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All Journal HAYATI Journal of Biosciences Jurnal Gizi dan Pangan ACTA VETERINARIA INDONESIANA Jurnal Pengolahan Hasil Perikanan Indonesia Hemera Zoa Jurnal Sain Veteriner Buletin Peternakan Jurnal Veteriner BIOTROPIA - The Southeast Asian Journal of Tropical Biology Medical Journal of Indonesia Current Biochemistry Jurnal Ilmu Ternak Veteriner Warta IHP (Warta Industri Hasil Pertanian) Historika Jurnal Medik Veteriner ARSHI Veterinary Letters FITOFARMAKA: Jurnal Ilmiah Farmasi Majalah Patologi Indonesia Indonesian Journal of Applied Research (IJAR) International Journal of Multidisciplinary: Applied Business and Education Research Journal of Education Research Jurnal Pusat Inovasi Masyarakat Jurnal Kedokteran Hewan Journal of Research and Publication Innovation VALUTA Jurnal Administrasi Bisnis, Manajemen dan Ekonomi Policy Brief Pertanian, Kelautan, dan Biosains Tropika Health Gate Jurnal Agripet Seminar Nasional Riset dan Teknologi (SEMNAS RISTEK)

Agus Setiyono

Departemen Klinik, Reproduksi Dan Patologi Fakultas Kedokteran Hewan, Institut Pertanian Bogor. Jl. Agatis Kampus IPB Dramaga 16680

Author-ID : 377503

Religion Agriculture, Biological Sciences & Forestry Humanities Biochemistry, Genetics & Molecular Biology Chemistry Civil Engineering, Building, Construction & Architecture Computer Science & IT Decision Sciences, Operations Research & Management Dentistry Earth & Planetary Sciences Economics, Econometrics & Finance Education Engineering Environmental Science Health Professions Immunology & microbiology Industrial & Manufacturing Engineering Languange, Linguistic, Communication & Media Law, Crime, Criminology & Criminal Justice Mathematics Medicine & Pharmacology Nursing Public Health Social Sciences Veterinary Other

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Enteritis necroticans pada ayam broiler akibat infeksi sekunder clostridium perfringeens Tipe A Agus Setiyono; Rachmat Nabib; Gatut Ashadi; Aisjah Girinda; Lily Natalia
Hemera Zoa Vol. 76 No. 1 (1993): Jurnal Hemera Zoa
Publisher : Hemera Zoa

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (406.531 KB)

The experiment was conducted to determine the relationship between conccidiosis vaccination and secondary infection of Clostridium perfringens type-A as a cause of Enteritis Necroticans in broiler chickens.The completely randomized design was used in this experiment. In this study 102 Arbor Acres strain unsexed chicken were randomly assigned to 8 treatment groups with 12 chicks each. Feed and water were given ad libitum.Treatment factors in this experiment were coccidiostat (Salinomycin 6%), coccidiosis vaccine and Clostridium perfringens type-A isolate, and their combinations.The anatomy pathology (AP) and histopathology (HP) figures of the chicken intestines were evaluated.Histopathology results indicated that the relationship between coccidiosis vaccination and secondary infection of Clostridium perfringens type-A was highly significant (p < 0.01) but the anatomy pathology figures showed that the relationship was not significant (p < 0.05).

Kasus infeksi cacing saluran pencernaan pada ayam hutan hijau (Gallus varius) yang diperiksa di laboratorium patologi unggas FKH_IPB Agus Setiyono
Hemera Zoa Vol. 76 No. 2 (1993): Jurnal Hemera Zoa
Publisher : Hemera Zoa

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (400.56 KB)

Telah ditemukan kasus inifeksi cacing saluran pencernaan pada ayam hutan hijau (Callus varius) yang diperiksa di Laboratorium Patologi Unggas, Fakultas Kedokteran Hewan lnstitut Pertanian Bogor, periode bulan Agustus 1992 - Januari 1993.Dari 62 ekor ayam hutan hijau yang mati, 26 ekor (41,93%) diantaranya positif terinfeksi cacing setelah dilakukan nekropsi. Dari 26 ekor ayam yang dinyatakan positif terinfeksi, 22 ekor terinfeksi oleh cacing pita (Cestoda), 3 ekor terinfeksi cacing gilik (Nematoda) dan 1 ekor ayam terinfeksi oleh kedua macam cacing tersebut.Hasil pemeriksaan terhadap parasit ini menunjukkan bahwa cacing pita yang menginfeksi ayam adalah Raillietina sp. dan cacing gilik yang ditemukan adalah Ascaridia galli.

Perubahan patologis dan gambaran lekosit pada itik yang diinfeksi Pasteurella multocida Wiwin Winarsih; Ekowati Handharyani; Agus Setiyono
Hemera Zoa Vol. 77 No. 1 (1995): Jurnal Hemera Zoa
Publisher : Hemera Zoa

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (502.912 KB)

Enam puluh ekor itik yang berumur lima minggu dipergunakan dalam penelitian ini. ltik dibagi secara acak menjadi dua kelompok yaitu kelompok infeksi dan kontrol.Kelompok pertama diinfeksi dengan Pasteurelhz multocida (Isolat lapangan) secara intramuskular. Itik diambil darahnya dan dinekropsi 1, 2, 4 dan 8 jam setelah infeksi untuk pemeriksaan diferensial lekosit dan perubahan makroskopik.Itik yang diinfeksi mulai menunjukkan lesio empat jam setelah infeksi. Itik yang diinfeksi dan mati menunjukkan gejala septikemi dengan hiperemi umum. Pemeriksaan gambaran lekosit menunjukkan peningkatan jumlah heterofil dan penurunan jumlah limfosit dan monosit 2,4 dan 8 jam setelah infeksi.

PAT-5 Update: Q Fever in Indonesia Agus Setiyono; Elok Puspita Rini; Handayu Untari; Eko Prasetyo Nugroho
Hemera Zoa Proceedings of the 20th FAVA & the 15th KIVNAS PDHI 2018
Publisher : Hemera Zoa

Show Abstract | Download Original | Original Source | Check in Google Scholar | Full PDF (561.027 KB)

Q Fever is zoonotic disease caused by Coxiella burnetii, an intracellular obligate and negative Gram bacterium with pleomorphic shape (Kaplan and Bertagna 1955). Centers for Disease Control and Prevention (CDC) classify Coxiella burnetii as potential bioterrorism agent within B rank for its ability and characteristics (CDC 2013).The main reservoir animals for Q fever are ruminants (Maurin and Raoult 1999). Q fever infection whether in animal or human generally occur through inhalation, vector bites, or through oral ingestion—the last two routes are considered as secondary routes (Angelakis and Raoult 2010). Clinical symptoms of Q fever in both animal and human generally asymptomatic. Q fever can cause abortion in the third trimester of pregnancy and pneumonia in ruminants. Whether in human, acute Q fever can cause flu like syndrome and can develop into hepatitis, endocarditis, and for some severe chronic case, it caused death (Fournier et al. 1998). Office international des epizooties (OIE) classify Q fever into a re-emerging disease group (OIE 2010). Based on OIE data in 2012, the distribution of Q fever in animals occur in almost all country in the world including ASEAN (OIE 2012).Q fever was first discovered in Australia in 1935 (Kaplan and Bertagna 1955). Q fever disease transmission in animal occurs almost in every country in the world. Based on OIE data, Indonesia was classified as no information region (OIE 2012). World Health Organization (WHO) reported that Q fever was first found in cow serologically in Indonesia in 1953 (Kaplan and Bertagna 1955). Q fever in ruminants was again reported by Indonesian researcher between 2006 to 2015 in Bali, West Java, Jakarta, and Medan (Mahatmi et al. 2007; Setiyono et al. 2008; Nasution et al. 2015).Positive Q fever result was obtained especially from ex imported cows, thus it was suspected that cows from abroad might play main role of transmission of Q fever in Indonesia. However, there was also report of positive result in local ruminants, such as Bali cattle, goat, and sheep (Mahatmi et al. 2007). Researches in various country also stated of potential local ruminant as reservoir animal, among them are Tibet sheep in China, Alpine and Saanen goat in Italia, camel (Camelus dromedaries) in Saudi Arabia, or Swedish dairy cattle in Swedia (Mohammed et al. 2014). Based on the historical study of Q fever in Indonesia, this research was performed by taken sample from both ex-import cattle and local ruminants in several region in East Java (Malang, Surabaya, Madura), Central Java (Boyolali), and West Java (Bogor, Bandung, Depok) which are known as regions with high population of ruminants in Indonesia. So far, surveillance data of Q fever has not been recorded from these regions.

Effectiveness of Gnetum gnemon Peel Extract as an Antihyperuricemic in White Rats Rattus norvegicus Akhmad Endang Zainal Hasan; Husnawati Husnawati; Chintia Ayu Puspita; Agus Setiyono
Current Biochemistry Vol. 7 No. 1 (2020)
Publisher : IPB University

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29244/cb.7.1.3

Melinjo (gnetum gnemon) peel extract is believed to have potential as antihyperuricemic agent, however, in vivo assays have yet to be conducted to prove the antihyperuricemic activity. The objectives of this research were to analyze antihyperuricemic activity of melinjo peel extract in Sprague Dawley male white rat. A total of 24 rats were divided into 6 groups, which are a normal, positive, and negative group, 70% ethanolic extract of melinjo peel dose 450 mg/kg BW (ET I) and dose 900 mg/kg BW (ET II), and water extract of melinjo peel dose 93.35 mg/kg BW (EA). The results show that antihyperuricemic potency of 70% ethanolic extract of melinjo peel (ET1 and ET2) was more effective than allopurinol dose 90 mg/kg BW with a percentage decrease of 50% and 54%. Ethanol extract of melinjo peel was able to reduce uric acid levels to be in the range of normal values (<5 mg / dL). Bodyweight gain of rats given ethanol extract of melinjo peel dose 450 mg/kg BW had values that were close to the normal group. Melinjo peel extract contained flavonoid, tannins, saponin, and triterpenoid.

Seroprevalence of Q fever in sheep and goat in West Java area Agus Setiyono; Ekowati Handharyani; Hapsari Mahatmi
Jurnal Ilmu Ternak dan Veteriner Vol 13, No 1 (2008): MARCH 2008
Publisher : Indonesian Center for Animal Research and Development (ICARD)

Q fever is a zoonotic disease caused by Coxiella burnetii, a species of bacteria that is distributed globally. Ruminant especially sheep and goats may play an important role in the transmission of the disease to human. The research of seroprevalence of Q fever in sheep and goats was carried out from August 2006 to March 2007 in West Java area. A total of 138 sera were collected; 69 sera from sheep and 69 sera from goats. The indirect immunoflourescent antibody test was used to determine the seroprevalence of Q fever. The seropositive based on the dilution of serum starting from 1 : 16. Seropositive were observed in 22 samples (31.88%) of sheep and 14 samples (20.28%) of goats. The highest titer of 1 : 128 was observed in 3 pregnant sheep. The results of the present study suggested that Q fever was endemic in West Java area. Key Words: Q Fever, Prevalence, Indirect Immunoflourescent Antibody Test

Potency of Medicinal Plants for Eradication of Avian Influenza : In Vitro Test on Vero Cells Agus Setiyono; Nurliani Bermawie
Jurnal Sain Veteriner Vol 31, No 1 (2013): JULI
Publisher : Fakultas Kedokteran Hewan Universitas Gadjah Mada bekerjasama dengan PB PDHI

Some of medicinal plants indicate their potency as anti-viral such as Sambiloto (Andrographis paniculata Nees), Temu Ireng (Curcuma aeruginosa L.), Beluntas (Pluchea indica L.) Sirih Merah (Piper crocatum) and Adas (Foeniculum vulgare). Avian Influenza (AI) H5N1 strain viruses used in this study was isolated from field in Cikole area, West Java in July 20th 2007. To explore the potency of medicinal plants as anti-viral substance, the consecutive assays were performed by virus infection inhibition test in in vitro study using Vero cells. After the Vero cells were growing confluently, they were treated with sterilized-extract of medicinal plants either in single or combination. Furthermore, the culture cells were infected with AI H5N1 strain virus, then incubated at 37oC and examined for cytopathic effect (CPE) microscopically. The result showed that extract of Sambiloto and combination of Sambiloto and Temu Ireng were stronger than others in inhibition of virus attachment and infection to the cells. The Vero cells still alive up to 3rd day post infection with AI H5N1 virus after treatment with Sambiloto and Temu Ireng. In conclusion, extract of Sambiloto and Temu Ireng showed their potency as candidate for anti-viral substances that may needed for eradicating AI infection.

Efek Protektif Andrografolid terhadap Kejadian Kardiotoksisitas Pasca Aplikasi Doksorubisin pada Tikus Sri Wahyuni Salam; Agus Setiyono; Vetnizah Juniantito
Jurnal Sain Veteriner Vol 34, No 2 (2016): Desember
Publisher : Fakultas Kedokteran Hewan Universitas Gadjah Mada bekerjasama dengan PB PDHI

Cardiotoxicity is one of the important side effects of doxorubicin, an anthracycline antibiotic and chemotherapeutic drug. The aim of this study was to explore the potential protective effect of andrographolide (Andro), an anti-inflammatory and anti-oxidant agents, against cardiotoxicity induced by doxorubicin (DXR). Thirty Sprague Dawley rats (80-100 g) were divided into four groups: (a) Control (b) DXR (4 mg/kg intraperitoneally (IP) were made weekly for 4 weeks), (c) DXR+Andro20 (low dose andro; 20 mg/kg IP weremade daily for 4 weeks, 24 h after DXR), (d) DXR+Andro100 (high dose andro; 100 mg/kg IP were made daily for 4 weeks, 24 h after DXR). Furthermore, at the end of experimental period, all rats were euthanized and hearts were removed for hispatological analyses. Hematoxylin-eosin (HE) and Masson Trichrome (MT) staining were used to observe the histomorphological alterations and fibrosis of hearts, respectively. Our results showed that andrographolide treatment (20 mg/kg) augmented the detrimental effects of DXR such as decreased body weight and heart weight, as compared with those in DXR-treated rats. Histopathologically, heart tissue from control group showed compact myocardial architecture without any noticeable lesions. Histopathological analysis fromDXR group showed severe inflammation and fibrosis, whereas DXR+Andro20 group showed almost normal heart morphology. Andrographolide at a dosage of 100 mg/kg did not show protective effects against doxorubicin,and even aggravated myocardial inflammation, as compared with DXR-treated rats. These results indicate that low dose of andrographolide compromised doxorubicin-induced decreased body weight, heart inflammation, andfibrosis.

Studi Reseptor Virus Infectious Bursal Disease (IBD) pada Organ Limfoid Ayam Pasca Vaksinasi dengan Metode Imunohistokimia Restu Librani; Agus Setiyono; Wiwin Winarsih
Jurnal Sain Veteriner Vol 37, No 1 (2019): Juni
Publisher : Fakultas Kedokteran Hewan Universitas Gadjah Mada bekerjasama dengan PB PDHI

Infectious bursal disease (IBD) affects economical impact for breeders due to it can cause damage lymphoid organ, especially bursa of Fabricius and causing failure the vaccination program. Infectious bursal disease virus receptors on lymphoid organs suspected contribute to subclinical and clinical IBD incidence in the chickens. The aim of this study was to determine and compare the distribution of IBDvirus receptors on lymphoid organ of the chicken which obtained different IBD vaccination program. The presence of virus receptors in bursa of Fabricius, spleen, and thymus were observed microscopically using immunohistochemical method and evaluated with Image J® software. Monoclonal anti LSCC-BK3 (Gifu University, Japan) antibodies as primary antibody was used in this study. The result showed that IBDvirus receptors found abudantly in the bursa of Fabricius, afterwards in the spleen and thymus. No significant differences of IBD virus receptor distribution within lymphoid organs between chicken which obtained once and twice IBD vaccination. Infectious bursal disease virus receptor distribution in bursa Fabricius chickens aged 23 days received twice IBD vaccination more than once IBD vaccination program.

BLOOD PROFILE, DIGESTIVE ORGAN AND FECAL AMMONIA OF LAYING DUCKS FED BY FERMENTED FEED Allaily Allaily; Muhammad Ridla; M. Aman Yaman; Agus setiyono; Nahrowi Nahrowi
Buletin Peternakan Vol 41, No 2 (2017): BULETIN PETERNAKAN VOL. 41 (2) MEI 2017
Publisher : Faculty of Animal Science, Universitas Gadjah Mada

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.21059/buletinpeternak.v41i2.13198

This study was conducted to determine how the fermented feed effect against blood profile, digestive organ and fecal ammonia of laying ducks. Feed materials were prepared according to laying ducks need as 21% crude protein, then mixed rations already given water until 45% the water content, then anaerobic fermented for 7 days. This study used a completely randomized design with 4 treatments of feed, such as: dry feed, wet feed, wet fermentation feed, and commercial dry feed. Each treatment was repeated 5 times, and each replication was consisted of four laying ducks. Laying duck were 80 heads Indramayu duck at 5 month of old. Blood observations were taken 3 times at 5 months, 6.5 months and 8 months old. Parameters measured were red blood cell (RBC), packed cell volume (PCV) = volume percentage of erythrocytes in the blood, hemoglobin (Hb), white blood cell (WBC), heterophile cell (H), lymphocytes (L), monosit (M) and the ratio of HL. Blood was taken from vena pectoralis vein under duck wings. Observation of digestive organs and fecal ammonia content was at 8 months old of duck. The results showed in pre treatment of the 5 months ducks significantly influenced the PCV and the WBC, but at 6.5 months and 8 months of treatment did not significantly affect blood profile of laying ducks. Digestive organs of liver in fermentation treatment showed significantly lower than in controls. The content of fecal ammonia were not significantly different.

Co-Authors Aditya Dharmawan, Aditya Aditya Wahyu Nugraha Afifah, Amalia Aisjah Girinda Akhmad Endang Zainal Hasan Akterono D. Budiyati Allaily Allaily Altaff Hendry Alvin Jefry Amrozi Andriyanto . Anita Esfandiari Arif Yahya Ario Damar Aulia Dina Kristina Bayu Sandi Chintia Ayu Puspita Daniel Latief Andre Darsono Darsono Desdiani Diah Nugrahani Pristihadi Dian Maulia Utami Dian Pratiwi DIMAS ANDRIANTO Dinar Arifianto Djumali Mangunwidjaja Dwi Astuti E. Mulyati Effend Effend, E. Mulyati Eko Prasetyo Nugroho Eko Suryanto Ekowati Handharyani Elok Puspita Rini Elpita Tarigan Eny Nur Aisyah Evy Damayanthi Fachriyan Hasmi Pasaribu Feri Kusnandar Gani, Ainal Gatut Ashadi Handayu Untari Handayu Untari Hanif Alamudin Manshur Hapsari Mahatmi Hardika Hardika, Hardika Hasim Hasim Hasim Hasim Hasim Hasim Hasim Hasim, . Hirawan Setiadi Husnawati, . I Ketut Suada I Komang Gede Wiryawan I wayan Teguh Wibawan Idqan Fahmi Ike Agustiani IPB, DGB Iris Rengganis Ismah Atika Salmah Japanis Permatasari, Retno Jusman, Sri Widia Azraki Kustiariyah Tarman Lala M Kolopaking Liesbetini Haditjaroko Lily Natalia M Agus Setiadi M. Aman Yaman M. Misbah, M. Ma'mun Sarma Maharani Maharani Maningtyas, Rosyidamyani Twinsari Masdiana C. Padaga Mega Safithri Miko Afrian Mochamad Alfinanda Santriagung Mohammad Irham Bagus Santoso Mohammad Sadikin Muhammad Daud Muhammad Ihsan Muhammad Luthfi Rahman Muhammad Ridla Muhammad Zuhdi Badruzzaman Muhammad Zulkifli Muharam Saepulloh Mutholaah Nadine Hanifa Permana Nahrowi Nahrowi Nahrowi Nathasia, Novena Neni Nuryani Nina Herlina Noviasari, Santi Nurjati Chairani Siregar Nurliani Bermawie Nurliani Bermawie Nurrahman, Arif Oktariza, Wawan Olive Wisepti Pratama Ono Suparno Pribadi, Yohanes Kusuma Purwiyatno Hariyadi Putri C. Eyanoer Rachmat Nabib Rahayu WoroWiranti Rahminiwati, Min Rayendra, Raendi Restu Librani Retno Damayanti Soejoedono Risa Tiuria Samsuridjal Djauzi Sandi, Bayu Sangko Sayuti Nasution Sekar Winahyu Ariadini Shila Rahmafia Putri SILMI MARIYA Siti Zulaikhah Slamet Budijanto Sri Purwaningsih, Sri Sri Wahyuni Salam Sudradjat Sumiati Suradi Suria Darma Tarigan Syahruddin Said Syifa Khaerani Teo Qin Yan Titi Candra Sunarti Tomas Iriyanto Tri Isyani Tungga Dewi Trioso Purnawarman Tsulatsy Puspitasari Purnamiharja Ulfah Juniarti Siregar Unang Patriana Upik Kesumawati Hadi Utamimah, Sri Vetnizah Juniantito Win Satya Rudramurti Winahyu Ariadini, Sekar Wiranda G Piliang Wiwin Winarsih Wiyono, W. Yeni Kartika Sari, Yeni Kartika

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