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RELATIONSHIP BETWEEN CLINICAL FINDINGS OF CARPAL TUNNEL SYNDROME WITH ENMG AND USG WRIST RESULT Masruroh Rahayu; Shahdevi Nandar Kurniawan; Neila Raisa; Farida Widyastuti
MNJ (Malang Neurology Journal) Vol. 8 No. 2 (2022): July
Publisher : PERDOSSI (Perhimpunan Dokter Spesialis Saraf Indonesia Cabang Malang) - Indonesian Neurological Association Branch of Malang cooperated with Neurology Residency Program, Faculty of Medicine Brawijaya University, Malang, Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.mnj.2022.008.02.12

Abstract

Background: Carpal tunnel syndrome is symptomatic compression neuropathy of median nerve in carpal tunnel on wrist causes functional decreased in that area. CTS is diagnosed from clinical, physical examination, also from ENMG and USG wrist. This research aims to know about CTS characteristics and the relationship between clinical findings of CTS with ENMG and USG wrist results. Objective: This study aims to determine whether there is a relationship between the clinical findings of patients diagnosed with CTS and the results of ENMG and wrist ultrasound at the Neurology Polyclinic of Saiful Anwar Hospital, Malang. Methods: This research was descriptive observational. Data were collected from anamnesis and physical examination at Neurology Polyclinic Saiful Anwar Malang Hospital (RSSA) from April to December 2017, then examined by ENMG and USG wrist at RSSA. There are 27 patients suspected of CTS who meet inclusion and exclusion criteria. Results: From 9 months (April-December 2017) found 27 patients suspected CTS, about 19 patients (70,37%) were female and 7 patients (19,63%) were male. The majority of the age group is 25-65 years old (66,67%). Most of the occupations were housewives (48,15%). Patients most came with chief complaint of tingling (88,89%) with positive tinnel sign (77,78%), and most concomitant diseases were hypertension (48,15%). The ENMG results confirmed CTS about 92,59%, compared to the USG that confirmed CTS about 85,19%. Statistical analysis with Pearson Chi-Square test showed no relationship between clinical findings of CTS (Tinnel, Phalen, Counter Phalen, Flick test) with ENMG and USG wrist results. Conclusion: This research concluded no relationship between clinical findings of CTS with ENMG and USG wrist results.
DANDY-WALKER VARIANT (DWV) IN 70 YEARS OLD WOMAN WITH DISEQUILIBRIUM AND CENTRAL VERTIGO : A CASE REPORT Ria Damayanti; Muhammad Welly Dafif; Shahdevi Nandar Kurniawan; Badrul Munir; Zamroni Afif
Journal of Pain, Headache and Vertigo Vol. 1 No. 1 (2020): March
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2020.001.01.2

Abstract

Dandy Walker Syndrome (DWS) and its Variants (DWV) is a congenital disorder that has prevalence of 1 in 25,000 to 30,000 births with the highest incidence occurs at age <1 year. However, patients may be diagnosed with DWS/DWV for the first time in adolescence and even old age. We reported the 70-year-old woman with a chief complaint of central type vertigo accompanied by disequilbrium. From history taking we obtained disequilibrium with gradual onset started in nine months and become settled since the last 6 months before she came to the neurology clinic. The patient also complained vertigo that have emerged since the last 3 month. A history of head trauma, stroke, dementia was denied. Physical examinaton show central nystagmus while other cranial nerve within normal range. Motor, sensory and autonomic status within normal range. Romberg test, tandem walking, disdiadokokinesia, and dismetria examination shows abnormal respons. MMSE results for patients within normal range (score:26). Head MRI shows Dandy Walker Variant in the accompanied by cerebral atrophy and degeneration serebelli Fazekas grade I. Patients receive symptomatic therapy betahistine mesylate 3x6 mg if necessary. Surgery management is not indicated because there is no sign of hydrocephalus or signs of increased intracranial pressure.
MYOFASCIAL PAIN SYNDROME Shahdevi Nandar Kurniawan; Nidia Suriani; Eko Arisetijono Marhaendraputro; Dessika Rahmawati
Journal of Pain, Headache and Vertigo Vol. 1 No. 1 (2020): March
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2020.001.01.5

Abstract

Pain is a sensory experience that is responded to as an unpleasant stimulus that is often caused by tissue damage. Perdossi defines pain as an unpleasant sensory and emotional experience due to tissue damage, both actual and potential, or which is described in the form of such damage. Pain can affect everyone regardless of sex, age, race, social status, and occupation. Myofascial pain syndrome (MPS) is a term used to describe the condition of acute or chronic pain in musculoskeletal soft tissue. This is indicated by the findings of sensory, motoric, and autonomic symptoms related to myofascial trigger points (MTRP). The symptoms can be local or far from MTRP. In MPS that are far from MTRP, the pain patterns that appear are usually pain is pervasive The most common location for MPS is the neck and back. The purpose of writing this literature review is to explain epidemiology, clinical manifestations, pathogenesis mechanisms, pathophysiology mechanisms, and management in patients with myofascial pain syndrome. Myofascial pain syndrome is a pain syndrome in muscles that includes musculoskeletal abnormalities and MPS management shows that most interventions, both medical therapy and non-medical therapy, still need studies to prove its effectiveness.
RADIOFREQUENCY AS PAIN INTERVENTIONAL THERAPY IN NEUROLOGY Badrul Munir; Widodo Mardi Santoso; Zamroni Afif; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 1 No. 2 (2020): September
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2020.001.02.3

Abstract

Radiofrequency (RF) intervention uses high frequency alternating current (AC) to inhibit and alter nociceptive pathway in various locations. Radiofrequency is divided into Continuous RF (CRF) and Pulsed RF (PRF). Continuous RF is a process in which the RF current is used to produce thermal lesions on the target nerve that will result in resistance to the afferent nociceptive pathway. PRF is a process whereby short bursts of RF are discharged toward a neural target that will produce a signal for lowering pain. PRF is a therapy of neural tissue with small neurodestructive possibility and is an alternative technique for continuing RF. RF therapy is a minimally invasive procedure that has been used for about three decades to treat various chronic pain syndromes such as trigeminal neuralgia, post herpes neuralgia, lower back pain (LBP), and complex regional pain syndromes or sympathetic reflex dystrophy. The mechanism action of the PRF involves cellular structure damage, neuronal activation, altered gene expression, a global continuation of evoked synaptic  activity in pain fibers through centrals and changes in synaptic strength and long-term potentiation.  PRF with its various characteristics has a better outcome than conventional RF.
CARPAL TUNNEL SYNDROME (DIAGNOSIS AND MANAGEMENT) Devi Annisa; Sri Budhi Rianawati; Masruroh Rahayu; Neila Raisa; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 2 No. 1 (2021): March
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.01.2

Abstract

Carpal Tunnel Syndrome (CTS) is a neuropathy disease that affects the median nerve with an incidence of around 90%. Carpal Tunnel Syndrome appears in 3.8% of the general population, with the highest prevalence occurring in women. There are several risk factors associated with CTS, namely medical and non-medical factors. The mechanism of carpal tunnel syndrome until now is still very complex and is not known with certainty, but compression and traction factors in the median nerve are thought to be the most common cause of CTS. Carpal Tunnel Syndrome can manifest clinically with subjective signs such as paresthesia, proprioceptive changes, and paresis, as well as objective signs, such as changes in motor sensitivity and function, positive Tinel and Phallen tests, and thenar muscle atrophy. The diagnosis of Carpal Tunnel Syndrome is based on the classic symptoms of pain, numbness, tingling, and/or burning sensation in the distribution of the median nerve in the hand, as well as the abnormal function of the median nerve based on nerve conduction studies. Conservative therapy is an option. Especially in Carpal Tunnel Syndrome patients with mild to moderate symptoms. Conservative therapy can be given in the form of corticosteroid and physical therapy. Patients with severe CTS or whose symptoms have not improved after four to six months of conservative therapy should be considered for surgical treatment.
DIABETIC NEUROPATHIC PAIN Reza Rachmantoko; Zamroni Afif; Dessika Rahmawati; Rodhiyan Rakhmatiar; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 2 No. 1 (2021): March
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.01.3

Abstract

Diabetic Neuropathy is the most common complication from diabetes, which experienced in almost 90% diabetes patient. Evenly pain is one of the most common symptoms of diabetic neuropathic, but the pathophysiology mechanism of pain is not clearly known. The hyptosesis of toxicity of hyperglycemia on development of pain complication has been widely accepted globally, but there is other proposed hypothesis. Basic concept in management of painful diabetic neuropathic is exclusion of the other cause of painful peripheral neuropathy, improving glycemic control for prophylaxis therapy and medication use for alleviating pain. The first choice drug of therapy for alleviating pain are anticonvulsant, like pregabalin and gabapentin, and antidepressant, mainly that work on inhibiting serotonine and noradrenaline reuptake. In conclusion, the better understanding of painful diabetic neuropathic underlying mechanism can help to find a better management that improving the guideline quality in optimalizing pain control.
LOW BACK PAIN Andaru Cahya S; Widodo Mardi Santoso; Machlusil Husna; Badrul Munir; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 2 No. 1 (2021): March
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.01.4

Abstract

Low back pain is the most common symptom found in the primary health care and is the number one cause of disability throughout worldwide. It is estimated that around 60 – 80% the world population will experience back pain during their lifespan. There are three different source of pain in the spine: axial-lumbosacral, radicular and reffered pain. All of these source brings different clinical presentations. Low back pain could be classified as acute, subacute and chronic low back pain. The pain could be nociceptive or neuropathic, the most common symptoms reported are “pressure pain” and “pain attack”.  The physician should be aware of “red flags” symptoms that lead into more serious condition beside back pain and, therefore the patient has to be investigated to further examination whenever these symptoms present. The management of low back pain consist of severe modalities, both therapeutic and rehabilitative procedure. Oftentimes, the management needed multidisciplinary approach. It is important to general practitioners to identify and treat low back pain appropriately to reduce the burden of the disease and to prevent the disabilties caused by this condition.
MENIERE’S DISEASE Helena Era Millennie; Badrul Munir; Zamroni Afif; Ria Damayanti; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 2 No. 1 (2021): March
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.01.5

Abstract

Meniere’s disease is a disorder of the inner ear resulting in symptoms of episodic vertigo, tinnitus, hearing loss and aural pressure. Although the exact etiology is uncertain, it is associated with raised pressure in the endolymph of the inner ear (endolymphatic hydrops). The diagnosis of Meniere's disease is based on the clinical setting of the patient. This disease usually presents with unilateral ear symptoms but can be also bilateral. Meniere's disease attacks are usually random and episodic (approximately  6-11 per year), with periods of remission that can last from months to years. Investigations are audiometry, electronystagmogram, vestibular evoked myogenic potentials (VEMPs) and imaging.The management consist pharmacological and non pharmacological. Meniere's disease is initially progressive but fluctuates unexpectedly. It is difficult to distinguish natural resolutions from treatment effects.
COMPLICATED MIGRAINE Kadek Putri Paramita Abyuda; Shahdevi Nandar Kurniawan
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.02.2

Abstract

Migraine is a chronic paroxysmal neurological disease characterized by attacks of moderate or severe headache accompanied by reversible neurologic and systemic symptoms. Although not life threatening, migraine can cause disability in the productive population. Migraine sufferers generally have a family history of migraine so that migraine is considered a genetic disease. Endogenous psychological factors such as stress or fatigue are the main triggers for migraine. Migraine pathophysiology involves various parts of the brain so that migraine symptoms are complex. Management of acute migraine can be done pharmacologically and non-pharmacologically. Migraine preventive management is needed if the patient has a chronic migraine or does not respond to abortive treatment.
VESTIBULAR NEURONITIS Shahdevi Nandar Kurniawan; Afiyfah Kaysa Waafi
Journal of Pain, Headache and Vertigo Vol. 2 No. 2 (2021): September
Publisher : Journal of Pain, Headache and Vertigo

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21776/ub.jphv.2021.002.02.5

Abstract

Vestibular neuronitis is an acute vestibular syndrome due to inflammation of the vestibular nerve characterized by the typical symptoms of acute rotatory vertigo accompanied by nausea, vomiting, and symptoms of balance disorders. The incidence of vestibular neuronitis is about 3.5 per 100,000 people. The exact etiology of this vestibular neuronitis is unknown. However, based on existing evidence, vestibular neuronitis is associated with viral infections of the upper respiratory tract and herpes zoster infection. The clinical manifestations of vestibular neuronitis are persistent rotatory vertigo accompanied by oscillopsia, horizontal-rotatory peripheral vestibular spontaneous nystagmus on the healthy side, and a tendency to fall on the affected side. Diagnosis of vestibular neuronitis can be made by clinical diagnosis, through history, physical examination, and special examinations. Through these examinations, the differential diagnosis of vestibular neuronitis should be excluded, such as Meniere's disease, labyrinthitis, benign paroxysmal positional vertigo, and vertigo due to central lesions such as cerebellar infarction. Management of vestibular neuronitis is in the form of symptomatic therapy with vestibular suppressants, antivertigo, and redirect to relieve the symptoms that arise, then causative therapy can be done by administering corticosteroids, and in patients, physiotherapy can be done to improve vestibular function.
Co-Authors Abdul Gofur Afiyfah Kaysa Waafi Al-Rasyid, Harun Aldita Husna Violita Aldita Husna Violita Andaru Cahya S Anggraini, Vivi Laras Anggraini, Vivid Prety Anisa Syahfitri Hanum Annisatul Hakimah Asmiragani, Syaifullah Auliya Nur Muthmainnina Badrul Munir Badrul Munir, Badrul Basya Adnani Basyar Adnani Chomsin Sulistya Widodo Chozin, Iin Noor Dalhar, Mochamad Damayanti, Ria DAMAYANTI, ZUHRIA PUSPITA Dessika Rahmawati Devi Annisa Devita Anggraeni Soeroso Dewi Permata Sari Dheka Sapti Iskandar Dhelya Widasmara Didi Candradikusuma Dini Jatiya Anggraini, Dini Jatiya Dwi, Pratiwi Suryanti Dyah Kusuma Wardhani Edi Widjajanto Eko Arisetijono Ekowati Retnaningtyas F, Fahimma Fahrani Yossa Prachika Farida Widyastuti Fitria Nikmahtustsani, Mulika Ade Gerry Gunawan, Gerry Gonius, Andry Gyang Hanandita Gusti Putri Hanestya Oky Hermawan, Hanestya Oky Hani Susianti Harbiyanti, Novita Titis Hardi Adiyatma Harun Al Rasyid Helena Era Millennie Heri, Sutanto Herwinda Brahmanti Husnul Khotimah Husnul Khotimah I Ketut Suada Irawan Satriotomo Irsyah Dwi Rohmayanti Izza Ayudia Hakim Janet, Karensa Abby Jatmiko, Sarazata Indi Rozaany Kadek Putri Paramita Abyuda Kartika Agustina Kinesya, Billi Lestari, Dwi Indriani Machlusil Husna Machlusil Husna, Machlusil Made Ayu Hariningsih Sunaga Masruroh Rahayu Masruroh Rahayu Masruroh Rahayu Masruroh Rahayu Masruroh Rahayu Masruroh Rahayu, Masruroh Masykur, Umar Jundullah Maziya, Yulianda Mega Yulia Rusmayanti Michelle Anisa Misnasari, Putri Priela Mochammad Istiadjid Eddy Santoso Mokhamad Fahmi Rizki Syaban Mondiani, Yeni Quinta Muhammad Welly Dafif Nadia Artha Dewi Nadiya Elfira Bilqis, Nadiya Elfira Nasution, Ali Napiah Nata Sanjaya Nectarine Natasya Regitta Yasmin Nidia Suriani Nurvia Andriani Petrarizky, Alfred Julius Pramesti, Fathia Annis Pratiwi, Made Dinda Pringga, Gutama Arya Pudji, Rosalyna Purbasari, Bethasiwi Putri, Dwi Sandhi Aulia Pramesti Putri, Laily Ardhianti Putri, Mutiara Kristiani R, Rahmad Rahma, Annisa Rahmasari, Herisa Rahmawati, Dessika Raisa, Neila Reza Rachmantoko Rislan Faiz Muhammad Rislan Faiz Muhammad Ristiawan Muji Laksono Rizki Rahamatullah Noer Rodhiyan Rakhmatiar Rulli Rosandi Safira Dita Arviana Sari, Atika Windra Sari, Diane Tantia Sari, Diane Tantia Sela Pricilia Siti Nurlaela Sri Budhi Rianawati Sri Budhi Rianawati, Sri Budhi Sri Budi Rianawati Suratmono, Mia Fajarningtyas Syafiatul Azizah Titin Andri Wihastuti Tri Wahju Astuti, Tri Wahju Vely Eva Meria Wa Ode Intan Nur Octina Wahyuni Ramadhani Suaib Widiatmoko, Arif Widodo Mardi Santoso Widodo Mardi Santoso, Widodo Mardi Wirathmawati, Andina Yudiansyah, Anggi Gilang Yuyun Yueniwati Zahra, Farah Shabri Alifia Zamroni Afif