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All Journal ACTA VETERINARIA INDONESIANA Jurnal Sain Veteriner Jurnal Veteriner Jurnal Ilmiah Mahasiswa Veteriner ARSHI Veterinary Letters Jurnal Kedokteran Hewan The International Journal of Tropical Veterinary and Biomedical Research Jurnal Medika Veterinaria
Etriwati E
Laboratorium Patologi Fakultas Kedokteran Hewan Universitas Syiah Kuala
Agriculture, Biological Sciences & Forestry Biochemistry, Genetics & Molecular Biology Immunology & microbiology Medicine & Pharmacology Public Health Veterinary

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Journal : Jurnal Veteriner

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STUDI HISTOPATOLOGI PADA LIMPA DAN BURSA FABRICIOUS AYAM NEWCASTLE DISEASE DARI KASUS LAPANG HISTOPATHOLOGY (STUDIES ON SPLEEN AND BURSA OF FABRICIUS OF NEWCASTLE DISEASE CHICKHENS FROM FIELD CASE) Etriwati Etriwati; Ekowati Handharyani; Surachmi Setiyaningsih
Jurnal Veteriner Vol 18 No 4 (2017)
Publisher : Faculty of Veterinary Medicine, Udayana University and Published in collaboration with the Indonesia Veterinarian Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (145.822 KB) | DOI: 10.19087/jveteriner.2017.18.4.510

Abstract

Newcastle Disease is a highly contagious and very virulent avian disease in all of poultry. The aims of this study were to determine the lesions and distribution of Newcastle disease virus on spleen and bursa of Fabricous of Newcastle disease chickhens from field cases. The sample used in this study were the spleen and bursa Fabricious organ of ten cases of suspect Newcastle disease. Haematoxylin and eosin staining examination was performed determine of cell morphology and distribution of the virus by immunohistochemical staining. The distribution of the lesions and the degree of reaction were determined by the category immunopositive mild, moderate and severe. Gross lesion on spleen, including swollen, fragile and necrotic multifocal. Histopathological lesions showed hyperemia/congestion and depletion of lymphoid cells. Immunopositive reaction found in lymphoid cells of red pulp and lymphoid cells in lymphoid follicles. Bursa of Fabricious showed atrophy and hyperemia/congestion with histopatological lesions hemorrhages, lymphoid cells in lymphoid follicles undergo necrosis and depletion of lymphoid follicles. Immunopositif reaction was observed in lymphoid cells in the mantle zone of lymphoid follicles. Newcastle disease from field cases cause damage to the spleen and bursa of Fabricius with degrees lesions and virus distribution in the organs are severe.
Biokimia Darah Hati dan Ginjal Setelah Implan Wire SS316L dan Wire Alternatif (BLOOD BIOCHEMISTRY OF LIVER AND KIDNEY AFTER IMPLANT WIRE SS316L AND ALTERNATIVE) Erwin Erwin; Rusli Rusli; Amiruddin Amiruddin; Etriwati Etriwati; Muhammad Isa; Abdul Harris; Yula Astuti
Jurnal Veteriner Vol 21 No 1 (2020)
Publisher : Faculty of Veterinary Medicine, Udayana University and Published in collaboration with the Indonesia Veterinarian Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (147.187 KB)

Abstract

The blood biochemistry changes of liver and kidney are indicative of disorder in these organs. The objective of our experimental approach was to examine the level serum glutamic pyruvic transaminase (SGPT), serum glutamic oxaloacetic transaminase (SGOT), blood urea nitrogen (BUN) dan creatinine after implant wire SS316L and alternative. Ten rats wistar strain aged 2-3-month-old, weighing 250-300 g were divide into two groups. Group I (G-I) implanted wire SS316L and group II (G-II) implanted alternative wire in os femur with each had a diameter of 0,5 mm. Blood sample was taken from sinus orbitalis on day 0 before implant, on day seven, 14, 21, 42 and 84 after implant. The results showed an increase in levels SGPT, SGOT, BUN and creatinine in the first few weeks after implantation. The levels of SGPT, SGOT, BUN and creatinine decreased again in the last week of observation with significant differences between the two treatment groups (P<0.05). The implant wire SS316L and alternative does not cause increased levels SGPT, SGOT, BUN dan creatinine for long time. Thus, it can be concluded that alternative wire has potential as internal fixation in fracture of small animal.
Temuan Kejadian Penyakit Tetelo pada Kadaver Unggas yang Dikirim ke Laboratorium Patologi Fakultas Kedokteran Hewan Universitas Syiah Kuala Etriwati Etriwati; Muhammad Nur Salim; ummu Balqis; Siti Aisyiah; Nazaruddin Nazaruddin; Aminuddin Aminuddin; Rusli Rusli; Divina Dinda Hayati
Jurnal Veteriner Vol 24 No 3 (2023)
Publisher : Faculty of Veterinary Medicine, Udayana University and Published in collaboration with the Indonesia Veterinarian Association

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.19087/jveteriner.2023.24.3.313

Abstract

Penyakit tetelo (Newcastle disease) merupakan penyakit penting yang sangat mudah menular dan menyebabkan kematian tinggi pada unggas rentan. Penelitian ini bertujuan untuk mengetahui kejadian penyakit tetelo pada unggas yang dinekropsi di Laboratorium Patologi, Fakultas Kedokteran Hewan (FKH), Universitas Syiah Kuala (USK), Banda Aceh. Sampel penelitian dikoleksi dari 239 ekor cadaver unggas terdiri dari ayam pedaging/broiler, ayam petelur/layer, ayam kampung/buras dan unggas lainnya yang dinekropsi di Laboratorium Patologi FKH USK periode Juli sampai November 2019. Sampel yang dikoleksi yaitu trakhea, proventrikulus, seka tonsil dan otak. Sampel dipilih berdasarkan perubahan patologi anatomi diduga penyakit tetelo, selanjutnya dibuat sediaan histopatologi dan imunohistokimia dalam parafin blok. Hasil penelitian menunjukkan bahwa unggas yang dinekropsi dan mati akibat penyakit tetelo sebanyak 106 ekor, diduga fowl cholera 52 ekor, infectious bronchitis 25 ekor, avian influenza 22 ekor, colibacillosis 14 ekor, chronic respiratory disease 7 ekor, coccidiosis 5 ekor, infectious laryngotrcheitis 4 ekor, infectious bursal disease 2 ekor dan coryza 2 ekor. Simpulan dari penelitian ini bahwa cadaver unggas yang dinekropsi di Laboratorium Patologi FKH USK periode Juli sampai November 2019 sebanyak 44,4% (106/239) terdiagnosa mati akibat terinfeksi penyakit tetelo.
Co-Authors . Amiruddin . Erwin . Erwin . Muttaqien . Rusli . Rusli Abdul Harris Abdullah Hamzah Abellisa Abellisa Abellisa, Abellisa Afif Yuda Kusuma Ajis, Fikri Alsidik Aminuddin Aminuddin Amiruddin . Amiruddin Amiruddin Amiruddin Amiruddin Anak Agung Gede Sugianthara Angreini, Meliyantika Annisa, Aiza Arman Sayuti Asnita Purnama Awaluddin Awaluddin Azhari Azhari Baehaqi Cut Erika Ramadhana Daniel Daniel Darmawi . Dasrul Dasrul Dian Masyitha Dirga Rizki Imanda divina dinda hayati Dwinna Aliza Ekowati Handharyani Erina Erina Erwin E Erwin Erwin Erwin Erwin Erwin Erwin Erwin Erwin Evan Kurniawan Faisal Jamin Faisal Mustafa Ferdian, Riyan Hafifatul Aini Hanif Fadli Hanni Aninaidu Hasan, Denny Irmawati HEFRI YUNALDI Helmi, T. Zahrial Hilmi, Mulkiah Jessica Anggun Safitri Langga Mora Lian Varis Riandi M Daud AK M Hasan M Isa M Jalaluddin M Jalaluddin M Nur Salim M Nur Salim M. Nur Salim Mahdi Abrar Maulina, Siti Raudha Muhammad Hasan muhammad isa Muhammad Isa Mulyadi Adam Muslim Akmal Muttaqien M Muttaqien Muttaqien N. Nazaruddin Nazaruddin Nazaruddin Nazaruddin Nazaruddin Novredha Rahmadita Nuzul Asmilia Nuzul Asmilia pani noveliani lubis Rahmi Eka Putri Rahmi, Ulfa Rastina Rastina Razali Daud Razali Daud Rezeki Indah Sari Roslizawaty Roslizawaty Rusli r Rusli r Rusli Rusli Rusli Rusli Rusli Rusli Sari, Wahyu Eka Seftia Yolanda Sirait, Muhammad Alfajri Erutama Sitarina Widyarini Siti Aisyah Siti Aisyah Siti Aisyiah Sugito - Sugito Sugito Surachmi Setiyaningsih Syah, Nada Yulian t fadrial karmil T Reza Ferasyi Teuku Fadrial Karmil Teuku Reza Ferasyi Teuku Zahrial Helmi Tri Wahyu Pangestiningsih Ummu Balqis Winaruddin Winaruddin Winaruddin Winaruddin Yula Astuti Zainuddin Zainuddin Zamzami, Rumi Sahara Zuhrawaty Zuhrawaty