 males and females respectively. The main clinical features consists of the absence of spontaneous puberty, partial or total loss of the sense of smell (anosmia). In this case report, we describe a 14 year old boy with Kallmann syndrome who was refferred with delayed puberty and lack of smell function. Physical examination revealed Tanner stage I and proven anosmia from olfactory test. Laboratory test showed low titer of testosteron. Testicular ultrasonography (USG) revealed small testicles. Treatment of this particular patient was with a 25 mg of intramuscular testosterone injection and were then increased by 25 mg every two weeks. Proper management of patients with Kallmann syndrome usually allows them to attain normal reproductive health.
Isolasi dan Seleksi Bakteri Antagonis untuk Pengendalian Penyakit Busuk Batang Panili (Vanilla planifolia Andrews) Secara In Vitro I MADE ARIMBAWA; GUSTI NGURAH ALIT SUSANTA WIRYA; I MADE SUDANA; I MADE WINANTARA
Jurnal Agroekoteknologi Tropika (Journal of Tropical Agroecotechnology) Vol.8, No.2, April 2019
Publisher : Program Studi Agroekoteknologi, Fakultas Pertanian, Universitas Udayana

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Abstract

Isolation and Selection of Antagonistic Bacteria to Control Stem Rot Disease on Vanilla Plant (Vanilla planifolia Andrews) by In Vitro Test. Isolation, selection, and identification of effective microbes are important steps to obtain biological agents. The perpose of this research was to get potential bacteria as controlling agents for stem rot of vanilla. The research was conducted at Plant Disease Laboratory, Faculty of Agriculture, Udayana University, Denpasar, from December 2017 to February 2018. The research comprised of isolation and identification the pathogen of stem rot of vanilla, pathogenicity test, isolation and identification of antagonistic bacteria, in vitro test of antagonistic bacteria ability to controled the pathogen of stem rot of vanilla, and hypersensitive response of antagonistic bacteria on tobacco plant. The result showed that the pathogen of stem rot on vanilla plants was identified as Fusarium oxysporum. There of 21 isolates of bacteria that has been isolated from field, there were 7 isolates could suppress of F. oxysporum growth are isolates P001, P002, P003, P004, P005, BM02, and BM03. In vitro test of antagonistic bacterial with F. oxysporum showed that isolate P002 could suppress pathogen growth with the highest percentage (98,36%), P005 (98,34%), P001 (92,78%), P003 (82,02%), P004 (80,34%), BM02 (71,80%), and BM03 (66,70%). Hypersensitive respons test showed that isolates P001, P002, P003, P004, P005, BM02, and BM03 weren’t pathogenic for plants.
Karakteristik Sindrom Turner di Jakarta I M Arimbawa; Jose RL Batubara; Bambang Tridjaya AAP; Aman B Pulungan
Sari Pediatri Vol 9, No 6 (2008)
Publisher : Badan Penerbit Ikatan Dokter Anak Indonesia (BP-IDAI)

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.14238/sp9.6.2008.386-90

Abstract

Latar belakang. Sindrom Turner adalah kumpulan gejala dengan karakteristik fisik dan hilangnya satukromosom X baik secara komplit maupun parsial, dan sering pula berupa sel mosaik.Tujuan. Untuk mengetahui karakteristik pasien sindrom Turner yang meliputi kariotip, umur saatdiagnosis, perawakan, kelainan penyerta, status pubertas, kadar follicle stimulating hormone (FSH), danusia tulang.Metode. Penelitian merupakan studi deskriptif. Data diperoleh dari Perkumpulan Turner Jakarta dancatatan medik pasien yang berkunjung ke Poliklinik Endokrin Anak RSCM dari tahun 1997-2006.Hasil. Dari 20 kasus yang berhasil dikumpulkan, 17 di antaranya dengan kariotip 45,X, sisanya mosaik.Rerata umur saat diagnosis adalah 7,75 tahun (rentang 0-15 tahun); rerata berat lahir 2590 gram; perawakanpendek 18 pasien (18/20). Terdapat 8 pasien dengan kelainan penyerta yaitu 4 anak kelainan jantung, 3gangguan telinga, dan 1 orang dengan hipertensi. Saat diagnosis tujuh pasien, mengalami pubertas terlambat.Rerata kadar FSH dari 16 pasien adalah 82,94 IU/liter (rentang 13,8-188 IU/liter). Data usia tulang (16pasien) menunjukkan retarded (11 pasien), dan sisanya average.Kesimpulan. Pada penelitian ini karakteristik utama sindrom Turner adalah kariotip 45,X dengankarakteristik fisik perawakan pendek dan pubertas terlambat disertai kelainan penyerta
Prevalensi dan Faktor Risiko Glikosuria pada Remaja Sekolah Menengah Pertama Swasta di Kota Denpasar Made Dwi Purnami; Made Arimbawa; Wayan Bikin Suryawan
Sari Pediatri Vol 17, No 2 (2015)
Publisher : Badan Penerbit Ikatan Dokter Anak Indonesia (BP-IDAI)

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.14238/sp17.2.2015.129-35

Abstract

Latar belakang. Pemeriksaan urin merupakan salah satu alat skrining yang penting pada DM untuk menentukan glikosuria.Tujuan. Mengetahui prevalensi glikosuria pada sekolah menengah pertama (SMP) swasta di kota Denpasar dan faktor-faktor terkaitDM pada kejadian glikosuria.Metode. Penelitian ini menggunakan desain potong lintang analitik yang dilakukan pada anak usia 12-14 tahun. Pengukuranantropometri berat badan dan tinggi badan dilakukan dan informasi mengenai riwayat DM pada keluarga menggunakan kuesioner.Glikosuria ditentukan menggunakan urin dipstik reagen strip One Med®. Hubungan beberapa faktor terkait glikosuria dianalisisdengan uji chi-square dan uji multivariat.Hasil. Total didapatkan 431 subyek dari 10 SMP yang memenuhi kriteria inklusi. Prevalensi glikosuria 3%. Proporsi obesitas lebihbesar pada subyek glikosuria dibandingkan subyek yang tidak (76,9 vs 23,1%). Rasio laki-laki yang glikosuria berbanding perempuanadalah 2:1. Analisis regresi logistik mendapatkan risiko subyek obes dengan glikosuria bermakna signifikan secara statistik [RO 5,8(IK95% 1,6 -21,3), p=0,008].Kesimpulan. Obesitas merupakan faktor risiko terjadinya glikosuria. Indeks glikosuria massa tubuh (IMT) 􀁴p95 merupakan faktorrisiko kejadian glikosuria.
Profil Pertumbuhan, Hemoglobin Pre-transfusi, Kadar Feritin, dan Usia Tulang Anak pada Thalassemia Mayor Arimbawa Made; Ariawati Ketut
Sari Pediatri Vol 13, No 4 (2011)
Publisher : Badan Penerbit Ikatan Dokter Anak Indonesia (BP-IDAI)

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (238.109 KB) | DOI: 10.14238/sp13.4.2011.299-304

Abstract

Latar belakang. Thalassemia adalah kelainan bawaan sintesis hemoglobin, dan salah satu penyakit monogenetik paling banyak dijumpai. Di Indonesia diperkirakan akan lahir 2500 anak dengan thalassemia mayor setiap tahunnya. Berkat kemajuan penanganan medis, sebagian besar pasien akan mengalami pertumbuhan normal pada masa anak-anak namun selanjutnya akan terjadi gangguan pertumbuhan dan keterlambatan pubertas secara signifikan.Tujuan. Mengetahui gambaran tinggi badan, kecepatan tumbuh, usia tulang, kadar hemoglobin pretranfusi, dan kadar feritin serum pasien thalassemia.Metode. Laporan serial kasus pada anak yang menjalani rawat inap di Sub-bagian Hematologi Bagian Ilmu Kesehatan Anak FK UNUD/RSUP Sanglah Denpasar dari bulan Desember 2010-Februari 2011. Data yang diperoleh disajikan dalam bentuk tabel.Hasil. Limabelas subyek thalassemia mayor, berumur antara 1,9 tahun – 13,5 tahun, 7 laki-laki dan 8 perempuan. Dua anak berumur kurang dari 3 tahun dan 7 anak telah memasuki usia pubertas. Semua pasien telah menjalani terapi kelasi besi deferioksamin namun kualitasnya tidak memadai. Perawakan pendek ditemukan pada 4 anak (26%), semua subjek mempunyai kecepatan tumbuh <5 cm/tahun. Secara klinis satu orang dikategorikan sebagai pubertas terlambat. Kadar hemoglobin rata-rata pre-transfusi dapat dipertahankan ≥8 mg/dl (10), sisanya (5) memiliki hemoglobin rata-rata di bawah 8 mg/dl. Empat anak dengan feritin serum di atas 3000 ng/ml, dan semua subjek mempunyai perawakan pendek. Pada evaluasi radiologi manus sinistra 5 anak memiliki usia tulang terlambat. Kesimpulan. Perawakan pendek didapatkan pada 26% kasus dan semua subjek telah memasuki usia pubertas. Semua subjek mempunyai perawakan pendek dan memiliki kadar feritin serum >3000 ng/ml. Sari Pediatri2011;13(4):299-304.
Co-Authors Alice Indradjaja, Alice Aman B Pulungan Anak Agung Ngurah Ketut Putra Widnyana Ayu Diah Perdana Paramita Bambang Tridjaya AAP Boy Arief Fachri Damastra, Garda Bagus Dewi, Ni Nyoman Gita Kharisma Fadilah, Siska Nuri Felix Arie Setiawan Gusti Ayu Putu Sri Satya Mahayani Gusti Ngurah Alit Susanta Wirya H Salim Hendy Halim Herman Saputra Herman Saputra I Gde Ardika Nuaba I Gde Ardika Nuaba I Gde Raka Widiana I Gusti Agung Trisna Windiani I Gusti Lanang Sidiartha I Gusti Ngurah Sanjaya Putra I K Kari I Made Darma Yuda I Made Kardana I MADE SUDANA I MADE WINANTARA I Nyoman Budi Hartawan I Putu Sudiarta I Wayan Bikin Suryawan I Wayan Gustawan Ida Bagus Gede Suparyatha Ida Bagus Ramajaya Sutawan, Ida Bagus Ramajaya Imanuel Yulius Malino Jose RL Batubara Kadek Adi Suryamulyawan Kartika Yantie, Ni Putu Veny Ketut Ariawati Ketut Suarta Khamil, Achri Isnan Komang Ayu Trisna Dewi Komang Ayu Witarini Leni Lukman Lestari, Afrila Tutut Dwijati Luh Gede Yuliadewi NS Luh Gede Yuliadewi NS Made Ayu Krishna Levina, Made Ayu Made Dwi Purnami Made Gede Dwi Lingga Utama Melisa Anggraeni Muharja, Maktum Ni Made Adi Tarini Ni Putu Siadi Purniti NP Veny Kartika Yantie Paramita, Ayu Diah Perdana Prastika, Andika Putri, Navisa Ayudia Putu Gede Karyana, Putu Gede Putu Meitri Nirmala Utami Rizki Fitria Darmayanti SANJAYA, I GUSTI NGURAH PRABU WIRA Selangga, Dewa Gede Wiryangga Setiyawan, I Made Karma Sholeha, Irdatus Soetjiningsih Soetjiningsih Wardhani, Maharani Tri Wega Upendra Sindhughosa Wibowo, Jihan Nafila Zakwan bin Ahmad