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Impact of Coma Duration on Functional Outcomes After Septic Encephalopathy: A Literature Review Shafitri, Shira Shalsabina; Sugiono, Muhammad Rafly Adrian; Paradiesta, Andi Frieskha Naurah; Aulia, Siti Zahroni; Ilmiah, Janiya Abdila; Hunaifi, Ilsa
Jurnal Biologi Tropis Vol. 25 No. 4a (2025): Special Issue
Publisher : Biology Education Study Program, Faculty of Teacher Training and Education, University of Mataram, Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29303/jbt.v25i4a.10516

Abstract

Sepsis-associated encephalopathy (SAE) is a neurological complication often found on sepsis patients and is associated with high morbidity and mortality rates. It arises from systemic inflammation, blood-brain barrier disruption, microglial and astrocytic activation, and oxidative stress which collectively contribute to neuronal injury and long-term cognitive impairment. This literature review aims to analyze the impact of coma duration on functional outcomes at discharge and long-term survival among patients with SAE. Literatures were searched through PubMed, ScienceDirect, and Google Scholar with a 10-year publication limit and selected based on topic relevance, study design, and data completeness and were analyzed using a narrative synthesis approach. Findings indicate that prolonged coma duration is strongly associated with poor neurological recovery, increased risk of disability, and early mortality. Conversely, early recovery of consciousness tends to correlate with better short-term functional outcomes. However, among survivors discharged from intensive care, coma duration doesn’t consistently predict long-term survival, although cognitive and quality of life impairments often persist. In conclusion, coma duration serves as an important prognostic factor in SAE, but it should be interpreted alongside other factors such as age, comorbidity, and the severity of sepsis itself. The scientific implications highlight the need for , infection control, and rehabilitative strategies to shorten periods of altered consciousness and improve neurological outcomes in the long term.
Cerebral Stroke Secondary to Decompression Ilness (DCI): Pathophysiology, Clinical Presentation, Diagnosis, and Management Khairifathiyyah, Khairifathiyyah; Aryasta, Ida Bagus Putra Bayu; Prawira, I Nyoman Dio Yudha; Untari, Lania Pradiva; Pambudi, Balqis Prudena Kurnia; Hunaifi, Ilsa
Jurnal Biologi Tropis Vol. 25 No. 4a (2025): Special Issue
Publisher : Biology Education Study Program, Faculty of Teacher Training and Education, University of Mataram, Indonesia

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.29303/jbt.v25i4a.10783

Abstract

Decompression illness (DCI) encompasses two major diving-related disorders: decompression sickness (DCS) and arterial gas embolism (AGE). Both conditions are characterized by the formation of gas bubbles that act as the primary mechanism of tissue injury, resulting in overlapping clinical manifestations and similar therapeutic approaches. This study aims to review the pathophysiology, clinical presentation, diagnosis, and management of DCI that presents with stroke-like neurological symptoms. The method used was a literature review approach based on current evidence from recent scientific publications. Findings show that DCS occurs when dissolved inert gases, such as nitrogen, become supersaturated during ascent and form bubbles within tissues and the venous circulation. AGE typically arises from pulmonary barotrauma, allowing trapped gas to enter the pulmonary veins and reach the arterial circulation, with the brain as the most vulnerable target organ. DCI is considered a medical emergency requiring definitive management with hyperbaric oxygen therapy (HBOT). Neurological manifestations of DCI often resemble conventional ischemic stroke but demonstrate distinct clinical and temporal patterns related to pressure changes. Therefore, in addition to recompression therapy, patients may require standard acute stroke management. Prognosis depends heavily on the promptness of diagnosis and treatment, as early intervention is critical to preventing permanent neurological injury.
PERANAN NEUROHORMONAL DALAM NEUROINFLAMASI PADA STROKE ISKEMIK AKUT Felicia Theddy; Fadila Olivia; Sang Ayu Nyoman Putri Pradnyasari; Ilsa Hunaifi
Jurnal Inovasi Pendidikan dan Sains Vol 3 No 3 (2022): December
Publisher : Lembaga Penelitian dan Pengabdian kepada Masyarakat Universitas Nahdlatul Wathan Mataram

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.51673/jips.v3i3.1208

Abstract

Stroke is the second most common cause of death and the third most common cause of disability in the world. Globally, 68% suffer from ischemic stroke. Post-ischemic stroke occurs a neuroinflammatory process which is a natural response of the immune system to tissue ischemia. The inflammatory process develops into three phases, namely the acute phase which begins shortly after the ischemic stroke, the subacute phase which begins several days after the ischemic stroke, and the final phase of astrocyte and microglia recovery. Neuroinflammation is a complex process involving inflammatory cells, inflammatory mediators, and hormones. Hormones that play a role in the neuroinflammatory process either increase or decrease the inflammatory response, namely estrogen, progesterone, testosterone, oxytocin, glucocorticoids, and cortisol.
THE EFFECT OF MLC901 ON RED CELL DISTRIBUTION WIDTH (RDW) AS A PREDICTOR OF ACUTE ISCHEMIC STROKE OUTCOME IN ACUTE ISCHEMIC STROKE MODEL IN RATS Ilham, Muhammad Arya Rifqi; Hunaifi, Ilsa; Dirja, Bayu Tirta
Folia Medica Indonesiana Vol. 61, No. 1
Publisher : Folia Medica Indonesiana

Show Abstract | Download Original | Original Source | Check in Google Scholar

Abstract

Stroke is the second leading cause of death globally, with ischemic stroke being the most prevalent type. Thrombolysis is the standard treatment for ischemic stroke; however, the rate of thrombolysis administration remains low. Therefore, neuroprotective therapies such as MLC901 are considered promising alternatives. The role of MLC901 in influencing red cell distribution width (RDW) during pathological ischemic stroke remains unclear. Hence, research on the effect of MLC901 on RDW is warranted. Male rats (n = 15) were divided into three groups: (1) acute ischemic stroke treated with MLC901 at a dose of 43.2 mg, (2) acute ischemic stroke treated with MLC901 at 21.6 mg, and (3) acute ischemic stroke treated with CMC-Na (positive control). Stroke was induced using the unilateral carotid artery occlusion (UCAO) method. RDW was measured before stroke induction and at 24 hours, 72 hours, and day 14. Neurological deficits were assessed using the foot fault score, and infarct volume was measured using ImageJ software. Following stroke induction, the positive control group had the highest mean RDW value, followed by the 21.6 mg and 43.2 mg MLC901 groups. However, there were no significant differences between the groups (p > 0.05). The positive control group exhibited greater neurological deficits compared to the MLC901 groups, with significant differences observed on days 7 and 14 (p < 0.05). Additionally, the positive control group had a significantly larger infarct volume than the MLC901 groups (p < 0.05). In conclusion, MLC901 did not affect RDW values but significantly reduced neurological deficits and infarct volume.
MLC 901 Decreases HSP-70, MMP-9, Cerebral Infarction Volume and Functional Outcome in Acute Ischemic Stroke Rat Model Ilsa Hunaifi; Andi Kurnia Bintang; Jumraini Tammasse; Isra&#039; Wahid; Mohammad Hatta; Andi Asadul Islam; Andi Alfian Zainuddin; Paulus Sugianto
The Indonesian Biomedical Journal Vol 17, No 3 (2025)
Publisher : The Prodia Education and Research Institute (PERI)

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.18585/inabj.v17i3.3610

Abstract

BACKGROUND: Acute ischemic stroke (AIS) is usually treated with thrombolysis, however the percentage of patients receiving this therapy is not quite low. Therefore, it is necessary to find alternative therapy using neuroprotective agent such as Moleac (MLC) 901. Heat shock proteins (HSP)-70 and matrix metalloproteinase (MMP)-9 are usually related to AIS due to the triggered stroke-induced physiological stress. However, the effect of MLC 901 on Hsp70 mRNA expression, HSP-70 and MMP-9 remains unclear. This study was conducted to determine the effect of MLC 901 on those three parameters in relation to cerebral infarction volume and functional outcomes in an AIS model.METHODS: Rats were induced with AIS using unilateral common carotid artery occlusion (UCAO) and received three different treatments: 43.2 mg/200 gBW MLC 901, 21.6 mg/200 gBW MLC 901, and sodium carboxymethyl cellulose (CMC-Na), that were administered orally for 14 days. HSP-70 and MMP-9 protein levels were assessed using enzyme-linked immunosorbent assay (ELISA), and Hsp70 mRNA expression was assessed using quantitative real-time polymerase chain reaction (qRT-PCR). Foot fault scores for evaluation functional outcome and infarction volume were assessed by ImageJ.RESULTS: AIS-induction increased HSP-70, MMP-9, and Hsp70 mRNA expression within 24-48 h. MMP-9, HSP-70 , and Hsp70 mRNA expression were reduced by MLC 901. MLC 901 at dose of 43.2 mg/200 gBW and 21.6 mg/200 gBW were effective in reducing these levels compared to the control. MLC 901 improved functional outcomes and decreased cerebral infarction volume. Moreover, a dosage of 43.2 mg/200 gBW was more effective in reducing Hsp70 mRNA expression and HSP-70, improving functional outcomes, and reducing cerebral infarction volume than a dosage of 21.6 mg/200 gBW, but not MMP-9 protein.CONCLUSION: MLC 901 effectively decreased Hsp70 mRNA expression, HSP-70 and MMP-9 protein levels, infarct volume, and functional outcomes. MLC 901 could be a potential therapeutic agent for AIS.KEYWORDS: MLC 901, HSP-70, MMP-9, acute ischemic stroke
PENYULUHAN PEMANFAATAN TANAMAN KATANG-KATANG (IPOMOEA PES-CAPRAE) SEBAGAI ALTERNATIF PENGOBATAN TRADISIONAL DI MASYARAKAT DESA TAMAN AYU, GERUNG, LOMBOK BARAT Muhammad Naufal Farras Ananta; Varelia Angelina; Baiq Irzana Putri Alamanda; Muhammad Iqbal Farobbi; Imasayu Nuralyza; Ilsa Hunaifi; Raisya Hasin; Dewi Suryani; Nisa Isneni Hanifa
Community Development Journal : Jurnal Pengabdian Masyarakat Vol. 6 No. 1 (2025): Volume 6 No. 1 Tahun 2025
Publisher : Universitas Pahlawan Tuanku Tambusai

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.31004/cdj.v6i1.40706

Abstract

Kegiatan pengabdian masyarakat ini bertujuan untuk meningkatkan pemahaman masyarakat Desa Taman Ayu, Gerung, Lombok Barat, mengenai manfaat Ipomoea pes-caprae sebagai alternatif pengobatan tradisional. Metode kegiatan meliputi pretest, ceramah, sesi tanya jawab, pembagian leaflet, dan posttest, yang dilakukan secara langsung kepada 30 peserta. Penyampaian materi mencakup pengenalan tanaman, manfaat tradisionalnya, dan cara pengolahannya. Hasil kegiatan menunjukkan peningkatan pengetahuan dan keterampilan masyarakat dalam memanfaatkan tanaman ini untuk pengobatan, seperti mengatasi nyeri perut, kram, dan sengatan ubur-ubur. Diskusi interaktif juga memperkuat pemahaman peserta. Kesimpulan dari kegiatan ini adalah bahwa penyuluhan efektif dalam membangun kesadaran masyarakat tentang pemanfaatan sumber daya lokal untuk kesehatan holistik, mendukung pengelolaan lingkungan berkelanjutan, serta meningkatkan kapasitas mereka dalam pengolahan obat tradisional.
Preliminary In Vitro Antiplatelet Potential of Ipomoea pes-caprae from North Lombok with Adenosine Diphosphate-Induced Platelet Aggregation Ilsa Hunaifi; Dewi Suryani; Lalu Husnul Hidayat; Muhammad Naufal Farras Ananta; Muhammad Iqbal Farobbi; Nisa Isneni Hanifa; Raisya Hasina
JURNAL FARMASI DAN ILMU KEFARMASIAN INDONESIA Vol. 12 No. 2 (2025): JURNAL FARMASI DAN ILMU KEFARMASIAN INDONESIA
Publisher : Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/jfiki.v12i22025.229-246

Abstract

Background: Cardiovascular diseases, particularly ischemic stroke, remain a global health burden, necessitating potential candidate for further antiplatelet with fewer side effects. Objectives: This study aimed to evaluate the antiplatelet potential of ethanolic extracts from the leaves and stems of Ipomoea pes-caprae (Katang-katang) from North Lombok, Indonesia, through ADP-induced platelet aggregation. Phytochemical screening, total tannin quantification, and in vitro antiplatelet assays were conducted. Methods: The leaves and stems were macerated with 96% ethanol, followed by qualitative phytochemical tests, Folin-Ciocalteu-based tannin analysis, and platelet aggregation inhibition assays using human platelet-rich plasma. Results: The extracts contained alkaloids, flavonoids, and tannins, with higher tannin levels in leaves (4.02 ± 0.02 mgEAT/g) than stems (3.67 ± 0.17 mgEAT/g). Concentration-dependent antiplatelet activity was observed, with leaf extracts showing inhibition (85.9% at 2000 μg/mL) compared to stems (79.5%) and aspirin (77.3%). IC₅₀ values were 727.78 μg/mL (leaves) and 349.95 μg/mL (stems). Statistical analysis confirmed significant differences across concentrations (p < 0.05). Conclusion: These findings demonstrate that Ipomoea pes-caprae exhibits potent antiplatelet activity, attributed to its tannin and phytochemical content, with leaves being more effective. Although these findings suggest preliminary antiplatelet potential, further analysis is required to validate the method using aspirin IC₅₀, and subsequent in vivo and pharmacological investigations are necessary before therapeutic application can be claimed.
Determinants of Executive Dysfunction: Serum NRF2 and Sociodemographic Profiles in Lombok Island’s Coastal Population Inayah Wulandari; Nurhidayati Nurhidayati; Ilsa Hunaifi; Arina Windri Rivarti; Herpan Syafii Harahap; Legis Octaviana Saputri; Dini Suryani; Fitriannisa Faradina Zubaidi; Rohadi Muhammad Rosyidi
AKSONA Vol. 6 No. 1 (2026): JANUARY 2026
Publisher : Universitas Airlangga

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.20473/aksona.v6i1.74449

Abstract

Highlight: Coastal communities show high prevalence of cognitive decline. There is no correlation between NRF2 levels and executive function. Gender and education level were significantly associated with executive dysfunction.   ABSTRACT Introduction: Elderly people frequently suffer from neurodegenerative conditions that affect cognitive function. Redox homeostasis dysregulation plays a significant role in neurodegenerative diseases. Oxidative stress activates the transcription factor called nuclear factor erythroid 2-related factor 2 (NRF2), which is essential for regulating the expression of genes involved in cellular defense mechanisms. Objective: This study aimed to determine the relationship between serum NRF2 levels and executive function, as well as to identify other variables that may influence executive function among residents of coastal villages on Lombok Island. Methods: The study used a cross-sectional design with consecutive sampling. Data on sociodemographic characteristics, hypertension, diabetes mellitus, smoking status, and physical activity were collected. Statistical analyses employed Spearman's rank correlation test, Fisher's exact test, and logistic regression test. Results: A total of 39 participants were included, with an average age of 53.38 years. Most participants were female, had a low educational level, did not have hypertension or diabetes mellitus, had high physical activity levels, and were non-smokers. The average NRF2 level was 0.54 ng/mL. The correlation test showed no significant relationship between serum NRF2 levels and executive function. Fisher's exact test showed a significant relationship between gender and education with executive function. Furthermore, logistic regression analysis identified education level as a predictor of executive dysfunction. Conclusion: Serum NRF2 levels were not correlated with executive dysfunction. Educational level was identified as a predictive factor for executive dysfunction.
Hubungan antara Penggunaan Smartphone pada Malam Hari dan Insomnia pada Remaja di Kota Mataram Mohammad Sany Rosafi Amanullah; Ilsa Hunaifi; I Putu Diatmika
Majalah Kedokteran Indonesia Vol 75 No 3 (2025): Journal of The Indonesian Medical Association - Majalah Kedokteran Indonesia, Vo
Publisher : PENGURUS BESAR IKATAN DOKTER INDONESIA (PB IDI)

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.47830/jinma-vol.75.3-2025-1868

Abstract

Introduction: Smartphone use among adolescents, particularly in Mataram City, West Nusa Tenggara, is highly prevalent. Nighttime smartphone useis associated with delayed sleep onset, as it extends the time adolescents more time to engage in various activities. Several studies have shown that smartphones use can contribute to sleep disturbances, including insomnia. This study aims to investigate the relationship between nighttime smartphone use and insomnia among adolescents in Mataram City.Methods: This study employed a cross-sectional design and included 362 students from Mataram City, selected through non-probability consecutive sampling method. Participants completed questionnaires assessing nighttime smartphone use and insomnia symptoms using Insomnia Severity Index. Data were analyzed using Spearman’s rank correlation test.Result: Of the total sample (44.5% male), the majority were 16 years old (62.4%), enrolled in Grade 11 (82.6%), and most (66.9%) were in the natural science program. The majority of participants were classified as having subclinical insomnia (43.9%), while only 2.5% experienced severe insomnia. The findings revealed a positive correlation between the frequency of nighttime smartphone and insomnia among adolescents (r=0.104; p=0.049).Conclusion: There is a significant relationship between the frequency of nighttime smartphone use and insomnia among adolescents in Mataram City.
The Role of Microglia and Astrocytes in the Neuroinflammation Process in Acute Ischaemic Stroke Dianing Aluh Qomala; I Ketut Wisnuaji Jayawardhana; Lalu Hizrian Abtartu; Rabsanjani Rabsanjani; Ilsa Hunaifi
Research of Service Administration Health and Sains Healthys Vol. 6 No. 2 (2025): Research of Service Administration Health and Sains Healthys (Desember)
Publisher : Lembaga Penelitian dan Pendidikan (LPP) Mandala

Show Abstract | Download Original | Original Source | Check in Google Scholar | DOI: 10.58258/rehat.v6i2.9784

Abstract

Acute ischemic stroke triggers a complex neuroinflammatory response involving resident glial cells, particularly microglia and astrocytes. Microglia rapidly activate following ischemia and polarize into two main phenotypes: the pro-inflammatory M1 phenotype, which exacerbates neuronal damage through the release of cytokines like TNF-α, IL-1β, and reactive oxygen species (ROS), and the anti-inflammatory M2 phenotype, which promotes tissue repair via IL-10 and IL-4 secretion. Astrocytes also play a dual role: in the acute phase, they exert neuroprotective effects by regulating glutamate homeostasis, producing antioxidants (e.g., glutathione and superoxide dismutase), and supporting neurogenesis and angiogenesis; however, in the chronic phase, reactive astrocytes contribute to glial scar formation, which inhibits axonal regeneration. The dynamic interplay between microglia and astrocytes critically shapes the neuroinflammatory milieu—determining whether the response is protective or detrimental. Understanding these dual roles offers promising avenues for developing neuroprotective therapies targeting neuroinflammation after acute ischemic stroke.
Co-Authors Adhima, Daffa Aulia Faza Agistany, Nur Feby Febiana Amalia, Azka Amaliya, Annisa Risqi Amatullah, Tazkiyah Arafah Ammar Rusydi, Muhammad Ananda Karunia Ramadhan Anandito Prabuningrat Andi Alfian Zainuddin Andi Asadul Islam Andi Kurnia Bintang andi, Silmi chairan Andiyani, Dinda Zahra Putri Anggoro, Joko Anjely Doni Lasmi Annafi, Muhammad Ibnu Annisa, Wardha Novia Arina Windri Rivarti Aryasta, Ida Bagus Putra Bayu Asri Buana Citra Dewi Asriningrum, Asriningrum Atari, Sicillia Putri Ath-Thariq Ramadhan Atmaja, I Made Brama Aulia, Siti Zahroni Baiq Annisa Pratiwi Baiq Hilya Kholida Baiq Hilya Kholida Baiq Hilya Kholida Baiq Holisatul Ismiana Baiq Irzana Putri Alamanda Baiq Rizqika Catur Putri Bambang Priyanto Bharata, Irsyadina Hasana Binti Amir, Nurul Aulia Briliansy Briliansy Briliansy, Briliansy Decky Aditya Zulkarnaen Dewi Suryani Dewi Suryani Dewi, Putri Krishna Kumara Dianing Aluh Qomala Diayanti Tenti Lestari Diayanti Tenti Lestari Diayanti Tenti Lestari Didit Yudanto Didit Yudhanto Dini Suryani Dini Suryani, Dini Dira Kurnia Rizki Dirja, Bayu Tirta Dwi Febrian, Haldy Dwi Rahmat Enny Ratna Indriyani Fadila Olivia Faizah, Lillah Fauzi, Yanuar Rahmat Febianisa, Tabitha Afifah Felicia Theddy Fesmia, Herodya Lajunee Fito Danang Sofiandi Fitriannisa Faradina Zubaidi Fitriannisa Faradina Zubaidi Fitriantoro, Santo Fitroningtyas, Elvika Aulia Gea, Endah Irnanda Ulfa Ghalvan sahidu ghalvan sahidu Ginuluh, Gintis Dhimar Habib, Phillip Hadinata, Lalu Gde Gilang Alid Helmiana, Putri Fatimah Herpan Syafi'i Harahap Herpan Syafii Harahap Herpan Syafii Harahap Herpan Syafii Harahap Herpan Syafii Harahap Herpan Syafii Harahap Herpan Syafii Harahap Herpan Syafii Harahap Herpan Syafii Harahap Herpan Syafii Harahap Hidayati, Eriska Humam, Anang Muh Naufal I Gede Andre Putra Rio I Gede Yasa Asmara I Ketut Wisnuaji Jayawardhana I Made Arya Yogiswara Mahayasa I Made Brama Atmaja I Made Wikrama Resindra I Putu Diatmika Ika Nur Fitria Ika Primayanti Ilham, M. Arya Rifqi Ilham, Muhammad Arya Rifqi Ilmiah, Janiya Abdila Imasayu Nuralyza Inayah Wulandari Indah Sapta Wardani Indrayana, Yanna Indrayana, Yanna Ismiana, Baiq Holisatul Isna Kusuma Nintyastuti Isra&#039; Wahid Jacob, Lusye Diana Joko Anggoro Joko Anggoro Jumraini Tammasse Justitia, Ica Kamila, Alya Syafa Kamilah, Nilam Nurmilatun Khairifathiyyah, Khairifathiyyah Khaula Karima kholida, Baiq Hilya Krisna , I Gusti Lanang Kurniawan, Theophany Margareta Kusumadewi, Rima Cahyati Kuta, Putu Cicilia Rarasati Lalu Ahmad Gamal Arigi Lalu Hizrian Abtartu Lalu Husnul Hidayat Lalu Wahyu Alfian Muharzami Landapa, Raditya Rachman Lasmi, Anjely Doni Lazuardi Arsy, Lazuardi Legis Octaviana Saputri Lestari, Diayanti Tenti Lestari, Rizka Vidya Lillah Faizah M Galvan Sahidu M Galvan Sahidu Maharani, Baiq Nadya Putri Marthin Fernandes Pasaribu Maulida, Rizka Ayu Mega Adwiatin Miraditi, Ni Komang Moestafa, Rizka Febriya Mohammad Hatta Mohammad Rizki Mohammad Sany Rosafi Amanullah monalisa nasrul Muhammad Galvan Sahidu Muhammad Ghalvan Sahidu Muhammad Ghalvan Sahidu Muhammad Ghalvan Sahidu Muhammad Ghalvan Sahidu Muhammad Ghalvan Sahidu Muhammad Ghalvan Sahidu Muhammad Ghalvan Sahidu Muhammad Ghalvan Sahidu Muhammad Ghifari Rifanhsa Muhammad Ibnu Annafi Muhammad Iqbal Farobbi Muhammad Iqbal Farobbi Muhammad Naufal Farras Ananta Muhammad Naufal Farras Ananta Mulayanatul Aliyyah Mutiara Syifa, R.R. Ditya Ngakan Putu Proudy Laksmana Ni Komang Miraditi Ni Made Amelia Ratnata Dewi Ni Made Metrya Savita Gera Ni Nyoman Ayu Susilawati Ni Nyoman Ayu Susilawati Ni Nyoman Ayu Susilowati Ni Nyoman Ayu, Susilawati Nisa Isneni Hanifa Nisa Isneni Hanifa Novrita Padauleng Nugraha, Gede Rama Hardy Nur Feby Febiana Agistany Nurfadia, Muhammad Fabian Nurhayati, Putri Nurhidayati Nurhidayati Nurhidayati Nurhidayati Nurhidayati Nurhidayati Nurhidayati Nurhidayati Nyoman Ayu Susilowati Ocktaviana Saputri, Legis Oktavianus Prayitno Padauleng, Novrita Padma Puspita, Ni Made Sri Paerdoe, Lalu Dane Pemban Pambudi, Balqis Prudena Kurnia Paradhini, Tri Sastra Paradiesta, Andi Frieskha Naurah Paulus Sugianto Pramudia, Prananda Rizki Pratiwi, Rukmanggana Satya Prawira, I Nyoman Dio Yudha Primayanti , Ika Pujiarohman Pujiarohman Pujiarohman Puspasari Mahadewi, Ni Made Putri Nurhayati Putri, Atina Rizki Putri, Siti Noururrifqiyati Juna Putu Diah Ananda Putri Atmaja Qindi, Ahmad Sa’bi Al Qurrata’yuni Pratiwi Rabsanjani Rabsanjani Raditya Bayu Farizil Akhyar Rahmadini, Nasyada Fadhila Rahmat, Dwi Raisya Hasin Raisya Hasina Ramzi, Ali Rian Pramudya Riantiani, Baiq Prita Rifansha, Muhammad Ghifari Rina Lestari Rizka Vidya Lestari Rizki, Mohammad Rohadi Rosmalasari Rosmalasari Rusdi, Yudika Ilhami Safat Wahyudi Safat Wahyudi Safat Wahyudi Safitri, Nasywa Aulia Sahidu, Muhammad Ghalvan Sahly, Aliza Raudatin Salsabila, Rika Kamila Sampe, Ester Sang Ayu Nyoman Putri Pradnyasari Sapta Wardhani, Indah Saraswati, Ni Wayan Citra Ayu Sepestian, Ghina Shadrina Rienti Setyawati Asih Putri Setyawati Asih Putri Setyawati Asih Putri Setyawati Asih Putri Setyawati, Indri Setyo Pambudi , Arif Shafira Adinda Putri Shafitri, Shira Shalsabina Shahab, Fatimah Silmi Chairan Andi Stefani Elizabeth Gunawan Stephanie Elizabeth Gunawan Stephanie Elizabeth Gunawan Stephanie Elizabeth Gunawan Stephanie Elizabeth Gunawan Subagiartha, I Wayan Sugiono, Muhammad Rafly Adrian Suryani, ⁠Dini Susilawati Ni Nyoman Ayu Teguh Budi Wicaksono Triana Dyah Cahyawati Triana Dyah Cahyawati Triana Dyah Cahyawati Ulfa Gea, Indah Ernanda Untari, Lania Pradiva Varelia Angelina Wahyu Maharani, Amelia Wardi, Baiq Prita Riantiani Wardi, Bq. Prita Riantiani Widad, Najwa Wiracakra, I Gusti Lanang Krisna Wulandari, Dwi Astuti Yanna Indrayana Yudika Ilhami Rusdi Yulianti, Arwinda Febri Yusika Saftari Handini Zubaidi, Fitriannisa Faradina